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1 o be a part of the infection process of this pathogenic microorganism.
2 he detection of nucleic acids derived from a pathogenic microorganism.
3 e growth, differentiation and virulence of a pathogenic microorganism.
4 s for immediate and robust identification of pathogenic microorganisms.
5 residence to both beneficial and potentially pathogenic microorganisms.
6 sponds to diverse structures associated with pathogenic microorganisms.
7 nes encoding the enzyme are found in several pathogenic microorganisms.
8 abel-free detection of low concentrations of pathogenic microorganisms.
9 n effective defense response against various pathogenic microorganisms.
10 en to obtain information about commensal and pathogenic microorganisms.
11 ater and food quality monitoring for various pathogenic microorganisms.
12 epithelial surfaces colonized by potentially pathogenic microorganisms.
13 the pectin/pectate lyases from several plant pathogenic microorganisms.
14 tors for use as antibacterial agents against pathogenic microorganisms.
15 ) is a naturally occurring Ag common to many pathogenic microorganisms.
16 lity to infections caused by a wide range of pathogenic microorganisms.
17 important in the innate host defense against pathogenic microorganisms.
18 s the main reason for antigenic variation in pathogenic microorganisms.
19 sed molecular basis for silver resistance in pathogenic microorganisms.
20 e another important virulence determinant in pathogenic microorganisms.
21 ve been evaluated for their activity against pathogenic microorganisms.
22 ce determinants is an important attribute of pathogenic microorganisms.
23  in cytotoxicity and cytostasis against many pathogenic microorganisms.
24 to afford protection against colonization by pathogenic microorganisms.
25 y contribute to the survival of commensal or pathogenic microorganisms.
26 tracellular spaces of plants challenged with pathogenic microorganisms.
27  these flies may play in the transmission of pathogenic microorganisms.
28  safer because they did not contact the live pathogenic microorganisms.
29 oach to overcome antimicrobial resistance in pathogenic microorganisms.
30 host-microbiota commensalism and immunity to pathogenic microorganisms.
31 and indirect protection against infection by pathogenic microorganisms.
32 y gland and usually caused by infection with pathogenic microorganisms.
33 le in human disease by serving as vectors of pathogenic microorganisms.
34 ombined with (2) predominance of potentially pathogenic microorganisms.
35 in many important physiological functions in pathogenic microorganisms.
36 otection to the developing offspring against pathogenic microorganisms.
37 impact plant stability and interactions with pathogenic microorganisms.
38 -brain barrier (BBB) protects the brain from pathogenic microorganisms.
39 rmaceutical prescriptions and the underlying pathogenic microorganisms.
40 he human body from invasion by exogenous and pathogenic microorganisms.
41 issues due to toxic impurity such as dye and pathogenic microorganisms.
42      We have never halted the exploration of pathogenic microorganisms.
43 related to the presence of food spoilage and pathogenic microorganisms.
44 rs for the direct or indirect recognition of pathogenic microorganisms.
45  as a novel ecological niche for potentially pathogenic microorganisms.
46 o mediate protection against a wide range of pathogenic microorganisms.
47 nsing and distinguishing pathogenic from non-pathogenic microorganisms.
48 and cytotoxic activities with regard to some pathogenic microorganisms.
49 l activity against clinically isolated human pathogenic microorganisms.
50 MS) can be applied for the identification of pathogenic microorganisms.
51 R3-independent killing of a diverse array of pathogenic microorganisms.
52 iving leads to reduced childhood exposure to pathogenic microorganisms.
53 cies and cultivars and the identification of pathogenic microorganisms.
54 el to discourage ingestion of food harboring pathogenic microorganisms.
55 he determination of microcystin residues and pathogenic microorganisms.
56 ssential for innate immune responses against pathogenic microorganisms.
57 us commensal, environmental, and potentially pathogenic microorganisms.
58 esis of Clostridium difficile and many other pathogenic microorganisms.
59 nization (and more importantly infection) by pathogenic microorganisms.
60 complex array of defensive responses against pathogenic microorganisms.
61  the innate and adaptive immune responses to pathogenic microorganisms.
62     The same peptide motifs are contained in pathogenic microorganisms.
63 sks to human health, including chemicals and pathogenic microorganisms.
64 al surfaces serve as transmission routes for pathogenic microorganisms.
65 long-lived Ab responses that protect against pathogenic microorganisms.
66 t against the attack of foreign, potentially pathogenic, microorganisms.
