ライフサイエンスコーパス: particulate matter

1 ative potential (OP) of ambient PM(2.5) (PM, particulate matter).

2 ry OAs (SOAs) and filter extracts of ambient particulate matter.

3 nts for a large fraction of the tropospheric particulate matter.

4 for analytes ranging from small molecules to particulate matter.

5 resence of interfering agents such as diesel particulate matter.

6 ark of the fragility proposed for jamming in particulate matter.

7 enzene, nitrogen dioxide, 1,3-butadiene, and particulate matter.

8 ion and corresponding ground-level ozone and particulate matter.

9 imate the annual mean concentrations of fine particulate matter.

10 y inflammation following exposure to ambient particulate matter.

11 icles, contributing substantially to organic particulate matter.

12 ff-road sector and (b) an increase in PM2.5 (particulate matter 2.5 mum or less in diameter) emission

13 States of America feed yards, the resulting particulate matter (669,000 kg) could contain enough ins

14 Airborne particulate matter accommodates rich and dynamic microbi

15 Our goal was to evaluate fine particulate matter air pollution ([Formula: see text]) a

16 quivocal evidence of the causal role of fine particulate matter air pollution (PM(2.5), or particulat

17 shop, Reducing the Cardiopulmonary Impact of Particulate Matter Air Pollution in High Risk Population

18 erventions and viable approaches directed at particulate matter air pollution.

19 by an average of 27% through interception of particulate matter and absorption of gaseous pollutants.

20 l fibroblasts to different concentrations of particulate matter and analyzing the effects over time u

21 causal relationship between exposure to fine particulate matter and cardiovascular morbidity and mort

22 This scientific commentary refers to 'Particulate matter and episodic memory decline mediated

23 burning of tobacco in terms of emissions of particulate matter and gases, and the mechanisms by whic

24 ompounds are a significant component of fine particulate matter and haze in national parks and wilder

25 MLE to nicotine-free dry particulate matter and nicotine was significantly reduce

26 stimates of annual average exposures to fine particulate matter and nitrogen dioxide at the residenti

27 We examined multipollutant models (with fine particulate matter and nitrogen dioxide) and effect meas

28 models and adjustment for copollutants (fine particulate matter and nitrogen dioxide), although some

29 ngs provide new insights into how wood smoke particulate matter and other transient receptor potentia

30 elds annual premature deaths related to fine particulate matter and ozone (95CI: 25 000-120 000), hea

31 rk has examined the association between fine particulate matter and risk of kidney disease; however,

32 and release hazardous air pollutants such as particulate matter and volatile organic compounds in add

33 ricity generation are expected to reduce WTW particulate matters and sulfur oxide emissions for furth

34 emits more nitrogen oxides, sulfur dioxide, particulate matter, and heavy metals per unit of energy

35 ess of molecules, supramolecular structures, particulate matter, and pathogens into it.

36 cribe the variation of size spectrum of fine particulate matter, and the respective contributions of

37 dverse human health effects upon exposure to particulate matter are still poorly understood.

38 rating that methods originally developed for particulate matter can also successfully be applied to N

39 rescence microscopy was used to determine if particulate matter caused activation of the aryl hydroca

40 per unit mass or organic carbon) of sinking particulate matter collected in the North Pacific Subtro

41 ow that a nationwide 1 mug/m(3) reduction in particulate matter concentration would generate nearly $

42 Consequently, future fine particulate matter control efficiency will not be underc

43 sociation between long-term exposure to fine particulate matter, defined as particulate matter with a

44 physiology due to exposure to diesel exhaust particulate matter (DEP).

45 es of allergens, detergents, tobacco, ozone, particulate matter, diesel exhaust, nanoparticles, and m

46 thalate (DEHP), mainly through production of particulate matter during cooking events.

