ライフサイエンスコーパス: pentose phosphate pathway

1 glucose-6-phosphate dehydrogenase or in the pentose phosphate pathway.

2 focused on the importance of glycolysis and pentose phosphate pathway.

3 asing dependence upon glutaminolysis and the pentose phosphate pathway.

4 iphosphatase 4 (PFKFB4), drives flux through pentose phosphate pathway.

5 y by glycolysis and to a minor extent by the pentose phosphate pathway.

6 sotope effect is only found in the reductive pentose phosphate pathway.

7 o glycolysis and to the oxidative arm of the pentose phosphate pathway.

8 te dehydrogenase (G6PDH), a regulator of the pentose phosphate pathway.

9 increase in glucose metabolic flux into the pentose phosphate pathway.

10 by high levels of nutrient flux through the pentose phosphate pathway.

11 tributor to cytosolic NADPH is the oxidative pentose phosphate pathway.

12 onance assigned to 6-phosphogluconate in the pentose phosphate pathway.

13 o alterations in glucose metabolized via the pentose phosphate pathway.

14 trated defects in the oxidative phase of the pentose phosphate pathway.

15 by the Calvin-Benson-Bassham (CBB) reductive pentose phosphate pathway.

16 metabolism, including several mRNAs from the pentose phosphate pathway.

17 d failed to predict the use of the oxidative pentose phosphate pathway.

18 trate for NADPH generation via the oxidative pentose phosphate pathway.

19 ydrogenase (6-PGDH), the third enzyme of the pentose phosphate pathway.

20 nvolved in starch turnover and the oxidative pentose phosphate pathway.

21 mark of transformed cells, is to support the pentose phosphate pathway.

22 ated flux through the antioxidant-generating pentose phosphate pathway.

23 pgl), which encodes the second enzyme in the pentose phosphate pathway.

24 transport, a glucokinase and enzymes of the pentose phosphate pathway.

25 y between the TGF-beta signaling pathway and pentose phosphate pathway.

26 ed for 4-carbon saccharides arising from the pentose phosphate pathway.

27 -phosphate and d-xylulose 5-phosphate in the pentose phosphate pathway.

28 carbon dioxide assimilation via a reductive pentose phosphate pathway.

29 cycle and its upstream contributors, and the pentose phosphate pathway.

30 e groups comprising either glycolysis or the pentose phosphate pathway.

31 fts from primarily glycolytic to include the pentose phosphate pathway.

32 arboxylic acid (TCA) cycle and an incomplete pentose phosphate pathway.

33 ion of the reduced form of NADPH through the pentose phosphate pathway.

34 e ATP generating roles of glycolysis and the pentose phosphate pathway.

35 eins missing from current annotations of the pentose phosphate pathway.

36 PH and ribose 5-phosphate production via the pentose phosphate pathway.

37 ot the tricarboxylic acid cycle, and not the pentose phosphate pathway.

38 osphogluconolactonase (6Pgl) involved in the pentose phosphate pathway.

39 pool and renders such cells dependent on the pentose phosphate pathway.

40 ogenase and metabolically integrated via the pentose phosphate pathway.

41 lytic-tricarboxylic acid cycle route and the pentose phosphate pathway.

42 s the tricarboxylic acid (TCA) cycle and the pentose phosphate pathway.

43 se 6-phosphate and by intermediates from the pentose phosphate pathway.

44 ne metabolism, glutamate metabolism, and the pentose phosphate pathway.

45 and diversion of glucose metabolites to the pentose phosphate pathway.

46 metabolism in the non-oxidative part of the pentose phosphate pathway.

47 idation via the tricarboxylic acid cycle and pentose-phosphate pathway.

48 nd is linked through its product Xu5P to the pentose-phosphate pathway.

49 iting enzyme in the nonoxidative part of the pentose-phosphate pathway.

50 ctate even in the presence of oxygen via the pentose-phosphate pathway.

51 drolytic enzymes and also enzymes key to the pentose-phosphate pathway.

52 upper Embden-Meyerhof-Parnas pathway nor the pentose-phosphate pathway.

53 metabolites through the oxidative arm of the pentose-phosphate pathway.

