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1 red for elevated adenosine-induced prolonged penile erection.
2 established model of electrically stimulated penile erection.
3 nd peripherally, notably through its role in penile erection.
4 m by the NO-cGMP pathway is important during penile erection.
5 thalamic and spinal cord regions involved in penile erection.
6 vivo efficacy in anesthetized dog model for penile erection.
7 annot be ignored as a potential regulator of penile erection.
8 g corpus cavernosum pressure to culminate in penile erection.
9 olonged immobility, 22-kHz vocalization, and penile erections.
10 that adenosine was induced during sustained penile erection and contributes to PI3K/AKT activation a
11 ctive dopamine receptor agonist, facilitates penile erection and is effective in patients suffering f
12 apism is featured with prolonged and painful penile erection and is prevalent among males with sickle
13 spliced forms of nNOS are major mediators of penile erection and so may be targets for therapeutic in
14 vernosum is the most important structure for penile erection, and its dysfunction causes many physiol
15 gulating male reproductive behavior, such as penile erection, and NOS inhibitors prevent erection.
17 ncreased propensity for sustained and robust penile erections as a potential underlying mechanism.
18 ions, including micturition, defecation, and penile erection, as well as to brain networks controllin
20 ted by neuronal NO synthase (nNOS) initiates penile erection, but has not been thought to participate
22 Viagra), a specific PDE5 inhibitor, promotes penile erection by blocking the activity of PDE5, which
25 NOS phosphorylation, and subsequent impaired penile erection featured with the reduction of ratio of
26 mediates both initiation and maintenance of penile erection, implying unique approaches for treating
27 f Krause corpuscle afferent terminals evoked penile erection in male mice and vaginal contraction in
30 ry potent and efficacious in vivo, eliciting penile erections in rats at a dose of 0.03 micromol/kg,
31 g, that Rho-kinase antagonism stimulates rat penile erection independently of nitric oxide, introduce
35 Nitric oxide (NO), a mediator involved in penile erection, is synthesized by the nitric oxide synt
39 C6' treatment reversed abnormalities in key penile erection signaling molecules, including phosphodi
40 istent inability to obtain and/or maintain a penile erection sufficient for adequate sexual relations
42 gnaling is implicated in normal and abnormal penile erection, the exact role and the underlying mecha
44 itric oxide (NO) pathway is known to mediate penile erection under normal conditions, we hypothesized
46 aling via A(2B)R activation, contributing to penile erection via PI3K/AKT-dependent eNOS activation.