47 stent with the substantial tightening of the particulate matter emission limit value that typically f

48 s suspended nanoscale cerium oxide to reduce particulate matter emissions and increase fuel economy,

49 Particulate matter emissions from agricultural livestock

50 cular morbidity and mortality, and that fine particulate matter exposure is a modifiable cardiovascul

51 n (nitrogen dioxide and nitrogen oxides) and particulate matter (fine particulate matter with an aero

52 e sought to assess the role exposure to fine particulate matter ([Formula: see text]) during differen

53 The effects of exposure to fine particulate matter ([Formula: see text]) during wildland

54 frequently studied exposures in humans were particulate matter ([Formula: see text]), mercury ([Form

55 d 5-y running average concentrations of fine particulate matter ([Formula: see text]), nitrogen dioxi

56 ee text]), ozone ([Formula: see text]), fine particulate matter [Formula: see text] ([Formula: see te

57 ed that tides played a major role in forming particulate matter from estuarine sources at the creek m

58 ave received airborne virus, carried by fine particulate matter, from infected farms within the state

59 ion (TRAP) is made up of complex mixtures of particulate matter, gases and volatile compounds.

60 ncentrations of PM(10), PM(2.5), PM(coarse) (particulate matter >2.5-10 mum diameter), nitrogen dioxi

61 nstraints on this critical component of fine particulate matter; however, no previous collection meth

62 cidification, freshwater eutrophication, and particulate matter impacts.

63 gases such as methane and the production of particulate matter important for climate and human healt

64 ced severe hazes with high concentrations of particulate matter in recent years.

65 found indoors in the gas phase, in airborne particulate matter, in dust, and on surfaces.

66 to organic extracts of air pollution-derived particulate matter, including diesel or biodiesel exhaus

67 y aimed to evaluate whether exposure to fine particulate matter interferes with lung recovery and rem

68 Numerous studies have indicated that ambient particulate matter is closely associated with increased

69 of the size spectrum evolution of submicron particulate matter is critical to making efficient remed

70 Understanding the route by which particulate matter is formed from these reactions is a k

71 and it is a major contributor to atmospheric particulate matter known to have a deleterious respirato

72 community-level ozone, nitrogen dioxide, and particulate matter less than 10 mum (PM10) and less than

73 ioxide, sulfur dioxide, carbon monoxide, and particulate matter less than 2.5 mum were assessed by we

74 posure to traffic-related air pollutants and particulate matter less than 2.5 um in diameter (PM(2.5)

75 A likely bimodal association of particulate matter less than or equal to 10 mum in aerod

76 We analyzed the effect of preceding ambient particulate matter less than or equal to 10 mum in aerod

77 Nitrogen dioxide, sulfur dioxide, and particulate matter less than or equal to 2.5 mum in aero

78 Unexpectedly, extreme particulate matter levels simultaneously occurred in nor

79 ng-term ambient residential exposure to fine particulate matter < 2.5 microns (PM2.5) and physical ac

80 annual average ambient residential levels of particulate matter < 2.5 mum and < 10 mum in aerodynamic

81 Acute exposure to ambient particulate matter < 2.5 mum in aerodynamic diameter (PM

82 Chronic exposure to particulate matter < 2.5u (PM(2.5)) has been linked to c

83 Elevated levels of fine particulate matter <2.5 microm in aerodynamic diameter (

84 arios targeting nitrogen dioxide (NO(2)) and particulate matter <2.5 mum (PM(2.5)) in separate interv

85 ies, and personal exposure to air pollution (particulate matter <2.5 mum in aerodynamic diameter [PM(

86 First, O(3) and PM(2.5) (particulate matter <2.5 mum in diameter) are elevated at

87 Long-term exposure to particulate matter <2.5 um in diameter (PM(2.5)) and ozo

88 Physical activity and ambient particulate matter <= 2.5 mum in aerodynamic diameter (P

89 ld air pollution (HAP) in terms of estimated particulate matter <= 2.5 mum in aerodynamic diameter (P

