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1 al disease was diagnosed before the onset of pemphigoid.
2 e autoimmune skin blistering disease bullous pemphigoid.
3 ent testing in patients with mucous membrane pemphigoid.
4 as found in patients with ocular cicatricial pemphigoid.
5 blood of some patients with mucous membrane pemphigoid.
6 skin of patients affected by mucous membrane pemphigoid.
7 rency in mid- to advanced ocular cicatricial pemphigoid.
8 een achieved in the field of mucous membrane pemphigoid.
9 s consistent with a diagnosis of cicatricial pemphigoid.
10 inhibitor-associated noninflammatory bullous pemphigoid.
11 ouse model of the autoimmune disease bullous pemphigoid.
12 are involved in the pathogenesis of bullous pemphigoid.
13 opment of the autoimmune response in bullous pemphigoid.
14 r surface disease such as ocular cicatricial pemphigoid.
15 rly lichen planus and benign mucous membrane pemphigoid.
16 istering disease that closely mimics bullous pemphigoid.
17 identified as an autoantigen in cicatricial pemphigoid.
18 able medications in the treatment of bullous pemphigoid.
19 ibodies (AAb) from patients with cicatricial pemphigoid.
20 ed with the blistering skin disease, bullous pemphigoid.
21 nt in the diagnosis and treatment of bullous pemphigoid.
22 id and help distinguishing them from bullous pemphigoid.
23 opan as a new therapeutic option for bullous pemphigoid.
24 haracteristics of a patient with orf-induced pemphigoid.
25 n are oral lichen planus and mucous membrane pemphigoid.
26 3 IgG-induced mouse model of mucous membrane pemphigoid.
27 phigoid/cicatricial pemphigoid, or anti-p200 pemphigoid.
28 clinical clue to the diagnosis of anti-p200 pemphigoid.
29 stering skin diseases, pemphigus and bullous pemphigoid.
30 on hands and feet, resembling dyshidrosiform pemphigoid.
31 cquisita or anti-laminin-332 mucous membrane pemphigoid.
32 he clinical benefit of patients with bullous pemphigoid.
33 ues to facilitate the diagnosis of anti-p200 pemphigoid.
34 the treatment of refractory mucous membrane pemphigoid.
35 ers had immunopathologic findings of bullous pemphigoid.
36 n lesions can be the only symptom of bullous pemphigoid.
37 common disease presenting as DG, followed by pemphigoid.
38 ses were diagnosed as benign mucous membrane pemphigoid, 10 as lichen planus or lichenoid mucositis (
39 le; specifically, 6951 patients with bullous pemphigoid, 1669 patients with pemphigus, and 79 patient
40 directed against the NC16A domain of bullous pemphigoid 180 (collagen XVII), a transmembrane protein
41 ind to this immunodominant region of bullous pemphigoid 180 are capable of inducing a skin disease th
42 ts, all of whom had circulating anti-bullous pemphigoid 180 autoantibodies, showed a specific prolife
44 ses were diagnosed as benign mucous membrane pemphigoid; 2 cases each of linear IgA disease and pemph
47 ld also be resistant to experimental bullous pemphigoid, a disease with a pathogenesis thought to be
49 sive drugs used for treating mucous membrane pemphigoid and certain definitive categories of mucous m
50 d characterization of subsets of cicatricial pemphigoid and help distinguishing them from bullous pem
51 antibody-reactive site recognized by bullous pemphigoid and herpes gestationis sera) and the other in
53 ithelium of patients with ocular cicatricial pemphigoid and increased levels of BiP/GRP78, sXBP1 and
54 ients with other variants of mucous membrane pemphigoid and mAb GoH3 and BQ16 to integrin alpha6.
56 case of treatment-refractory mucous membrane pemphigoid and propose a mechanism to explain the lack o
57 ne for short-term blister control in bullous pemphigoid and significantly safer in the long-term.
