ライフサイエンスコーパス: periapical

1 ance in the microbiological findings between periapical abscess and the maxillary sinus flora was fou

2 ial clinical diagnosis and the presence of a periapical abscess at surgery and at pathologic examinat

3 Clinical examination ruled out periapical abscess, periodontal abscess, and lateral per

4 patient underwent incision and drainage of a periapical abscess.

5 firm the importance of anaerobic bacteria in periapical abscesses and demonstrate their predominance

6 nificant association between the presence of periapical abscesses and oral viridans streptococci DNA-

7 Four patients had periapical abscesses at pathologic analysis, and the fif

8 Aspirate of pus from 5 periapical abscesses of the upper jaw and their correspo

9 ery, one of five calibrated examiners viewed periapical and bitewing radiographs of the surgical site

10 Periapical and panogram radiographs were taken.

11 raditionally performed using two-dimensional periapical and panoramic radiographs.

12 The regeneration of periodontal, periapical, and pulpal tissues is a complex process requ

13 es successful, stimulating bleeding from the periapical area of the tooth can be challenging and in t

14 es in modulating the microenvironment of the periapical area.

15 designed to enhance detection of crestal or periapical bone density changes and to help evaluate car

16 cient C3H/HeJ mice had significantly reduced periapical bone destruction compared to wild-type C3H/He

17 Periapical bone destruction occurs as a consequence of p

18 In the present study, we compared periapical bone destruction, sepsis, and inflammatory cy

19 or laborious histologic analyses to quantify periapical bone destruction.

20 the rapid and non-invasive quantification of periapical bone destruction.

21 results in the development of gingivitis and periapical bone loss, which apparently are associated wi

22 RC7 gene expression to simultaneously target periapical bone resorption and periapical inflammation.

23 Periapical bone resorption occurs following infection of

24 ted mice (40%; P < 0.05) exhibited increased periapical bone resorption, compared to wild-type contro

25 dental pulp result in tissue destruction and periapical bone resorption.

26 infected wild-type mice, which also had more periapical bone resorption.

27 ith inflammatory periapical lesions and with periapical cyst formation represents an interesting but

28 vestigate and compare the potential of human periapical-cysts mesenchymal stem cells (hPCy-MSCs) and

29 or saline, after which we induced ONJ using periapical disease and tooth extraction.

30 fore, the development of a new class of anti-periapical disease therapies is necessary and critical f

31 Periapical disease, an inflammatory disease mainly cause

32 ty to treat this disease in a mouse model of periapical disease.

33 ar nerve and mental foramen on panoramic and periapical films prior to implant placement; use of CT s

34 package to detect and number teeth in dental periapical films.

35 ts of canal anastomosis on the generation of periapical fluid pressure at different fluid flow rates

36 ession of DNA methylation machinery genes in periapical granuloma and to assess longitudinal changes

37 as unique metabolites in radicular cysts and periapical granuloma, respectively.

38 normal periodontal ligament (PDL) and of 12 periapical granulomas or cysts.

39 Normal PDL and periapical granulomas with scant inflammatory infiltrati

40 er 6 and 12 months for the evaluation of the periapical healing based on the established clinical and

41 clinical attachment level (CAL), pulpal and periapical healing, root resorption, and radiographic bo

42 Periapical images obtained with a storage phosphor plate

43 Periapical imaging is relatively accurate when standardi

44 l structures were assessed using the complex periapical index (COPI).

45 port, a 54-year-old patient presented with a periapical infection involving the mesial root of the ma

46 e canal roof is discovered, enucleation of a periapical infection or subsequent implant placement can

47 i-implantitis (RPI) is a rapidly progressing periapical infection that forms around the implant apex.

48 s to be protective against infection-induced periapical inflammation and bone destruction via suppres

49 e knockdown to study the function of Ac45 in periapical inflammation and bone resorption.

50 esign effective therapies that could prevent periapical inflammation and revolutionize current treatm

51 Periapical inflammation may also exacerbate inflammation

52 plays an important role in the chronicity of periapical inflammation via induction of inflammatory ce

53 e root canal system of the tooth, leading to periapical inflammation, bone erosion, severe pain, and

54 In the development of mouse periapical inflammation, SAA1.1/2.1 was elevated locally

55 terial infection-stimulated bone erosion and periapical inflammation, which confirms the potential th

56 eously target periapical bone resorption and periapical inflammation.

