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1  of LRRK2 expression in the substantia nigra pars compacta.
2 as infused locally into the substantia nigra pars compacta.
3 droxylase expression in the substantia nigra pars compacta.
4 idus, the thalamus, and the substantia nigra pars compacta.
5  and ventral tegmental area/substantia nigra pars compacta.
6 t remarkably, little to the substantia nigra pars compacta.
7 dopaminergic neurons of the substantia nigra pars compacta.
8 opamine (DA) neurons in the substantia nigra pars compacta.
9 opamine (DA) neurons in the substantia nigra pars compacta.
10 cortex, caudate-putamen and substantia nigra pars compacta.
11 dopaminergic neurons in the substantia nigra pars compacta.
12 ral tegmental area (VTA) or substantia nigra pars compacta.
13 rsal raphe nucleus, and the substantia nigra pars compacta.
14  the substantia nigra pars reticulata and/or pars compacta.
15 ence of degeneration of the substantia nigra pars compacta.
16 dopaminergic neurons in the substantia nigra pars compacta.
17 sis and inflammation in the substantia nigra pars compacta.
18 dopaminergic neurons in the substantia nigra pars compacta.
19  expression is noted in the substantia nigra pars compacta.
20  of dopaminergic neurons in substantia nigra pars compacta.
21 dopaminergic neurons in the substantia nigra pars compacta.
22 dopaminergic neurons in the substantia nigra pars compacta.
23  to a lesser extent, in the substantia nigra pars compacta.
24 the dopamine neurons of the substantia nigra pars compacta.
25 olfactory tract, and within substantia nigra pars compacta.
26 ral tegmental area, and the substantia nigra pars compacta.
27  of dopamine neurons in the substantia nigra pars compacta.
28 dopaminergic neurons of the substantia nigra pars compacta.
29  ventral tegmental area and substantia nigra pars compacta.
30 mine neuron activity in the substantia nigra pars compacta.
31  in dopamine neurons of the substantia nigra pars compacta.
32 dopaminergic neurons in the substantia nigra pars compacta.
33 dopamine neurons from mouse substantia nigra pars compacta.
34 e dorsal GL or in the right substantia nigra pars compacta.
35 nergic interneurons and the substantia nigra pars compacta.
36 rol vs. 36+/-2.6 O.D. BMP-7-treated, p<0.05; pars compacta: 29.0+/-4.9 O.D. control vs. 64.4+/-6.9 O.
37 dopaminergic neurons in the substantia nigra pars compacta 60 days after infection.
38  of dopamine neurons of the substantia nigra pars compacta, a deficit in dopamine neurotransmission a
39 ource of serotonin, and the substantia nigra pars compacta, a major source of dopamine, while monkeys
40 s are most numerous in the substantia nigra, pars compacta, a region badly affected in homozygous wea
41 dopaminergic neurons of the substantia nigra pars compacta, a susceptible brain region in PD.
42 al tegmental area (VTA) and substantia nigra pars compacta activates inhibitory postsynaptic D2-recep
43 jections of 6-OHDA into the substantia nigra pars compacta and 1 week later were tested for startle a
44 opaminergic (DA) neurons in substantia nigra pars compacta and age-dependent L-DOPA-sensitive motor d
45 ne-producing neurons in the substantia nigra pars compacta and decreased striatal dopamine levels are
46 the dopaminergic neurons of substantia nigra pars compacta and in the CA1/2 pyramidal cells of hippoc
47 dopaminergic neurons in the substantia nigra pars compacta and intracellular inclusion called Lewy bo
48 dopaminergic neurons in the substantia nigra pars compacta and locus ceruleus, without Lewy body path
49 in pigmented neurons of the substantia nigra pars compacta and locus coeruleus in all four DYT1 dysto
50 holaminergic neurons of the substantia nigra pars compacta and locus coeruleus, among other regions.
51 ntaining cell levels in the substantia nigra pars compacta and nigrostriatal terminal density in vivo
52 paminergic neurons from the substantia nigra pars compacta and noradrenergic neurons from the locus c
53 clusions) in neurons of the substantia nigra pars compacta and other brain areas.
