ライフサイエンスコーパス: perfusate

1 < 0.001), and E-selectin (P < 0.001) in the perfusate.

2 educed the ATP concentration detected in the perfusate.

3 ximately 20 mM when NAD+ was included in the perfusate.

4 ycle intermediates in tissue versus effluent perfusate.

5 f other transporters were independent of the perfusate.

6 A levels, NSD-1015 20 microM was included in perfusate.

7 rin, and concentration of fibronectin in the perfusate.

8 cence over time after dyes were removed from perfusate.

9 (2.8 +/- 1.0 microM) were identified in the perfusate.

10 1 to 5 micromol/L) was added to the coronary perfusate.

11 essed when SR11302 (100 nM) was added in the perfusate.

12 moval of HCO3-/CO2 from the pulmonary artery perfusate.

13 lated intact cardiomyocytes through the cell perfusate.

14 rate of the respective isotope added to the perfusate.

15 nium, an inhibitor of SACs, was added to the perfusate.

16 motic solutions or a reduced level of normal perfusate.

17 g/dL bovine serum albumin or a protein-free perfusate.

18 han twice as fast if HDL was included in the perfusate.

19 by lowering the calcium concentration in the perfusate.

20 1-hour period of NMP using a red cell-based perfusate.

21 successfully detected and quantified in the perfusate.

22 (flavin mononucleotide, FMN) in the machine perfusate.

23 with reference to biochemical changes in the perfusate.

24 ion was performed using an erythrocyte-based perfusate.

25 challenges with or without treatments to the perfusate.

26 perfusion with a plasma-free red cell-based perfusate.

27 1 mg/mL) or MnTBAP (0.3 mg/mL), added to the perfusate.

28 total of 51 analytes, 34 were measurable in perfusates.

29 after transplantation and the NMR data from perfusates.

30 opionic acid from [5,6,7-(13)C(3)]heptanoate perfusates.

31 were increased in the nonstreptokinase group perfusates.

32 were 10-fold higher than that in the kidney perfusates.

33 ration of BK stabilised at 378 +/- 48 pg (ml perfusate)(-1), that of trypsin-activated BK precursor w

34 e groups (n = 4 each) were differentiated by perfusate: (1) isolated red blood cells (RBCs) (current

35 Addition of enalaprilat to the perfusate (10 mM) prevented the conversion of exogenousl

36 ction in lactate dehydrogenase levels in the perfusate (333+/-22 vs.103+/-8 U/L) and an increase in b

37 With [U-13C16] palmitate in the perfusate, agmatine significantly increased the output o

38 minutes of ex situ perfusion, at which point perfusate alanine transaminase level was 1152 IU/L and u

39 The use of an autologous whole donor blood perfusate allowed 24H of preservation without functional

40 In whole liver, H/R significantly increased perfusate ALT.

41 Perfusate analysis demonstrated reduced fibrin generatio

42 to determine the predictive value of machine perfusate analysis on graft outcome.

43 ficial cerebrospinal fluid and the collected perfusate analyzed for ATP and SP content using the fire

44 s in the interstitial fluid diffuse into the perfusate and are collected.

45 and PCO2 electrodes simultaneously measured perfusate and effluent pH and PCO2.

46 Net flux should equal zero when perfusate and interstitial concentrations are equal.

47 ed increases in proinflammatory cytokines in perfusate and lung lavage fluid, compared to control.

48 rfused rat kidney model using a protein-free perfusate and perfusates containing bovine serum albumin

49 Perfusate and urine samples were obtained at baseline an

50 resence of extracellular histones in machine perfusates and deceased donor kidney viability.

51 tion strategies, and the impact of different perfusates and leukocyte filters.

52 increased ATP concentrations in the bladder perfusate, and also increased voiding frequency; these e

53 ection limits for DOX extracted from buffer, perfusate, and lung tissue were 40, 100, and 3700 mug L(

54 tors VII and X were evaluated in pre-bypass, perfusate, and pericardial wound blood before and during

55 Protein binding was measured in each of the perfusates, and the venous outflow was collected to dete

56 nts of acetic acid and propionic acid in the perfusate are comparable to the labeling pattern of acet

57 irst kidney was perfused with red cell-based perfusate at 37 degrees C for 75 min, mean renal blood f

58 ents by removal of bicarbonate ions from the perfusate at this point, which resulted in further swell

59 Furthermore, we measured perfusate ATP concentration with a luciferin-luciferase

60 Urocortin 10(-8)M was added to the perfusate before I, before I and during R, and during R

61 ion of a specific caspase-9 inhibitor to the perfusate before ischemia prevented endothelial apoptosi

62 When added to the perfusate, benzamil almost completely inhibited the elev

63 n and conjugates were measured in intestinal perfusates, bile, plasma, and urine.