58 desquamative gingivitis secondary to IgG/IgA pemphigoid and the management of this challenging varian
59 -epidermal-junction is a hallmark of bullous pemphigoid and was shown to be important for pathogenesi
60 gus foliaceous, pemphigus, vulgaris, bullous pemphigoid, and benign chronic pemphigus, however, tPA i
61 aniridia, chemical/thermal burn, cicatrizing pemphigoid, and chronic ocular surface inflammation and
62 ng Stevens-Johnson syndrome, mucous membrane pemphigoid, and congenital disorders) who underwent KPro
64 were diagnosed; there was 1 case of bullous pemphigoid; and 1 suspected case of paraneoplastic pemph
67 l transmembrane protein, the 180 kDa bullous pemphigoid antigen (BP180), also known as type XVII coll
68 ing primers specific for the 230 kDa bullous pemphigoid antigen (BPAG1), the 180 kDa bullous pemphigo
70 phigoid antigen (BPAG1), the 180 kDa bullous pemphigoid antigen (BPAG2), the alpha6 and beta4 chains
82 utoreactive T cells to an epitope of bullous pemphigoid antigen 180 kilodaltons were identified in th
83 integrin-associated plakin protein, bullous pemphigoid antigen 1e (BPAG1e) functions as a scaffold f
86 in grafts from mice expressing human bullous pemphigoid antigen 2 (hBPAG2) in epidermal basement memb
87 .05), and in skin from patients with bullous pemphigoid antigen 2 mutations (n = 3) the counts were 5
88 such as the integrin alpha6beta4 and bullous pemphigoid antigen 2 within the hemidesmosomes along the
92 of zinc finger protein 185 (ZNF185), bullous pemphigoid antigen gene (BPAG1), and prostate secretory
93 est to the importance of the 180-kDa bullous pemphigoid antigen in the attachment of the epidermis to
97 proteins: one enriched with the two bullous pemphigoid antigens (BP230, BP180) and one enriched with
98 (cell adhesion molecules in desmosomes), and pemphigoid antigens are found in hemidesmosomes (which m
102 confirmed the clinical diagnosis of IgG/IgA pemphigoid as the cause of desquamative gingivitis and t
103 + cells-issues of relevance to patients with pemphigoid as well as individuals undergoing gene replac
105 ntly demonstrated that reactivity of bullous pemphigoid autoantibodies to the BP180 ectodomain is alm
107 techniques for detecting circulating bullous pemphigoid autoantibodies, including other enzyme-linked
108 0 and 230 kDa proteins recognized by bullous pemphigoid autoantibodies, LABD97 has been thought to re
109 hemidesmosome plaque is the 230-kDa bullous pemphigoid autoantigen (BP230/BPAG1), which connects dir
111 this area: inflammatory process - cicatrical pemphigoid, benign neoplastic process - chondroma, malig
115 ed Hospitals with a new diagnosis of bullous pemphigoid (BP) between May 1, 1997 and September 1, 200
117 Although predisposing factors for bullous pemphigoid (BP) have been recently established, no clini
138 from the sera of some patients with bullous pemphigoid (BP) react with a 180 kDa protein termed BPAg
140 s long been a histologic hallmark of bullous pemphigoid (BP), a subepidermal autoimmune blistering di
142 d by autoantibodies in patients with bullous pemphigoid (BP), and absent in patients with one type of
143 om patients with pemphigus vulgaris, bullous pemphigoid (BP), and chronic atopic and chronic ocular r
144 ith a blistering skin disease called bullous pemphigoid (BP), is a transmembrane component of the hem
145 ents with MMP, 1 of 50 patients with bullous pemphigoid (BP), none of 7 with pemphigus, and 3 of 32 o
146 gG passive transfer murine models of bullous pemphigoid (BP), pemphigus foliaceus (PF), and pemphigus
151 ding an experimental murine model of bullous pemphigoid by showing that the plasminogen/plasmin signa
152 pion repair in patients with mucous membrane pemphigoid can be achieved if control of inflammation is
153 quamative gingivitis associated with IgG/IgA pemphigoid can be challenging to diagnose and treat.
155 ermolysis bullosa acquisita, mucous membrane pemphigoid/cicatricial pemphigoid, or anti-p200 pemphigo
158 e compared the functional characteristics of pemphigoid conjunctival fibroblasts to normal conjunctiv
163 derwent clinical evaluation with the Bullous Pemphigoid Disease Activity Index (BPDAI); and 36 patien
164 s experimental mouse models of pemphigus and pemphigoid disease are increasingly being used to unders
165 ized by sera from 20 untreated patients with pemphigoid disease limited to the oral cavity, and to de
169 esterase 4, and leukotriene B4 inhibitors in pemphigoid disorders, and chimeric antigen receptor T ce
170 e compared with those in the nonflare state (pemphigoid [emotions: flare, 52.4 (IQR, 38.1-69.0); nonf
171 sis of pemphigus vulgaris/foliaceus, bullous pemphigoid, epidermolysis bullosa acquisita, mucous memb
175 utoantibodies, whereas the autoantibodies in pemphigoid fix complement and mediate inflammation.
176 ropose the unifying term pruritic nonbullous pemphigoid for all patients with immunopathologic findin
178 of grade 3 fatigue, one case each of grade 3 pemphigoid, grade 3 hypothyroidism, and grade 3 peripher
179 rmis to epidermal basement membrane (eg, the pemphigoid group of disorders [bullous, gestational, and
180 re-noncicatrizing disease>ocular cicatricial pemphigoid>chemical burns>Stevens-Johnson syndrome.