54 nsmission, particularly the substantia nigra pars compacta and other dopamine-synthesizing cell group
55 ed dopamine (DA) neurons in substantia nigra pars compacta and restored DA levels in striatum using t
56 essed in neurons within the substantia nigra pars compacta and striatum, two regions critically invol
57 dopaminergic neurons in the Substantia Nigra pars compacta and terminal dopamine fiber density in the
58 dopaminergic neurons in the substantia nigra pars compacta and the accumulation of the protein alpha-
59 paminergic neurons from the substantia nigra pars compacta and the presence, in affected brain region
60 trafficking, in vivo in the substantia nigra pars compacta and the serum of 1-methyl-4-phenyl-1,2,3,6
61 nergic projections from the substantia nigra pars compacta and the ventral tegmental area to the basa
62 mine neurons located in the substantia nigra pars compacta and the ventral tegmental area, which form
63  dopamine by neurons in the substantia nigra pars compacta and ventral tegmental also influences basa
64                         The substantia nigra pars compacta and ventral tegmental area contain the two
65 ular nucleus, raphe nuclei, substantia nigra pars compacta and ventral tegmental area homologs, super
66     Dopamine neurons in the substantia nigra pars compacta and ventral tegmental area regulate behavi
67 inergic (DA) neurons in the substantia nigra pars compacta and ventral tegmental area regulate extrap
68 responses from cells in the substantia nigra pars compacta and ventral tegmental area.
69 ll bodies of neurons in the substantia nigra pars compacta and ventral tegmental area.
70 homologous to the mammalian substantia nigra pars compacta and was renamed accordingly; a group of ga
71 ulum (PT; homologous to the substantia nigra pars compacta and/or ventral tegmental area of mammals)
72 n dopaminergic neurons, A9 (substantia nigra pars compacta) and A10 (ventral tegmental area), have di
73 lated within neurons of the substantia nigra pars compacta, and dual labeling and confocal imaging co
74 us, ventral tegmental area, substantia nigra pars compacta, and nucleus of the solitary tract.
75  in the ventral tier of the substantia nigra pars compacta, and the lowest levels in the ventral tegm
76 n of Mn accumulation in the substantia nigra pars compacta, and thus, can represent a link between Mn
77 sion was upregulated in the substantia nigra pars compacta, and trkB was elevated in the striatum in
78 Dopaminergic neurons of the substantia nigra pars compacta are autonomous pacemakers.
79 Dopaminergic neurons of the substantia nigra pars compacta are defective in Parkinson's disease, but
80 opamine neurons in the substantia nigra (SN) pars compacta are selectively lost during the progressio
81         Because the VTA and substantia nigra pars compacta are the sole sources of striatal and limbi
82 thalamic nucleus (STN), and substantia nigra pars compacta, are conserved throughout vertebrate phylo
83 he A9 and A10 region of the substantia nigra pars compacta as well as the technical and surgical step
84 neurons of the adult rodent substantia nigra pars compacta, as well their development in vitro.
85 was a robust gliosis in the substantia nigra pars compacta associated with significant upregulation o
86 dopaminergic neurons of the substantia nigra pars compacta, become transduced.
87 munoreactive neurons in the substantia nigra pars compacta (both in total and in subregions) were per
88 f brain neurons not only in substantia nigra pars compacta but also in other extrastriatal areas of t
89  of dopamine neurons in the substantia nigra pars compacta, but not in the adjacent ventral tegmental
90 ctive, was observed in the substantia nigra, pars compacta, but not in the ventral tegmental area.
91 munoreactive neurons in the substantia nigra pars compacta by 2-fold (p < 0.05) in animals lesioned w
92 opamine terminal damage and substantia nigra pars compacta cell loss.
93 ections to the striatum and substantia nigra pars compacta compared with PV-GPe neurons.
94 rain ventral tegmental area-substantia nigra pars compacta complex.