67 er(13), host responsiveness to CDN-producing pathogenic microorganisms(11) and-potentially-for some i
68                               Interestingly, pathogenic microorganisms (28%) affected root lifespan m
69 to introduce a protective immune response to pathogenic microorganisms after mucosal colonization.
70 ic bottle sets (systems) were compared, more pathogenic microorganisms (again with the exception of S
71 serve as a habitat for breeding and refuging pathogenic microorganisms against stresses.
72    Mucosal sites are continuously exposed to pathogenic microorganisms and are therefore equipped to
73          Plant-mediated interactions between pathogenic microorganisms and arthropod herbivores occur
74 loped to identify and measure target DNAs of pathogenic microorganisms and eliminated the need of PCR
75 ence of periodontal pockets which can harbor pathogenic microorganisms and evoke a host response coul
76 e that occurs due to the interaction between pathogenic microorganisms and host defenses.
77 bations (East London cohort) for analysis of pathogenic microorganisms and inflammatory indices (sput
78  antibiotics that target iron acquisition in pathogenic microorganisms and is especially effective ag
79 d NO work together in mediating responses to pathogenic microorganisms and microbe-associated molecul
80 ing technologies for the characterization of pathogenic microorganisms and monitoring of their global
81        Skin protects humans from invasion by pathogenic microorganisms and provides a home for divers
82 s the approaches to the genome annotation of pathogenic microorganisms and the available popular geno
83 ctions reveal the evolutionary properties of pathogenic microorganisms and the dynamic relationships
84 rsatile role of mucins at the interface with pathogenic microorganisms and the microbiome, and sparks
85 and particularly in the relationship between pathogenic microorganisms and their host that involves p
86 mes, glycopeptide fragments of collagen, and pathogenic microorganisms and thus reduces damage follow
87  innate immune system senses the invasion of pathogenic microorganisms and tissue injury through Toll
88 nced to simultaneously prevent infections by pathogenic microorganisms and tolerate the endogenous fl
89                                Intracellular pathogenic microorganisms and toxins exploit host cell m
90                                              Pathogenic microorganisms and toxins have evolved a vari
91 ctivities for antimicrobial activity against pathogenic microorganisms, and assessed structure-functi
92  debris, allergens, pollutants, commensal or pathogenic microorganisms, and respiratory viruses.
93 ve immune response against a wide variety of pathogenic microorganisms, and they represent the protot
94                                              Pathogenic microorganisms are considered to a major thre
95 cin (1) resistance proteins FosA and FosX in pathogenic microorganisms are related to a catalytically
96                  The germ theory states that pathogenic microorganisms are responsible for causing in
97                                              Pathogenic microorganisms are responsible for many infec
98 y, presumed to contribute in defense against pathogenic microorganisms as plants with higher levels o
99 n many species as a receptor for potentially pathogenic microorganisms as well as IgE.
100                                         Some pathogenic microorganisms as well as members of the comm
101     The human body is exposed to potentially pathogenic microorganisms at barrier sites such as the s
102 nfirmatory, and label-free identification of pathogenic microorganisms at the single-cell level.
103  orchestrating host immune responses against pathogenic microorganisms at this site.
104 o heavy metals, organic micropollutants, and pathogenic microorganisms attract stakeholder concern.
105 in microorganisms, and also the virulence of pathogenic microorganisms, because cooperative traits su
106 otic resistance are not only associated with pathogenic microorganisms, but are also found in non-pat
107 hoglycerate isomers is catalysed in numerous pathogenic microorganisms by a cofactor-independent muta
108 g extracellular effector proteins from plant pathogenic microorganisms by combining data mining of ex
109 d by neutrophils that inhibits the growth of pathogenic microorganisms by sequestering essential meta
110            It inhibits the growth of various pathogenic microorganisms by sequestering the transition
111 the human host, the fitness of commensal and pathogenic microorganisms can be constrained by both met
112         The cellular attachment and entry of pathogenic microorganisms can be facilitated by the expr
113 ex gastrointestinal microbial community by a pathogenic microorganism causes reproducible and signifi
114 nactive against a wide range of commensal or pathogenic microorganisms comprising panels of 25 aerobi
115 epatocyte mass, and freedom from potentially pathogenic microorganisms could be assured.
