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1 amygdala codes neither intensity nor valence per se, but a combination that we suggest reflects the o
2 sphorylation were not a result of anesthesia per se, but a consequence of anesthesia-induced hypother
3 ia is therefore not an abreaction to sounds, per se, but a manifestation of activity in parts of the
4                   However, it is not poverty per se, but a multitude of risk factors associated with
5 trate that indomethacin did not inhibit GSIS per se, but activation of GPR40 in the presence of indom
6  long debated OPA1 variant is not pathogenic per se, but acts as a phenotypic modifier if it encounte
7 studies did not examine athletic performance per se, but all studies assessed one or more physical fi
8  by the allergen and its biological function per se but also by other small molecular compounds, such
9 not only by dysregulation of reward circuits per se but also by recruitment of brain and hormonal str
10 icine as cargo of drugs or as species active per se but also in a variety of other fields ranging fro
11 of AM are related not only to the AM process per se but also to external factors such as ecosystem pr
12 at GABA is unnecessary for synapse formation per se but also uncover a novel facet of GABA in regulat
13 se quality of life, not just due to the pain per se, but also because of the depression that can ofte
14  for sleep is not only driven by wakefulness per se, but also by specific neuronal-glial circuits tha
15  We not only consider the imaging techniques per se, but also how the construction of new fluorescent
16 ormational landscape of the membrane protein per se, but also increases the gelation of lipids, both
17    Treatment should address not only obesity per se, but also its effects on self-esteem in a hostile
18 is due not only to the growth arrest signals per se, but also to signals elicited by specific growth
19  conclusion, SAH did not affect adipogenesis per se but altered adipocyte functionality through epige
20  the localization of Sec31A to ER exit sites per se but appeared to acutely regulate the stability an
21  maintain the weak or strong [PSI+] variants per se, but appears to maintain them in an 'undifferenti
22 these alterations are not related to uremia, per se, but are dependent on the presence of excess para
23 t the result of failure in allelic exclusion per se, but arise through receptor editing.
24  construe social robots not as social agents per se, but as depictions of social agents.
25 the Cys residue at position 122 upon binding per se but at a subsequent step in the overall transport
26 d oxidized glutathione (GSSG), were inactive per se but attenuated the effect of both GSH and mercapt
27       NICD did not affect TGF-beta signaling per se but blocked induction of the Cdk inhibitor p15(IN
28            Again, troglitazone had no effect per se but blocked the dexamethasone-induced increase in
29 opioid receptors with U-50,488 had no effect per se, but blocked quinelorane-induced increases in med
30 ntazocine, a sigma ligand was without effect per se, but blocked the ability of rimcazole to increase
31 ion is determined not by the local curvature per se but by the packing of lipid headgroups (i.e., are
32 the stereotypical transcriptional trajectory per se but can stall, delay or accelerate progression al
33 ellum is not involved in language processing per se, but cognitive processing more generally.
34 that is not required for the fission process per se but contributes to its efficiency, likely by prom
35 ptor and that the D2 domain is not essential per se but contributes to receptor function by promoting
36  does not impair the activity of this enzyme per se but corrupts both the fidelity of the cofactor-lo
37 IN components is not essential for septation per se, but critical for septation to take place in a ti
38 did not impair anti-I-C or anti-I-F function per se but did exacerbate inhibition defects during comp
39 in the identity of the tumor progenitor cell per se but differences imposed on a common progenitor by
40  native association of hsp90 with p50(cdc37) per se, but does result in the formation of salt-labile
41  with fluids in microchannels is not useful, per se, but does suggest broader application for microfl
42 ion did not affect incidental taste learning per se, but effectively suppressed aversive taste memory
43  cells are not activated by allogeneic cells per se, but expand, both in vitro and in vivo, in the pr
44 is not the management of care at end of life per se, but facilitating the transition from active inte
45  BLA is not necessary for prosocial behavior per se, but fine-tunes the degree of prosociality as a f
46 y, ARF is not necessary for p53 accumulation per se but for altering the spectrum of genes activated
47 quired not for the death of bursCCAP neurons per se but for the removal of neural projections.
48 , but not exclusively from groove bound ions per se, but from a balance of competing factors in the e
49 ssant effects resulted not from hyperthermia per se, but from nonspecific aspects of the intervention
50 es is predicted to result not from diversity per se, but from the uniformly low levels of resources t
51 little effect on IR tyrosine phosphorylation per se but greatly reduced IR downstream signaling.
52  strand had no effect on transesterification per se but had synergistic effects when combined with 8-
53 did not impair spatial learning or retrieval per se, but impaired the ability to follow changing spat
54 PI3K signaling, not in lung tumor initiation per se, but in both the rate of tumor growth and the pro
55 rounded in acoustic attributes of the voices per se, but in how these features are perceptually inter
56 orylation at Y68 did not change its activity per se, but induced increased ubiquitination of cofilin
57  no apparent effect on blood vessel function per se, but induces increased apoptosis and decreased pr
58 ent on metastasis depends not on integration per se but instead on expression of the mVL30-1 RNA.