95 minergic neurons within the substantia nigra pars compacta coupled with depletion of striatal dopamin
96 dopaminergic neurons in the substantia nigra pars compacta coupled with intracytoplasmic inclusions k
97  of dopamine neurons in the substantia nigra pars compacta, culminating in severe motor symptoms, inc
98  ventral tegmental area and substantia nigra pars compacta DA neurons in the post-reward period had a
99 ning neurons of the brain's substantia nigra pars compacta die in Parkinson's disease has been an end
100 vo recordings of identified substantia nigra pars compacta dopamine neurons in R1441C LRRK2 transgeni
101 sion of torsinA mRNA in the substantia nigra pars compacta dopamine neurons, the locus ceruleus, the
102 d thereby properly maintain substantia nigra pars compacta dopaminergic neurons and their innervation
103              Studying mouse substantia nigra pars compacta dopaminergic neurons both in brain slice a
104        We show that, in rat substantia nigra pars compacta dopaminergic neurons, the voltage dependen
105 stance from the soma in rat substantia nigra pars compacta dopaminergic neurons.
106  PAR1 is expressed in human substantia nigra pars compacta glia as well as tyrosine hydroxylase-posit
107 els, forming an ultra-short substantia nigra pars compacta --&gt; SNr dopamine pathway that regulates th
108 n of alpha-synuclein in the substantia nigra pars compacta impacts visual processing in a well-establ
109 ydopamine (6-OHDA) into the substantia nigra pars compacta in 10 rats.
110 nd neurodegeneration in the substantia nigra pars compacta in aged VMAT2 LO mice.
111 of nigrostriatal neurons of substantia nigra pars compacta in Parkinson's disease represents the best
112 or the vulnerability of the substantia nigra pars compacta in PD, why the disorder is age related, an
113 ctivity (TH-ir) in the substantia nigra (SN) pars compacta, in the lesioned hemisphere [31.7+/-5.2 (o
114  ventral tegmental area and substantia nigra pars compacta innervate the majority of the cortex, incl
115 dopaminergic neurons of the substantia nigra pars compacta is a cardinal feature of Parkinson's disea
116 generation of DA neurons in substantia nigra pars compacta is a key neuropathological feature in Park
117 dopaminergic neurons in the substantia nigra pars compacta is the primary cause for motor symptoms ob
118 dopaminergic neurons of the substantia nigra pars compacta leading to abnormal activity within the ba
119 dopaminergic neurons in the substantia nigra pars compacta, leading to nigrostriatal degeneration.
120 neuron subtypes outside the substantia nigra pars compacta may be compensating at a molecular level f
121 ted nucleus and dorsomedial substantia nigra pars compacta) mesodiencephalic dopaminergic neuron subs
122 are characterized by severe substantia nigra pars compacta neuronal loss.
123 B (trkB) receptor occurs in substantia nigra pars compacta neurons and is required for neuroprotectio
124       Unilateral lesions of substantia nigra pars compacta neurons created rats with hyperexcitable t
125 g the spontaneous firing of substantia nigra pars compacta neurons, isolated from transgenic mice in
126 able dopaminergic (DAergic) substantia nigra pars compacta neurons, only select downregulation of the
127 on finger tapping is a clinical correlate of pars compacta nigral degeneration.
128 t is characterised by the presence of severe pars-compacta nigral-cell loss, and accumulation of aggr
129 eurons in culture or in the substantia nigra pars compacta of 5-wk-old mice.
130 ive cell body counts in the substantia nigra pars compacta of 6-OHDA- and free-3NT-treated mice (inje
131 vo data from the cortex and substantia nigra pars compacta of aged LRRK2 transgenic animals revealed
132  single DA neurons from the substantia nigra pars compacta of controls and subjects with idiopathic P
133  in dopamine neurons of the substantia nigra pars compacta of mice following systemic administration
134  of dopamine neurons in the substantia nigra pars compacta of monkeys (Macaca mulatta) during a reach
135  of p21(ras) in vivo in the substantia nigra pars compacta of MPTP-intoxicated mice.
136 fects of glial cells in the substantia nigra pars compacta of Parkinson's disease.
137 have been identified in the substantia nigra pars compacta of patients with sporadic PD.
