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1 er adhesin that is conserved within the ETEC pathovar.
2 hat some antigens are restricted to the ETEC pathovar.
3 0%), but absent in the other major X. oryzae pathovar.
4 to diverse TAL effectors from both X. oryzae pathovars.
5 ree Secretion System in Pseudomonas syringae pathovars.
6 issue specificity for individual species and pathovars.
7 dividual host species coevolving with single pathovars.
8 d that blurs the distinction between E. coli pathovars.
9 but not complete, overlap between these two pathovars.
10 to (Pto) representing particularly divergent pathovars.
11 tors that contribute to the formation of new pathovars.
13 e" confirmed the diverse history of the ETEC pathovar and provides a finer resolution of the E. coli
15 fector suites from two sequenced P. syringae pathovars and show that type III effector protein suites
16 cluster but before the divergence of modern pathovars and that some EELs underwent transpositions yi
19 sed by uropathogenic Escherichia coli (UPEC) pathovars belong to the most frequent infections in huma
20 sed by uropathogenic Escherichia coli (UPEC) pathovars belong to the most frequent infections in huma
24 wever, the recently emerged ST313 lineage II pathovar commonly causes systemic bacteremia in sub-Saha
25 cterium Pseudomonas syringae is divided into pathovars differing in host specificity, with P. syringa
26 he family of agn alleles in Escherichia coli pathovars encodes autotransporters that have been implic
27 nts and unique features of three unsequenced pathovars, enterotoxigenic E. coli, enteropathogenic E.
30 phenotypic markers, but only two of the six pathovars have been subjected to any genome sequencing p
31 protein in the T3SS of different P. syringae pathovars, hrpP from P. syringae pv. syringae 61 and P.
32 stence and virulence potential of an E. coli pathovar hybrid that blurs the distinction between E. co
33 ng its adaptation into a virulent pathogenic pathovar infecting Brassica vasculature and mesophyll ti
34 other distinguishing feature between the two pathovars is their distinctive sets of transposable elem
35 f infection with either Pseudomonas syringae pathovar japonica (Psj) or Xanthomonas translucens patho
36 rast, the resistance to Pseudomonas syringae pathovar maculicola (RPM1) bacterial resistance gene is
37 ation with HR-eliciting Pseudomonas syringae pathovars measured by laser photoacoustic detection was
38 ultiple effector proteins from an individual pathovar more frequently and more intensely than host sp
39 e majority of deaths are attributable to one pathovar of E. coli, namely, enterotoxigenic E. coli.
42 newly analyzed and eight known PFPs from 12 pathovars of P. syringae, which belong to four genomospe
46 ne (COR) is a phytotoxin produced by several pathovars of Pseudomonas syringae and consists of corona
47 t only compromised nonhost resistance to few pathovars of Pseudomonas syringae and Xanthomonas campes
49 portant virulence factor produced by several pathovars of the bacterial pathogen Pseudomonas syringae
50 ance to another race of H. parasitica and to pathovars of the bacterial pathogen Pseudomonas syringae
52 i (ETEC) is an important pathogenic variant (pathovar) of E. coli in developing countries from a huma
53 al blight (BB), caused by Xanthomonas oryzae pathovar oryzae (Xoo), is a major rice disease in Asia a
54 e analyses with a phylogenetically divergent pathovar, P. syringae pv. tomato DC3000, revealed a stro
55 hF gene was cloned from Pseudomonas syringae pathovar phaseolicola (PPH:) races 5 and 7, based on its
57 tomato hrpW were found in other P. syringae pathovars, Pseudomonas viridiflava, Ralstonia (Pseudomon
59 psis thaliana pathogens Pseudomonas syringae pathovar (pv.) tomato and pv. maculicola were made by in
60 (serotypes A-C) and two sexually transmitted pathovars; serotypes D-K and lymphogranuloma venereum (L
64 ess and its prevalence in many X. campestris pathovars suggests that the Bs2 gene may be durable in t
67 invasive Escherichia coli (EIEC) is a unique pathovar that has a pathogenic mechanism nearly indistin
69 were more resistant to Pseudomonas syringae pathovar tomato (Pst) DC3000 infection implicating SCD1
71 pe Pi-0 is resistant to Pseudomonas syringae pathovar tomato (Pst) strain DC3000 expressing the T3S e
73 the bacterial pathogens Pseudomonas syringae pathovar tomato and Erwinia amylovora, respectively, and
74 odel bacterial pathogen Pseudomonas syringae pathovar tomato DC3000 (DC3000), which is pathogenic on
75 of the foliar pathogen, Pseudomonas syringae pathovar tomato DC3000 (hereafter PstDC3000), into the p
77 s in the plant pathogen Pseudomonas syringae pathovar tomato strain DC3000 possess characteristic pat
78 lants were resistant to Pseudomonas syringae pathovar tomato strain DC3000-induced stomatal reopening
79 RK1 OE plants were insensitive to P syringae pathovar tomato strain DC3118 (coronatine deficit)-induc
80 lence protein AvrPto of Pseudomonas syringae pathovar tomato, the agent of bacterial speck disease, a
82 e resistance in response to Pto (P. syringae pathovars tomato) DC3000(avrB), but not against Pto DC30
83 dentified as putative orthologs in these two pathovars using a reciprocal best-hit method, with 3,941
85 virulence factor from Xanthomonas campestris pathovar vesicatoria (Xcv) that is translocated into tom
86 ype III effector from Xanthomonas campestris pathovar vesicatoria (Xcv), suppresses symptom productio
87 -dependent promoters of Pseudomonas syringae pathovars were identified with a consensus sequence of 5
89 ted AvrBs2 proteins from the two Xanthomonas pathovars were strongly conserved and had predicted sequ