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1 amine transporter occurs as part of an early pathological process.
2 ging, and is usually evident years after the pathological process.
3 edicted to be a significant component of the pathological process.
4 formal schooling, can counteract the ongoing pathological process.
5 ate tumors and that Src kinases mediate this pathological process.
6 plantation of the cell types affected by the pathological process.
7 kinase activity plays a critical role in the pathological process.
8 regulating the acetylome play a role in this pathological process.
9 vity of the C1s protease not involved in the pathological process.
10 ate that such structures are involved in the pathological process.
11 onse after MI and after other cardiovascular pathological processes.
12 mmon to many developmental, regenerative and pathological processes.
13 gration is central to many developmental and pathological processes.
14 es to functions in various physiological and pathological processes.
15 reduced gene expression in physiological and pathological processes.
16 yet this damage initiates and potentiates in pathological processes.
17 to be essential in lots of physiological and pathological processes.
18 plays an important role in physiological and pathological processes.
19 fects against a variety of physiological and pathological processes.
20 ntral role in a variety of physiological and pathological processes.
21 a sensitive test for many physiological and pathological processes.
22 ators of most, if not all, physiological and pathological processes.
23 , is highly dynamically regulated in several pathological processes.
24 wn to take part in several physiological and pathological processes.
25 y pivotal roles in diverse physiological and pathological processes.
26 ays an important role in many biological and pathological processes.
27 trocytes appear to mediate or exacerbate the pathological processes.
28 of genes involved in both physiological and pathological processes.
29 verns diverse developmental, homeostatic and pathological processes.
30 gene regulation of several physiological and pathological processes.
31 tems, involved in numerous physiological and pathological processes.
32 define key developmental, physiological and pathological processes.
33 w understood to mediate numerous healthy and pathological processes.
34 s a route to a plausible mechanistic link to pathological processes.
35 ate changes to EMT in many developmental and pathological processes.
36 n the presence of anatomical variability and pathological processes.
37 HC O3/ Cl may mediate many physiological and pathological processes.
38 ction in kidney function caused by different pathological processes.
39 osely associated with many physiological and pathological processes.
40 ng issue is how they modulate biological and pathological processes.
41 ediates a variety of other physiological and pathological processes.
42 by different glycoforms during biological or pathological processes.
43 a crucial role in various physiological and pathological processes.
44 nvolved in a wide range of physiological and pathological processes.
45 more accurate cellular and animal models of pathological processes.
46 an important role in both physiological and pathological processes.
47 of the role of endothelial metabolism in key pathological processes.
48 lay critical roles in many physiological and pathological processes.
49 stroke also highlights their involvement in pathological processes.
50 exacerbate or mitigate injury during various pathological processes.
51 d mediate a broad range of physiological and pathological processes.
52 of circulating plasma holding insights into pathological processes.
53 rtant roles in a number of physiological and pathological processes.
54 have been related to both physiological and pathological processes.
55 small changes in neurodegenerative and other pathological processes.
56 n the study of FA associated with normal and pathological processes.
57 d as a crucial pathway in several normal and pathological processes.
58 rences in toxicity and consequent effects on pathological processes.
59 mportant roles in multiple physiological and pathological processes.
60 in regulation of physiological functions and pathological processes.
61 ionally implicated in diverse biological and pathological processes.
62 es involved in the insurgence/development of pathological processes.
63 y mediate a broad range of physiological and pathological processes.
64 ediator during a number of physiological and pathological processes.
65 lls strongly regulate many physiological and pathological processes.
66 and controls a variety of physiological and pathological processes.
67 ene markers that monitor individual hallmark pathological processes.
68 ir significance in various physiological and pathological processes.
69 distortions arising from adaptive as well as pathological processes.
70 ion implicated in numerous physiological and pathological processes.
71 ation that autophagy affects many normal and pathological processes.
72 d play vital roles in many physiological and pathological processes.
73 rucial pathways underlying physiological and pathological processes.
74 fibrosis, but was also able to reverse these pathological processes.
75 important consequences for physiological and pathological processes.
76 standing of their roles in physiological and pathological processes.
77 erproduction of ROS is a hallmark of various pathological processes.
78 s is important for various physiological and pathological processes.
79 cid dependent roles during physiological and pathological processes.
80 mTOR) participates in many physiological and pathological processes.
81 productive infection but also trigger unique pathological processes.
82 participating in multiple physiological and pathological processes.
83 mechanistic basis of many physiological and pathological processes.
