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1 s, globus pallidus and the substantia nigra (pars reticulata).
2 of the basal ganglia (i.e. substantia nigra pars reticulata).
3 neuronal migration into the substantia nigra pars reticulata.
4 nterpositus nucleus and the substantia nigra pars reticulata.
5 d ventral pallidum and the substantia nigra, pars reticulata.
6 ne reticular formation, and substantia nigra pars reticulata.
7 iatum, globus pallidus, and substantia nigra pars reticulata.
8 in the globus pallidus and substantia nigra pars reticulata.
9 on, as did the striatum and substantia nigra pars reticulata.
10 ication of D-amphetamine in substantia nigra pars reticulata.
11 botenic acid lesions of the substantia nigra pars reticulata.
12 cond microdialysis probe in substantia nigra pars reticulata.
13 with PD and controls in the substantia nigra pars reticulata.
14 ed GABAergic neurons in the substantia nigra pars reticulata.
15 sh pars compacta ventralis and dorsalis from pars reticulata.
16 ectly to the homolog of the substantia nigra pars reticulata.
17 to subthalamic nucleus and substantia nigra pars reticulata.
18 ganglia output neurons in substantia nigra, pars reticulata.
19 ion from globus pallidus or substantia nigra pars reticulata.
20 igra pars compacta (SNC) or substantia nigra pars reticulata.
21 in slices from rat or mouse substantia nigra pars reticulata.
22 rgic neurons of the VTA and substantia nigra pars reticulata.
23 hydroxylase (TH) positive cell bodies in the pars reticulata.
24 idus, the entopeduncular nucleus, and the SN pars reticulata.
25 he brainstem, including the substantia nigra pars reticulata.
26 la, and caudal parts of the substantia nigra pars reticulata.
27 nificantly increased in the substantia nigra pars reticulata (13.5+/-4.1 and 26.3+/-2.9%, respectivel
28 ir in the SN of the non-lesioned hemisphere (pars reticulata: 14.8+/-1.19 O.D. control vs. 36+/-2.6 O
29 found that activity in the substantia nigra pars reticulata, a basal ganglia output, predictably dif
30 12 GABAergic neurons in the substantia nigra pars reticulata also were recorded and filled with bioti
31 eptors (D1+) project to the substantia nigra pars reticulata and are thought to facilitate movement.
32 atergic transmission in the substantia nigra pars reticulata and entopeduncular nucleus, two major ta
34 ernus, subthalamic nucleus, substantia nigra pars reticulata and neurons of the indirect pathway.
36 s from 684 globus pallidus, substantia nigra pars reticulata and subthalamic neurons recorded in inta
37 ethane anaesthesia from the substantia nigra pars reticulata and subthalamic nucleus along with corti
38 munoreactive nuclei were concentrated in the pars reticulata and the majority of labeled nigral neuro
41 and globus pallidus interna/substantia nigra pars reticulata, and infusion of recombinant glial deriv
42 urons in the substantia nigra pars compacta, pars reticulata, and pars lateralis), and 51% in nucleus
43 line output is initiated in substantia nigra pars reticulata, and this influence contributes to the e
44 riatum, globus pallidus and substantia nigra pars reticulata, and was not detectable in the subthalam
45 thesis that D1 receptors in substantia nigra pars reticulata are involved in the excitatory component
46 entopeduncular nucleus, and substantia nigra pars reticulata, areas of the basal ganglia receiving st
47 n of GABAergic cells in the substantia nigra pars reticulata blocks signaled active avoidance by inhi
48 the activity of GABAergic neurons in the SN pars reticulata, but does so indirectly via D1 dopamine
51 ng GABAergic neurons of the substantia nigra pars reticulata cannot be differentiated on the basis of
52 -dorsal-lateral part of the substantia nigra pars reticulata (cdlSNr), directly or indirectly through
54 ojection neurons target the substantia nigra pars reticulata (direct pathway) or the lateral globus p
56 ntal area, caudate putamen, substantia nigra pars reticulata, entorhinal cortex, central amygdala, la
57 in the globus pallidus and substantia nigra pars reticulata following lesions of the nigrostriatal t
58 afferents from striatum, globus pallidus, or pars reticulata have been shown to be mediated predomina
59 In the globus pallidus and substantia nigra pars reticulata, however, mGluR1a-ir was tightly cluster
61 ordings from neurons in the substantia nigra pars reticulata in rat brain slices and labeled them wit
62 reflex blinking is (1) the substantia nigra pars reticulata inhibits SC neurons, (2) the SC excites
64 in the globus pallidus and substantia nigra pars reticulata is caused by abnormal striatal activity.
