ライフサイエンスコーパス: pathological condition

1 vascular resistance under physiological and pathological condition.

2 l for understanding neural plasticity in any pathological condition.

3 control of ghrelin response under normal and pathological conditions.

4 S-nitrosylation under both physiological and pathological conditions.

5 is, but RM s rescued mice from AAM -mediated pathological conditions.

6 isequilibrium in transcriptomes resulting in pathological conditions.

7 e signaling and its regulation in normal and pathological conditions.

8 mitigate hyperactive stress responses under pathological conditions.

9 oring an organ's biochemical omic changes in pathological conditions.

10 vascular remodeling, under physiological and pathological conditions.

11 of the human brain, under physiological and pathological conditions.

12 ain barrier (BBB), claudin-1 is expressed in pathological conditions.

13 vity that may be relevant in both normal and pathological conditions.

14 s are often early indicators of the onset of pathological conditions.

15 ution views of Tau in both physiological and pathological conditions.

16 rimary rat neurons in both physiological and pathological conditions.

17 pmental stages, under both physiological and pathological conditions.

18 prevent excessive sprouting angiogenesis in pathological conditions.

19 y be relevant to control BBB permeability in pathological conditions.

20 and dynamics under various physiological and pathological conditions.

21 tions for neuromuscular integrity in various pathological conditions.

22 neurons are sufficient to produce pain under pathological conditions.

23 ular metabolism under both physiological and pathological conditions.

24 neurodegenerative diseases as well as other pathological conditions.

25 ns are substantially reorganized during many pathological conditions.

26 ainst pathogens and whose imbalance leads to pathological conditions.

27 the therapeutic spectrum of HU to additional pathological conditions.

28 hondria and released into bloodstream during pathological conditions.

29 hese RNAs must remain unrecognized under non-pathological conditions.

30 e mTORC2 kinase under both physiological and pathological conditions.

31 n be targeted to ameliorate dendrite loss in pathological conditions.

32 ggable target to modulate angiogenesis under pathological conditions.

33 may be inadequate and misleading under many pathological conditions.

34 y thus be a useful tool to study healthy and pathological conditions.

35 during a broad spectrum of physiological and pathological conditions.

36 programs of CNS myelination under normal and pathological conditions.

37 ally modulating a wide-range of inflammatory pathological conditions.

38 bute to prefrontal function under normal and pathological conditions.

39 e cell injury or activation in patients with pathological conditions.

40 involved in a plethora of physiological and pathological conditions.

41 ological processes in both physiological and pathological conditions.

42 e microenvironment of both physiological and pathological conditions.

43 mitochondrial function to physiological and pathological conditions.

44 onses to a wide variety of physiological and pathological conditions.

45 resulting in considerable tissue damage and pathological conditions.

46 s for memory storage under physiological and pathological conditions.

47 or regulators of T cell responses in several pathological conditions.

48 ancer and an increasing number of identified pathological conditions.

49 dvanced glycation end products under certain pathological conditions.

50 ession and activity in OA chondrocytes under pathological conditions.

51 -cell communication in normal physiology and pathological conditions.

52 ecular role of these antibodies in different pathological conditions.

53 th following a thrombotic event in normal or pathological conditions.

54 nt cellular lineages under physiological and pathological conditions.

55 erstanding of how these factors change under pathological conditions.

56 distances, are imperative in both normal and pathological conditions.

57 e is known about how this profile changes in pathological conditions.

58 tides significantly associated with discrete pathological conditions.

59 d metabolism signifies a key element in many pathological conditions.

60 lose during normal development as well as in pathological conditions.

61 involved in executive function in normal and pathological conditions.

62 enesis of delayed mucosal healing in certain pathological conditions.

63 nd emerges as an important regulator of many pathological conditions.

64 pendent regulation in both physiological and pathological conditions.

65 ates anti-inflammatory actions under various pathological conditions.

66 pment and their implications in a variety of pathological conditions.

67 tein secretion and autophagy under stress or pathological conditions.

68 c lineage in vivo, both in physiological and pathological conditions.

69 lar activities, physiological processes, and pathological conditions.

70 ing the cardiac cycle under physiological or pathological conditions.

71 of the Kv7.4 channel under physiological and pathological conditions.

72 omes for degradation under physiological and pathological conditions.

73 for beta-A detection under physiological and pathological conditions.