84 e of elements as cofactors in biological and pathological processes.
85 at plays a pivotal role in physiological and pathological processes.
86 es mediate developmental, physiological, and pathological processes.
87 BPs) mediate a wide variety of essential and pathological processes.
88 pe in RA and have an important role in these pathological processes.
89 his PTM regulates various key biological and pathological processes.
90 lopment and provided important insights into pathological processes.
91 ed to important cellular, physiological, and pathological processes.
92 te in glycans refers to different normal and pathological processes.
93 tabolic changes underlying a wide variety of pathological processes.
94 activities in a variety of physiological and pathological processes.
95 egenerative diseases, providing insight into pathological processes.
96 ar communication in physiological as well as pathological processes.
97 ay a critical role in many immunological and pathological processes.
98 umerous human cancers and hyperproliferative pathological processes.
99 tor family, implicated in a diverse array of pathological processes.
100 it is involved in several physiological and pathological processes.
101 and modulators of numerous physiological and pathological processes.
102 tal role in several important biological and pathological processes.
103 vo NE transmission in both physiological and pathological processes.
104 n associated with numerous physiological and pathological processes.
105 role of heme in important physiological and pathological processes.
106 e of FXIIa and thrombin in physiological and pathological processes.
107 sis and angiogenesis during developmental or pathological processes.
108 native alphaSyn can impact a variety of its pathological processes.
109 ization, and its disruption may characterize pathological processing.
111 strates implicate RHBDL2 in physiological or pathological processes affecting epithelial homeostasis.
112 ough infiltrating macrophages influence many pathological processes after spinal cord injury (SCI), t
113 titatively and non-invasively monitoring the pathological process and the efficacy of arthritis treat
114 tation, but does not identify the underlying pathological process and therefore cannot be considered
115 olved in a wide variety of physiological and pathological processes and are best described by ensembl
116 he regulation of divergent physiological and pathological processes and are increasingly recognized t
117 cular, the centrality of mitochondria in the pathological processes and clinical phenotypes associate
118 rs are involved in several physiological and pathological processes and constitute therefore an impor
119 odegenerative disorders that involve similar pathological processes and could also be useful for the
120 t importance for understanding physiological/pathological processes and developing intracellular drug
121 numerous NAD(+)-associated physiological and pathological processes and facilitate screening for drug
123 rging as key regulators of physiological and pathological processes and therefore have been identifie
124 to a better understanding of the individual pathological processes and to a personalized diagnosis a
125 disease but rather an active player in many pathological processes and, therefore, a genuine target
126 Ks) play critical roles in physiological and pathological processes, and are important anticancer dru
128 s, discuss their impact on physiological and pathological processes, and highlight important areas of
129 is a modulator of various physiological and pathological processes, and it mediates its effects via
130 se mechanisms, their impact on physiological/pathological processes, and potential therapeutic applic
131 ic gene-regulatory programs, mutant-specific pathological processes, and technical effects, and provi
132 ing tissues during normal development and in pathological processes are often due to modifications of
135 rtant mediator for various physiological and pathological processes associated with acidosis and alka
136 buting role of monosodium urate (MSU) to the pathological processes associated with the induction of
137 heimer's disease (AD) is crucial because the pathological process begins decades before dementia onse
138 ant insights into multiple physiological and pathological processes but is hindered by a lack of auto
139 ssociated with invasion in developmental and pathological processes, but how netrin signaling is coor
140 cell type involved in many physiological and pathological processes, but remain poorly-characterised.
141 of proteome change in various biological and pathological processes by quantitative mass spectrometry
143 iggers a complex cascade of inflammatory and pathological processes, culminating in formation of a sc
145 emonstrates that even in presence of ongoing pathological processes, education may facilitate both br
146 rall prevalence of HANDs, suggesting ongoing pathological process even when viral replication is supp
149 ver injury in mice and the discrimination of pathological processes from normal liver processes with
150 er brain functions and is altered in various pathological processes, from chronic immunopathological
151 ts involvement in many key physiological and pathological processes has been documented in numerous s
152 mechanistic role that glycans play in these pathological processes has driven the development of the
153 Autophagy is essential in physiological and pathological processes, however, the role of autophagy i
154 cans play crucial roles in physiological and pathological processes; however, it is laborious to stud
155 est that sleep defects may reflect a primary pathological process in HD, and that measurements of sle
159 focal inflammatory lesions is the underlying pathological process in relapsing-remitting multiple scl
160 ks the disease in the exocrine pancreas to a pathological process in the endocrine pancreas, suggesti
161 y in psoriatic skin represents an additional pathological process in this complex inflammatory skin d
162 ss of retinal ganglion cells (RGCs) is a key pathological process in TON, yet mechanisms responsible
164 atherosclerosis via phenotypic switching, a pathological process in which SMC dedifferentiation, mig
165 ter involvement may be linked to progranulin pathological processes in a subset of GRN mutation carri
169 Although techniques used for quantifying pathological processes in different regions of the CNS h
170 by the timing of their separate and distinct pathological processes in disrupting healthy cerebral de
171 ed factors play a central role in normal and pathological processes in every tissue in the body.