116 ogical community of commensal, symbiotic and pathogenic microorganisms, defined as the gut microbiota
117                          The fitness of most pathogenic microorganisms depends on transmission from h
118         In addition, we have achieved simple pathogenic microorganism detection without a laborious s
119 regulated by interactions with commensal and pathogenic microorganisms, environmental exposures, and
120  inactivation of enveloped viruses and other pathogenic microorganisms for vaccine application.
121 ented aerobic FA (FA) medium for recovery of pathogenic microorganisms from adult patients with bacte
122 BACTEC Plus Aerobic/F medium for recovery of pathogenic microorganisms from adult patients with bacte
123 ove drinking water quality by removing human pathogenic microorganisms from contaminated water.
124      This limits biological insight, and for pathogenic microorganisms hampers the development of new
125 ection of single-nucleotide polymorphisms in pathogenic microorganisms has normally been carried out
126     Although a wealth of studies focusing on pathogenic microorganisms has revealed much about the ra
127                                              Pathogenic microorganisms have apparently evolved defenc
128 asing number of complete genome sequences of pathogenic microorganisms have become available.
129                           Several species of pathogenic microorganisms have developed strategies to s
130                                         Many pathogenic microorganisms have evolved hemoglobin-mediat
131                 To cause diseases in plants, pathogenic microorganisms have evolved mechanisms to del
132                                         Many pathogenic microorganisms have evolved tactics to modula
133 tem cells as a defense mechanism against the pathogenic microorganisms, have the ability to damage nu
134  the patients with neutrophilic asthma had a pathogenic microorganism in BAL culture, which suggested
135 L) was sufficient to mimic the 95% growth of pathogenic microorganisms in 24 h.
136 esponses can in turn affect the commensal or pathogenic microorganisms in a feed-forward circle.
137 isite for reliable and specific detection of pathogenic microorganisms in a microfluidic chip.
138 toring to detect carbapenemase activity from pathogenic microorganisms in a rapid and quantitative ma
139     The efficient and sensitive detection of pathogenic microorganisms in aqueous environments, such
140 be part of inflammatory responses induced by pathogenic microorganisms in cancer, but not nonmalignan
141 ve for applications such as the detection of pathogenic microorganisms in food and water.
142 or the rapid extraction and concentration of pathogenic microorganisms in food samples, providing a m
143  antimicrobial activity against spoilage and pathogenic microorganisms in food.
144 [LYKs]) mediate recognition of symbiotic and pathogenic microorganisms in plants.
145 se but also humoral immune responses against pathogenic microorganisms in several animal models.
146                        Long-term survival of pathogenic microorganisms in streams enables long-distan
147                               In addition to pathogenic microorganisms in the biofilm, genetic and en
148 al, and oral mucosa, whereas the presence of pathogenic microorganisms in the dermis or lungs elicits
149 with quite an efficient defence against some pathogenic microorganisms in the event of their penetrat
150 em will respond to diet, the microbiota, and pathogenic microorganisms in the future.
151 imary physical barrier against commensal and pathogenic microorganisms in the gastrointestinal (GI) t
152 users, such as the potential inactivation of pathogenic microorganisms in waste and the recovery of r
153 concentration together with the emergence of pathogenic microorganisms including Haemophilus, Moraxel
154 al activity against twelve common nosocomial pathogenic microorganisms including Staphylococcus aureu
155  are susceptible to contamination of various pathogenic microorganisms, including bacteria and viruse
156                              For a subset of pathogenic microorganisms, including Streptococcus pneum
157 ats are known reservoirs for a wide range of pathogenic microorganisms, including viruses, bacteria,
158 udies on the interactions between plants and pathogenic microorganisms indicate that the processes of
159                   Moreover, the diversity of pathogenic microorganisms inhibited and/or killed by chl
160            However, during an infection when pathogenic microorganisms invade the host, our knowledge
161         The multidrug resistance of numerous pathogenic microorganisms is a serious challenge that ra
162 ring the earliest possible identification of pathogenic microorganisms is critical for selecting the
163                  Antibiotic resistance among pathogenic microorganisms is emerging as a major human h
164 istance gene-dependent disease resistance to pathogenic microorganisms is mediated by genetically sep
165 responses to polysaccharides associated with pathogenic microorganisms is of importance for improving
166 a depuration step that aims to nullify their pathogenic microorganism load and decrease chemical cont
167     When transitioning from the environment, pathogenic microorganisms must adapt rapidly to survive
168                                     As such, pathogenic microorganisms must evade recognition by TLRs
169                                              Pathogenic microorganisms must precisely regulate morpho
170   Transgenic plants expressing antigens from pathogenic microorganisms offer many advantages as low-c
171                                              Pathogenic microorganisms often have the ability to atta
172 stress response mechanisms used by different pathogenic microorganisms often involved in food-borne d
173                                              Pathogenic microorganisms often reside in glycan-based b
174  infections as a result of the dependence of pathogenic microorganisms on iron.