54 nas, the Entner-Doudoroff, and the reductive pentose phosphate pathways.

55 : the Embden-Meyerhof, Entner-Doudoroff, and pentose phosphate pathways.

56 s in both the oxidative and the nonoxidative pentose phosphate pathways.

57 A high flux was found through the oxidative pentose phosphate pathway (19.99 +/- 4.39 micromol d(-1)

58 of the Calvin-Benson-Bassham (CBB) reductive pentose phosphate pathway, a scheme that does not appear

59 as the transition from the glycolysis to the pentose phosphate pathway-a conserved first-line respons

60 C-null strains show indications of oxidative pentose phosphate pathway activation as well as increase

61 te elevated activities of glycolysis and the pentose phosphate pathway, activation of macrophages wit

62 on of Nrf2 and the ARE, coupled with reduced pentose phosphate pathway activity and decreased generat

63 Fasting stimulated pentose phosphate pathway activity and metabolism of [U-

64 etylglucosamine, S. marcescens exhibits high pentose phosphate pathway activity and nucleotide synthe

65 that CP12 is essential to separate oxidative pentose phosphate pathway activity from Calvin-Benson cy

66 NADPH, a substrate for NOX, and pentose phosphate pathway activity increased with glucos

67 nic state, cells exhibiting a contrary, high pentose phosphate pathway activity state, spontaneously

68 ose uptake, glucose metabolism and oxidative pentose phosphate pathway activity were similarly repres

69 tabolites provides an important biomarker of pentose phosphate pathway activity, triacylglycerol synt

70 scription factors repress glucose uptake and pentose phosphate pathway activity, while their low numb

71 sponse in the clinic and of monitoring tumor pentose phosphate pathway activity.

72 cose and demonstrated reduced glycolysis and pentose phosphate pathway activity.

73 nd most abundant sugar in nature, is via the pentose phosphate pathway after a two-step or three-step

74 d an increase in the oxidative branch of the pentose phosphate pathway and (13)C incorporations sugge

75 rovided the first link between the bacterial pentose phosphate pathway and activation of host IFN-bet

76 cemia by increasing glucose flux through the pentose phosphate pathway and enhancing fatty acid synth

77 fluxes in the pentose phosphate pathway and gluconeogenesis were stabl

78 th a shift in glucose metabolism between the pentose phosphate pathway and glycolysis, (2) interactio

79 between the oxidative steps of the oxidative pentose phosphate pathway and glycolysis.

80 ection against hydrogen peroxide insult in a pentose phosphate pathway and GSH-dependent manner.

81 a fructose-2,6-bisphosphatase, promoting the pentose phosphate pathway and helping to lower intracell

82 inhibition of MCT1 suppressed the oxidative pentose phosphate pathway and increased levels of reacti

83 yrophosphate production by the non-oxidative pentose phosphate pathway and late steps by modulating a

84 nsated by activation of anabolic metabolism (pentose phosphate pathway and lipogenesis) allowing live

85 glucose is preferentially metabolized by the pentose phosphate pathway and mitochondria, as opposed t

86 glutathione biosynthesis or in the oxidative pentose phosphate pathway and other NADPH-producing enzy

87 of genes associated with photosynthesis, the pentose phosphate pathway and primary metabolism, but lo

88 We identified the pentose phosphate pathway and pyrimidine metabolism - bo

89 gene deletion mutants from upper glycolysis, pentose phosphate pathway and the Entner-Doudoroff pathw

90 ls further showed enhanced activation of the pentose phosphate pathway and the glutathione system, wh

91 se (G6PD) is the rate-limiting enzyme of the pentose phosphate pathway and the principal source of NA

92 is required to divert glucose flux into the pentose phosphate pathway and thereby generate sufficien

93 biosynthetic genes and discrete steps in the pentose phosphate pathway and tricarboxylic acid cycle t

94 The oxidative and nonoxidative pentose phosphate pathways and the ratio between them al

95 ificant increase in rate-limiting enzymes of pentose-phosphate pathway and 1-carbon metabolism in pos