138 aB was activated within the substantia nigra pars compacta of PD patients and MPTP-intoxicated mice.
139 re also up-regulated in the substantia nigra pars compacta of post-mortem PD brains as compared with
140 as also demonstrated in the substantia nigra pars compacta of post-mortem PD brains.
141 minergic neurons within the substantia nigra pars compacta of the midbrain.
142 erikarya and primary dendrites, while in the pars compacta of the PPN (PPNc) axosomatic synapses were
143 uno-positive neurons in the substantia nigra pars compacta of wild-type mice.
144 se-positive neurons in the substantial nigra pars compacta one week after the first dose of MPTP.
145 the dopamine neurons of the substantia nigra pars compacta, other regions of the midbrain, and also t
146 ilar in injured IL-6 (+/+) and IL-6 (-/-) SN pars compacta (pc), microgliosis was severely compromise
147 ioris amygdalopallii pars basalis (PoAb) and pars compacta (PoAc), the nucleus taeniae amgygdalae (Tn
148 dopaminergic neurons in the substantia nigra pars compacta portion of the brain.
149 dopaminergic neurons of the substantia nigra pars compacta preferentially terminate in patch-like reg
150 ne-releasing neurons of the substantia nigra pars compacta produce an extraordinarily dense and expan
151 ergic (DA) neurons in their substantia nigra pars compacta, progressing to bilateral degeneration of
152 ateralis, r. subcuneiformis, r. peduncularis pars compacta, r. subcoeruleus pars alpha, raphe obscuru
153 dopaminergic neurons in the substantia nigra pars compacta, reproducing an important pathological fea
154      Neuronal counts in the substantia nigra pars compacta revealed that neuronal loss in the parkin
155 yrosine hydroxylase mRNA in substantia nigra pars compacta (SC) (93%; P<0.05) compared with control r
156 f cell proliferation in the substantia nigra pars compacta (SN(C)) with a time-dependent adoption of
157  form the laterally-located substantia nigra pars compacta (SN) and medially-located ventral tegmenta
158 dopaminergic neurons in the substantia nigra pars compacta (SN) leads to debilitating motor dysfuncti
159 hionin-stained cells in the substantia nigra pars compacta (SN-PC)].
160 dopaminergic neurons in the substantia nigra pars compacta (SNc) and consequent depletion of striatal
161 e lacks the majority of the substantia nigra pars compacta (SNc) and experiences striatal denervation
162 opaminergic neurones in the substantia nigra pars compacta (SNc) and may contribute to excitotoxic ce
163 ation ([DA](o)) between the substantia nigra pars compacta (SNc) and striatum.
164 cation between the midbrain substantia nigra pars compacta (SNc) and the CeA.
165 the dopamine neurons of the substantia nigra pars compacta (SNc) and the importance of protein aggreg
166 dopaminergic neurons in the substantia nigra pars compacta (SNc) and the presence of intracytoplasmat
167   Dopamine neurons from the substantia nigra pars compacta (SNc) and ventral tegmental area (VTA) are
168 brain dopamine centers, the substantia nigra pars compacta (SNc) and ventral tegmental area (VTA), de
169  of dopamine neurons in the substantia nigra pars compacta (SNC) and ventral tegmental area (VTA).
170 neurons that project to the substantia nigra pars compacta (SNc) are activated by a visual CS for foo
171 al tegmental area (VTA) and substantia nigra pars compacta (SNc) are not significantly modulated by a
172 al tegmental area (VTA) and substantia nigra pars compacta (SNC) contain dopamine neurons intermixed
173 al tegmental area (VTA) and substantia nigra pars compacta (SNc) convey distinct signals.
174 ticulata (SNr), which in turn inhibits nigra pars compacta (SNc) DAergic neurons.