59 s are not likely to be designed for splicing per se, but instead have been adopted in yeast for that
60  stability of the newly synthesized histones per se, but instead histone binding by Asf1 promotes the
61 ctivity has little influence on excitability per se, but instead influences axonal conduction velocit
62  may not influence developmental progression per se, but instead serves to perpetuate those developin
63 nhance the representation of target features per se, but instead to optimize performance on the curre
64 ren may not view bragging as a male behavior per se, but instead, as associated with males in certain
65 89U mutation does not inhibit tmRNA activity per se but interferes with an upstream step intermediate
66      IalphaI is not produced by the ES cells per se but is added to the cells via the cell culture me
67 c domain is not required for kinase activity per se but is essential to couple B-Raf catalytic activi
68 is not required for Ca2+-dependent secretion per se but is essential to the mechanism by which cAMP p
69  not simply a direct result of transcription per se but is most likely the result of the concerted ac
70 or cAMP sensing, chemotaxis, and development per se but is part of a signal transduction cascade esse
71 ession is not essential for the switch event per se, but is essential for a subset of phenotypic char
72 hydrolysis is not required for translocation per se, but is essential for both active and unidirectio
73 infants' rule learning is not tied to speech per se, but is instead enhanced more broadly by communic
74  is not required for polarized hyphal growth per se, but is involved in septum formation.
75 n iPSC clones is not caused by reprogramming per se, but is rather a consequence of cloning individua
76  is not critical for regulating tumor growth per se, but is required for the early stages of cancer c
77  is not activated by the control of movement per se, but is strongly engaged during the acquisition a
78 iency is not a risk factor for WNV infection per se, but it is a risk factor for both early and late
79 expression may not be required for migration per se, but it may be play a role in either stabilizing
80 the high-affinity site does not activate TG2 per se, but it protects against oxidative enzyme deactiv
81                   Troglitazone had no effect per se, but it was able to normalize FFA suppressibility
82 ation is not essential for VSV RNA synthesis per se but likely plays an important role in vivo in fac
83  may not be required for telomerase activity per se but may be conserved as a regulatory site for the
84  seems not to be required for nuclear import per se but may instead regulate its efficiency and/or ki
85 atal DA is not associated with obesity class per se but may nevertheless confer an increased risk of
86 the outer membrane in the eps mutant strains per se but may provide osmoprotective functions.
87 ins are not necessary for leukocyte adhesion per se, but may facilitate downstream biologic events.
88 etic variants (which are not disease-causing per se, but may increase susceptibility to ventricular f
89  require the monkey to attend to orientation per se but merely to foveate the receptive field stimulu
90 nrelated to changes in cytosolic redox state per se, but more probably by increased production of sup
91                    However, the PS particles per se but not the residual styrene in both PS particle
92 ese results indicate that membrane targeting per se, but not necessarily myristoylation, is sufficien
93 Our results demonstrate that phosphorylation per se, but not the interaction with a specific GRK, is
94  as a consequence not of the virus infection per se but of the hyperactivated immune response.
95 s not dependent on the biotype of the strain per se but on the tcpPH promoter itself.
96 ow number of molecules expressed from a gene per se but originate in the random, rare events of gene
97  not just on associative learning and memory per se, but other more complex aspects of cognition such
98 ate that YidC is not necessary for insertion per se, but plays an important role in folding of LacY.
99  the maintenance of stimulus representations per se but points to deficits in more associative compon
100         TNFalpha did not induce constriction per se but potentiated the constriction with serotonin a
101 vation failed to affect AMPK phosphorylation per se but potentiated the stimulation by either muscari
102 or forskolin activation of adenylate cyclase per se but prevented D2 receptor-mediated sensitization
103 The D2/3 antagonist raclopride had no effect per se, but prevented the quinelorane-induced decrease i
104  NGF on memory is not perikaryal hypertrophy per se but rather an increased density of terminals, whi
105  not well reflected by cognitive performance per se but rather are much better indexed by the deviati
106  Atf1 is not required for activation of Atf1 per se but rather for modulating its stability.
107 s resulted not from pyruvate kinase activity per se but rather from the formation of an active tetram
108 ly not involved in the computation of thirst per se but rather is activated during changes in thirst/
109  is not because of deglycosylation of Asp-39 per se but rather is likely because of the change of thi
110 that loss of ODC does not affect cell growth per se but rather is required for survival of the plurip
111 show that it is not mitochondrial morphology per se but rather Mff and Drp1 that directly control MAR
112 ious studies, we focus not on lateralization per se but rather on patterns of left-hemisphere (LH) an
113 ependent on the removal of hydrogen peroxide per se but rather on preventing the generation of hydrox
114 hosphatidylinositol 3-kinase is not required per se but rather plays a role in signal amplification.
