ライフサイエンスコーパス: percussion

1 of these fragments also preserve evidence of percussion.

2 ury in hippocampal slices subjected to fluid percussion.

3 flakes by hitting stone on stone, free hand percussion.

4 g the evolutionary origins of human rhythmic percussion.

5 d rainbow trout killed by asphyxia in air or percussion.

6 d models induce head injury by lateral fluid percussion, a controlled cortical impact or impact accel

7 f traumatic brain injury, parasagittal fluid percussion, analysis of the metal load of metallothionei

8 Furthermore, the presence of bones with percussion and cut-marks near to several hammerstones su

9 o classify spectrogram images generated from percussion and palpation signals.

10 ected to diffuse brain injury (midline fluid percussion), and cognition, gliosis, and neuroinflammati

11 ts underwent normothermic parasagittal fluid-percussion brain injury (1.8-2.1 atmospheres) followed b

12 transplanted at 24 h following lateral fluid percussion brain injury (FP) in rats.

13 Fluid percussion brain injury (FPI) elevates the CSF concentra

14 sensitive K (K(ca)) channel following fluid percussion brain injury (FPI) in newborn pigs equipped w

15 sitive K(+) (K(ca)) channels following fluid percussion brain injury (FPI) in newborn pigs equipped w

16 After fluid percussion brain injury (FPI) in the newborn pig, pial a

17 were then subjected to moderate/severe fluid percussion brain injury (FPI).

18 lation during hypotension after piglet fluid percussion brain injury (FPI).

19 on of superoxide anion (O(-)(2)) after fluid percussion brain injury (FPI).

20 artery dilation was impaired following fluid percussion brain injury (FPI).

21 cats were subjected to moderate/severe fluid percussion brain injury after intrathecal administration

22 Rats (n=45) were subjected to lateral fluid percussion brain injury and euthanized at 3 h to 28 days

23 l artery vasodilation was unchanged by fluid percussion brain injury and phenylephrine.

24 ented DNA at 12 or 24 hr after lateral fluid percussion brain injury in anesthetized rats.

25 hypotensive pial artery dilation after fluid percussion brain injury in females, but paradoxically ca

26 reased markedly during hypotension and fluid percussion brain injury in males but less in females.

27 oxically caused vasoconstriction after fluid percussion brain injury in males.

28 hanges were investigated using lateral fluid percussion brain injury in mice.

29 and reduces cerebral blood flow after fluid percussion brain injury in piglets.

30 n kinase C were measured after lateral fluid percussion brain injury in rats.

31 e and motor deficits following lateral fluid percussion brain injury in rats.

32 etrograde memory dysfunction following fluid percussion brain injury in rats.

33 index were determined before and after fluid percussion brain injury in untreated, preinjury, and pos

34 severity was induced using the lateral fluid percussion brain injury model in anesthetized rats (n =

35 24 hours to 2 months following lateral fluid percussion brain injury of moderate severity (2.4-2.6 at

36 The effect of fluid percussion brain injury on hippocampal long-term potenti

37 lots were conducted in rats subject to fluid percussion brain injury or sham injury.

38 At 2 weeks after moderate fluid-percussion brain injury or sham surgery, adult male Spra

39 utoregulation during hypotension after fluid percussion brain injury through modulation of extracellu

40 utoregulation during hypotension after fluid percussion brain injury through modulation of extracellu

41 d elevated intracranial pressure after fluid percussion brain injury were greater in males, which wer

42 During hypotension and fluid percussion brain injury, pial artery dilation was impair

43 experiment 1, 15 min prior to central fluid percussion brain injury, rats (n=8 per group) were injec

44 sham surgery or moderate parasagittal fluid-percussion brain injury.

45 ein kinase upregulation in males after fluid percussion brain injury.

46 eased more in males than females after fluid percussion brain injury.

47 e blunted by phenylephrine pre- or postfluid percussion brain injury.

48 cerbates histopathologic outcome after fluid-percussion brain injury.

49 t, male rats were subjected to midline fluid percussion brain or sham injury and evaluated between 1d

50 differentiate wheel-throwing, coil-building, percussion-building, and coil-wheeling techniques.

51 ng to coil-wheeling, but also coil-building, percussion-building, percussion-wheeling, and drawing.