175 neurons (DAs) of the rodent substantia nigra pars compacta (SNc) display varied electrophysiological
176 ve disease characterized by substantia nigra pars compacta (SNc) dopamine (DA) neuron loss and subseq
177                             Substantia nigra pars compacta (SNc) dopamine neurons and their targets a
178 udies have established that substantia nigra pars compacta (SNc) dopamine neurons are a key node in t
179 ctor underlying the loss of substantia nigra pars compacta (SNc) dopaminergic neurons in Parkinson's
180            Burst spiking in substantia nigra pars compacta (SNc) dopaminergic neurons is a key signal
181 layed increased activity of substantia nigra pars compacta (SNc) dopaminergic neurons, elevated basel
182 hat depend on the firing of substantia nigra pars compacta (SNc) dopaminergic neurons, we identified
183 homologous to the mammalian substantia nigra pars compacta (SNc) evokes increasing activation of MLR
184 Dopaminergic neurons of the substantia nigra pars compacta (SNc) exhibit functional heterogeneity tha
185 inergic (DA) neurons of the substantia nigra pars compacta (SNc) govern movements requires a detailed
186  neurons (TH-IR) within the substantia nigra pars compacta (SNc) in both young and aged animals.
187 egmental area (VTA) and the substantia nigra pars compacta (SNc) mDA neurons.
188 this receptor in regulating substantia nigra pars compacta (SNc) neuron physiology in both mice and r
189 e recordings were made from substantia nigra pars compacta (SNC) neurons in horizontal brain slice pr
190 eurons differs from that in substantia nigra pars compacta (SNc) neurons, where subthreshold calcium
191 g was much stronger in monkey than in rat SN pars compacta (SNc) neurons, whereas a moderate level of
192 l dendrites of dopaminergic substantia nigra pars compacta (SNc) neurons.
193 nal heterogeneity among the substantia nigra pars compacta (SNc) neurons.
194 with rAAV2/5 vectors in the substantia nigra pars compacta (SNc) on one side of the brain; the other
195 ion was seen in neighboring substantia nigra pars compacta (SNC) or substantia nigra pars reticulata.
196 and have been implicated in substantia nigra pars compacta (SNc) pathology in Parkinson's disease.
197  extent dopamine neurons in substantia nigra pars compacta (SNc) play such roles.
198                         The substantia nigra pars compacta (SNc) projects specifically into the rostr
199 , dopaminergic cells in the substantia nigra pars compacta (SNc) respond immediately to unexpected co
200 opamine (DA) release in the substantia nigra pars compacta (SNc) shows a limited dependence on extrac
201 dDA) neurons, including the substantia nigra pars compacta (SNc) subpopulation that preferentially de
202 pamine neuron in the monkey substantia nigra pars compacta (SNc) that retains past learned reward val
203 on of viral vector into the substantia nigra pars compacta (SNc) to investigate its influence on nigr
204 al tegmental area (VTA) and substantia nigra pars compacta (SNc) to that of axonal dopamine release i
205 ostriatal pathway, from the substantia nigra pars compacta (SNc) to the dorsal striatum, and on relat
206 injections of rAAV into the substantia nigra pars compacta (SNc) transduced both dopaminergic and non
207 o Yellow or Fluorogold into substantia nigra pars compacta (SNc) were combined with larger injections
208 itive neuron numbers in the substantia nigra pars compacta (SNC) were estimated using stereological m
209 opamine (DA) neurons of the substantia nigra pars compacta (SNc) were found to exhibit sustained resp
210 ior colliculus (SC), to the substantia nigra pars compacta (SNc) where direct synaptic contacts are m
211 dopaminergic neurons in the substantia nigra pars compacta (SNc), but not in ventral tegmental area o
212 , substantia nigra pars reticulata (SNr) and pars compacta (SNc), but not the thalamus.
213 re first established in the substantia nigra pars compacta (SNc), but their validity in the VTA is un
214 dopaminergic neurons of the substantia nigra pars compacta (SNc), in addition to many other regions,
215 al tegmental area (VTA) and substantia nigra pars compacta (SNc), no clear evidence for separate stru
216 ergic neurons (DaNs) of the substantia nigra pars compacta (SNc), resulting in the characteristic hyp
217 (PD)-related changes in the substantia nigra pars compacta (SNc), the key pathological loci.