64 These perfusate biomarkers can be potentially used for more pr

65 We analyzed the diagnostic accuracy of the perfusate biomarkers glutathione S-transferase, lactate

66 The diagnostic accuracy of the perfusate biomarkers glutathione S-transferase, LDH, hea

67 viable; however, current tools and urine and perfusate biomarkers to identify these viable kidneys ar

68 Although TTX (1 microm) or a calcium-free perfusate both caused reductions in the power amplitude

69 PPi levels in the perfusates both in the liver and kidney of Abcc6(-/-) ra

70 to allow the use of biologically appropriate perfusate buffers containing high salt content.

71 eticulum Ca(2+)-ATPase inhibition or reduced perfusate [Ca(2+)], which indicates a Ca(2+)-mediated me

72 ificantly higher systolic performance at low perfusate [Ca2+] compared with R278C-Tg hearts, which we

73 The NMR spectroscopy of the perfusate can identify differences in the metabolomic pr

74 lin-dibutyryl cAMP (db-cAMP) cocktail to the perfusate caused no significant change in Jv in either w

75 Thus, perfusate cell profiles could serve as potential biomark

76 nd hypertonic shock, and rapid withdrawal of perfusate chloride.

77 he maximum prednisolone concentration in the perfusate (Cmax) by 3.0 and 2.2 fold, respectively.

78 Impermeant CA inhibitors abolished net perfusate CO2 loss and increased net HCO3- gain, whereas

79 quilibrated under flow for 30 min, using the perfusate collected during the final 10 min of the equil

80 jected to push-pull perfusion of the MPA and perfusates collected at 30 min intervals were analyzed f

81 Perfusates collected from a separate group of rats were

82 50% (NH4)2SO4 saturation of perfusates collected from Langendorff rat heart preparat

83 s of the putative algogen endothelin (ET) in perfusates collected from the tumor sites of hyperalgesi

84 a) perfusion increased net CO2 loss from the perfusate compared with controls (pH 6.4 saline, PCO2 ap

85 Perfusate comparison analysis of fresh livers and cold i

86 ls considering duration of NMP and different perfusate compositions in experimental and clinical mode

87 The minimum perfusate concentration of NMDA needed to elicit feeding

88 d not be discarded because of high biomarker perfusate concentration.

89 yclohexyladenosine (10 microM) in the tubule perfusate confirmed the ability of the afferent arteriol

90 The perfusate consisted of either (1) plasma alone, (2) rt-P

91 ed decorticate rat preparation, hyperosmotic perfusate consisted of either 135 mm NaCl, or a non-NaCl

92 minutes with unmodified perfusate (control), perfusate containing 20 nM dopamine, dopamine+2,3-butane

93 ased when 1 micro mol/l GLY was added to the perfusate containing 5 mmol/l glucose.

94 Perfusate containing either 4 microM oxyHb or SNO-Hb inc

95 Addition of L-glutamate or sucralose to a perfusate containing low glucose (20 mM) each activated

96 Electroosmosis was used to pull perfusate containing secreted insulin into 4-cm-long rea

97 Excess glutathione (GSH) added to perfusate containing SNO-Hb resulted in a 20 to 40% fall

98 Then 7.5 microM VIP was added to the perfusate containing VIP(10-28) at the three concentrati

99 al function during ischemia-reperfusion with perfusates containing 145 or 155 mM Na(+) in Langendorff

100 ney model using a protein-free perfusate and perfusates containing bovine serum albumin.

101 e reperfused for 120 minutes with unmodified perfusate (control), perfusate containing 20 nM dopamine

102 om a monocentric series of consecutive liver perfusates could help identify risk factors portending a

103 Liver perfusate cytofluorimetric phenotyping may contribute as

104 sulated clodronate, significantly attenuated perfusate cytokine levels, especially tumor necrosis fac

105 In contrast, atelectasis had no effect on perfusate cytokines compared to control but did induce s

106 Liver graft perfusate-derived KCs and in vitro-generated monocyte-de

107 r zinc (Zn2+ as 7 micromol l-1 ZnSO4) to the perfusate did not affect reabsorption of water, Na+ or K

108 Removing magnesium from the perfusate did not affect spontaneous subthreshold oscill

109 Interestingly, IL-6 levels in the perfusate did not increase after the lungs were removed

110 al of NO from Hb via transnitrosation to the perfusate did not reduce augmentation of HPV by SNO-Hb o

111 t rate (HR), coronary perfusion pressure and perfusate distribution to the myocardium.

