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1 tent varied from 3% (Surinam cherry) to 39% (peach).
2 The highest ash content was 3.9% (melon and peach).
3 and sorbitol accumulation in flower buds of peach.
4 tool to assist fruit quality improvement in peach.
5 rsification of almond and its close relative peach.
6 hrimp, peanut, hazelnut, celeriac, apple and peach.
7 ergic subjects that tolerate both peanut and peach.
8 ulatory networks for fruit quality traits in peach.
9 ved clinical association between cypress and peach.
10 .3% of GB positive patients were positive to peach.
11 scale post-harvest physiological disorder in peach.
12 pression quantitative trait loci (eQTLs) for peach.
13 for future genomic research and breeding in peach.
14 breeding for extending the harvest season in peach.
15 ieved to regulate some aspect of maturity in peach.
16 to environmental stresses in flower buds of peach.
17 o a dwarf phenotype similar to that of dw/dw peaches.
18 esent in both cultivated and wild grapes and peaches.
19 ng after school lunch, which included canned peaches.
20 nd color are important quality attributes in peaches.
21 linking organic acid and color regulation in peaches.
22 us Prunus, including cherries, apricots, and peaches.
23 kg) and pollen (18-28 ug/Kg), thiacloprid in peaches (21-35 ug/kg) and acetamiprid was identified in
25 ciation mapping population consisting of 132 peach accessions was phenotypically evaluated for MD and
29 es to pollen. We found that more than 90% of peach-allergic patients in both populations evolved to L
30 ization profile and severity of reactions in peach-allergic patients sensitized to nsLTP from two Med
31 nut allergy and tolerance to peach; Group 3, peach allergy and tolerance to peanut; Group 4, nonaller
34 Mediterranean patients: Group 1, peanut and peach allergy; Group 2, peanut allergy and tolerance to
37 gic: those that presented reaction only with peach and (b) LTP-Allergy: those that presented reaction
40 -Allergy: those that presented reaction with peach and at least another plant-food containing LTP.
41 the DORMANCY ASSOCIATED MADS-BOX genes from peach and hypothesize that it may play a direct role in
43 yphenolic extracts and fractions of selected peach and plum genotypes were evaluated for cell viabili
44 created from cold-acclimated bark tissue of peach and selectively probed using an antibody directed
46 y important species; however, SV profiles of peach and their functional impacts remain largely unexpl
48 y of enhancing phenolic compound contents in peaches and nectarines by post-harvest irradiation with
49 ompound was also tested in freshly harvested peaches and oranges, exhibiting promising control profil
50 plant Arabidopsis thaliana, Prunus persica (peach) and four most popular assemblers, ABySS, SOAPdeno
52 sibling deciduous and evergreen genotypes of peach, and also inducible by water deficit in cv. Rio Os
53 strongly correlated with IgE against walnut, peach, and apple and against Chenopodium, plane tree, an
55 commercialized in Brazil (strawberry, mango, peach, and orange) were analyzed and indicated the absen
58 presence/absence variants between almond and peach, and we show that the recent history of TE movemen
61 persicae Sulzer, commonly known as the green peach aphid (GPA), which is an important phloem sap-cons
63 Arabidopsis (Arabidopsis thaliana) and green peach aphid (GPA; Myzus persicae Sulzer), we found that
64 PAD4) is essential for defense against green peach aphid (GPA; Myzus persicae) and the pathogens Pseu
66 ana shoots by the phloem sap-consuming green peach aphid (GPA; Myzus persicae), an agronomically impo
67 globally important economic pest - the green peach aphid (Myzus persicae) - growing on 34 plant taxa,
69 8 also show enhanced resistance to the green peach aphid (Myzus persicae) compared with wild-type con
71 se pathways was investigated following green peach aphid (Myzus persicae) feeding on Arabidopsis.
73 ays with the generalist phloem-feeding green peach aphid (Myzus persicae), and in weight-gain assays
74 se to feeding by its aphid vector, the green peach aphid (Myzus persicae), and increases aphid fecund
75 w (Hyaloperonospora arabidopsidis) and green peach aphid (Myzus persicae), but retained susceptibilit
76 oEL displayed reduced fecundity of the green peach aphid (Myzus persicae), indicating enhanced resist
77 is gap we exposed four naive clones of green peach aphid (Myzus persicae), maintained on the model cr
81 e took advantage of the ability of the green peach aphid Myzus persicae to colonize divergent plant s
85 ial diet both decrease Myzus persicae (green peach aphid) reproduction, suggesting a direct toxic or
87 eding herbivore-host plant system: the green peach aphid, Myzus persicae, feeding on multiple brassic
92 Here we provide the transcriptome of green peach aphids (Myzus persicae) carrying PLRV and virus-fr
94 sis (cotton mealybug), Myzus persicae (green peach aphids) and Bemisia tabaci (silver leaf whitefly).