218 been suggested that, in the substantia nigra pars compacta (SNc), the pacemaking relies more on Ca(2+
219 ject to dopamine neurons in substantia nigra pars compacta (SNc), whereas matrix neurons receive sens
220 opamine (DA) neurons in the substantia nigra pars compacta (SNc).
221 dopaminergic neurons in the substantia nigra pars compacta (SNc).
222 dopaminergic neurons in the substantia nigra pars compacta (SNc).
223 ic) cell bodies in the substantia nigra (SN) pars compacta (SNc).
224 opamine (DA) neurons in the substantia nigra pars compacta (SNc).
225 ut not in DA neurons of the substantia nigra pars compacta (SNc).
226  of dopamine neurons in the substantia nigra pars compacta (SNc).
227 opaminergic neurones of the substantia nigra pars compacta (SNc).
228 dendrites of neurons in the substantia nigra pars compacta (SNc).
229 expressed GFRalpha-1 in the substantia nigra pars compacta (SNC).
230 al tegmental area (VTA) and substantia nigra pars compacta (SNc).
231 dopamine neurons within the substantia nigra pars compacta (SNc).
232 dopamine neurons within the substantia nigra pars compacta (SNc).
233 dopaminergic neurons in the substantia nigra pars compacta (SNc).
234 dopaminergic neurons of the substantia nigra pars compacta (SNc).
235 sterior, a homologue of the substantia nigra pars compacta (SNc)/ventral tegmental area (VTA) is pres
236 dopaminergic neurons of the substantia nigra pars compacta (SNpc) against 6-OHDA and MPTP.
237 opaminergic neurons of the substantia nigra, pars compacta (SNpc) akin to what is observed in Parkins
238 inergic (DA) neurons in the substantia nigra pars compacta (SNpc) and of noradrenergic neurons in the
239 ificantly diminished in the substantia nigra pars compacta (SNpc) and striatum, regions most affected
240 to these neurons in the rat substantia nigra pars compacta (SNpc) and ventral tegmental area (VTA), a
241  of dopamine neurons in the substantia nigra pars compacta (SNpc) and widespread aggregates of the pr
242 r centers, compromising the substantia nigra pars compacta (SNpc) and, later, the cerebral cortex.
243 ore dopamine neurons in the substantia nigra pars compacta (SNpc) are greatly needed to effectively c
244 dopaminergic neurons in the substantia nigra pars compacta (SNpc) as seen in Parkinson's disease.
245 for dopamine neurons of the substantia nigra pars compacta (SNpc) by regulating the magnitude of the
246 ctive (THir) neurons in the substantia nigra pars compacta (SNpc) compared with saline treatment.
247 rosine hydroxylase-immunoreactive (TH-ir) SN pars compacta (SNpc) DA neurons are immunoreactive for e
248 gic (DA) neurons within the substantia nigra pars compacta (SNpc) display a differential vulnerabilit
249 on/degeneration of midbrain substantia nigra pars compacta (SNpc) dopaminergic (DA) neurons contribut
250  characterized by a loss of substantia nigra pars compacta (SNpc) dopaminergic (DA) neurons, and can
251 neurons of the ventral tier substantia nigra pars compacta (SNpc) failed to demonstrate a preponderan
252        Neuronal loss in the substantia nigra pars compacta (SNpc) in Parkinson disease (PD) is not un
253 t optogenetic activation of substantia nigra pars compacta (SNpc) neurons alleviates parkinsonism in
254 dopaminergic neurons of the substantia nigra pars compacta (SNpc) of human PD patients.
255  degeneration occurs in the substantia nigra pars compacta (SNpc) of patients with Parkinson's diseas
256 ation has been shown in the substantia nigra pars compacta (SNpc) of PD models when there has been a
257  transfer of RGS10 into the substantia nigra pars compacta (SNpc) of rats reduced microgliosis and pr
258 (DA) neurons at the ventral substantia nigra pars compacta (SNpc) preferentially degenerate in Parkin
259 inergic (DA) neurons in the substantia nigra pars compacta (SNpc) remains to be answered.
