ライフサイエンスコーパス: perfusion index

1 and thenar) and finger photoplethysmographic perfusion index.

2 all measurement sites (except cerebral) and perfusion index.

3 plasma lactate levels and inversely with the perfusion index.

4 tically generated and were used to calculate perfusion indexes.

5 an the least significant change observed for perfusion index (1.62% 0.80%).

6 s 166.83 +/- 204.44; P = .002) rate, wash-in perfusion index (-42.29 +/- 59.21 vs 50.96 +/- 71.13; P

7 Perfusion indexes accurately depicted stress-induced hyp

8 ng previous results, the relative changes in perfusion index also reliably detected a positive passiv

9 The derived perfusion indexes also correlated highly with (r up to 0

10 asis of a newly proposed Anatomic Myocardial Perfusion index, AMP) were each determined.

11 Relative changes in perfusion index and cardiac index observed during all in

12 ies, there was linear correlation between MR perfusion indexes and the microsphere flow measurements

13 esence of pain, tissue oxygen saturation and perfusion index are further reduced by hypovolemia (lowe

14 Tissue oxygen saturation and peripheral perfusion index are proposed as early indirect markers o

15 when evaluating tissue oxygen saturation and perfusion index as markers of hypovolemia in trauma pati

16 atory occlusion-perfusion index at baseline]/perfusion index at baseline x 100) detected a positive p

17 fusion index during end-expiratory occlusion-perfusion index at baseline]/perfusion index at baseline

18 STAI score, vital signs (oxygen saturation, perfusion index, blood pressure, heart rate, and tempera

19 sive leg raising increased cardiac index and perfusion index by 17% 7% and 49% 23%, respectively, In

20 ratory occlusion increased cardiac index and perfusion index by 6% 2% and 11% 8%, respectively.

21 perfusion was monitored with the peripheral perfusion index, capillary refill time, tissue oxygen sa

22 eover, the perfusion area deficit and spinal perfusion index correlated with the injury severity at i

23 Perfusion index could be used as a reliable surrogate of

24 end-expiratory occlusion-induced changes in perfusion index could detect a positive passive leg rais

25 The spinal perfusion index decreased with increasing injury severit

26 ts with a negative passive leg raising test, perfusion index did not significantly change during pass

27 We assessed whether the doppler perfusion index (DPI; ratio of hepatic arterial to total

28 fusion index greater than or equal to 2.5% ([perfusion index during end-expiratory occlusion-perfusio

29 ssue oxygenation (all measurement sites) and perfusion index during hypovolemia and pain than during

30 ral capillary refill time and the peripheral perfusion index, followed by a significant increase at T

31 sion of alveolar mucosa, called the gingival perfusion index (GIPI).

32 atory occlusion-induced relative increase in perfusion index greater than or equal to 2.5% ([perfusio

33 The perfusion index is the ratio between the pulsatile and t

34 entify using existing modalities such as the perfusion index measured using a pulse oximeter.

35 peripheral perfusion, as reflected by a low perfusion index or a high core-peripheral temperature gr

36 significantly reduced peak enhancement (PE), perfusion index (PI), and area under the curve (AUC) of

37 ficantly improved (p = 0.002); however, mean perfusion index, postductal saturation, and mean renal t

38 We measured perfusion index (Radical-7 device; Masimo Corp., Irvine,

39 Resting RV:LV perfusion index ranged from 0.32 to 0.34 in each group,

40 nductive plethysmography, oxygen saturation, perfusion index, regional cerebral and perirenal tissue

41 nal explanation for elevation in the Doppler perfusion index that occurs prior to tumor formation.

42 The spinal perfusion index was calculated by normalizing the contra

43 n survivors than in nonsurvivors; peripheral perfusion index was similar in the two groups.

44 Both the perfusion area deficit and spinal perfusion index were different between motor-complete an

45 Capillary refill time and peripheral perfusion index were measured at the same time points on

46 ssue oxygen saturation (except cerebral) and perfusion index were reduced by pain during normovolemia

47 genation in all measurement sites and finger perfusion index were reduced during hypovolemia/sham com

48 capillary segment density and the capillary perfusion index, which was prevented by both starches.

49 d heart rate, blood pressure, and peripheral perfusion index without affecting the corrected QT inter