96 a-dystroglycan, all consistent with enhanced pentose-phosphate pathway and 1-carbon metabolism that c

97 ppears to direct glycolytic metabolites into pentose-phosphate pathway and 1-carbon metabolism, which

98 oprotective and repair pathways, such as the pentose-phosphate pathway and 1-carbon metabolism, which

99 factor that may regulate serum urate via the pentose-phosphate pathway and MRPS7 and IDH2 that encode

100 bstrates, such as sedoheptulose-6-phosphate (pentose phosphate pathway) and serine and glycine (1-car

101 pathways (the Embden-Meyerhof-Parnas or the pentose-phosphate pathway) and in dependence of Na(+) av

102 ncluding glycerol oxidation, glycolysis, the pentose phosphate pathway, and carbon sources of stored

103 cluding glycolysis, the oxidative arm of the pentose phosphate pathway, and de novo lipid biosynthesi

104 ses including fatty acid esterification, the pentose phosphate pathway, and gluconeogenesis through t

105 lism, reactive oxygen species clearance, the pentose phosphate pathway, and glutathione homeostasis.

106 f glycolysis, gluconeogenesis, the oxidative pentose phosphate pathway, and glycogen metabolism were

107 ical pathways, such as glycolysis, oxidative pentose phosphate pathway, and glycogen metabolism, were

108 ycolysis, through the oxidative/nonoxidative pentose phosphate pathway, and into the general phenylpr

109 way, nucleotide sugars, intermediates of the pentose phosphate pathway, and lipogenesis, including pr

110 through up-regulation of glucose influx, the pentose phosphate pathway, and NAD salvaging pathways.

111 for the complete non-oxidative phase of the pentose phosphate pathway, and others predicted to media

112 rom erythrose 4-phosphate, generated via the pentose phosphate pathway, and phosphoenolpyruvate.

113 tion of glucose flux between glycolysis, the pentose phosphate pathway, and serine biosynthesis seems

114 ed reduced glucose metabolism in glycolysis, pentose phosphate pathway, and sorbitol pathway, which m

115 r phosphates that constitute glycolysis, the pentose phosphate pathway, and the RNA and DNA backbone,

116 ent decrease in flux through glycolysis, the pentose phosphate pathway, and the tricarboxylic acid (T

117 LC7A11-associated cystine metabolism and the pentose phosphate pathway, and uncover an accompanying m

118 lites of glycolysis, TCA cycle, amino acids, pentose phosphate pathway, and urea cycle, from LRMS and

119 ioning of glucose carbons into C1/folate and pentose phosphate pathways, and increased tricarboxylic

120 Thus the first two steps of the pentose phosphate pathway are catalysed by a single nove

121 When reactions of the oxidative pentose phosphate pathway are taken into consideration,

122 nd fatty acid-utilizing, upregulation of the pentose phosphate pathway as a source of antioxidant NAD

123 ase step, promoting carbon overflow into the pentose phosphate pathway as evidenced by the increased

124 ional view of the Calvin cycle and oxidative pentose phosphate pathway as separate systems, they are

125 s of NADPH reflecting a higher activation of pentose phosphate pathway as this is accompanied with hi

126 nclude up-regulated NADPH production via the pentose phosphate pathway as well as activation of the N

127 etabolic intermediates in the glycolytic and pentose phosphate pathways as well as abnormal mitochond

128 h glycolysis and the oxidative branch of the pentose phosphate pathway, as well as to stimulate de no

129 Results suggested that the deinococcal pentose phosphate pathway augmented the DNA excision rep

130 TORC1 directs increased glucose flux via the pentose phosphate pathway back into glycolysis, thereby

131 idative and the nonoxidative branches of the pentose phosphate pathway blocked the stimulation of glu

132 Glycolysis and the pentose phosphate pathway both play a central role in th

133 Its major source is considered to be the pentose phosphate pathway, but cytosolic NADP(+)-depende

134 bolism through the citric acid cycle and the pentose phosphate pathway by 240 and 90%, respectively,

135 er occurs through diverting glucose into the pentose phosphate pathway by ADPr inhibition of glyceral