260 Dopaminergic neurons in the substantia nigra pars compacta (SNpc) undergo natural cell death during d
261 eurons containing CR in the substantia nigra pars compacta (SNpc) were relatively spared compared to
262      Here, we show that the substantia nigra pars compacta (SNpc), a brain region where dopamine (DA)
263 ced dopaminergic neurons in substantia nigra pars compacta (SNpc), and SNCA mice were more vulnerable
264  of dopaminergic neurons in substantia nigra pars compacta (SNpc), and there was no loss of dopaminer
265 had more TH-ir cells in the substantia nigra pars compacta (SNpc), but this difference was significan
266 ergic neurons (DANs) in the substantia nigra pars compacta (SNpc), decrease of dopamine (DA) transmit
267 sive degeneration of DNs in substantia nigra pars compacta (SNpc), decreased striatal dopamine levels
268 rgic neurons located in the substantia nigra pars compacta (SNpc), expression of monoamines and indol
269 l intensity loss within the substantia nigra pars compacta (SNpc), locus coeruleus, and ventral tegme
270 ecting GDF5 into either the substantia nigra pars compacta (SNpc), the lateral ventricle (LV) or the
271 f dopaminergic cells in the substantia nigra pars compacta (SNpc), ventral tegmental area (VTA), and
272 dopaminergic neurons of the substantia nigra pars compacta (SNpc).
273 inergic (DA) neurons in the substantia nigra pars compacta (SNpc).
274 opaminergic (DA) neurons in substantia nigra pars compacta (SNpc).
275 rmation was detected in the substantia nigra pars compacta, striatum, hippocampus, or selected region
276 ein accumulation within the substantia nigra pars compacta, suggesting that nigrostriatal dopamine dy
277 ell accumulation within the substantia nigra pars compacta, suppression of microglial activation, and
278 t of the BG, and dopaminergic neurons of the pars compacta that modulate thalamic excitability.
279 dopaminergic neurons in the substantia nigra pars compacta, the exact mechanism involved is still poo
280                      In the substantia nigra pars compacta, there was a 50-90% loss of tyrosine hydro
281 ion from frontal cortex and substantia nigra pars compacta tissue, isolated by several filtration and
282 bus pallidus, thalamus, and substantia nigra pars compacta to various environmental or genetic insult
283 eeks and a 42% reduction in substantia nigra pars compacta tyrosine hydroxylase-positive neurons at 8
284 e-containing neurons in the substantia nigra pars compacta use pacemaking mechanisms common to neuron
285 bgroups of neurons, such as substantia nigra pars compacta, ventral tegmental area and retrorubal fie
286 pocampus and dentate gyrus, substantia nigra pars compacta, ventral tegmental area, geniculate nucleu
287 on of the SN allowing readers to distinguish pars compacta ventralis and dorsalis from pars reticulat
288 ic terminals in the ventral substantia nigra pars compacta (vSNc) or to the ventral tegmental area (V
289 wn as the nucleus tegmenti pedunculopontinus pars compacta was recognized as homologous to the mammal
290 n of TRPM2 and TRPM4mRNA in substantia nigra pars compacta.We propose that ICAN is selectively activa
291 ted dopamine neurons in the substantia nigra pars compacta were immunonegative.
292 e-containing neurons in the substantia nigra pars compacta were located within the calbindin-rich zon
293 ost-commissural putamen and substantia nigra pars compacta were processed for tyrosine hydroxylase an
294 e dopaminergic cells in the substantia nigra pars compacta were unaffected by METH or haloperidol alo
295  more dorsolaterally in the substantia nigra pars compacta, whereas neurons inhibited by the stimuli
296 inergic (DA) neurons of the substantia nigra pars compacta, which are preferentially lost in human Pa
297  specify mDA neurons of the substantia nigra pars compacta while the late Shh and Gli1 lineages maint
298 dopaminergic neurons in the substantia nigra pars compacta, with more than 40% of these neurons lost
299 nal loss and gliosis in the substantia nigra pars compacta, without Lewy bodies.
300 educed neuron counts in the substantia nigra pars compacta, yet striatal medium spiny neuron dendriti

 
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