57 arkedly elevated IL-1alpha production within periapical inflammatory tissues (>10-fold) compared with

58 Radiographic evaluation revealed that the periapical lesion extended from the apex of the tooth to

59 progressive and significant increase in the periapical lesion size in both strains was observed.

60 During surgical endodontic treatment, periapical lesion tissue was collected and used for rela

61 hage, and dendritic cell infiltration in the periapical lesion was dramatically reduced, and the peri

62 lt in bone-resorptive cytokine production in periapical lesion.

63 ess susceptible to develop bacterial-induced periapical lesion.

64 o and decreased the number of T cells in the periapical lesion.

65 To investigate this question we compared periapical-lesion pathogenesis in RAG-2 severe combined

66 those of exposed immunocompetent controls in periapical-lesion size.

67 itis-module, caries-localization-module, and periapical-lesion-localization-module.

68 ckly and accurately detecting and segmenting periapical lesions (PALs) associated with AP on cone bea

69 current knowledge concerning periodontal and periapical lesions activity and the underlying molecular

70 ed using MTA and TotalFill in the healing of periapical lesions after endodontic surgery.

71 kening (odds ratio: 3.02, P <0.001), whereas periapical lesions and root canal fillings were not.

72 orptive cytokines IL-1alpha and IL-1beta, in periapical lesions and with decreased expression of the

73 l proliferation associated with inflammatory periapical lesions and with periapical cyst formation re

74 Periodontal and periapical lesions are infectious inflammatory osteolity

75 SAA1/2 was locally expressed in human periapical lesions at the mRNA and protein levels.

76 PCR) showed KGF expression in 4 specimens of periapical lesions but low or undetectable levels in nor

77 There was heavy cytokine staining in periapical lesions from both strains, especially in area

78 tion regulates the transcriptomic profile of periapical lesions in a mouse model of apical periodonti

79 The development of periapical lesions in IL-17RA KO mice was significantly

80 Forty non-molar teeth with periapical lesions in need of extraction and ARP from 33

81 of root coverage, and extent of inflammatory periapical lesions in relation to adjacent anatomical st

82 crosis and the histomorphometric features of periapical lesions in scid vs. normal mice.

83 on of KGF expression in the stromal cells of periapical lesions may play an important role in stimula

84 preclinical studies of both periodontal and periapical lesions points to a high receptor activator o

85 preclinical studies of both periodontal and periapical lesions points to a high receptor activator o

86 scessed RAG-2 teeth had significantly larger periapical lesions than did nonabscessed RAG-2 teeth (P

87 The cross-sectional area of periapical lesions was determined by image analysis of c

88 Periapical lesions were induced in 24 canine teeth of 6

89 Periapical lesions were induced in the lower first molar

90 y has been implicated in the pathogenesis of periapical lesions, although the extent to which these m

91 mine the influence of periodontal bone loss, periapical lesions, and root canal fillings on these sin

92 rior teeth, including periodontal bone loss, periapical lesions, and root canal fillings, were assess

93 diagnosis of endodontic pathologies such as periapical lesions, fractures and resorptions, as well a

94 e had redundant attenuation of the extent of periapical lesions, these animals showed strikingly impr

95 ether SAA is involved in the pathogenesis of periapical lesions, using human periapical surgical spec

96 ue to dental caries, periodontal disease, or periapical lesions.

97 reatment, is a persistent inhabitant of oral periapical lesions.

98 nly within vasculature structures located in periapical lesions.

99 roup exhibited internal root resorption with periapical lesions.

100 n infection caused by periodontal disease or periapical lesions.

101 Periapical lucency and sinus opacification were seen in

102 CT findings, including periapical lucency suggesting abscess, sinus opacificati

103 Abnormal periapical lucency, widening of the periodontal ligament

104 re needed to focus on histologic data around periapical microbiota to establish specific etiology and

105 Visible periapical or periodontal changes in dentition were anal

106 Asymptomatic periapical osteolysis, periodontal disease or dead teeth

107 s regulate the transcriptomic profile of the periapical osteolytic lesion in a mouse model of apical

108 cone beam computed tomography (CBCT) versus periapical (PA) radiographs in detecting PA changes at b

109 including 7814 radiographs of 12,373 molars (periapical, panoramic, cone-beam computed tomography), w

110 of abrogation of T- and B-cell mechanisms on periapical pathogenesis were then assessed.