112 After 24 hr of preservation, perfusate distribution was assessed, and oxygen consumpt

113 ol or isoprenaline had no effect on coronary perfusate distribution.

114 Effects of CsA addition to the perfusate during reperfusion were time dependent, with b

115 (NMR) to predict graft outcome by analyzing perfusates during machine perfusion time.

116 ed with significant metabolic differences in perfusate (eg, lactate, succinate, flavin mononucleotide

117 six percent of the dose was recovered in the perfusate either as unchanged (-)-epicatechin (22 mg) or

118 perfusion characteristics (flow, resistance, perfusate electrolytes, and pH) were serially measured.

119 metabolic tracer universally labeled glucose.Perfusate, end-point renal cortex, and medulla samples u

120 4377 ng/mL, CK: 1442 U/L), peaking 6 h after perfusate exchange (medians: myoglobin: 9206 ng/mL, CK:

121 milk-like secretion, which was dependent on perfusate flow and contained a concentration of BK which

122 PP but not NPP significantly improved renal perfusate flow and urine production and significantly in

123 cutoff of 30 kDa in a concentric probe and a perfusate flow rate of 2.0 muL min(-1), microdialysis re

124 After 10 mins, the perfusate flow was resumed at 20% of baseline flow and m

125 e perfusion pressure was linearly related to perfusate flux between 60 and 210 ml min(-1) and the flo

126 s factor-alpha (300 mug) was injected in the perfusate, followed 5 minutes later by melphalan at 1.5

127 icropipette electrodes and the collection of perfusate for analysis are other possibilities.

128 30 hour period with hourly sampling from the perfusate for measurement of general metabolism and clin

129 e able to (1) perfuse tissue and collect the perfusate for quantitative analysis of the solutes intro

130 High K+ (80 mM) was included in perfusate for two 30 min periods, 2.5 h apart.

131 s in smooth muscle cells downstream from the perfusate from an endothelium-intact arteriole was exami

132 py to examine the metabolomic profile of HMP perfusate from human cadaveric kidneys awaiting transpla

133 ng electrophoresis-based immunoassays of the perfusate from islets.

134 neutrophil recruitment when stimulated with perfusate from MAPC-treated kidneys (P < .001).

135 so decreased basal ATP concentrations in the perfusate from non-distended bladders and inhibited incr

136 acid, acetic acid, and propionic acid in the perfusate from rat liver exposed to Krebs-Ringer bicarbo

137 Perfusates from HMP kidneys were sampled at 45 min and 4

138 ver-specific immune cells were released into perfusates from Lewis rat livers as a result of cold isc

139 dynamic variables of circulation, as well as perfusate gases and electrolytes (pH, pCO2, pO2, O2 satu

140 Furthermore, we demonstrated that liver perfusates, generated by isolated liver perfusion system

141 se less than 16 mmol/L (P = 0.013), and bile/perfusate glucose ratio less than 0.67 (P = 0.013).

142 As for cholangiocyte function, bile/perfusate glucose ratio was significantly lower (<0.6) i

143 r between or through endothelial cells where perfusate had direct access to the basement membrane.

144 Restoring bicarbonate to the perfusate halted this swelling, and the corneas then thi

145 Since using blood as a clinical perfusate has limitations, including matching availabili

146 rospinal fluid was used as the microdialysis perfusate, Hcrt-1 no longer produced an increase in glut

147 Our data suggest no superiority of novel perfusates HTK-N and TiProtec in terms of tissue preserv

148 inistration of acriflavine directly into the perfusate immediately before IPC.

149 Urea was added to the perfusate in concentrations of 0.83, 2.5, 5.0, and 13.33

150 effects of hypothermic MP with an acellular perfusate in human upper extremities and compare with th

151 soluble complement receptor 1 (sCR1) to the perfusate in one further group (hDAF(+/-)/AbAbs/sCR1).

152 or Bis-Mal-PEGHb (100 micromol/L) to buffer perfusate increased normoxic PAP and augmented HPV in si

153 ow-inflow) and the urea concentration in the perfusate (inflow).