90 ods: Individual exposures to source-specific particulate matter <=10 mum in aerodynamic diameter (PM(

91 llutant models accounting for coexposures to particulate matter <=2.5 mum in aerodynamic diameter (PM

92 l data were used to determine annual average particulate matter <=2.5 mum in aerodynamic diameter (PM

93 Exposure to ambient fine particulate matter (particulate matter <=2.5 mum in aerodynamic diameter (PM

94 articulate matter air pollution (PM(2.5), or particulate matter <=2.5 mum in diameter) in cardiovascu

95 t-term exposures to fine particulate matter (particulate matter <=2.5 um aerodynamic diameter; PM(2.5

96 majority of species observed in the sampled particulate matter may be described as benzenoids, consi

97 results highlight, for the first time, that particulate matter may contaminate gas condensates with

98 However, it is not known whether nanoscale particulate matter (nPM) with aerodynamic diameter <=200

99 smoke and are excluded from the nonvolatile particulate matter (nvPM) standard.

100 We investigate whether exposure to particulate matter of a diameter of <=2.5 mum (PM(2.5))

101 erage and maximal daily exposure to fine air particulate matter of diameter <=2.5 um (PM(2.5)) was ob

102 fects of a known component of air pollution (particulate matter) on human primary dermal fibroblasts.

103 or detrimental effects on health is for fine particulate matter (particles <=2.5 um in diameter (PM(2

104 st health impact assessments of ambient fine particulate matter (particles with an aerodynamic diamet

105 Exposure to ambient fine particulate matter (particulate matter <=2.5 mum in aero

106 arrhythmias and short-term exposures to fine particulate matter (particulate matter <=2.5 um aerodyna

107 The impact of chronic exposure to fine particulate matter (particulate matter with an aerodynam

108 ship between relatively low exposure to fine particulate matter (particulate matter with an aerodynam

109 tween short-term (or acute) exposure to fine particulate matter [particulate matter with aerodynamic

110 ndia, which contributes significantly to the particulate matter (PM < 2.5 mum) and CO levels in ambie

111 nd-use regression (LUR) models for submicron particulate matter (PM(1)) components from an urban area

112 Fine particulate matter (PM(2.5)) air pollution exposure is t

113 Fine particulate matter (PM(2.5)) air pollution has been reco

114 ty generation is a large contributor to fine particulate matter (PM(2.5)) air pollution.

115 isting scientific literature focuses on fine particulate matter (PM(2.5)) and carbon monoxide (CO).

116 nts of airborne metal concentrations in fine particulate matter (PM(2.5)) are important for understan

117 Accurate data concerning historical fine particulate matter (PM(2.5)) concentrations are needed t

118 study estimated annual average ambient fine particulate matter (PM(2.5)) concentrations at 1 km reso

119 easures achieved an average decrease of fine particulate matter (PM(2.5)) concentrations of ~14% in B

120 erican decreases in population-weighted fine particulate matter (PM(2.5)) concentrations since 2000 h

121 The ground-level particulate matter (PM(2.5)) downwind over New Delhi sho

122 Here we project ambient fine particulate matter (PM(2.5)) exposure in India for the y

123 We evaluate fine particulate matter (PM(2.5)) exposure-response models to

124 into the global public health burden of fine particulate matter (PM(2.5)) exposures frequently requir

125 sions of most pollutants that result in fine particulate matter (PM(2.5)) formation have been decreas

126 Exposure to fine particulate matter (PM(2.5)) from fuel combustion signif

127 obally regionalized approach for quantifying particulate matter (PM(2.5)) health impacts.

128 ations suggest that exposure to ambient fine particulate matter (PM(2.5)) is associated with increase

129 Exposure to ambient air particulate matter (PM(2.5)) is well established as a ri

130 g chronic health effects independent of fine particulate matter (PM(2.5)) mass is inconclusive.