74 ity, leading to autoimmune diseases or other pathological conditions.

75 ortical circuit function under normal and/or pathological conditions.

76 ead to uncontrolled cell division in several pathological conditions.

77 chemerin/ChemR23 system in physiological and pathological conditions.

78 However, this relationship is reversed under pathological conditions.

79 tors of immune responses in cancer and other pathological conditions.

80 maging due to its upregulation under various pathological conditions.

81 lly validated therapeutic target for several pathological conditions.

82 sculature have now been uncovered in several pathological conditions.

83 d proteases in patient samples from multiple pathological conditions.

84 at are present in the CNS in homeostatic and pathological conditions.

85 meability, with potential implications under pathological conditions.

86 P dynamics under different physiological and pathological conditions.

87 support or increase sGC activity in various pathological conditions.

88 citation-contraction coupling in healthy and pathological conditions.

89 ge dynamics during various physiological and pathological conditions.

90 in but is also secreted in physiological and pathological conditions.

91 l component of the baroreflex is affected in pathological conditions.

92 release under a variety of physiological and pathological conditions.

93 hondrial homeostasis under physiological and pathological conditions.

94 logy in the healthy nervous system and under pathological conditions.

95 atable by a broad range of physiological and pathological conditions.

96 ocifensive behaviour under physiological and pathological conditions.

97 hromboembolic events under physiological and pathological conditions.

98 sense and adapt to dynamic physiological and pathological conditions.

99 RNA networks in vivo under physiological and pathological conditions.

100 t-output properties of neurons in normal and pathological conditions.

101 complex microglial activation process under pathological conditions.

102 s, and its aberrant uptake is linked to many pathological conditions.

103 has been implicated in several noninfectious pathological conditions.

104 ession programs under both physiological and pathological conditions.

105 bases of consciousness in physiological and pathological conditions.

106 biological filaments under physiological and pathological conditions.

107 ons of adult-born neurons and their roles in pathological conditions.

108 tarsometatarsal joint (Lisfranc) complexes, pathological conditions affecting capsuloligamentous str

109 phy (MRN) has been used extensively to study pathological conditions affecting the peripheral nervous

110 progression and undesired inflammation under pathological conditions, against reference datasets that

111 hereas virulent B. pertussis caused a severe pathological condition and death in these mice, even at

112 However, the distinction between the pathological condition and the benign one is very challe

113 tion of LIF in the pancreas is restricted to pathological conditions and correlates with PDAC pathoge

114 ture of redox stress in cancers and in other pathological conditions and could potentially be used fo

115 ervous system (CNS) that become activated in pathological conditions and determine the fate of other

116 rial dysfunction has been implicated in many pathological conditions and diseases.

117 curs in a diverse range of physiological and pathological conditions and is driven by a conserved set

118 to the role of mitochondria under normal and pathological conditions and point to spatially regulated

119 rophages develop mixed phenotypes in complex pathological conditions and polarize to a predominant ph

120 mechanism to reduce intracellular tau under pathological conditions and that effective tau immunothe

121 essential since it underlies a multitude of pathological conditions and therapeutic interventions.

122 perties of a cell reflect its biological and pathological conditions and there have been active resea

123 genesis in mice during development and under pathological conditions and to explore the underlying mo

124 ulating angiogenesis in physiological versus pathological conditions and to the alternative compensat

125 signaling is attenuated but activated under pathological conditions, and how low-level signaling und

126 ptations to changes in external environment, pathological conditions, and nutrient deprivation.

127 holamines also play important roles in other pathological conditions, and specific agonists and antag

128 uclear alterations are often associated with pathological conditions as in Hutchinson-Gilford progeri

129 ism and contribute to both physiological and pathological conditions as second messengers.

130 very suggests a continuous role of EVs under pathological conditions as well as during routine cognit

131 A complete understanding of pathological conditions as well as normal brain function

132 s, which has been implicated in diseases and pathological conditions associated with delayed OP toxic

133 s involved as a radical scavenger in several pathological conditions associated with oxidative stress

134 Under pathological conditions, ATP synthase hydrolyzes ATP to

135 cell release may have applications in other pathological conditions beyond organ transplantation.

