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1 plex-partial seizures) or unimpaired (simple-partial seizures).
2  for tonic-clonic seizures than they are for partial seizures.
3 temporal and parietal regions than in simple partial seizures.
4  of 56 patients with drug-resistant, complex partial seizures.
5 y complex partial seizures and lastly simple partial seizures.
6  during complex-partial compared with simple-partial seizures.
7 ly greater in complex-partial than in simple-partial seizures.
8 set of obvious chronic recurrent spontaneous partial seizures.
9 cquired non-progressive cerebral lesions and partial seizures.
10 ity in up to 80% of patients with refractory partial seizures.
11 e epilepsy and medically intractable complex partial seizures.
12  lobe structures of 21 patients with complex partial seizures.
13 ages in 36 patients with intractable complex partial seizures.
14 ity in a rat model of focally evoked complex partial seizures.
15                          Among patients with partial seizures, 35% respond poorly to available medica
16           Epilepsies were most often complex partial seizures (47%) and more than half were nonconvul
17 tion, of cerebral tissue in 10 patients with partial seizures and acquired lesions and 30 patients wi
18 vulsive state expressed mainly by continuous partial seizures and even new bouts of generalized seizu
19  in opioid neurotransmission associated with partial seizures and higher cognitive function, we inves
20 reat infantile spasms and refractory complex partial seizures and is in clinical trials to treat addi
21 ly generalized seizures, followed by complex partial seizures and lastly simple partial seizures.
22 vely tissue organization in 22 patients with partial seizures and MCD, with 30 control subjects.
23  of pregabalin, a drug used for treatment of partial seizures and neuropathic pain.
24 es and acquired lesions and 30 patients with partial seizures and normal MRI.
25  Individual analyses of the 30 patients with partial seizures and normal optimal MRI identified a sig
26 st recently presented with continuous simple partial seizures and occasional breakthrough complex par
27 ed with SE during an unprovoked seizure were partial seizures and previous craniotomy.
28 imaging methods for identifying the cause of partial seizures, and can contribute to pre-surgical eva
29 , cingulate, and retrosplenial cortex during partial seizures, and increases in all of these regions
30 al and ictal states in patients with complex partial seizures, and the importance of the transition b
31 n, measured 2 hours after the first stage 2 (partial) seizure, appeared in neurons of the ipsilateral
32   Patients presenting with a history of only partial seizures are at low risk of subsequent tonic-clo
33 f the 10 patients with DNT had postoperative partial seizures arising in the ipsilateral hemisphere,
34 Epilepsy and Single Seizures) study had only partial seizures at randomisation.
35  and following seizures that impair (complex partial seizures) but not those that preserve (simple pa
36 onal activity and cerebral metabolism during partial seizures, but found increased neuronal activity
37 ed episodes of epigastric pain), and complex partial seizures consistent with temporal lobe epilepsy.
38 th no impairment of awareness (auras, simple partial seizures) continue, if there is a prior history
39  the frontal cortex during behaviorally mild partial seizures, contrasted with fast polyspike activit
40 sence, generalised tonic-clonic, and complex partial seizures, converge on the same set of anatomical
41  into temporal lobe structures cause complex partial seizures (CPS) and pathological changes observed
42                       We studied 328 complex partial seizures (CPS) in 63 consecutive patients with t
43 have also been proposed as models of complex partial seizures (CPSs) following traumatic brain injury
44                   We compared daily times of partial seizures determined by continuous electroencepha
45     The group did, however, continue to have partial seizures during follow-up, and modelling showed
46 ient developed and continues to have complex partial seizures every 2-3 months that are persistent de
47 seizures evoked by pentylenetetrazole (PTZ), partial seizures evoked from the forebrain, absence seiz
48 er presentation), one or more generalised or partial seizures (excluding febrile seizures), a new-ons
49 Secondarily generalized seizures and complex partial seizures exhibited increased slow waves distribu
50 between seizure type (generalized or complex partial), seizure frequency, age of onset and duration o
51                        Patients with complex partial seizures had lower GABA levels (1.03 mmol/kg of
52 ents who present with only simple or complex partial seizures have a poorly documented prognosis.
