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1 l response to a GABA(A) receptor antagonist, pentylenetetrazole.
2 ures induced by pilocarpine, kainic acid, or pentylenetetrazole.
3 istration of the GABA(A) receptor antagonist pentylenetetrazole.
4 n vivo administration of the chemoconvulsant pentylenetetrazole.
5 convulsant drugs: caffeine (250mg/L; 1.3mM), pentylenetetrazole (1.5g/L; 11.0mM) and picrotoxin (100m
6 sh revealed an exacerbated motor response to pentylenetetrazole, a pro-convulsive drug, consistent wi
7 Ss upon injection of a subconvulsive dose of pentylenetetrazole after 6 weeks.
8 d severity of seizures induced by kainate or pentylenetetrazole, and both necrosis and apoptosis of h
9  mice where the effects of the proconvulsant pentylenetetrazole are attenuated by acutely increasing
10                                        Using pentylenetetrazole as a chemoconvulsant, SST(2), SST(3),
11 ic rat brain regions by the convulsive drug, pentylenetetrazole, as well as by the anticonvulsant dru
12 , did not interfere with seizures induced by pentylenetetrazole, bicucculine, picrotoxin, and strychn
13 at electrographic seizure events, induced by pentylenetetrazole, can be reliably distinguished from e
14 uration or scores of the seizures induced by pentylenetetrazole, DHA significantly prolonged the seiz
15 norhabditis elegans (Lis-1) and Danio rerio (pentylenetetrazole) highlight a reductionist approach.
16  electrically-induced limbic seizures and to pentylenetetrazole induced tonic-clonic seizures.
17 ound persistently enhanced susceptibility to pentylenetetrazole-induced convulsions 15 weeks after TB
18 25 (LY3130481), was fully protective against pentylenetetrazole-induced convulsions in rats without t
19 and delayed the onset of chemical convulsant pentylenetetrazole-induced generalized convulsive seizur
20 ad no effect on Kv2.1 channels, and moderate pentylenetetrazole-induced seizure activity in adult mic
21 ompound 33 was also found to be effective in pentylenetetrazole-induced seizure model (ED50 PTZ = 123
22 is the most effective anticonvulsant against pentylenetetrazole-induced seizures (ED50, 37 mg/kg; PI
23 of mice), and lower threshold for developing pentylenetetrazole-induced seizures (hypoactivity, myocl
24 sing the number and reducing the severity of pentylenetetrazole-induced seizures in rats.
25                            Susceptibility to pentylenetetrazole-induced seizures was assessed by elec
26 enotypes, including increased sensitivity to pentylenetetrazole-induced seizures.
27  and a substantially decreased threshold for pentylenetetrazole-induced seizures.
28 e tested for susceptibility to flurothyl- or pentylenetetrazole-induced seizures.
29 ned using a completely independent paradigm, pentylenetetrazole-induced tonic-clonic seizures, exclud
30  kainic acid model of status epilepticus and pentylenetetrazole model and in male and female mice in
31                                       In the pentylenetetrazole mouse seizure model, 3,3-diethyl lact
32  KO mice to seizures induced by kainic acid, pentylenetetrazole, or flurothyl, although DKO mice were
33 rent convulsant mechanisms (4-Aminopyridine, Pentylenetetrazole, Pilocarpine and Strychnine) resulted
34 tion patterns, transient seizure activity by pentylenetetrazole provoked only a brief c-Jun phosphory
35    However, exposure to acute stressors like pentylenetetrazole (PTZ) compromises galanin's sedative
36 NA interference (RNAi) was performed using a pentylenetetrazole (PTZ) exposure paradigm to induce con
37                          Bath application of pentylenetetrazole (PTZ) or glutamate reduced the stimul
38 s in vivo in 2 and 14-day old piglets during pentylenetetrazole (PTZ) seizures using 31P nuclear magn
39  which we exposed Xenopus laevis tadpoles to pentylenetetrazole (PTZ), a known convulsant.
40 pr suppressed generalized seizures evoked by pentylenetetrazole (PTZ), partial seizures evoked from t
41 4E-BP1, in mice increases the sensitivity to pentylenetetrazole (PTZ)- and kainic acid (KA)-induced s
42      We show that ABHD6 inhibition decreases pentylenetetrazole (PTZ)-induced generalized tonic-cloni
43 manifest increased aggressiveness and higher pentylenetetrazole (PTZ)-induced seizure propensity.
44 significantly more potent protection against pentylenetetrazole (PTZ)-induced seizures compared to VA
45 ed the effects of intestinal inflammation on pentylenetetrazole (PTZ)-induced seizures in mice and th
46 bility, we administered the GABAA antagonist pentylenetetrazole (PTZ; 0.3 or 1.0 mg/kg/day) to arrhyt
47  controls and their ictal activity following pentylenetetrazole (PTZ; 85 mg/kg i.p.) was compared to
48 al electroshock (MES) test, the subcutaneous pentylenetetrazole (scPTZ) test, and the six-hertz (6 Hz
49 imulating electrodes, and then injected with pentylenetetrazole to induce seizures.
50 vity induced by intraperitoneal injection of pentylenetetrazole was recorded from microwire electrode
51  oxidation in the cerebral cortex induced by pentylenetetrazole was significantly attenuated by DHA,