131 We estimated fine particulate matter (PM(2.5)) mortality in the 250 most p

132 acts on carbon dioxide emissions and ambient particulate matter (PM(2.5)) related premature mortality

133 Exposures to ambient and household fine-particulate matter (PM(2.5)) together are among the larg

134 urban form determinants of decadal-long fine particulate matter (PM(2.5)) trends in all 626 Chinese c

135 Data on ambient fine particulate matter (PM(2.5)) was obtained from the Natio

136 surface of the chemical composition of fine particulate matter (PM(2.5)) would offer valuable inform

137 armful air pollutants such as ozone and fine particulate matter (PM(2.5))(4).

138 , particle-bound aromatic hydrocarbons, fine particulate matter (PM(2.5)), and ultrafine particles (r

139 We monitored particulate matter (PM(2.5)), black carbon (BC), A-weigh

140 n-use emissions of pollutants including fine particulate matter (PM(2.5)), organic and elemental carb

141 (CO(2)), carbon monoxide (CO), methane, fine particulate matter (PM(2.5)), PM(2.5) elemental carbon (

142 Therefore, we measured outdoor fine particulate matter (PM(2.5)), species of polycyclic arom

143 re increasingly a significant source of fine particulate matter (PM(2.5)), which has been linked to a

144 land fire is a major emission source of fine particulate matter (PM(2.5)), which has serious adverse

145 ntials, slightly lower than those of ambient particulate matter (PM(2.5)).

146 often dominant component of atmospheric fine particulate matter (PM(2.5)).

147 n increase in the atmospheric burden of fine particulate matter (PM(2.5)).

148 h-damaging pollutants such as ozone and fine particulate matter (PM(2.5)).

149 The impact of inhalable fine particulate matter (PM(2.5), aerodynamic diameter <2.5 m

150 Fine particulate matter (PM(2.5), aerodynamic diameter <=2.5

151 (NO(2): 60% with 95% CI 48 to 72%), and fine particulate matter (PM(2.5): 31%; 95% CI: 17 to 45%), wi

152 Particulate matter (PM) air pollution causes cardiopulmo

153 Exposure to ambient particulate matter (PM) air pollution is a leading risk

154 Long-term exposure to particulate matter (PM) air pollution is associated with

155 ssesses the effect of short-term exposure to particulate matter (PM) air pollution on human cognitive

156 ately linear short-term associations between particulate matter (PM) and mortality in Western communi

157 ent systems for control of emissions such as particulate matter (PM) and nitrogen oxides (NO(x)) to c

158 Our focus is on particulate matter (PM) and the associated low molecular

159 th risks associated with exposure to ambient particulate matter (PM) are a major concern around the w

160 Regulations on ambient particulate matter (PM) are becoming more stringent beca

161 Chemical forms of phosphorus (P) in airborne particulate matter (PM) are poorly known and do not corr

162 ology studies rely on concentrations of fine particulate matter (PM) as a smoke tracer.

163 xide (CO(2)), tropospheric ozone (O(3)), and particulate matter (PM) can be reduced by urban trees th

164 Knowing the damage that particulate matter (PM) can cause in skin is important f

165 Rationale: Higher indoor particulate matter (PM) concentrations are linked with i

166 Clean and particulate matter (PM) contaminated filters of a range

167 Particulate matter (PM) deposited on Platanus acerifolia

168 Exposure to fine particulate matter (PM) during pregnancy is associated w

169 Spatiotemporally resolved particulate matter (PM) estimates are essential for reco

170 The link between particulate matter (PM) exposure and adverse health outc

171 cts are well known, but associations between particulate matter (PM) exposure and mental illness have

172 fication of the association between maternal particulate matter (PM) exposure and preterm delivery (P

173 The metabolome of World Trade Center (WTC) particulate matter (PM) exposure has yet to be fully def

174 on of sulfate in cloudwater, contributing to particulate matter (PM) formation.

