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1  or equal to 3:1, and intrastent doubling of peak-systolic velocity.
2 P=0.36), despite increased blood flow (Delta peak systolic velocity, 6.3 cm/s, 3.5-9.07; P<0.001; Del
3                                              Peak systolic velocity after administration of topical a
4 processed to reconstruct mitral annulus (MA) peak systolic velocity and displacement.
5                           During stress, the peak systolic velocity and early diastolic velocity incr
6   beta values of the relationship between MA peak systolic velocity and LV end-diastolic volume and m
7 ional left ventricular function, assessed by peak systolic velocity and strain rate in 12 paired, non
8          The best-performing parameters were peak systolic velocity and V(MCA)/V(ICA) ratio.
9  lumen area increased significantly, whereas peak systolic velocity and wall shear stress decreased.
10                      Average volume of flow, peak systolic velocity, and diameter of residual lumen w
11 en kidneys and between regions was found for peak systolic velocity, but the magnitude of this variat
12                     Increasing age decreased peak systolic velocity by 0.34 cm/sec/y.
13 2 months, defined as >/=2.5-fold increase in peak systolic velocity by duplex ultrasonography.
14                      Internal carotid artery peak systolic velocities decrease with advancing age and
15 the implementation of middle cerebral artery peak systolic velocity Doppler measurements to detect fe
16                 Children with SCA had higher peak systolic velocities, end-diastolic velocities, and
17 r or if flow approached 1,300 mL/min without peak systolic velocity greater than 400 cm/sec.
18 ive diagnostic US criteria for stenosis were peak-systolic velocity greater than 1.25 m/sec, internal
19 ive criteria for stenosis were also applied: peak-systolic velocity greater than 1.7 m/sec, ICA end-d
20  patients with isolated, asymptomatic AS and peak systolic velocity &gt; or =4 m/s by Doppler echocardio
21 ort-/long-axis ratio <0.6, tricuspid annulus peak systolic velocity &gt;/= 8 cm/s, and peak systolic lon
22 gh measurement of the middle cerebral artery peak systolic velocity has led to a paradigm shift in an
23 eroseptal-posterior delay, and SD in time to peak systolic velocity in the 12 left ventricular segmen
24                            The SD in time to peak systolic velocity in the 12 left ventricular segmen
25 -/long-axis ratio >/= 0.6, tricuspid annulus peak systolic velocity &lt;8 cm/s, and peak systolic longit
26 .06 cm s(-1) and -0.50 +/- 2.55 cm s(-1) for peak systolic velocity, mean flow velocity, and end dias
27 icuspid annular plane systolic excursion and peak systolic velocity, myocardial performance (expresse
28 ses in TDI indices occurred with dobutamine: peak systolic velocity of 4.41 +/- 1.07 to 6.67 +/- 1.07
29 significant decreases occurred with esmolol: peak systolic velocity of 4.46 +/- 0.94 to 2.31 +/- 0.81
30 arget artery of at least 70%, diagnosed by a peak systolic velocity of at least 3.0 m/s.
31                            Duplex ultrasound peak systolic velocity of the cavernosal arteries increa
32 - 0.47 mm; p < 0.05) and correlated with the peak systolic velocity of the second palmar digital arte
33               Doppler-defined pre-procedural peak systolic velocities (PSV) and end-diastolic velocit
34                                      (c) ICA peak systolic velocity (PSV) and presence of plaque on g
35                   Point, non-angle-corrected peak systolic velocity (PSV) and resistive index (RI) va
36 elocity (MFV), end diastolic velocity (EDV), peak systolic velocity (PSV) and resistive index (RI).
37                                          ICA peak systolic velocity (PSV) and the ratio of the PSV in
38 s was a focal twofold or higher elevation of peak systolic velocity (PSV) compared with the PSV immed
39                                 The range of peak systolic velocity (PSV) measurement (maximum minus
40 ic parvus tardus waveforms) with and without peak systolic velocity (PSV) thresholds (determined with
41 stically significant changes observed in the peak systolic velocity (PSV), end diastolic velocity (ED
42           Early systolic acceleration (ESA), peak systolic velocity (PSV), end diastolic velocity (ED
43                                              Peak systolic velocity (PSV), end-diastolic velocity (ED
44                             The range of CCA peak systolic velocities (PSVs) and end diastolic veloci
45 ss, defined as primary patency at 12 months (peak systolic velocity ratio <2.4 by duplex ultrasound w
46 t core laboratory-assessed duplex ultrasound peak systolic velocity ratio <=2.4 in the absence of cli
47  as evidenced by a higher mean graft/femoral peak systolic velocity ratio (1.6 vs. 0.90, P=0.006).
48 inal narrowing was assessed by graft/femoral peak systolic velocity ratio.
49 lic velocity greater than 0.4 m/sec, ICA/CCA peak-systolic velocity ratio greater than 2.0, and ICA/C
50  artery (ICA) to common carotid artery (CCA) peak-systolic velocity ratio of greater than or equal to
51 g-axis/length-area ratios, tricuspid annulus peak systolic velocity, RV peak longitudinal global syst
52 assessed by measuring differences in time-to-peak systolic velocity (T(SV)) between the RV free wall,
53                                          The peak systolic velocity threshold for moderate (>=50%) st
54 surgical cohort, 1 in 10 (9.8%) patients had peak systolic velocity values that warranted the diagnos
55 g-axis myocardial function was quantified by peak systolic velocity (Vs) and strain rate (SR) respons
56     ARAS was severe (60% to 99% stenosis) if peak systolic velocity was >200 cm/s.
57                                              Peak systolic velocity was additionally found to be inve
58                                              Peak systolic velocity was influenced by age (P =.008),
59                                              Peak systolic velocity was measured by Doppler velocimet
60                          Preoperative portal peak systolic velocity was uniformly around 10 cm/sec.
61                      Internal carotid artery peak systolic velocity was used by all centers to assess
62 rity, pulsatility index, resistive index, or peak-systolic velocity, was associated with malignancy.
63                               Changes in TDI peak systolic velocity were correlated with changes in f
64        Resistive and pulsatility indexes and peak systolic velocity were documented.