115 hed hDNA by responding to mismatches in hDNA per se but rather rejection of mismatched hDNA appears t
116 ement does not actually affect frameshifting per se but rather serves to limit the fraction of riboso
117 dimerization or viruslike filament formation per se but rather the ability of M to assemble in ordere
118 ery, suggesting that it is not the operation per se but rather the absence of physiological stress or
119 en mating and virulence is not due to mating per se but rather to certain mating-pathway components t
120 n stroke may not relate to semantic deficits per se, but rather an inability to use semantics to supp
121 itory may not necessarily represent the hand per se, but rather any other body part that shares the f
122 not necessary for stable binding of U2 snRNP per se, but rather are necessary for accessibility of co
123 umulation is not caused by diminution of ECS per se, but rather by increased net K+ efflux due in par
124 h direct potentiation of the afferent signal per se, but rather by reducing the intrinsic noise of th
125 esults not from the loss of SMARCB1 function per se, but rather from DCAF5-mediated degradation of SW
126 inant race stereotypes may not reflect race, per se, but rather inferences about how one's ecology in
127 inhibitors that do not block enzyme activity per se, but rather interfere with targeting of the enzym
128 uence of p16(INK4a) activation or senescence per se, but rather is a damage response that is separabl
129 duced longevity is not the result of obesity per se, but rather is influenced by other metabolic path
130  does not affect bacterial replication rates per se, but rather mediates intra-cytosolic survival and
131  of mTOR does not induce cellular senescence per se, but rather promotes the survival of senescent ce
132  be due to a defect in activating end growth per se, but rather reflects a failure of one of the cell
133 hat LIPC does not mediate semantic retrieval per se, but rather subserves the selection of task-relev
134 not related to internally directed attention per se, but rather to a higher level of self-generated t
135 as a means of generating the natural product per se, but rather to enable access to analogues contain
136  is not directly toxic to GSE jejunal mucosa per se, but rather toxicity requires the participation o
137 ntage is not associated with the polydactyly per se, but rather with the hemimelia which is a common
138 ot involve modifications of the 5-HT pathway per se, but rather, changes in the response of SNs to th
139 ations did not prevent transcript initiation per se but, rather, generated transcripts that were unst
140 B receptors do not regulate dendritic growth per se but, rather, the mode of such growth.
141 t a model in which sigma(E) is not essential per se but, rather, we propose that rpoE mutant cells di
142 he cross-sectional antiviral antibody levels per se but, rather, with the delayed kinetics of NAb pro
143 tion is not required for mesoderm production per se, but, rather unexpectedly, in the absence of Smad
144 rotubule reorganization are not pathological per se but reflect a return to an earlier developmental
145 gous promoter did not activate this promoter per se but rendered it responsive to PU.1.
146 vo does not cause hepatic injury or fibrosis per se but renders the livers more susceptible to the in
147 hat does not rely on "high affinity" binding per se, but requires a high-potency agonistic interactio
148 uired for the catalytic function of PKCalpha per se, but serves to control the duration of activation
149 nel block has no effect on PVEM inducibility per se, but shifts it to later time points by delaying r
150 is detected no increases in response to heat per se, but showed transient elevations in response to r
151 roduced no change in reactive oxygen species per se but significantly potentiated menadione-induced e
152 t that it is not the mere presence of Glu69, per se, but specific Glu69-containing alleles and their
153  that ATR pathway proteins are not antiviral per se but that activation of ATR signaling may have neg
154 el traits that do not alter the manipulation per se but that alleviate its detrimental fitness conseq
155 trol is not needed to be honest or dishonest per se but that it depends on an individual's moral defa
156 for transduction of the mating factor signal per se but that it is essential for proper mating factor
157 ction is required for curved cell morphology per se but that production of the altered O-polysacchari
158 thical values are brought to bear on science per se but that those values are highly questionable or
159 l to the network supporting motor adaptation per se but that, within this network, M1 may be importan
160 daptation is not the ionic-liquid conditions per se but the evolutionary barrier of crossing its phys
161 this article are not with information theory per se, but the assumptions surrounding it.
162 e nucleotide sequence or secondary structure per se, but the critical clustering of specific redundan
163 e purine base catabolism, or the lack of UOX per se, but the elevated uric acid concentration in the
164         Furthermore, it is not JEV infection per se, but the inflammatory cytokines/chemokines induce
165  SCA1 allele is not the result of cell death per se, but the result of cellular dysfunction and morph
166 fferences we found were caused by trade-offs per se, but they are consistent with predictions derived
167 This implies that T6P is not a growth signal per se, but through SnRK1, T6P primes gene expression fo
168  in CI studies do not correspond to contours per se but to filled-in surface regions contained by tho
169 se (ANR) activity in soil is not due to NH4+ per se but to glutamine formed by microbial assimilation
170 n of ANR activity by NH4+ is due not to NH4+ per se but to products formed by microbial assimilation
171 s not due to unreliability of the array data per se, but to the design of the experiments analyzed.
172 fficient export does not require methylation per se, but unmethylated arginine residues lead to reten
173 not regulated by the mobilization of the ICE per se, but was due directly to inactivation of the rela
174 d to variation in demographic susceptibility per se, but were possibly driven instead by finer-scale
175 FDG uptake was not associated with adiposity per se but with higher visceral fat mass.

 
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