95 with this hypothesis, Myzus persicae (green peach aphids) prefer to settle on Nicotiana benthamiana
96 context, the syntenic regions identified in peach, apple and strawberry might be useful to interpret
99 ma perception defining Chardonnay character (peach, apricot and gun flint); ii) did not impact the ar
101 characterised by floral and fruity flavours (peach/apricot, Muscat, melon, banana and strawberry) whi
102 hod to small RNA sequence data from soybean, peach, Arabidopsis and rice and provide experimental val
105 e one or more allergies to fruits other than peaches, as in these cases, and relevant fruits differ d
106 gene for the br mutation in Prunus persica (peach) associated with vertically oriented growth of bra
109 It is demonstrated that LTPs from wheat and peach bind a range of lipids in a variety of conditions,
114 enbuconazole, propiconazole, or pyridaben in peaches; carbendazim, imazalil, terbutryn, and thiabenda
115 myl was quantified in stone fruits (apricot, peach, cherry) and nuts (pistachio, almond, walnut) with
116 Since matrix effects were observed in the peach commodity, organic acids were quantified by the st
118 mediate sorbitol synthesis in flower buds of peach concomitantly with specific chromatin modification
121 tionship between susceptibility of different peach cultivars (cvs) to the Mediterranean fruit fly (me
123 esults indicate that the phenolic extract of peach cultivars inhibits Abeta and alphaS fibril formati
125 tonic crude phenolic extracts of six Georgia peach cultivars were prepared and separated into low- an
129 al dehydrins (V. riparia YSK2, 60 kilodalton peach dehydrin [PCA60], barley dehydrin5 [Dhn5], Thellun
132 tial number of SVs have been selected during peach domestication and improvement, which together affe
136 Our results elucidate the genetic basis of peach evolution and provide new resources for future gen
137 Here, we report a comprehensive analysis of peach evolution based on genome sequences of 480 wild an
138 ow that China has been a critical region for peach evolution since long before human presence, much l
140 en compared to other phenolic classes in the peach extract, in these two biologically relevant assays
141 t assays indicated that the HMW fractions of peach extracts were major contributors to the antioxidan
142 cs revealed that UV-B has a strong impact on peach flesh metabolome, determining an initial decrease
144 ble (except alpha-carotene and zeaxanthin in peach) for 13, 9.7, 5.7, 2.5 and 7.5months, respectively
147 o analyse 238 kaki, cashew apple, guava, and peach fruit and pulp samples, which were also analysed f
150 driven modulation of secondary metabolism in peach fruit by enhancing the biosynthesis of specific ph
151 roperties, such as sweetness and acidity, in peach fruit by mid infrared spectroscopy is of interest
152 ic content and the antioxidant capacities of peach fruit extracts was found, indicating that phenolic
155 o increase the health-promoting potential of peach fruits and indirectly to ameliorate the aesthetic
157 -FTIR) was tested here on two populations of peach fruits issued from contrasting genitors providing
159 regions in the eight pseudomolecules of the peach genome (Peach v2.0), with an average of 53% locate
160 82, annotated as hypothetical protein in the peach genome sequence, was identified as a candidate gen
161 ecular bases of how environments have shaped peach genomes by natural selection and adds candidate ge
163 no acid sequence of LTP was identical in all peach genotypes but, for the first time, peel LTP was fo
164 g selection (MASS) targeted towards widening peach germplasm for maturity, particularly early maturit
165 he conservation and exploitation of European peach germplasm resources and, ultimately, as a true her
167 gy; Group 2, peanut allergy and tolerance to peach; Group 3, peach allergy and tolerance to peanut; G
171 p 3 in vivo, a mouse model of anaphylaxis to peach has been produced and changes in the humoral and b
172 Allergic cross-reactions between cypress and peach have been reported, including an oral allergy synd
176 on is very bright but shows subtle yellow to peach hues which probably arise from the production of c
178 cted in the related species, Prunus persica (peach), indicating that artificial selection during dome
181 in clarification of apple, kiwi, orange and peach juices and enhanced their reducing sugar content.
188 nation of As, Cd, Hg and Pb in NIST SRM 1547 peach leaves and SRM 1573a tomato leaves reference mater
194 osis of LTP allergy and sensitization to the peach LTP allergen Pru p 3, were compared to UK subjects
196 onditions do not disrupt the 3D structure of peach LTP, explaining why LTPs retain their ability to b
198 cross-reactive allergen between cypress and peach might be responsible for the observed clinical ass
199 our physical and eight sensory properties of peach nectar were explored using the best-fit multiple l
207 h oranges (OR = 0.18; 95% CI: 0.06-0.51) and peaches (OR = 0.30; 95% CI: 0.13-0.67) had a decreased o
210 (P = .023), fresh oranges (P = .002), fresh peaches (P = .002), and collard greens/kale (P = .014).