175 omonas syringae and P. digitatum, the common pathogenic microorganisms on pears.
176 by the consumption of food contaminated with pathogenic microorganisms or their toxins have very seri
177                                  Invasion of pathogenic microorganisms or tissue damage activates inn
178 e epithelial layer and in protection against pathogenic microorganisms, overproduction of NO has been
179  a key role in host resistance to a range of pathogenic microorganisms, particularly during the initi
180 al diseases (PDT) and (antibiotic-resistant) pathogenic microorganisms (PDI).
181 Food resources contaminated with spoilage or pathogenic microorganisms pose severe problems to all hi
182 s (HR, 1.2 [95% CI, 1.1-1.5]), and number of pathogenic microorganism (PPM) isolations (HR, 1.1 [95%
183 antimicrobial resistance to cancer cells and pathogenic microorganisms, respectively.
184                                     Numerous pathogenic microorganisms secrete small molecule chelato
185                                         Many pathogenic microorganisms subvert TLR signaling pathways
186                                              Pathogenic microorganisms such as Bordetella pertussis a
187      Infectious plant diseases are caused by pathogenic microorganisms such as fungi, bacteria, virus
188 ate immune response is a key barrier against pathogenic microorganisms such as human immunodeficiency
189                                        Since pathogenic microorganisms such as Staphylococcus aureus
190      Infectious plant diseases are caused by pathogenic microorganisms, such as fungi, oomycetes, bac
191                                              Pathogenic microorganisms, such as herpes simplex virus,
192                                      Indeed, pathogenic microorganisms target many of these molecules
193 , sometimes redundant, mechanisms to contain pathogenic microorganisms that are always evolving to ev
194                                              Pathogenic microorganisms that form biofilms are very di
195  virulence factor expression is critical for pathogenic microorganisms that must sense and adapt to a
196 re evaluated against a panel of prototypical pathogenic microorganisms: the Gram-positive Enterococcu
197 ated microbiota dispersal generally focus on pathogenic microorganisms; the dispersal of beneficial m
198  to mediate the direct killing of a range of pathogenic microorganisms through an as-yet-undefined me
199 ng edge of locomoting cells and rocketing of pathogenic microorganisms through host cell cytoplasm.
200  also subject to intrusions of allochthonous pathogenic microorganisms through pollution and runoff.
201                   Humans have coexisted with pathogenic microorganisms throughout its history of evol
202                            The attachment of pathogenic microorganisms to host cells and tissues is o
203 requires modification to improve recovery of pathogenic microorganisms to make it competitive with ot
204                 Type IV secretion is used by pathogenic microorganisms to transfer effector macromole
205 and pharmaceuticals) and in the detection of pathogenic microorganisms, toxic agents, and pesticides
206                                              Pathogenic microorganisms use Darwinian processes to cir
207                                    Growth of pathogenic microorganisms was detected earlier in VITAL
208                                    Growth of pathogenic microorganisms was detected earlier in VITAL
209 h a density and surface charge comparable to pathogenic microorganisms were found to be mobile in gro
210                                              Pathogenic microorganisms were isolated in 42.3% of pati
211  more often from VITAL aerobic bottles, more pathogenic microorganisms were recovered from BACTEC NR6
212                                         More pathogenic microorganisms were recovered from VITAL anae
213 significantly decreased, but OTU richness of pathogenic microorganisms were significantly increased i
214 nd iron availability is a signal that alerts pathogenic microorganisms when they enter the hostile ho
215 f virulence-associated surface structures on pathogenic microorganisms, which prevents host humoral i
216 vironments, there are numerous phenotypes of pathogenic microorganisms, which vary considerably in ch
217 ed fat, cholesterol, lactose, estrogens, and pathogenic microorganisms, while displacing fiber, compl
218                 Previous studies have linked pathogenic microorganisms with decreased allograft toler
219 is largely due to the increasing presence of pathogenic microorganisms with resistance to existing an
220        MacB represents an ABC transporter in pathogenic microorganisms with unique structural feature
221  host strategy for domesticating potentially pathogenic microorganisms without killing them.

 
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