154 showed decreasing lactate production in the perfusate (initial: 0.031 +/- 0.004 vs final: 0.007 +/-

155 Without fatty acids in the perfusate, insulin output in the Lard group (135 +/- 22

156 e (the sink, outer pipet) a buffer solution (perfusate) into each of the two pools.

157 During hypoxia the flow of blood (or perfusate) is maintained and, while there is a substanti

158 synaptic blockade, to identify EBs active at perfusate K(+) concentrations ([K(+)](o)) of 3, 6, and 9

159 ect out contribution of fatty acid uptake, a perfusate-lacking fatty acids was used.

160 ity was assessed, using predefined criteria: perfusate lactate <1.7 mmol/L, pH 7.35 to 7.45, bile pro

161 ygen efficiency (P = 0.01), with lower blood perfusate lactate (P = 0.007).

162 The vascular flow parameters and the perfusate lactate clearance were similar in both groups.

163 Perfusate lactate concentration also decreased (0 hour,

164 Perfusate lactate concentration decreased from baseline

165 Perfusate lactate level fell from 7.2 to 0.3 mmol/L with

166 The evaluation protocol consisted of perfusate lactate, bile production, vascular flows, and

167 livers displaying good function during NMP, perfusate levels of ALT and D-dimers were low (</=3500 n

168 and significantly increased the reduction of perfusate levels of creatinine and urea during reperfusi

169 Perfusate levels of fatty acid binding protein, a marker

170 de synthase, along with significantly higher perfusate levels of the endogenous vasodilator nitric ox

171 air control, exposure to 70% N(2)O increased perfusate levels of the NO metabolites nitrite and nitra

172 ven when DMI (20 microM) was administered in perfusate, LNAA- still lowered DMI-induced DA and NE lev

173 ine in an organ bath, allowing access to the perfusate (luminal) and the bath (serosal) solutions.

174 ter-investigator variability using different perfusates makes comparisons difficult.

175 as present in human aqueous humor and in the perfusate medium of perfusion-cultured human eyes.

176 fusate PO2, compared with low perfusate PO2, perfusate metabolic profile analysis suggests that aerob

177 sothiol], in association with an increase in perfusate [metHb].

178 When FFAs in the perfusate mimicked the quantity and composition of plasm

179 fusion (MPanox) or active oxygenation of the perfusate (MPox).

180 However, perindoprilat (10 mM in the perfusate, n = 7) significantly decreased RIF AngII conc

181 Interstitial infusion of AngI (100 nM in the perfusate, n = 7) significantly increased the RIF AngII

182 h the microdialysis probe (1 or 10 mM in the perfusate; n = 5 and 8, respectively) significantly incr

183 Bile/perfusate Na+ ratio was significantly higher (>=1.2) aft

184 A significant (P < 0.05) difference in the perfusate nitrate concentration was observed in each loc

185 The liver perfusate NK cell subset was strongly associated with bi

186 Perfusate of 26 transplanted cadaveric kidneys was analy

187 Addition to the perfusate of a medium-chain fatty acid (caprylic acid) t

188 ) are often used as an oxygen carrier in the perfusate of ex situ NMP.

189 n to increase glutamate concentration in the perfusate of hippocampal slices and in purified rat hipp

190 rotein was markedly increased in serum or in perfusate of isolated heart following ischemia/reperfusi

191 ong decrease of APAP-GLUC secretion into the perfusate of Mrp3-/- livers.

192 orbic acid (0.2 mM) was administrated in the perfusate of the ascorbic acid + electrical shock and as

193 Machine perfusates of 390 donations after circulatory death kidn

194 concentrations were significantly higher in perfusates of kidneys with posttransplant graft dysfunct

195 ment of mitochondrial FMN release in machine perfusates of livers disclosed a strong correlation with

196 tly reduced, but the PPi levels in the liver perfusates of wild-type rats were 10-fold higher than th

197 omized to receive 3.0 mM supplemental GSH to perfusate or no supplementation (control) and were prese

198 Stimulus was then added to either the perfusate or the bath and the perfusate was collected fo

199 erfusate), to inhibit (hyperoxic, hypocapnic perfusate) or to stimulate (hypoxic, normocapnic perfusa

200 The first 6 livers were perfused at high perfusate oxygen tensions, and the subsequent 6 at near-

201 The effect of HOPE was dependent on perfusate oxygenation in the cold.

202 At 50% perfusate oxygenation, APD and LVDP were significantly h

203 s assessing correlations of easily available perfusate parameters (PP) (aspartate aminotransferase, a

204 The optimal perfusate partial pressure of oxygen (PO2) during hypoth

205 n metabolism at hypothermia to the design of perfusates, perfusion machine technology, and drug thera

206 o influence MP characteristics when added to perfusate: PGE1, trifluoperazine, verapamil, and papaver

207 Increasing perfusate pH buffers decreased surface pH toward perfusa

208 en consumption and significantly neutralized perfusate pH.