52 Microscopic examination of percussion damage suggests the bones were used as knappi

53 a closed cranial window were connected to a percussion device consisting of a saline-filled cylindri

54 A parasagittal fluid percussion (FP) brain injury (1.8-2.1 atm) was induced i

55 c protein were measured after lateral fluid percussion (FP) brain injury in rats.

56 free fatty acids (FFAs) after lateral fluid percussion (FP) brain injury in the rat.

57 ation following lateral (parasagittal) fluid-percussion (FP) brain injury in the rat.

58 free fatty acids (FFAs) after lateral fluid percussion (FP) brain injury in the rat.

59 s in the adult rat brain after lateral fluid-percussion (FP) brain injury was characterized using ter

60 (1.8-2.1 atm), or severe (2.2-2.7 atm) fluid percussion (FP) injury (or sham surgery) and processed f

61 Because the lateral fluid-percussion (FP) model of experimental brain injury produ

62 hort- and long-term effects of lateral fluid percussion head injury on the perisomatic inhibitory con

63 Mild to moderate (1.7-2.1 atm) lateral fluid percussion head injury or sham operation was produced in

64 lity in the dentate gyrus 1 week after fluid percussion head trauma.

65 brain injury (TBI) induced by lateral fluid percussion in adult rats, using 2 new ligands for PET: (

66 derate injury was induced with midline fluid percussion in some of the rats.

67 xpression.(1) Key features of human rhythmic percussion include individual and regional variation, as

68 All patients had percussion-induced muscle rippling and half had percussi

69 sorder characterized by electrically silent, percussion-induced muscular contractions.

70 sion- or stretch-induced muscle rippling and percussion-induced rapid muscle contraction with or with

71 he temporal profile of apoptosis after fluid percussion-induced traumatic brain injury (TBI) in rats

72 The late EPSCs in granule cells from fluid percussion-injured rats were not blocked by the NMDA rec

73 Animals were subjected to fluid percussion injury (2.2-2.4 atm; 1 atm = 101.3 kPa) and,

74 xperimental TBI was induced in vivo by fluid percussion injury (25 psi) in male and female wild-type

75 We utilized a model of central fluid percussion injury (CFPI) in adult male rats treated with

76 sham injury, lateral (LFPI) or central fluid percussion injury (CFPI) only, or to combined LFPI or CF

77 isoflurane, prepared for parasagittal fluid percussion injury (FPI) and randomly assigned to receive

78 Lateral fluid percussion injury (FPI) and sham-operated (Sham) rats we

79 22, a CSF1R antagonist, before midline fluid percussion injury (FPI) in male mice and cortical neurop

80 to dilator opioids were blunted after fluid percussion injury (FPI) in newborn pigs.

81 y taken up by the injured brain, after fluid percussion injury (FPI) in the rat.

82 Using a fluid percussion injury (FPI) model and patch-clamp recordings

83 ontribute to TBI, we studied a lateral fluid percussion injury (FPI) model in mice.

84 This study used the rodent fluid percussion injury (FPI) model, in combination with elect

85 estigated the early effects of in vivo fluid percussion injury (FPI) on hippocampal synaptic potentia

86 o weeks after they had received a mild fluid percussion injury (FPI) or sham surgery.

87 rats (n=19) were performed mild brain fluid percussion injury (FPI) prior to exercise.

88 behavioral function following lateral fluid percussion injury (FPI) to the brain in male rats.

89 (ECoG) of rats weeks and months after fluid percussion injury (FPI), a model of traumatic brain inju

90 se (GAD) positive neurons between sham fluid percussion injury (FPI), FPI with sham Vagus Nerve Simul

91 del of closed head injury, the lateral fluid percussion injury (FPI), in the rat.

92 of cognitive recovery after a lateral fluid percussion injury (FPI).

93 s appear following a single episode of fluid percussion injury (FPI).

94 Lateral fluid percussion injury (LFP), a model of mild-moderate concus

95 iffuse-focal model of TBI, the lateral fluid percussion injury (LFPI), on interneurons, 8 weeks post-

96 Male mice received a midline fluid percussion injury (mFPI) and 7 d later were subjected to

97 ogy over 28 days following rat midline fluid percussion injury (mFPI) as a first step in exploiting m

98 Rats were given a sham or mild fluid percussion injury (mFPI), and behavioral testing, MRI, a

99 ce received a sham (n = 52) or midline fluid percussion injury (TBI; n = 57).

100 (controlled cortical impact [CCI] and fluid percussion injury [FPI]).

