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1 should share a common haplotype spanning the paternal gene.
2 tion and coordinately regulate expression of paternal genes.
3 s the paternal epigenotype and expression of paternal genes.
4 nd spatial divergence (FLS) for maternal and paternal genes are derived.
5 ved to permit control of whether maternal or paternal genes are expressed.
6 ernal versus paternal ratio, suggesting that paternal genes are nearly fully activated by 7 DAP.
7  inactivating the PWS-SRO in oocytes so that paternal genes are silenced on the future maternal allel
8  between a maternal DDK factor and a non-DDK paternal gene, both of which have been mapped to the Ovu
9 e define a 465-kb candidate interval for the paternal gene by recombinant progeny testing.
10  reciprocal interaction between maternal and paternal genes, coordinating the allocation of resources
11        Low effective population size (Ne) of paternal genes due to polygyny and female-biased adult s
12             PWS is caused by a deficiency of paternal gene expression and AS is caused by a deficienc
13 utation paternally showed a complete loss of paternal gene expression and died neonatally.
14 eletion of 4.8 kb showed only a reduction in paternal gene expression and incomplete penetrance of ne
15                                   Absence of paternal gene expression from this region results in Pra
16  F1 crosses, suggesting a role for disrupted paternal gene expression in seed abortion that varies in
17 WS-IC mediates activation and maintenance of paternal gene expression in the 15q11-q13 region, with r
18 suggest that maternal epigenetic factors and paternal gene expression play important roles in the pos
19 restoration of feeding larval structures and paternal gene expression that have been lost in the evol
20 terns, resulting from predominantly European paternal gene flow.
21                      These data suggest that paternal genes have little, if any, effect on preterm de
22 sms include de novo gene mutations, aberrant paternal gene imprinting, or telomere/telomerase biology
23                  The lack of normally active paternal genes in 15q11-q13, as an outcome of either a p
24  allocation early in life, with silencing of paternal genes in offspring soliciting increased materna
25                                  The role of paternal genes in the etiology of preeclampsia appears t
26                Likewise, effective sizes for paternal genes may be higher than for diparentally inher
27                Our results indicate that the paternal gene pool of both groups is shaped by several s
28 t the majority of the Lingayat and Vokkaliga paternal gene pools are composed of four Y-chromosomal h
29  The results support the hypothesis that the paternal gene pools of Jewish communities from Europe, N
30  predicts that the individual's maternal and paternal genes will evolve separate infraorganismal iden
31 anisms by which imprinting and expression of paternal genes within the AS/PWS domain - such as MKRN3