175 Air was sampled as particulate matter (PM) greater than 10 mum (PM > 10) an

176 Exposure to trace metals in airborne particulate matter (PM) has been linked to various adver

177 physical and chemical properties of airborne particulate matter (PM) have been extensively studied, t

178 Early life exposure to fine particulate matter (PM) in air is associated with infant

179 Particulate matter (PM) in atmospheric pollution contain

180 to improve our understanding of exposure to particulate matter (PM) in residences because of associa

181 arction in association with elevated ambient particulate matter (PM) in the previous hour(s).

182 Particulate matter (PM) is a complex mixture.

183 of respiratory infections to source-specific particulate matter (PM) is an area of active research.

184 Analysis of particulate matter (PM) is important for the assessment

185 , GDIs had, on average, a factor of 2 higher particulate matter (PM) mass emissions than PFIs due to

186 The adverse effects of ambient particulate matter (PM) on human health have been well d

187 Particulate matter (PM) pollution has become a serious p

188 Ambient particulate matter (PM) pollution is a major environment

189 tower PMS A003 sensors when exposed to eight particulate matter (PM) sources (i.e., incense, oleic ac

190 ize that the deposition velocity of airborne particulate matter (PM) to leaves is species-specific an

191 tress is a potential mechanism of action for particulate matter (PM) toxicity and can occur when the

192 tudies address health effects of atmospheric particulate matter (PM) using mass-based measurements as

193 Annual mean concentrations of particulate matter (PM) with a diameter < 2.5 mug/m3 (PM

194 ht noise (Lden); nitrogen dioxide (NO2); and particulate matter (PM) with aerodynamic diameter < 2.5

195 lution (nitrogen dioxide, nitrogen monoxide, particulate matter (PM) with an aerodynamic diameter <=1

196 els by adjusting for copollutants, including particulate matter (PM) with an aerodynamic diameter of

197 te associations between prenatal exposure to particulate matter (PM) with diameter [Formula: see text

198 ass burning is a major source of atmospheric particulate matter (PM) with impacts on health, climate,

199 pollutants including nitrogen oxides (NOX), particulate matter (PM), and components of PM were estim

200 information is available on total arsenic in particulate matter (PM), but little is known about the r

201 al exposure to mixtures of air pollutants of particulate matter (PM), PM(2.5), PM(10), nitrogen oxide

202 effects associated with exposure to ambient particulate matter (PM).

203 human health effects and exposure to ambient particulate matter (PM).

204 ole in adverse health effects of atmospheric particulate matter (PM).

205 bon monoxide (CO), carbon dioxide (CO2), and particulate matter (PM).

206 e atmosphere as mixtures attached to ambient particulate matter (PM).

207 proposed as a measure of toxicity of ambient particulate matter (PM).

208 e gas phase and adsorbed onto the surface of particulate matter (PM).

209 dent and joint effects of heatwaves and fine particulate matter [PM < 2.5 mum in aerodynamic diameter

210 We are measuring fine particulate matter [PM < 2.5 mum in aerodynamic diameter

211 we measured 48-h personal exposures to fine particulate matter [PM [Formula: see text] in aerodynami

212 hose with a diameter of approximately 1 mum (particulate matter, PM(1)) are released from fossil fuel

213 by 38.3 Tg carbon dioxide (CO(2)) and 0.6 Tg particulate matter (PM1), or 12.9% and 11.5%, respective

214 ever, the association between ambient coarse particulate matter (PM10; </=10 mum in aerodynamic diame

215 Pollutants measured included PM2.5 (PM = particulate matter), PM10, ultrafine particles, black ca

216 h has identified an association between fine particulate matter (PM2.5) air pollution and lung cancer

217 between short-term exposures to ambient fine particulate matter (PM2.5) and ozone, and at levels belo

218 ur personal exposure to log-transformed fine particulate matter (PM2.5) concentrations among cooks (b

219 nd long-term exposure to a low level of fine particulate matter (PM2.5) concentrations.