136 te (GFR) of individual kidneys in normal and pathological conditions, broadening the biomedical appli

137 olic shifts in physiological adaptations and pathological conditions, but may be influenced by the nu

138 ylation has been observed in podocytes under pathological conditions, but the molecular mechanism lin

139 rsors of neuromelanin (NM) pigment and under pathological conditions can modify and damage macromolec

140 In different pathological conditions cardiac lymphatics undergo signi

141 s on the effects of periodontal disease on 3 pathological conditions: cardiovascular disease, diabete

142 Alcoholic liver disease is a pathological condition caused by overconsumption of alco

143 s, cell competition may occur in a number of pathological conditions.Cell competition causes the remo

144 A cataract is a pathological condition characterized by the clouding of

145 pharmacological agents for the treatment of pathological conditions characterized by impaired mitoch

146 or novel pharmacological approaches in those pathological conditions characterized by persistent immu

147 ocytes (control condition) and glioma cells (pathological condition) clearly distinguished normal ver

148 released from organs under non-proliferative pathological conditions, correlating with the degree of

149 lated by calcium, and it can be perturbed by pathological conditions (e.g., myopathies), physiologica

150 ation pathway that protects cells exposed to pathological conditions from stress-induced tissue damag

151 s to nuclear mechanical state in physio- and pathological conditions, furthering our understanding of

152 se-specific patterns of retinal cell loss in pathological conditions has been highlighted by the emer

153 and TFH cells to autoantibody profiles under pathological conditions has not been thoroughly investig

154 has been observed in many physiological and pathological conditions; however, a simple method to sor

155 ammatory diseases whose activity can lead to pathological conditions if unregulated.

156 How a pathological condition in a given tissue influences syst

157 napse development and function in normal and pathological conditions in a cell-specific manner.SIGNIF

158 helial Wnt signaling and BBB integrity under pathological conditions in adult mice.

159 that anti-MPER specificity arises only under pathological conditions in autoantibodies endowed with s

160 inical tests to assess the manifestations of pathological conditions in blood.

161 hysiological perturbations and play roles in pathological conditions in macrophages.

162 al, mechanical, or chemical means alleviated pathological conditions in rat colitis and hypertension

163 tes generated from stem cells and exposed to pathological conditions in rodent brain or cell culture

164 HDAC6 dysfunction is associated with several pathological conditions in the central nervous system.

165 glia markers at some developmental stages or pathological conditions, in particular during chronic ne

166 nd microRNAs are involved in numerous physio-pathological conditions included cancer.

167 erapeutic applicability in a wide variety of pathological conditions including asthma, cancer, erecti

168 biogenesis have been associated with various pathological conditions including cancers.

169 is correlated with the incidence of various pathological conditions including depression, diabetes a

170 at contributes to the progression of several pathological conditions including diabetes, cancer, neur

171 to various detrimental cellular functions in pathological conditions including diabetic retinopathy (

172 serve as non-invasive biomarkers in several pathological conditions including inflammatory diseases.

173 cumulative stressors can lead to an array of pathological conditions including posttraumatic stress d

174 sses, and have also been implicated in human pathological conditions (including cancer).

175 n this mechanism are responsible for several pathological conditions, including acute pancreatitis.

176 roteasome system, is associated with various pathological conditions, including acute tissue damage,

177 etimes NDM appears in association with other pathological conditions, including autoimmune diseases.

178 TGF-beta1 is involved in many pathological conditions, including autoimmune disorders,

179 namics play an important role within several pathological conditions, including cancer and neurologic

180 ction has been associated with several human pathological conditions, including cancer, aging, and ne

181 uption of these blood vessels as observed in pathological conditions, including cancer, diabetes, str

182 leading to development without the onset of pathological conditions, including cancer.

183 tion of protein under many physiological and pathological conditions, including cancer.

184 e been implicated in the development of many pathological conditions, including cancer.

185 erous physiological processes and in various pathological conditions, including cardiovascular diseas

186 srupted sleep increases the risk of multiple pathological conditions, including cardiovascular diseas

187 Hypoxia occurs in many pathological conditions, including chronic inflammation

188 reciate the role of necroptosis in different pathological conditions, including critical illnesses.

189 of genes involved in many physiological and pathological conditions, including development, metaboli

190 ing is involved in various physiological and pathological conditions, including development, tumorige

191 and oxygen species is implicated in certain pathological conditions, including diabetes mellitus.