182 In the literature, patients with bullous pemphigoid have been reported to have itch without blist
184 such as Stevens-Johnson syndrome, and ocular pemphigoid have little hope of visual recovery from conv
185 here are now three bullous diseases, bullous pemphigoid, herpes gestationis, and cicatricial pemphigo
186 go (HR, 0.534; 95% CI, 0.254-1.123), bullous pemphigoid (HR, 0.524; 95% CI, 0.140-1.956), and Grover
187 t is, chronic spontaneous urticaria, bullous pemphigoid, IgA (Henoch-Schonlein purpura) and IgM/IgG i
189 her patients with anti-laminin-5 cicatricial pemphigoid immunoprecipitated both laminin-6 and laminin
192 port describes the management of cicatricial pemphigoid in a patient with multiple sites of gingival
193 English literature have reported cicatricial pemphigoid in children, primarily as oral mucosal lesion
195 in the three different clinical variants of pemphigoid, involves the recognition by T cells of a cla
200 Mucous membrane pemphigoid or cicatricial pemphigoid is a mucocutaneous blistering disease charact
208 catricial pemphigoid (benign mucous membrane pemphigoid) is an autoimmune vesiculobullous disease tha
211 isted of pemphigus vulgaris, mucous membrane pemphigoid, lichen planus, linear immunoglobulin A disea
212 phigoid, pemphigus vulgaris, and cicatricial pemphigoid-like diseases did not recognize the 205-kDa p
214 ch as the use of topical corticosteroids for pemphigoid may have played a pivotal role in improving t
216 immunobullous disease ocular mucous membrane pemphigoid (MMP) causes blindness; the pathogenesis of s
218 t International Consensus on Mucous Membrane Pemphigoid (MMP) guidance, which recommends that clinica
225 An accurate diagnosis of mucous membrane pemphigoid (MMP) is essential to reduce diagnostic and t
226 diagnosis and management of mucous membrane pemphigoid (MMP) published in the English literature sin
227 A case of anti-laminin-332 mucous membrane pemphigoid (MMP) that developed shortly after a diphther
231 lupus erythematosus (BSLE) (n = 3), bullous pemphigoid (n = 16), pemphigus (n = 11), and normal cont
232 igus vulgaris (n=40, USA and Japan), bullous pemphigoid (n=40, USA), and healthy donors (n=55, USA) s
238 junctiva of patients with ocular cicatricial pemphigoid (OCP), also labeled as ocular mucous membrane
239 patients with BP, 17 with ocular cicatricial pemphigoid (OCP), and 22 with oral pemphigoid (OP) to a
245 Immune checkpoint inhibitor-induced bullous pemphigoid often occurred during the course of anti-PD-1
249 the sera of patients with MMP, OCP, and oral pemphigoid (OP) recognize only their target antigens, an
250 catricial pemphigoid (OCP), and 22 with oral pemphigoid (OP) to a panel of 218 haplotypes of normal i
253 cious anemia (OR = 1.91; 95% CI, 1.29-2.84), pemphigoid (OR = 1.90; 95% CI, 1.62-2.24), psoriasis (OR
254 tural proteins that promote cell matrix (eg, pemphigoid) or cell-to-cell (eg, pemphigus) adhesion in
257 d malposition resulting from mucous membrane pemphigoid, particularly cicatricial entropion, that req
258 source of infection in patients with bullous pemphigoid, particularly in the setting of high-dose imm
259 upus erythematosus patients (n = 3), bullous pemphigoid patients (n = 15), and normal humans (n = 12)
267 ah Health autoimmune dermatology clinic with pemphigoid, pemphigus, dermatitis herpetiformis, and lin
268 cle is to review articles on mucous membrane pemphigoid published in the English literature from 2006
272 predominant epitope identified with bullous pemphigoid sera is located in the noncollagenous region
273 sera recognize beta4 integrin subunit, oral pemphigoid sera recognize alpha6 integrin subunit, and a
276 Interestingly, all three of the bullous pemphigoid sera that were negative in this assay had bee
278 g a skin disease that closely mimics bullous pemphigoid, supporting the hypothesis that epitopes with
279 per presents a case of childhood cicatricial pemphigoid that clinically manifested as necrotizing ulc
280 phigoid, herpes gestationis, and cicatricial pemphigoid, that are known to be associated with an auto
281 us membranes lose cell-cell adhesion, and in pemphigoid, the basal keratinocytes lose adhesion to the
282 ay be indistinguishable from that of bullous pemphigoid, the latter being the most common autoimmune
283 ised controlled trial of adults with bullous pemphigoid (three or more blisters at two or more sites
284 th a complement-targeting therapy in bullous pemphigoid, to our knowledge, and supports further studi
285 elderly patients, including scabies, bullous pemphigoid, transient acantholytic dermatosis, and mycos
288 ocular disorders, including ocular cicatrial pemphigoid, vernal keratoconjunctivitis, and pterygium.
289 ial role of Staphylococcus aureus in bullous pemphigoid was explored by examining the colonization ra
290 ng IgA against an antigen in mucous membrane pemphigoid was found in about 20% of patients, without p
291 oglobulin as treatment of ocular cicatricial pemphigoid was found to be superior to conventional immu
292 well-known target antigen in mucous membrane pemphigoid, was a major autoantigen in orf-induced immun
293 ed phenotype exist in ocular mucous membrane pemphigoid, we compared the functional characteristics o
294 reover, patients with ocular mucous membrane pemphigoid were characterized by marginal N-acetylglucos
295 nger onset (<60 years) of ocular cicatricial pemphigoid were found to have disease evolution similar
296 [range] age, 75 [55-85] years) with bullous pemphigoid were included in the trial, of whom 5 were wo
297 r preliminary studies in humans with bullous pemphigoid, which is also associated with excessive leve
298 The most common of these diseases is bullous pemphigoid, which mainly affects older people and the re