136 Inhibition of the pentose phosphate pathway by glucose-6-phosphate dehydro

137 Adiponectin also reversed induction of the pentose phosphate pathway by HFD.

138 te of the cells with regard to the oxidative pentose phosphate pathway, Calvin cycle, tricarboxylic a

139 tration of metabolites of glycolysis and the pentose phosphate pathway, central metabolic players in

140 ne phosphate and fructose-1,6-bisphosphate), pentose phosphate pathway components and Kreb's cycle in

141 non-photosynthetic conditions, the oxidative pentose phosphate pathway contributes to basic metabolis

142 Idn1 act together to shunt glucose into the pentose phosphate pathway, creating an alternative route

143 Further, pentose phosphate pathway deregulation and impaired fatt

144 uiescent mutant 1 (NQM1), a paralogue to the pentose phosphate pathway enzyme transaldolase (TAL1), a

145 3.6% of glycolysis, and three non-oxidative pentose phosphate pathway exchange fluxes were calculate

146 lycolysis, the tricarboxylic acid cycle, the pentose phosphate pathway, fatty acid and nucleotide bio

147 down and mitochondrial processing toward the pentose phosphate pathway, favoring anabolic over catabo

148 is upon Gpi1 inactivation was compensated by pentose phosphate pathway flux and increased mitochondri

149 The oxidative pentose phosphate pathway flux was 3.6% of glycolysis, a

150 associated with a corresponding increase in pentose phosphate pathway flux, assessed using (13)C-lab

151 owever, LPS also induced a small decrease in pentose phosphate pathway fluxes and an increase in glut

152 e identified increased citric acid cycle and pentose phosphate pathway fluxes as consistent markers o

153 ol g(-1)h(-1) that was partly caused by high pentose phosphate pathway fluxes.

154 Embden-Meyerhof-Parnas, Entner-Doudoroff, or pentose phosphate pathway for glycolytic carbon metaboli

155 acilitate metabolic reprogramming toward the pentose-phosphate pathway for achieving redox balance an

156 reduced by NADPH generated via the oxidative pentose phosphate pathway, functions as a signal in germ

157 Genes involved in glycolysis (PFKP), pentose phosphate pathway (G6PD), and defense against li

158 lytic RA T cells diverted glucose toward the pentose phosphate pathway, generated more NADPH, and con

159 nase (G6PD), the rate-limiting enzyme in the pentose phosphate pathway, generates NADPH in a reaction

160 We show that pentose-phosphate-pathway generation of NADPH is critica

161 nd metabolomics revealed that glycolytic and pentose phosphate pathway genes are induced by dERR, and

162 se, ferredoxin reductase, and enzymes in the pentose phosphate pathway), genes encoding novel metabol

163 disorders, we focused on the glycolytic and pentose phosphate pathways (GPPPs).

164 Loss of the pentose phosphate pathway had no effect on plaque format

165 d zwf1, which encode the first enzyme in the pentose phosphate pathway, have a more severe growth phe

166 es necessary to reduce NADP by the oxidative pentose phosphate pathway (hexokinase, glucose-6-phospha

167 rains cell proliferation, glycolysis and the pentose phosphate pathway in a catalytic-activity-indepe

168 x from glucose-6-phosphate (G6P) through the pentose phosphate pathway in egg extracts maintains NADP

169 dogenously produced from glucose through the pentose phosphate pathway in humans.

170 of the Calvin-Benson-Bassham (CBB) reductive pentose phosphate pathway in Rhodobacter capsulatus.

171 r supply from the Calvin cycle and oxidative pentose-phosphate pathway in primary metabolism to pheny

172 Physiological analysis indicated that the pentose-phosphate pathway, in particular, was poisoned b

173 Fru-2,6-BPase), and through promotion of the pentose phosphate pathway, increases NADPH production to

174 te) was associated with perturbations in the pentose phosphate pathway induced initially by colistin

175 ogenase (H6PD) is the initial component of a pentose phosphate pathway inside the endoplasmic reticul

176 the two-step conversion of glucose into the pentose phosphate pathway intermediate 6-phosphogluconat