65 ically increases the medial substantia nigra pars reticulata neuron activity and has a preferential a
66 via nigral D2 receptors, perhaps located on pars reticulata neurons themselves, to regulate basal ga
69 nnabinoid agonists, applied locally into the pars reticulata of substantia nigra (SNpr), could modula
71 pine-7,8-diol (SKF 38393) hydrochloride into pars reticulata of substantia nigra elicited a significa
72 profiles of neurons in the substantia nigra pars reticulata of the basal ganglia and in the superior
76 region of the hippocampus, substantia nigra pars reticulata, pallidum, and deep cerebellar nuclei.
79 timulation was identical in substantia nigra pars reticulata slices prepared from TKO and wild-type m
80 to inhibit output from the substantia nigra pars reticulata (SNpr) and internal pallidal segment (GP
81 nge in LFPs recorded in the substantia nigra pars reticulata (SNpr) and motor cortex (MCx) in the hem
82 hown that inhibition of the substantia nigra pars reticulata (SNpr) attenuates seizures, yet the circ
83 , increases in motor cortex-substantia nigra pars reticulata (SNpr) coherence emerged in the 8-25 Hz
84 rior colliculus (SC) or the substantia nigra pars reticulata (SNpr) contralateral to the cortical les
86 rance of APP domains in rat substantia nigra pars reticulata (SNpR) neurons targeted for delayed dege
87 cular nucleus (EPN) and the substantia nigra pars reticulata (SNpr) on the intact side of the brain,
89 nculopontine nucleus to the substantia nigra pars reticulata (SNr) act on muscarinic acetylcholine re
90 he monkey, but not the rat, substantia nigra pars reticulata (SNr) also harbored a significant level
91 dy, we demonstrate that the substantia nigra pars reticulata (SNr) also provides a massive input to P
92 nch LSt projects to the GP, substantia nigra pars reticulata (SNr) and pars compacta (SNc), but not t
93 unbalanced responses in the substantia nigra pars reticulata (SNr) and suggesting a mechanism for the
94 expressed GFRalpha-1 in the substantia nigra pars reticulata (SNR) and the ventral tegmental area (VT
95 ated the involvement of the substantia nigra pars reticulata (SNr) and the ventromedial nucleus of th
96 th GABAergic neurons in the substantia nigra pars reticulata (SNr) and with distal dendrites (in SNr)
97 c projection neurons in the substantia nigra pars reticulata (SNr) are key basal ganglia output neuro
98 evelopmental changes in the substantia nigra pars reticulata (SNr) associated with the expression of
101 he light microscope level, neurons of the SN pars reticulata (SNr) displayed moderate to strong immun
102 tory glutamatergic input to substantia nigra pars reticulata (SNr) from the subthalamic nucleus (STN)
104 argets of the striatum, the substantia nigra pars reticulata (SNr) has been hypothesized to play a ro
105 measured BG output from the substantia nigra pars reticulata (SNr) in mice while monitoring their mov
108 nt evidence that the monkey substantia nigra pars reticulata (SNr) in the basal ganglia represents st
113 ia (BG) output nucleus, the substantia nigra pars reticulata (SNr) is well positioned to impact behav
114 l, a GABA agonist, into the substantia nigra pars reticulata (SNr) markedly reduced blink amplitude.
116 p recordings were made from substantia nigra pars reticulata (SNr) neurones in rat midbrain slices.
