ライフサイエンスコーパス: penicillin resistance

1 ivity is essential for clinical pneumococcal penicillin resistance.

2 enicillin resistant and 20% had intermediate penicillin resistance.

3 ls without branches and also completely lost penicillin resistance.

4 ides from the cell wall and complete loss of penicillin resistance.

5 jor part is played by the acylation steps in penicillin resistance.

6 strains caused a virtually complete loss of penicillin resistance.

7 ecific amplicons was effective for screening penicillin resistance.

8 (P < .05) were significantly associated with penicillin resistance.

9 fically beta-lactam use were associated with penicillin resistance.

10 ency pde1 mutations conferring S. pneumoniae penicillin resistance.

11 an evolutionary gateway conferring low-level penicillin resistance.

12 els of cyclic-di-adenosine monophosphate and penicillin resistance.

13 quire further genetic changes and high-level penicillin resistance.

14 The cost of penicillin-resistance acquisition for the Streptococcus

15 tures, three of which had a known history of penicillin resistance and were thought to originate from

16 m susceptible recipient cells to donor level penicillin resistance, and a resistant transformant was

17 types of S. pneumoniae, independent of their penicillin resistance, and it is very specific for this

18 cell wall structure, original high level of penicillin resistance, and normal sensitivity to lysis.

19 and 54/243 isolates tested were resistant to penicillin (resistance breakpoint >/= 2 mug/ml).

20 inding proteins (PBPs) can confer high-level penicillin resistance but other poorly understood geneti

21 us detection of S. pneumoniae and high-level penicillin resistance can be accurately performed with t

22 of Streptococcus pneumoniae with high-level penicillin resistance collected during a national survei

23 ses per 100,000 population), whereas sero19A penicillin resistance (defined as a minimum inhibitor co

24 We found no evidence of penicillin resistance due to either serotype switching o

25 d the percentage of isolates with high-level penicillin resistance from cultures taken from children

26 005-2007, which paralleled increased sero19A penicillin resistance (from 28.7% [163 of 567 isolates]

27 Prevalence of penicillin resistance has declined in the years since go

28 lationship between cell wall abnormality and penicillin resistance has remained obscure.

29 The level of penicillin resistance in clinical isolates of Streptococ

30 cell wall composition that often accompanies penicillin resistance in clinical strains of pneumococci

31 The major mechanism of dissemination of penicillin resistance in Iceland appears to be the repea

32 ganisms are tested locally, particularly for penicillin resistance in pneumococci and glycopeptide re

33 We show that the frequency of penicillin resistance in S. pneumoniae can be explained

34 Penicillin resistance in S. pneumoniae depends in part u

35 s plays a critical role in the expression of penicillin resistance in S. pneumoniae.

36 ants that may further attenuate the level of penicillin resistance in the bacteria.

37 d rsiP homologues are required for inducible penicillin resistance in these species.

38 Penicillin resistance increased from 1988 (8.4%) to 1991

39 esting that these isolates probably acquired penicillin resistance independently.

40 Penicillin resistance is rapidly increasing among S. pne

41 This data indicates that low-level penicillin resistance is sometimes conferred by determin

42 Low-concentration penicillin resistance (MICs = 0.06 microgram/ml to 0.25

43 Penicillin resistance occurred in one-fifth of all S. pn

44 ts encoding the low affinity of PBPs and the penicillin resistance of the bacteria are separable from

45 ux pump in conferring chromosomally mediated penicillin resistance on certain strains of Neisseria go

46 genes expressing chloramphenicol resistance, penicillin resistance, or gyrase A function can effectiv

47 <.001) and more likely to have isolates with penicillin resistance (P=.03).

48 Increasing rates of penicillin resistance, particularly in viridans group st

49 ts and population analyses to identify a new penicillin resistance pathway that is independent of PBP

50 Penicillin resistance peaked at 19.6% in 1991, then decl

51 Penicillin resistance was found in 16% (4/25) of S. pneu

52 Apparent prevalence of penicillin resistance was surprisingly high in human and

53 Those serotypes associated with penicillin resistance were 6B, 9V, 19A, and 19F.

54 acy of a six-primer PCR assay in identifying penicillin resistance were analyzed by using clinical is

55 We conclude that agr and hla (along with penicillin resistance) were essential for world dominanc