177 sphate cyclases are enzymes that utilize the pentose phosphate pathway intermediate, sedoheptulose 7-

178 chment of Embden-Meyerhof-Parnas pathway and pentose phosphate pathway intermediates indicated high a

179 ted glucose uptake and metabolism, increased pentose phosphate pathway intermediates, with a complime

180 ase (Tps1) is responsible for regulating the pentose phosphate pathway, intracellular levels of NADPH

181 rangements in the nonoxidative branch of the pentose phosphate pathway involving transaldolase that p

182 in the endosperm and suggest the amyloplast pentose phosphate pathway is a heat-sensitive step in ma

183 The pentose phosphate pathway is a key route of glucose meta

184 The pentose phosphate pathway is a major site of NADPH produ

185 er these conditions, NADPH generation by the pentose phosphate pathway is impaired, but AMPK induces

186 Although the oxidative pentose phosphate pathway is important for tumor growth,

187 establish that the carbon overflow into the pentose phosphate pathway is mainly through its non-oxid

188 ial amounts of NADPH and do so even when the pentose phosphate pathway is operational.

189 ehydrogenase (G6PD), the first enzyme of the pentose phosphate pathway, is the principal intracellula

190 metabolism of glucose from glycolysis to the pentose phosphate pathway, it was hypothesized to have a

191 volving tricarboxylic acid cycle, polyol and pentose phosphate pathways, leading to improved redox ba

192 f cells in 5 mM glucose or inhibition of the pentose phosphate pathway maintained GSSG elevation and

193 Pentose-phosphate-pathway-mediated inhibition of cell de

194 ysis, and the Krebs cycle, but the levels of pentose phosphate pathway metabolites and of many free a

195 in carbohydrate or hydroxyl acid metabolism, pentose phosphate pathway metabolites, or free fatty aci

196 ugh lack of GLUT1 blunted glycolysis and the pentose phosphate pathway, MPhi were metabolically flexi

197 products of glycolysis, the Krebs cycle, the pentose phosphate pathway, nucleobases, UDP-sugars, glyc

198 luding glucose and glutamine metabolism, the pentose phosphate pathway, nucleotide and fatty acid bio

199 s to the less efficient Entner-Doudoroff and pentose phosphate pathways on algal-dominated reefs.

200 xamined the role of the plastidial oxidative pentose phosphate pathway (OPPP) in embryo development.

201 showing that HEXOKINASE1-mediated oxidative pentose phosphate pathway (OPPP) metabolism is required

202 The oxidative pentose phosphate pathway (OPPP) provides plants with im

203 rk on the irreversible part of the oxidative pentose phosphate pathway (OPPP) revealed comparable eff

204 Fluxes through the oxidative pentose phosphate pathway (OPPP) were the principal sour

205 tion network of glycolysis and the oxidative pentose phosphate pathway (OPPP).

206 f the carbon flux between glycolysis and the pentose phosphate pathway or the Kennedy pathway, respec

207 e in the flux of the oxidative branch of the pentose phosphate pathway (ox-PPP) in response to the PD

208 The oxidative pentose phosphate pathway (oxiPPP) contributes to cell m

209 that inhibition of the oxidative arm of the pentose phosphate pathway (oxPPP) is required for antima

210 a dependence on the oxidative branch of the pentose phosphate pathway (oxPPP), and oxPPP inhibition

211 abolites revealed a reduction in glycolysis, pentose phosphate pathway, polyamines and nucleotides, b

212 genation elevates glycolytic flux and lowers pentose phosphate pathway (PPP) activity in mammalian er

213 A sudden increase in pentose phosphate pathway (PPP) activity, the fastest kn

214 dehydrogenase (G6PD) is a key enzyme in the pentose phosphate pathway (PPP) and plays an essential r

215 R-ABL-ERK signaling in H929 cells drives the pentose phosphate pathway (PPP) and RNA biosynthesis, wh

216 analysis identified the up-regulation of the pentose phosphate pathway (PPP) and the antioxidant defe

217 two cytosolic NADPH-producing pathways, the pentose phosphate pathway (PPP) and the NADP-dependent m