209 activated in wound, but not in pre-bypass or perfusate plasma (monocyte chemotactic protein-1 = 29.5

210 nfusion time: 80.8+/-18.2 min; mean coronary perfusate pO(2): 631+/-235 mm Hg).

211 mized to receive 22-hour HMP with either low perfusate PO2 (30% oxygen, low oxygenated HMP [HMPO2]) (

212 low oxygenated HMP [HMPO2]) (n = 8) or high perfusate PO2 (90% oxygen, HMPO2high) (n = 8), before au

213 PO2 resulted in a 3- and 10-fold increase in perfusate PO2 compared with low HMPO2 and standard HMP,

214 bic mechanism is better supported under high perfusate PO2 conditions.

215 al, and flow dynamic effects of low and high perfusate PO2 during continuous HMP in a pig kidney isch

216 lungs with anoxic gas for 5 minutes reduced perfusate PO2 to 11+/-1.0 Torr.

217 advantages for early graft function for high perfusate PO2, compared with low perfusate PO2, perfusat

218 on for high perfusate PO2, compared with low perfusate PO2, perfusate metabolic profile analysis sugg

219 on enhanced the outflow facility and lowered perfusate pressure in biomimetic TM scaffolds populated

220 fter 4 h of perfusion was independent of the perfusate, probably because normal kidneys can maintain

221 th tBHQ (10 micro M), both outflow rates and perfusate proMMP-3 level increased significantly within

222 Perfusate protein expression during EVLP can differentia

223 er of mitochondrial complex I injury, in the perfusate provides a fast prediction of liver graft func

224 The protein binding of MAG3 in the albumin perfusates ranged from 87% to 95%, significantly higher

225 An appreciable increase in perfusate recovery due to a shift in the directionality

226 ory clearance, so an appreciable increase in perfusate recovery of these metabolites was not observed

227 /-); La3+ (1 micromol/L) addition to wt lung perfusate reduced the agonist effect to that observed in

228 ing superoxide dismutase and catalase in the perfusate reduced the ESR signals.

229 ption of pro-inflammatory mediators from the perfusate represents a potential intervention which may

230 ion of dmLSB (10 micromol/L) to the coronary perfusate restored the epicardial AP dome, reduced EDR a

231 NO-cyanometHb) on HPV, expired NO (eNO), and perfusate S-nitrosothiol (SNO) concentration in isolated

232 esulted in an approximately 50% reduction in perfusate [S-nitrosothiol], in association with an incre

233 ered randomly, all as a 1 mm bolus, into the perfusate: saline vehicle (control); sodium hydrogen sul

234 r the collection of a pretreatment blood and perfusate sample, rats were injected (i.p.) with the veh

235 c bead array assay, we evaluated analytes in perfusate samples collected at 1 hour and 4 hours of EVL

236 Perfusate samples from the MPA were collected at the rat

237 The perfusate samples were analyzed for NE and dopamine (DA)

238 Perfusate samples were collected every 30 min and analyz

239 In the period October 2018-February 2020, perfusate samples were obtained every 30 minutes during

240 an NMDA receptor antagonist was added to the perfusate shortly before and during LTP induction.

241 g SNO-Hb resulted in a 20 to 40% fall in the perfusate SNO concentration, with a concomitant increase

242 es revealed that relatively small changes in perfusate sodium ([Na(+)](o)) composition significantly

243 24 hours (n = 8) and exhibited physiological perfusate sodium levels (140.7 +/- 1.2 mmol/L), while ki

244 f 7.7 +/- 1.5 hours and significantly higher perfusate sodium levels (159.6 +/- 4.63 mmol/:, P < .01)

245 in UW or machine perfused (MP) in UW-machine perfusate solution (MPS).

246 herefore used to examine DOX extraction from perfusate solution and a homogenized lamb lung tissue.

247 abbit TNF-alpha antibody to cardioplegia and perfusate solutions restored postischemic function.