172 lites are involved in many physiological and pathological processes in human cells; thus, our results
174 )(+)]i) signaling mediates physiological and pathological processes in multiple organs, including the
175 of how DMF may act clinically to ameliorate pathological processes in neurodegenerative disease.
177 underlies a number of key physiological and pathological processes in situ, but has not been studied
178 which H2O2 contributes to physiological and pathological processes in the brain remain poorly unders
181 go phenotypic changes during development and pathological processes in vivo and during cell culture i
182 d these structures to both physiological and pathological processes, in particular to genome instabil
183 e involved in a variety of physiological and pathological processes, in which regulation of the Ca(2+
184 ary molecular defect, neuromuscular disorder pathological processes include disturbance of intramuscu
186 s plays a key role in many physiological and pathological processes including cancer metastasis.
187 c targets due to their contributions to many pathological processes including cancers, atherosclerosi
188 cial roles in a variety of physiological and pathological processes including embryogenesis, wound he
190 and have the capacity to initiate a PD-like pathological process, including intracellular and presyn
191 RNAs that affect numerous physiological and pathological processes, including cancer development and
193 important roles in several physiological and pathological processes, including cancers, infection, an
195 shown to be a key player in many physio- and pathological processes, including cell differentiation,
196 nstrated that circCCDC66 controlled multiple pathological processes, including cell proliferation, mi
197 x4), regulates a number of physiological and pathological processes, including cellular differentiati
200 se inhibitor (serpin) implicated in numerous pathological processes, including coronary heart disease
202 nvolved in a wide range of physiological and pathological processes, including fibrosis and tumor pro
203 GAMP-STING pathway in many physiological and pathological processes, including host defense against m
204 Defects in autophagy are implicated in many pathological processes, including inflammatory bowel dis
205 nal lymphangiogenesis contributes to various pathological processes, including inflammatory disease a
206 issue regeneration, it is also a hallmark of pathological processes, including malignancy and chronic
208 is and its contribution to physiological and pathological processes, including receptor function in i
209 ays critical roles in many physiological and pathological processes, including regulating organogenes
210 some release have been implicated in various pathological processes, including the progression of Alz
211 actor involved in numerous physiological and pathological processes, including tissue repair and infl
212 s been shown to play important roles in many pathological processes, including tumorigenesis and dise
213 mine early molecular events that may dictate pathological processes induced by exogenous cellular ins
214 stions have emerged, including how different pathological processes interact over the disease course
215 uroinflammation and neuronal dysfunction are pathological processes involved in Alzheimer's disease.
216 and perhaps incomplete understanding of the pathological processes involved in later stages of aorti
217 scular function, to assess physiological and pathological processes involved in the regulation of cer
218 ha2 delta subunits in both physiological and pathological processes involving calcium channels, and a
219 This article intends to review the various pathological processes involving the cerebellum along wi
220 ogenesis and the type of cells affected by a pathological process is needed to develop effective prop
221 thelial cells (EC) in many physiological and pathological processes is widely modeled using human EC
222 the role of the transmembrane form of PrP in pathological processes leading to neuronal death remains
223 iral infections have been proposed to elicit pathological processes leading to the initiation of T he
224 ges in the colonocyte luminal environment in pathological processes, like mucosal inflammation, prene
225 s suggest that beta-amyloid- and tau-related pathological processes may interact but not necessarily
227 (GFAP) is a measure of astrogliosis, a known pathological process of FTD, but has yet to be explored
228 indicate that inflammatory responses and the pathological process of hyperglycemia may influence each
229 However, small size is only a proxy for the pathological process of interest, intrauterine growth re
232 and other biomarker assays suggest that the pathological processes of Alzheimer's disease (AD) begin
233 seases and itching dermatitis, modulates the pathological processes of inflammation, vasculopathy, an
234 yzed to identify bioactive compounds against pathological processes of neurodegenerative diseases.
