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1 ional decision-making, such as addiction and pathological gambling.
2 ng gambling outcomes confer vulnerability to pathological gambling.
3 ing excessive risk-taking in humans, such as pathological gambling.
4 tions characterized by risky choices such as pathological gambling.
5 for increased striatal dopamine synthesis in pathological gambling.
6 disorders such as drug abuse, overeating, or pathological gambling.
7 st nalmefene in the treatment of adults with pathological gambling.
8 tically significant reduction in severity of pathological gambling.
9 curacy of the DSM-IV diagnostic criteria for pathological gambling.
10 characteristics of patients with and without pathological gambling.
11 ke inhibitor fluvoxamine in the treatment of pathological gambling.
12 ndividuals with a lifetime history of DSM-IV pathological gambling, 36%-39% did not experience any ga
13 d thoughts and behavior as measured with the pathological gambling adaption of the Yale-Brown Obsessi
14 The authors investigated the occurrence of pathological gambling among cocaine-dependent outpatient
15 ogical and nonpharmacological treatments for pathological gambling and is based on a review of the li
16 ribes cognitive-behavioral therapy (CBT) for pathological gambling and its general principles and pro
17 fects such as impulse control disorders (eg, pathological gambling and shopping, binge eating, and hy
18 ve-compulsive disorder, Tourette's syndrome, pathological gambling, and addictions remain poorly unde
19 disorders including substance use disorders, pathological gambling, and attention deficit hyperactivi
22 Impulse control disorders (ICD), including pathological gambling, are common in Parkinson's disease
24 -problem gambling twins and their problem or pathological gambling brothers, but adjusted co-twin ana
25 tors may be effective for some patients with pathological gambling, but those with comorbid condition
26 risk factor for addictive disorders such as pathological gambling, cigarette smoking, and drug and a
27 rown Obsessive Compulsive Scale Modified for Pathological Gambling, compared to the placebo group.
29 3) who reported a lifetime history of DSM-IV pathological gambling disorder (N=21 and N=185, respecti
31 tural recovery among individuals with DSM-IV pathological gambling disorder in two large and represen
35 ial portion of individuals with a history of pathological gambling eventually recover, most without f
37 The efficacy of naltrexone treatment for pathological gambling has been replicated in a double-bl
39 ted a role for amantadine as a treatment for pathological gambling in patients with Parkinson disease
47 results of large epidemiological surveys of pathological gambling may eventually overturn the establ
49 ses in their gambling behavior scores on the pathological gambling modification of the Yale-Brown Obs
53 highlights the need to identify subgroups of pathological gambling patients with bipolar spectrum con
56 between two behavioral addiction phenotypes: pathological gambling (PG) and binge eating disorder (BE
57 and impulsivity have both been implicated in pathological gambling (PG) and in reward processing.
62 rly half of all individuals in treatment for pathological gambling (PG), but relatively little is kno
63 mportant to both cocaine dependence (CD) and pathological gambling (PG), few studies have directly in
68 thium carbonate compared to placebo on total pathological gambling scores on the Yale-Brown Obsessive
69 pled with low rates of treatment-seeking for pathological gambling suggests that natural recovery mig
71 easure of the DSM-IV diagnostic criteria for pathological gambling was administered, along with other
74 rown Obsessive Compulsive Scale Modified for Pathological Gambling) were analyzed by using a linear m
76 he hypothesis that they are dysfunctional in pathological gambling with poor self-control, we studied
77 e replacement therapy, compulsive buying and pathological gambling, with eight patients exhibiting mo