248 red from donor urine at procurement and from perfusate soon after pump perfusion) were not different

249 PO2, PCO2 and pH in the venous effluent perfusate stabilised at 157 +/- 10 mmHg, 50.1 +/- 2.4 mm

250 responds to changes in the properties of the perfusate, such as the ionic strength ( ), pH, and catio

251 dition of copper or manganese to the luminal perfusate suggesting that these ions may compete with ir

252 etabolic perturbations, including changes in perfusate temperature, electromechanical uncoupling, and

253 n-coagulative, echogenic, and cytoprotective perfusate that promotes recovery from anoxia, reduces re

254 usate) or to stimulate (hypoxic, normocapnic perfusate) the CB chemoreflex, while the systemic circul

255 In the 2.5 g/dL albumin perfusate, the EF of MAG3 was 44%, significantly less th

256 However, in the protein-free perfusate, the EF of MAG3 was 64%, equal to or higher th

257 F of the three EC complexes; in the 7.5 g/dL perfusate, the MAG3 EF fell to 18% versus 39%-45% for th

258 Recirculation of the perfusate through the support rat diminishes the need to

259 y lipoprotein significantly increased in the perfusate throughout the perfusion in both perfusion gro

260 l of both dissolved gas and pH levels in the perfusate thus demonstrating applicability for a wide ra

261 ls, lowering the temperature of the coronary perfusate to induce mild hypothermia (32 degrees C-34 de

262 parative responses to cellular and acellular perfusates to identify these benefits.

263 normal CB blood gases (normoxic, normocapnic perfusate), to inhibit (hyperoxic, hypocapnic perfusate)

264 significant increases in alveolar lavage and perfusate tumor necrosis factor-alpha, inflammatory cell

265 In both systems, inflammatory cytokines on perfusate (tumor necrosis factor-alpha, interleukin [IL]

266 1)) or two (U(m+2)) (15)N was generated from perfusate [U-(15)N(4)]arginine.

267 d aspartate, respectively, were derived from perfusate [U-(15)N(4)]arginine.

268 Serial samples of perfusate, urine, and tissue biopsies were taken for com

269 ring release of acetylcholinesterase in cell perfusates using the Ellman reagent.

270 usate pH buffers decreased surface pH toward perfusate values.

271 Using fluorescence recovery, we determined perfusate velocity to calculate diameter changes under d

272 n was found to facilitate the maintenance of perfusate volume and homeostasis.

273 rfused using urine recirculation to maintain perfusate volume without fluid replenishment.

274 to either the perfusate or the bath and the perfusate was collected for another 30 min to measure th

275 The perfusate was continuously sampled by electroosmotic flo

276 Hearts were paced to increase workload and perfusate was deoxygenated to study the effects of myoca

277 )I-labelled albumin efflux into the vascular perfusate was determined.

278 NO(x) (NO(-)(2) + NO(-)(2) production in the perfusate was measured by chemiluminescence.

279 SAAP using an oxygen-carrying perfusate was more effective in this study than non-oxyg

280 Perfusate was plasma-based with a hemoglobin concentrati

281 The complement activity of the perfusate was reduced by CAB-2, as was the generation of

282 With the use of overlapping injections, the perfusate was sampled every approximately 10 s.

283 The WB perfusate was superior (vs RBC) for maintaining stabilit

284 This effect was lost when deoxygenated perfusate was used.

285 Mucin in perfusates was quantified by periodic acid-Schiff's base

286 nality of metabolite excretion, from bile to perfusate, was noted in knockout mice only for conjugate

287 H+ and CO2 loss from the perfusate were accompanied by increases of PV H+ and tra

288 Enzyme biomarkers present in the kidney perfusate were all significantly reduced by the use of s

289 in (IL)-6 levels in the lung grafts and EVLP perfusate were also significantly lower after EVLP with

290 Drug levels in the perfusate were analysed using ultra-high-performance liq

291 with isotonic solution and hourly samples of perfusate were collected and analyzed.

292 Ten-minute fractions of aCSF perfusate were collected from separate groups of room ai

293 Fractions of choroidal perfusate were collected, and fluorescence was measured

294 zure-like events (SLEs) in low-Mg2+/ high-K+ perfusate were measured in the CA3 region of the intact

295 and outflow facility and cAMP levels in the perfusate were measured simultaneously.

296 r pressure (IOP), and proMMP-3 levels in the perfusate were monitored.

297 Liver perfusates were collected during the back-table surgical

298 or by isotonic replacement of sodium in the perfusate with choline or tetramethylammonium greatly at

299 Pike Laboratories Inc, Eagle, PA) is a novel perfusate with enhanced vasodilatory and antioxidant cap

300 rther reduced on combination of calcium-free perfusate with octanol (1 mM) and was abolished using a