101 nduced in rats by rostral parasagittal fluid percussion injury and epilepsy patients evaluated with i

102 We used fluid percussion injury and in situ hybridisation to evaluate

103 -H mice were subjected to mild central fluid percussion injury and killed at various times between 15

104 ats were subjected to moderate central fluid percussion injury and killed between 30 min and 7 d afte

105 we used rats that underwent a lateral fluid percussion injury at acute and chronic time-points to in

106 mild (33 degrees C) hypothermia after fluid-percussion injury combined with secondary hypoxia.

107 After lateral fluid percussion injury in adult rats, oral treatment with EVT

108 Rostral parasagittal fluid percussion injury in rats reliably induces a perilesiona

109 d previously that rostral parasagittal fluid percussion injury induces different types of chronic rec

110 rkable feature of rostral parasagittal fluid percussion injury is the occurrence, in the early months

111 eated mild TBI (rmTBI) using a lateral fluid percussion injury model.

112 oorly characterized by comparison with fluid percussion injury models.

113 cts of mild (1.4-1.5 atm) parasagittal fluid-percussion injury on the electrophysiology of this circu

114 The effect of moderate, central fluid percussion injury on the subcellular distribution of thi

115 The data support the conclusion that fluid percussion injury results in redistribution of the enzym

116 In a rat model of lateral fluid percussion injury, combined pioglitazone (PG) and hMSC (

117 t the assessment of the time course of fluid percussion injury-induced epileptogenesis is dramaticall

118 ransgenic rodent model of parasagittal fluid percussion injury.

119 lcineurin activity increased following fluid percussion injury.

120 travenously at 30 min prior to midline fluid percussion injury.

121 significant benefits for sound quality of a percussion instrument (xylophone).

122 Rhythmic percussion is present across human cultures and has been

123 Using direct percussion, language-competent bonobo-chimpanzees Kanzi

124 brains after sham-operation or lateral fluid percussion (LFP) injury were scanned ex vivo in a 9.4 T

125 opogenic modification of the crania (cut and percussion marks), indicates that the carcasses of the c

126 ude deeply impressed lines, cross-hatchings, percussion marks, and other geometric shapes on flat wal

127 facilitate conchoidal fracture in free hand percussion may still have been a critical watershed for

128 movement (electrically induced myotonia) or percussion (mechanically induced myotonia).

129 The fluid-percussion model (FP) is one of the oldest and most comm

130 eviously characterized for the lateral fluid-percussion model of brain injury.

131 s it shows that a prerequisite for free hand percussion (namely, free hand hitting) is part of the sp

132 Rats received either two mild fluid percussion or sham injuries administered five days apart

133 6 mice received diffuse TBI by midline fluid percussion or were sham-injured.

134 cussion-induced muscle rippling and half had percussion- or stretch-induced muscle mounding.

135 wavelike muscle contractions (rippling) and percussion- or stretch-induced muscle mounding.

136 based on the presence of electrically silent percussion- or stretch-induced muscle rippling and percu

137 tachment level, width of keratinized tissue, percussion sensitivity, pulp vitality tests, radiographi

138 Rats were injured using lateral fluid percussion TBI (2.8 +/- .10 atm).

139 cted to moderate (1.8-2.2 atmospheres) fluid-percussion TBI and naive controls.

140 he hippocampus at the onset of central fluid percussion TBI and that the enhanced phospholipase C-cat

141 rus at 2 and 15 days following lateral fluid percussion TBI in adult rats.

142 selective hippocampal cell death after fluid-percussion TBI in rats, consistent with the reported red

143 hippocampal injury after experimental fluid percussion TBI in rats.

144 intraperitoneally 30 min after lateral fluid percussion TBI in the rat.

145 l deficits, in a moderate parasagittal fluid percussion TBI model.

146 ubjected to injury (INJ) consisting of fluid percussion TBI of 3 atm with concurrent 30 ml/kg graded

147 ithin a broader pattern of Palaeolithic bone percussion technology in Africa, Eurasia and North Ameri

148 He had no percussion tenderness of his spine, and a neurologic exa

149 nd lactate were measured after central fluid percussion traumatic brain injury (TBI) in rats.

150 nd 24 h after mild, moderate or severe fluid percussion traumatic brain injury (TBI).

151 but also coil-building, percussion-building, percussion-wheeling, and drawing.

152 cused injury than is seen with lateral fluid percussion which may have implications for the behaviora