218 ADP(+)/NADPH ratio controls flux through the pentose phosphate pathway (PPP) and the polyol pathway,

219 and miR-206 to direct carbon flux toward the pentose phosphate pathway (PPP) and the tricarboxylic ac

220 d and carbohydrate metabolites, particularly pentose phosphate pathway (PPP) and tricarboxylic acid (

221 d the flux allocation between glycolysis and pentose phosphate pathway (PPP) as potential mechanisms

222 al crosstalk between the PI3K/AKT signal and pentose phosphate pathway (PPP) branching metabolic path

223 The importance of the pentose phosphate pathway (PPP) during S. aureus infecti

224 cent case reports suggest a link between the pentose phosphate pathway (PPP) enzyme transaldolase (TA

225 domestic livestock that exclusively use the pentose phosphate pathway (PPP) for hexose catabolism, w

226 y mediating fluxes through glycolysis or the pentose phosphate pathway (PPP) in an oxidative stress-d

227 increased glucose flux through the oxidative pentose phosphate pathway (PPP) in erythrocytes.

228 Survival upon nutrient stress or pentose phosphate pathway (PPP) inhibition depends on co

229 The oxidative pentose phosphate pathway (PPP) is crucial for cancer ce

230 emonstrate that a metabolic shift toward the pentose phosphate pathway (PPP) is necessary for NET rel

231 Here we add the pentose phosphate pathway (PPP) of T. brucei to the glyc

232 The pentose phosphate pathway (PPP) plays a critical role in

233 on increases the flux of glucose through the pentose phosphate pathway (PPP) to increase nucleotide p

234 -phosphogluconate dehydrogenase (PGD) in the pentose phosphate pathway (PPP) were found to be the NAD

235 6PD) is the first, rate-limiting step in the pentose phosphate pathway (PPP), a key metabolic pathway

236 lucose-dependent nucleotide synthesis by the pentose phosphate pathway (PPP), along with sphingolipid

237 atabolic glycolysis but increased use of the pentose phosphate pathway (PPP), and an enhanced abundan

238 rythrose-4-phosphate, an intermediate of the pentose phosphate pathway (PPP), generates 4-phosphoeryt

239 sses and pathways, including glycolysis, the pentose phosphate pathway (PPP), KCl cotransport, ATP re

240 The pentose phosphate pathway (PPP), metabolism in the citri

241 revealed that metabolites in the glycolysis, pentose phosphate pathway (PPP), pyrimidine biosynthesis

242 potential by shunting nectar glucose to the pentose phosphate pathway (PPP), resulting in a reductio

243 s revealed that glucose is funneled into the pentose phosphate pathway (PPP), which is indispensable

244 sphogluconate dehydrogenase in the oxidative pentose phosphate pathway (PPP), while 2-PG activates 3-

245 e metabolism and increased NADPH levels in a pentose phosphate pathway (PPP)-dependent manner.

246 of ribose, leading us to hypothesize that a pentose phosphate pathway (PPP)-responsive regulator med

247 lase (TKT), a key enzyme of the nonoxidative pentose phosphate pathway (PPP).

248 cription factors or increasing NADPH via the pentose phosphate pathway (PPP).

249 with H2O2 rapidly induces glycolysis and the pentose phosphate pathway (PPP).

250 glycolysis and especially the non-oxidative pentose phosphate pathway (PPP).

251 cts carbon flux from the Calvin cycle to the pentose phosphate pathway (PPP).

252 s an anti-oxidant response by regulating the pentose phosphate pathway (PPP).

253 ylic acid cycle and the oxidative arm of the pentose phosphate pathway (PPP).

254 dative pathways and glucose flux through the pentose phosphate pathway (PPP).

255 -5-phosphate with NADP(+) as cofactor in the pentose phosphate pathway (PPP).

256 a cellular anti-oxidant response through the pentose phosphate pathway (PPP).

257 cose flux towards glycolysis relative to the pentose phosphate pathway (PPP).

258 sm in CDCP1+ CSCs is routed to the oxidative pentose phosphate pathway (PPP); multiple cycling of car

259 ediates into the hexosamine biosynthesis and pentose phosphate pathways (PPP).