192 ADAMTS-16 is implicated in a number of pathological conditions, including hypertension, cancer

193 e of various selenoproteins under normal and pathological conditions, including in pancreatic beta-ce

194 ing but also with several other, age-related pathological conditions, including inflammation, vascula

195 ty and brain repair, is altered by aging and pathological conditions, including metabolic disorders.

196 ronic, sustained inflammation underlies many pathological conditions, including neurodegenerative dis

197 PCAT activity may be potentially involved in pathological conditions, including nonalcoholic fatty li

198 ntal inflammation is associated with several pathological conditions, including stillbirth and fetal

199 articipate in a variety of physiological and pathological conditions, including tumor suppression [2]

200 ng evidence has demonstrated that peripheral pathological conditions induce physiological regulation

201 Under pathological conditions induced by chronic catecholamine

202 e 2 (MK2) pathway is involved in a series of pathological conditions (inflammation diseases and metas

203 Although multiple acute and chronic lung pathological conditions involve perturbations in sphingo

204 t of metabolic alterations on acetylation in pathological conditions is a significant challenge.

205 possible contribution of aberrant R-loops to pathological conditions is also discussed.

206 ion of secreted metabolites under normal and pathological conditions is critical for understanding th

207 hanism of how mTORC1 activation occurs under pathological conditions is not completely known.

208 r, its effect on cartilage homeostasis under pathological conditions is not known.

209 corneal tissue structure in homeostatic and pathological conditions is unknown.

210 deacetylation in normal brain functions and pathological conditions is unquestionable, yet the molec

211 let hyperreactivity, which is common in many pathological conditions, is associated with increased at

212 inger and/or consequence of stress, age, and pathological conditions, is emerging as a novel concept

213 In pathological conditions, it forms fibrils that are molec

214 hyaluronan in animal cells are indicators of pathological conditions like infection or malignancy.

215 invade the central nervous system only under pathological conditions like tumor formation.

216 HRH would be beneficial for life-threatening pathological conditions, like cardiac hypertrophy and he

217 Under pathological conditions, matrix metalloproteinases (MMPs

218 neous distribution of RyR cluster size under pathological conditions may potentiate Ca waves and thus

219 logy of this interface and how it changes in pathological conditions may provide new treatments for v

220 l ganglia output during motor learning or in pathological conditions may render this pathway effectiv

221 A wide spectrum of pathological conditions may result from the interaction

222 ation of tissues, cell divisions, aging, and pathological conditions observed in many cancers.

223 Apolipoproteins are involved in pathological conditions of Alzheimer's disease (AD), and

224 Under pathological conditions of dysregulated Ca(2+) cycling,

225 he CNS is relevant to many physiological and pathological conditions of the CNS.

226 epresents a potential therapeutic target for pathological conditions of the muscle in which the stem

227 nsitive to elevated extracellular ATP during pathological conditions of urinary bladder inflammation

228 heir contribution to an increasing number of pathological conditions, opens a plethora of medicinal c

229 ength of these currents becomes larger under pathological conditions or drug administrations.

230 cient bone vasculature is a key component in pathological conditions ranging from developmental skele

231 Diagnosis and therapeutic interventions in pathological conditions rely upon clinical monitoring of

232 membrane homeostasis under physiological and pathological conditions remain largely unknown.

233 tational activity is regulated in normal and pathological conditions remains largely unknown.

234 s, but its functions under physiological and pathological conditions remains to be defined.

235 strating how the shift from physiological to pathological conditions results from slight perturbation

236 l ganglia activity in both physiological and pathological conditions.SIGNIFICANCE STATEMENT Striatal

237 erent striatal territories during normal and pathological conditions.SIGNIFICANCE STATEMENT The nucle

238 understand VGLUT's involvement in normal and pathological conditions.SIGNIFICANCE STATEMENT VGLUT3 is

239 licable to the analysis of other tissues and pathological conditions, so it may further improve our u

240 their therapeutic implication under several pathological conditions, spanning from infections to can

241 xcitability, and have been linked to several pathological conditions such as absence epilepsy, cardio

242 r dysfunction and are associated with severe pathological conditions such as acute respiratory distre

243 levels, such as attention and arousal and in pathological conditions such as Alzheimer's disease.

244 ree miRISC/AGO2 complexes can be affected by pathological conditions such as amyotrophic lateral scle

245 re when tissues are overloaded or subject to pathological conditions such as aneurysms.