117 apped with non-dopaminergic substantia nigra pars reticulata (SNr) neurons and proximal dendrites of
118 ied altered the activity of substantia nigra pars reticulata (SNr) neurons of the basal ganglia (BG)
119 e activity and responses of substantia nigra pars reticulata (SNr) neurons to GABA, glutamate (GLU),
120 the operating room from the substantia nigra pars reticulata (SNr) of patients with PD undergoing dee
121 d the effects of DBS of the substantia nigra pars reticulata (SNr) on amygdala-kindled seizures.
122 , neurons downstream in the substantia nigra pars reticulata (SNr) only responded to Stop cues in tri
123 nglia output neurons in the substantia nigra pars reticulata (SNr) receive strong CARTir input from t
125 output neurons in the mouse substantia nigra pars reticulata (SNr) represent complex forelimb movemen
126 on dendrites located in the substantia nigra pars reticulata (SNr) than on those located in SNc, reve
127 histochemistry, in the rat substantia nigra pars reticulata (SNr) through an acute and persistent au
128 ation is transferred to the substantia nigra pars reticulata (SNr) through motor and medial prefronta
130 e, GABAergic neurons of the substantia nigra pars reticulata (SNr) tonically inhibit the target nucle
131 nd overlying brain, and the substantia nigra pars reticulata (SNr) were compared with contralateral b
132 lia model predicts that the substantia nigra pars reticulata (SNr) will inhibit locomotion and the gl
134 A projection neurons of the substantia nigra pars reticulata (SNr), a key basal ganglia output nucleu
135 pattern is expressed in the substantia nigra pars reticulata (SNr), a main target of striatal efferen
136 ve GABAergic neurons of the substantia nigra pars reticulata (SNr), a major output of the basal gangl
137 excitatory influence on the substantia nigra pars reticulata (SNR), a major output structure of the b
138 positive (PV(+)) neurons in substantia nigra pars reticulata (SNR), accompanied with anxiety-like beh
139 largest output nucleus, the substantia nigra pars reticulata (SNr), and delineate the organization an
140 a dual-output nucleus, the substantia nigra pars reticulata (SNr), and the presence of modulatory in
141 hin the basal ganglia [DLS, substantia nigra pars reticulata (SNr), and the thalamostriatal parafasci
142 peduncular nucleus (EP) and substantia nigra pars reticulata (SNr), as does the acute injection of le
143 their midbrain target, the substantia nigra pars reticulata (SNr), at E14 in the mouse with a robust
144 in GABAergic neurons of the substantia nigra pars reticulata (SNr), but not in DA neurons of the subs
145 their target neurons in the substantia nigra pars reticulata (SNr), internal pallidal segment, and su
146 output in the midbrain, the substantia nigra pars reticulata (SNr), shows movement-related neural act
147 ckground strain mice in the substantia nigra pars reticulata (SNr), subthalamic nucleus (STN), rostro
148 n of GABAergic cells in the substantia nigra pars reticulata (SNr), the main output of the basal gang
150 ), globus pallidus (GP) and substantia nigra pars reticulata (SNr), together with the ipsilateral fro
152 on 5-HT2C receptors in the substantia nigra pars reticulata (SNr), which in turn inhibits nigra pars
153 bil (Meriones unguiculatus) substantia nigra pars reticulata (SNr), whose "neuronal" phenotype was co
154 ~50% of GABA neurons in the substantia nigra pars reticulata (SNr), ~30% in the VTA, and ~70% in the
167 s of NS cell bodies in the pars compacta and pars reticulata (TH immunohistochemistry and Cresyl viol
168 ra (SN) consists of GABAergic neurons of the pars reticulata that inhibit thalamic neurons and provid
169 peractivity in both STN and substantia nigra pars reticulata, the main output structure of basal gang
170 output projections from the substantia nigra pars reticulata to the deep layers of the superior colli
171 ens and 5-HT release in the substantia nigra pars reticulata, using a common stimulation in a single
172 d to a lesser extent in the substantia nigra pars reticulata, while no hybridization signal was detec
173 A(DA) cells arises from the substantia nigra pars reticulata, with large contributions from the VTA a