213 n of carotenoids from the peel of tucuma and peach palm fruits and their carotenoid profile were inve
215 otenoid (7.8 and 7.3 mg/100 g for tucuma and peach palm, respectively), followed by gamma-carotene an
219 able Rosaceae ESTs, the genetically anchored peach physical map, Rosaceae genetic maps and comprehens
222 hibits diverse fruit types, including drupe (peach), pome (apple), drupetum (raspberry), and achenetu
224 abidopsis thaliana by the highly polyphagous peach-potato aphid (Myzus persicae), we identified cell
226 , we targeted the VGSC (MpNa(v)) gene in the peach-potato aphid Myzus persicae, by oral feeding of ar
230 se data define potential for improvements to peach production efficiency and fruit quality, particula
231 luate the allergenic properties of LTPs from peach (Pru p 3) and pellitory (Par j 1/Par j 2), major f
232 caused by lipid transfer protein (LTP) from peach (Pru p 3) is frequently associated with sensitizat
235 ivated in Tunisia: kaki (Diospyros kaki L.), peach (Prunus persica L.) and apricot (Prunus armeniaca
237 distant species we estimated that almond and peach (Prunus persica) diverged around 5.88 million year
239 uence-based genotyping, and the high-quality peach (Prunus persica) genome reference sequence for sin
241 sign, and realization of the first multisite peach (Prunus persica) reference collection (PeachRefPop
242 a recessive brachytic dwarfism trait (dw) in peach (Prunus persica) that has little or no effect on f
243 stone fruits such as cherry (Prunus avium), peach (Prunus persica), and apple (Malus domestica).
244 tein (LTP, Pru p 3) is the major allergen of peach (Prunus persica), and is in a greater abundance in
247 esentative crops such as apple (Malus spp.), peach (Prunus spp.), and strawberry (Fragaria spp.).
248 ADS-box transcription factors (DAM genes) in peach [Prunus persica (L.) Batsch] as potential candidat
251 d ripening stages in three climacteric (i.e. peach [Prunus persica] and two tomato [Solanum lycopersi
253 hree different cookies formulated with 10.5% peach pulp incorporation and 50% fat or added-sucrose re
254 ation showed that heavier crop loads reduced peach quality (DMC, SSC) and delayed maturity (I(AD)) an
256 , blueberries, sweet cherries, table grapes, peaches, raspberries, and strawberries) in a postharvest
257 n blot analysis, indicating transposition of peach rather than random integration of the plasmid DNA.
258 subjects had a positive SPT to LTP-enriched peach reagent, compared to 91% of the 35 UK LTP subjects
259 10,981,971-11,298,736 bp on chromosome 4 of peach reference genome used for haplotype analysis revea
260 wing two patterns: patients also allergic to peach, responding to Ara h 2 and Pru p 3, and patients a
263 of this region, BES were mapped against the peach scaffold_3 and BACs were anchored to the apricot m
264 ple 3-tiered risk stratification was formed: PEACH score 0 (in-hospital mortality 0.2%), 1-2 (3.6%),
265 al value these seeds, especially apricot and peach seeds, could be exploited to produce value-added p
267 and genomes of the rosid species poplar and peach, showed areas of conserved gene order, with overal
270 ettlement in the American Southeast and that peaches spread independently of interactions with Spanis
271 h event in the southwestern USA, the 18.8 Ma Peach Spring Tuff, were formed by pyroclastic flows that
275 es of intact fruit from six cultivars (three peaches, three nectarines) before and after storage at 1
278 upational respiratory diseases in workers of peach tree crops have been reported punctually and have
284 e eight pseudomolecules of the peach genome (Peach v2.0), with an average of 53% located in exonic re
285 rification of LTP from peel and pulp of four peach varieties [Gladys (white flesh), California (necta
287 g technologies should be designed to improve peach varieties with rich functional contents because of
290 ed peach SPT reagent and/or LTP allergens in peach, walnut, mugwort and plane tree may enhance diagno
291 rd and sensitization to the LTP allergens in peach, walnut, mugwort and plane tree These sensitizatio
293 nonspecific lipid transfer protein (LTP) of peach were compared with the homologous LTP1 of barley a
295 cal modeling, these early dates suggest that peaches were likely in the interior prior to permanent S
297 rst shown to directly impact endodormancy in peach where a deletion of a series of DAM resulted in lo
298 r two highly homologous genes are present in peach, whereas an additional member was detected under l
299 Robustness was demonstrated using fresh peaches, which provided recovery values within acceptabl
300 rases encoded by mycobacteriophages Bxz2 and Peaches with unusual and unpredictable specificities.