246 flammation and is prominent in most vascular pathological conditions such as atherosclerosis and rest

247 e regulators of immune responses during many pathological conditions such as cancer and transplantati

248 rganisms and has been implicated in numerous pathological conditions such as cancer.

249 mmune responses and is often activated under pathological conditions such as cancer.

250 Indeed, pathological conditions such as cellular senescence are

251 e neuronal volume regulation often occurs in pathological conditions such as glutamate excitotoxicity

252 hanisms may play a maladaptive role in human pathological conditions such as in the fatigue and anore

253 Upon cardiac pathological conditions such as ischemia, microenvironme

254 or sigma(2) receptors play a crucial role in pathological conditions such as pain, neurodegenerative

255 ng and memory, motor control and reward, and pathological conditions such as Parkinson's disease and

256 ssibly to increased susceptibility to common pathological conditions such as plantar fasciitis.

257 diate succinate and is implicated in various pathological conditions such as rheumatoid arthritis, li

258 l brain tissue that is linked to a number of pathological conditions such as stroke and migraine.

259 r processes such as cell differentiation and pathological conditions such as tumor cell migration.

260 Pathological conditions, such as a stroke, trigger neura

261 ytosolic accumulation of self-DNA results in pathological conditions, such as autoimmunity.

262 on the correlation between RAGE activity and pathological conditions, such as cancer, diabetes, cardi

263 nvolvement of human NAT enzymes in different pathological conditions, such as cancer, has encouraged

264 Under pathological conditions, such as continuous MAPK pathway

265 elp advance therapeutic avenues for managing pathological conditions, such as diseases arising from i

266 of the dural vasculature has been limited to pathological conditions, such as headaches, but little i

267 the clinical characterisation and grading of pathological conditions, such as pancreatitis or cancer.

268 lation are concurrent with physiological and pathological conditions, such as pregnancy and rheumatoi

269 y cognitive functions and to be disrupted in pathological conditions, such as schizophrenia.

270 ndria indicates loss of their function under pathological conditions, such as stroke and brain trauma

271 ase in extracellular ATP concentration under pathological conditions, such as tissue damage or viscer

272 ion of contact inhibition is associated with pathological conditions, such as tumor spreading.

273 Early pathological conditions-such as hypoxia, inflammation, o

274 glucose homeostasis under physiological and pathological conditions, suggesting that deeper understa

275 death that promotes inflammation in various pathological conditions, suggesting that it might be a p

276 wn to be transcribed under physiological and pathological conditions, suggesting that sophisticated r

277 Under pathological conditions, Tau dissociates from axonal mic

278 ay-Thurner syndrome or Cockett syndrome is a pathological condition that arises due to extrinsic comp

279 erfusion (I/R) injury is a relatively common pathological condition that can lead to multi-organ fail

280 Cardiac fibrosis is a pathological condition that occurs after injury and duri

281 cians evaluating the cerebellum and possible pathological conditions that affect it.

282 ied mitochondrial fragmentation during acute pathological conditions that mimic severe, moderate, and

283 rocytes under a variety of physiological and pathological conditions, the acute effects of seizures o

284 Under pathological conditions, the delivery of NGF enables neu

285 ellular vesicles (EVs) and physiological and pathological conditions, the interest in EVs is exponent

286 eutic direction.SIGNIFICANCE STATEMENT Under pathological conditions, the presynaptic protein alpha-s

287 anges in cardiomyocytes in physiological and pathological conditions, thereby providing novel therape

288 g normal brain aging, but also under certain pathological conditions, this crosstalk can go beyond ph

289 ing evidence of function in health and under pathological conditions, this review will outline the gl

290 2) regulates blood vessel remodeling in many pathological conditions through differential effects on

291 ted to be involved in some physiological and pathological conditions through processing substrates, i

292 bute to the development of many diseases and pathological conditions through the excessive production

293 aberrant activation of TRPC4 may occur under pathological conditions to cause cell damage.

294 d protein metabolism under physiological and pathological conditions, underscoring the potential of t

295 However, under pathological conditions, upregulation of death receptor

296 ircadian clock genes under physiological and pathological conditions; we focus on their roles in regu

297 hich could be of relevance in the context of pathological conditions where spine densities and neural

298 olved in a variety of cellular processes and pathological conditions, which makes it a promising phar

299 for therapeutic intervention in a variety of pathological conditions with reduced synaptic neuromuscu

300 alyzing their action against ROS produced by pathological conditions within the cells.