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1 pproximately 25% of LQTS remains unexplained pathogenetically.
2  a similar array of features might be linked pathogenetically.
3  to identify putative circulating biomarkers pathogenetically and prognostically linked to bicuspid a
4 ter cases suggest that skeletal myxoid CS is pathogenetically distinct from its extraskeletal counter
5 e results show that FGPs in FAP patients are pathogenetically distinct from sporadic FGPs.
6 of therapies tailored to this clinically and pathogenetically distinctive subgroup of DLBCL.
7 temic lupus erythematous is a clinically and pathogenetically heterogeneous disease that has long cha
8 s-Danlos syndrome (EDS) is a genetically and pathogenetically heterogeneous group of disorders of whi
9               Acute transverse myelitis is a pathogenetically heterogeneous inflammatory disorder of
10 support the concept that STAT3 activation is pathogenetically important in ALCL cells by deregulating
11            Kaposin B enhances the release of pathogenetically important proinflammatory cytokines by
12                      TOLS represent a novel, pathogenetically important type of TLO that are in situ
13                   Ganglionic antibody may be pathogenetically important while the paraneoplastic anti
14                   Ulcerations appeared to be pathogenetically important, because bacteria localized p
15 perties, whose alteration in leukemia may be pathogenetically important.
16 migraine and HS suggesting the syndromes are pathogenetically independent.
17 ased serotonin transporter functioning being pathogenetically involved in OCD.
18                                These results pathogenetically linked thrombin to subpopulations of AD
19 protein 43 (TDP-43) are RNA-binding proteins pathogenetically linked to amyotrophic lateral sclerosis
20 terizes PMF, while the clinical phenotype is pathogenetically linked to ineffective hematopoiesis and
21 chia coli (ECOLI) PDC-E2 mimic that has been pathogenetically linked to PBC but lacks this motif has
22 nt Ca release was much more prevalent in the pathogenetically pivotal CD28(null) subset compared with
23 sis (ET), and primary myelofibrosis (PMF) as pathogenetically related myeloproliferative disorders (M
24 ic pulmonary arterial hypertension (IPAH) is pathogenetically related to low levels of the vasodilato
25 rus 8, is the causal agent of KS but is also pathogenetically related to several lymphoproliferative
26                              CMN and CFS are pathogenetically related, and it is likely that they rep
27 ase-IV (DPPIV/CD26) as a novel, specific and pathogenetically relevant biomarker of CD34(+)/CD38( hor
28 ma and TNF-alpha) secretion and to a lack of pathogenetically relevant compensatory responses to othe
29                          Genetic variants of pathogenetically relevant cytokines may influence the de
30 ptor (CAR) named T1E28z that targets several pathogenetically relevant ErbB dimers.
31 ise positional mapping and identification of pathogenetically relevant genes, we analysed of human ge
32 cted recent data that provide clinically and pathogenetically relevant insights or identify potential
33 d at least two genes (c-kit and PDGFRA) with pathogenetically relevant mutations have been identified
34 and illustrate the importance of delineating pathogenetically relevant subgroups within the "recurren
35 fication of novel diagnostic biomarkers, and pathogenetically relevant therapeutic targets.
36 tive kinase activation and are thought to be pathogenetically relevant, implicating FLT3 as a plausib
37 n between these two HSP gene products may be pathogenetically relevant.
38 an preneoplastic and neoplastic lesions that pathogenetically resemble human ovarian cancer.