220 In adults, exposure to fine particulate matter (PM2.5) has been associated with redu

221 Reductions in ambient concentrations of fine particulate matter (PM2.5) have contributed to reduction

222 ion, up to 11 mug m(-3) for annual mean fine particulate matter (PM2.5) in northern Vietnam and up to

223 Fine particulate matter (PM2.5) is thought to be responsible

224 estimate premature mortality caused by fine particulate matter (PM2.5) pollution as a result of atmo

225 stigate the associations of exposure to fine particulate matter (PM2.5) with HRV as an indicator of c

226 reased inhalation exposures to PCBs and fine particulate matter (PM2.5), using disability-adjusted li

227 natal and childhood exposure to ambient fine particulate matter (PM2.5).

228 mal chow or a high-fat diet to airborne fine particulate matters (PM2.5), and then investigated the c

229 t-term exposure to main air pollutants (fine particulate matter [PM2.5] and ozone) and cause-specific

230 T(reg)) cells is necessary for allergens and particulate matter pollutants to promote airway inflamma

231 95% UI 113.1 to 247.7]), followed by ambient particulate matter pollution (88.5%, 66.4 to 116.4).

232 ptured a pattern related to urbanicity, with particulate matter pollution (PM(25)) inversely related

233 Exposure to fine particulate matter pollution (PM2.5) is hazardous to hea

234 Ambient fine particulate matter pollution (PM2.5) is one leading caus

235 ame time, evidence has accumulated that fine particulate matter pollution can exacerbate asthma sympt

236 rding to our estimates, recent reductions in particulate matter pollution in the USA have resulted in

237 sure, smoking, high-sodium diet, and ambient particulate matter pollution were among the leading four

238 Airborne particulate matter poses a serious threat to human healt

239 tions between increased respirable suspended particulate matter (RSPM) deposition and changes in bee

240 irborne microplastics >4.7 mum in an outdoor particulate matter sample obtained at an urban sampling

241 hrin was detected most frequently at >67% of particulate matter samples and at a mean concentration o

242 Of 16 different pesticides quantified in particulate matter samples collected from beef cattle fe

243 molecular-resolved comparison with airborne particulate matter samples from the nearby high-alpine r

244 ollected from a globally distributed surface particulate matter sampling network (SPARTAN) between Ja

245 Average concentrations of fine particulate matter smaller than 2.5 micrometers in diame

246 Air pollution involving particulate matter smaller than 2.5 mum in size (PM2.5)

247 bon dioxide (CO(2)), as well as carbonaceous particulate matter, specifically elemental carbon (EC) a

248 se of eDNA extracted from archived suspended particulate matter (SPM) for identifying fish species us

249 Suspended particulate matter (SPM) is present in the natural aquat

250 erosol (OOA) is a major component of ambient particulate matter, substantially impacting climate, hum

251 transport and pollutant chemistry of primary particulate matter, sulfur dioxide (SO(2)), nitrogen oxi

252 ns of acetic acid, formic acid, NO(2), O(3), particulate matter, sulfur dioxide, and total VOCs were

253 rom industrial cattle feeding operations via particulate matter, the objective of this study is to de

254 With the potential of air pollutants such as particulate matter to not only modulate the expression o

255 ersistent free radicals (EPFRs) in the total particulate matter (TPM) of mainstream and sidestream TP

256 cation of microplastics (>=2 mum) in ambient particulate matter, using different chemometric techniqu

257 Particulate matter was found to dose-dependently increas

258 osal exposes communities to heavy metals and particulate matter waste.

259 For fine particulate matter, we observed elevated and significant

260 pollution measurements (nitrogen oxides and particulate matter) were combined with data from permane

261 g carbon:phosphorus (C:P) ratios in settling particulate matter when APS declined.