260 ynthesized endogenously from glucose via the pentose-phosphate pathway (PPP) in stable isotope-assist

261 allow glucose to be metabolized through the pentose-phosphate pathway (PPP), which regenerates NADPH

262 nase (G6PD), the key enzyme of the oxidative pentose phosphate pathway, provides reducing equivalents

263 rate metabolism (glycolysis/gluconeogenesis, pentose phosphate pathway, pyruvate metabolism), proteas

264 ation of glyceraldehyde-3-phosphate into the pentose phosphate pathway; radC, which encodes a RecG-li

265 ion and may enhance glucose turnover via the pentose phosphate pathway rather than through glycolysis

266 demonstrates an essential role of oxidative pentose-phosphate pathway reactions in peroxisomes, like

267 as the first and rate-limiting enzyme in the pentose phosphate pathway, responsible for the generatio

268 lycine into the cytosol is observed; (b) the pentose phosphate pathways serve for biosynthesis only,

269 rnative route for directing glucose into the pentose phosphate pathway that bypasses hexokinase and t

270 otransposition, apoptosis, and the oxidative pentose phosphate pathway that these genes are involved

271 but instead of increasing the glycolysis and pentose phosphate pathway, the glucose is shunted throug

272 While NQM1 appears not to function in the pentose phosphate pathway, the interplay of NQM1 with VH

273 creased concomitant with an induction of the pentose phosphate pathway, the primary source of de novo

274 ently in clinical trials, on glycolysis, the pentose phosphate pathway, the tricarboxylic acid (TCA)

275 vity and redirected glucose flux through the pentose phosphate pathway, thereby conferring a selectiv

276 ow increased flux through glycolysis and the pentose phosphate pathway, thereby establishing a critic

277 UDP-glucose; an alternative to the textbook pentose-phosphate pathway therefore predominates in plan

278 ert energy production from glycolysis to the pentose phosphate pathway to generate NADPH.

279 tes shunting of glucose-6-phosphate into the pentose phosphate pathway to generate reduced glutathion

280 hosphate is then converted by enzymes of the pentose phosphate pathway to glyceraldehyde 3-phosphate

281 ydrogenase, the TCA cycle, and the oxidative pentose phosphate pathway to obtain NADPH.

282 ,6-bisphosphatase, potentially promoting the pentose phosphate pathway to produce NADPH for antioxida

283 nverted by known enzymes of the nonoxidative pentose phosphate pathway to ribose-5-phosphate.

284 ferentially metabolized via the nonoxidative pentose phosphate pathway to synthesize nucleic acids an

285 erating in a gluconeogenic fashion), and the pentose phosphate pathways to form an unforeseen metabol

286 stressed cells protected Rpe and enabled the pentose-phosphate pathway to retain function.

287 ase in TIGAR expression, which regulates the pentose phosphate pathway, treatment with the MUC1-C inh

288 pression of lipogenesis, glycolysis, and the pentose phosphate pathway triggered a strong growth rest

289 showed that: (i) flux through the oxidative pentose phosphate pathway varied independently of the re

290 Activation of the pentose phosphate pathway was identified from the list o

291 and ribulose 5-phosphate 3-epimerase) in the pentose phosphate pathway were overexpressed, and a gera

292 nitrate assimilation, energy metabolism, and pentose-phosphate pathway were most affected.

293 ologs, RpiRc is a potential regulator of the pentose phosphate pathway, which also regulates RNAIII l

294 a methionine auxotrophy was dependent on the pentose phosphate pathway, which is a major source of NA

295 amental routes, glycolysis and the oxidative pentose phosphate pathway, which produces NADPH and the

296 th was potentiated by its suppression of the pentose phosphate pathway, which resulted in inhibition

297 xylic acid (TCA) cycle intermediates and the pentose phosphate pathway, which results in increased gl

298 hat a concerted preemptive activation of the pentose phosphate pathway, which targets both mRNA trans

299 produce NADPH from glucose is the oxidative pentose phosphate pathway, with malic enzyme sometimes a

300 nerated by metabolism of glucose through the pentose phosphate pathway, would have an anticonvulsant