235 x, plays multiple roles in physiological and pathological processes of protein translation, signal tr
236 peutic strategies that interrupt progressive pathological processes offer an appealing approach for t
237 mportance for understanding the cellular and pathological processes on which TSPO PET imaging is repo
238 hough PTS affects numerous physiological and pathological processes, only a small fraction of the tot
240 e sclerosis, the roles that these individual pathological processes play in multiple sclerosis progre
241 ribution and the hypothesis of an outside-in pathological process possibly driven by CSF-mediated imm
243 widespread implications in physiological and pathological processes, ranging from neurological develo
245 transient is essential to physiological and pathological processes related to synaptic development,
246 help elucidate fundamental physiological and pathological processes related to the mycomembrane and m
247 ith their crucial role in many biological or pathological processes require the development of new hi
248 upted beta-amyloid homeostasis initiates the pathological process resulting in Alzheimer disease (AD)
250 of the histone-PHD interaction in normal and pathological processes, selective inhibitors of this ass
251 essential roles in various physiological and pathological processes such as blood coagulation, skelet
252 During migration of cells in vivo, in both pathological processes such as cancer metastasis or phys
253 extrusion to developmental, homeostatic and pathological processes such as cancer metastasis, its un
254 and repair, in wound healing and in numerous pathological processes such as cancer progression or dia
255 2A17) are involved in many physiological and pathological processes such as cell differentiation, apo
256 cumulates in tissues during many physio- and pathological processes such as embryonic development, wo
257 asis, and their dysregulation contributes to pathological processes such as fibrosis and tumor progre
258 r its involvement in multiple biological and pathological processes such as growth-related events, ce
259 P2) is involved in various physiological and pathological processes such as insulin secretion, stem c
261 ortance to understand many physiological and pathological processes such as morphogenesis, tissue hom
262 re involved in a myriad of physiological and pathological processes such as neurodegenerative disease
264 ted in a broad spectrum of physiological and pathological processes, such as Alzheimer disease or can
265 ional responses, and may even predispose for pathological processes, such as Alzheimer's disease and
266 development and tissue formation, as well as pathological processes, such as cancer metastasis and wo
268 ramount importance in both physiological and pathological processes, such as development, wound heali
270 ffers a natural, accelerated model of common pathological processes, such as inflammation, HIV-relate
273 s thought to play a critical role in several pathological processes, such as neurodegenerative diseas
274 eractions mediate critical physiological and pathological processes, such as neuronal plasticity, dev
275 ation of neuronal networks through different pathological processes, such as prion-like spread, may p
276 A large amount of evidence suggests that pathological processes taking place in early embryonic n
278 extremity predominant are consistent with a pathological process that preferentially affects lumbar
279 ive the rate of disease onset by eliciting a pathological process that ultimately claims vulnerable c
280 rocephalus can also be acquired, mostly from pathological processes that affect ventricular outflow,
281 nset, allows investigation of presymptomatic pathological processes that can identify a therapeutic w
282 plex syndrome involving diverse pathways and pathological processes that can manifest in circulation
283 s and grey matter neuronal injury are likely pathological processes that contribute to disease progre
285 critical for understanding physiological and pathological processes that involve changes in pH and ch
286 nt low back pain (LBP) are common and costly pathological processes that require improved treatment s
287 generation is defined by a complex series of pathological processes that ultimately lead to death.
288 hed a steady state and analyzed to determine pathological processes that were activated, inactivated,
289 y of studies implicating TSPO in a number of pathological processes through its actions on the PTP.
290 ic opportunities to regulate angiogenesis in pathological processes through the manipulation of peric
291 ely because of their potential importance in pathological processes, toxicology, and cancer therapy.
292 senescent or damaged mitochondria is a major pathological process underlying Parkinson disease and a
293 The many complex local and disseminating pathological processes underlying burn injury's clinical
295 s are slow in development because unlike the pathological processes underlying the motor deficits of
296 tion of proangiogenic factors induced by the pathological process was greatly neutralized by SCT inje
297 triggering Wallerian degeneration of axons-a pathological process which reliably activates microglia.
298 in cell-cell communication and modulation of pathological processes, which, however, are not yet well
299 ns in different cellular, developmental, and pathological processes will keep researchers busy for de
300 tead governed by a highly complex, regulated pathological process with similarities to skeletal bone