262 These pollutants can be adsorbed to particulate matter which is present in the air or deposi

263 tion of the ceramic spheres into micron-size particulate matter, which provided an abrasive interface

264 hemistry and long-distance transport of fine particulate matter with a diameter less than 2.5 um (PM(

265 ijing, negative emotions occur when PM(2.5) (particulate matter with a diameter less than 2.5 um) inc

266 We pooled estimates by pollutant type (particulate matter with a diameter of <2.5 mum [PM2.5] o

267 al average residential ozone (O(3)) and fine particulate matter with a diameter of 2.5 mum or less (P

268 des (including nitrogen dioxide [NO(2)]) and particulate matter with a diameter of less than 2.5 mum

269 nce-specific third-trimester black carbon or particulate matter with a diameter of less than 2.5 mum

270 Evidence suggests exposure to particulate matter with aerodynamic diameter <2.5 mum (P

271 investigated the associations of exposure to particulate matter with aerodynamic diameter <= 10 mum (

272 nal and kitchen exposure assessments of fine particulate matter with aerodynamic diameter <= 2.5 mum

273 e relationship between long-term exposure to particulate matter with aerodynamic diameter <= 2.5 mum

274 acute) exposure to fine particulate matter [particulate matter with aerodynamic diameter [Formula: s

275 e to nitrogen dioxide ([Formula: see text]), particulate matter with aerodynamic diameter [Formula: s

276 to the pathogenesis due to exposure to fine particulate matter with aerodynamic diameter [Formula: s

277 trast, neither benzo(a)pyrene (BaP) nor fine particulate matter with aerodynamic diameter [Formula: s

278 Coarse particulate matter with aerodynamic diameter between 2.5

279 on: individual residential outdoor levels of particulate matter with aerodynamic diameters less than

280 onal pollutants (nitrogen dioxide [NO(2)] or particulate matter with an aerodynamic diameter <=2.5 mu

281 ely low exposure to fine particulate matter (particulate matter with an aerodynamic diameter <=2.5 mu

282 osure to fine particulate matter, defined as particulate matter with an aerodynamic diameter less tha

283 hort-term (2-day moving average) exposure to particulate matter with an aerodynamic diameter less tha

284 n of rural residential emissions to PM(2.5) (particulate matter with an aerodynamic diameter less tha

285 chronic exposure to fine particulate matter (particulate matter with an aerodynamic diameter less tha

286 We investigated the impact of particulate matter with an aerodynamic diameter less tha

287 rm associations of air pollution, defined as particulate matter with an aerodynamic diameter less tha

288 odynamic diameter of <=2.5 mum [PM(2.5)] and particulate matter with an aerodynamic diameter of <10 m

289 ue medication and their exposure to PM(2.5) (particulate matter with an aerodynamic diameter of <2.5

290 Particulate pollutants-including particulate matter with an aerodynamic diameter of <= 2.

291 itrogen oxides) and particulate matter (fine particulate matter with an aerodynamic diameter of <=2.5

292 kel, sulfur, silicon, vanadium, and zinc) in particulate matter with diameter <2.5 mum (PM(2.5)) usin

293 ., nitrogen oxides ([Formula: see text]) and particulate matter with diameter [Formula: see text] ([F

294 ducation, smoking in pregnancy, and prenatal particulate matter with diameter of <2.5 micrometers (PM

295 ality index and PM(2.5) concentrations (fine particulate matter with diameters equal or smaller than

296 Seasonal-mean concentrations of particulate matter with diameters smaller than 2.5 mum (

297 ution in the developing world, mostly due to particulate matter with diameters smaller than 2.5 um (P

298 to a multitude of health-hazardous airborne particulate matter with notable deposition in the fragil

299 (HBECs) treated with pneumotoxic wood smoke particulate matter (WSPM) and chemical agonists of each

300 ng and dysfunction due to World Trade Center-Particulate Matter (WTC-PM) exposure are not well descri