53 zure types and the rate of generalization of partial seizures, however rapid-eye-movement sleep seems
54  electroencephalography recordings during 63 partial seizures in 26 patients with surgically confirme
55 roved as an add-on drug for the treatment of partial seizures in adults with epilepsy.
56 or seizures in animals, analogous to complex partial seizures in humans, result in a consistent activ
57 s model mild absence seizures and/or complex partial seizures in humans, then an opportunity may exis
58                        This remote effect of partial seizures may cause impaired cerebral functions,
59 ture hippocampus, possibly providing a novel partial seizure model in the developing rat.
60 eizures) but not those that preserve (simple partial seizures) normal consciousness and responsivenes
61  anxiety disorder and adjunctive therapy for partial seizures of epilepsy.
62                                    Migrating partial seizures of infancy is an early onset epileptic
63        The study has revealed that migrating partial seizures of infancy is associated with an expand
64 We report the case of a child with migrating partial seizures of infancy secondary to an activating m
65             Fourteen children with migrating partial seizures of infancy were reported during the 2 y
66                                    Migrating partial seizures of infancy, also known as epilepsy of i
67 roencephalograms in 35 patients with complex partial seizures of temporal lobe origin who were seizur
68                     The differing effects of partial seizures on neurobehavioral recovery following a
69 ndent bilateral temporal lobe (IBTL) complex partial seizures on the intracranial electroencephalogra
70 alysis, reflecting the most common region of partial seizure onset.
71 e the frequency of further simple or complex partial seizures or a combination of both.
72 seizures and classified as impaired (complex-partial seizures) or unimpaired (simple-partial seizures
73 three different time periods: (i) during the partial seizure phase prior to generalization; (ii) duri
74                                              Partial seizures produce increased cerebral blood flow i
75  tachycardia (VT) in bipolar disorder (BPD), partial seizures (PSZ) and generalized tonic-clonic seiz
76                        Specifically, complex partial seizures require evaluation of the frontal lobes
77                                However, when partial seizures secondarily generalize, there are often
78 nts remained seizure free (apart from simple partial seizures [SPS]) at 5 years after surgery, and 47
79  with pharmacologically intractable, complex-partial seizures, surgical excision of the involved temp
80 ng number of genes have been identified with partial seizure syndromes.
81 pilepsy begins in adults with short-duration partial seizures that originate in the hippocampus.
82 erent types of chronic recurrent spontaneous partial seizures that worsen in frequency and duration o
83 ologically extended the neural mass model of partial seizures, the Epileptor, by including two neuron
84 c-clonic seizures (all patients) and complex partial seizures (three patients).
85 ormations of cortical development (MCDs) and partial seizures, to quantify the GABA(A)-central benzod
86 nisms of intractable epilepsy of the complex partial seizure type.
87                     All patients had complex partial seizures, underwent amygdalohippocampectomy, and
88            Finally, we observed that complex-partial seizures were somewhat more common with onset in
89 nimal clonic, maximal tonic, or psychomotor (partial) seizures were determined in 16 different inbred
90 d in the same ictal sites as obvious complex partial seizures, were electrographically similar to rat
91                  In patients with refractory partial seizures who are candidates for surgical treatme
92 eral temporal lobe epilepsy characterized by partial seizures with auditory disturbances.
93 seizures and occasional breakthrough complex partial seizures with postictal migraines.
94            The brain regions involved during partial seizures with secondary generalization have not
95              In agreement with findings from partial seizures without secondary generalization, cereb
96 ons of cerebral blood flow increases than in partial seizures without secondary generalization.
97                         In the subset of 253 partial seizures without secondary generalized convulsio