111 the use of this model system for studies of periapical pathogenesis.

112 1 and 98.2 for implants placed in sites with periapical pathology and implants placed in sites withou

113 e implants placed in the sites demonstrating periapical pathology were followed in function for </=11

114 ts immediately placed in sites demonstrating periapical pathology yielded results comparable to those

115 te implant placement in a site demonstrating periapical pathology, and immediate implant placement in

116 thology and implants placed in sites without periapical pathology, respectively, according to publish

117 erapy present with hopeless teeth exhibiting periapical pathology.

118 Therefore, pulpal and periapical pathosis were independent of the presence of

119 For both models, periapical pressure increased with increasing irrigant f

120 Immediately post-operatively, a standardized periapical radiograph (PA) was taken using a standardize

121 T post-processing software and compared with periapical radiographs (PRs).

122 al parameters and radiographic findings from periapical radiographs and Cone Beam Computed Tomographi

123 D-speed vertical bitewing and periapical radiographs incorporating aluminum stepwedges

124 ECBL was assessed using periapical radiographs obtained during implant placement

125 ated from digitalized standardized intraoral periapical radiographs obtained from natural teeth and d

126 Periapical radiographs of 18 African Americans with sick

127 Periapical radiographs of mandibular incisors from subje

128 ly assessed in 450 adults using standardized periapical radiographs of maxillary central incisors.

129 Panoramic and periapical radiographs showed a circumscribed 0.8x0.9-cm

130 andibles were measured from non-standardized periapical radiographs taken at abutment connection and

131 of apical portion of implant, and subsequent periapical radiographs taken demonstrated a radiolucent

132 same reference just after loading by digital periapical radiographs to determine the marginal bone lo

133 ralized bone matrix were included, and 1,536 periapical radiographs were analyzed.

134 Periapical radiographs were evaluated before surgery, po

135 Periapical radiographs were evaluated immediately after

136 Periapical radiographs were obtained, as well as two inc

137 Clinical parameters and periapical radiographs were registered on the day of imp

138 Digital photographs and periapical radiographs were taken after restoration.

139 and bleeding on probing) were measured, and periapical radiographs were taken at the time of implant

140 Standardized periapical radiographs were taken immediately after impl

141 Periapical radiographs were taken using the long-cone te

142 evaluation was performed, and panoramic and periapical radiographs were taken.

143 Periapical radiographs were used to evaluate changes in

144 eolar bone loss (ABL), measured on intraoral periapical radiographs with a modified Schei ruler metho

145 The periapical radiographs yielded stable peri-implant bone

146 fety was evaluated by clinical examinations, periapical radiographs, and occurrence of adverse experi

147 The periapical radiographs, obtained in a standardized manne

148 detects dental conditions using bitewing and periapical radiographs, providing a detailed analysis of

149 d distal region of mandibular first molar on periapical radiographs.

150 nal bone levels using standardized intraoral periapical radiographs.

151 ant contact (distance bone-implant [DIB]) by periapical radiographs.

152 s with fractal dimension analysis on digital periapical radiographs.

153 pulp test for tooth #15, and complete mouth periapical radiographs.

154 CBCT scanning, periapical radiography (PA), and direct measurements usi

155 sociation between the presence of persistent periapical radiolucency and root wall thickness ( P = 0.

156 As there was a periapical radiolucency, an endodontic consultation was

157 volve manipulation of gingival tissue or the periapical region of teeth or perforation of the oral mu

158 We combined data of widened periapical spaces (WPSs) and apical rarefactions to a sc

159 Periapical surgeries, implants and maxillary sinus lift

160 region is an important area with respect to periapical surgery, implant placement, and sinus lifts.

161 hogenesis of periapical lesions, using human periapical surgical specimens and mice deficient in SAA

162 us bone fragments) on the healing process of periapical tissues after endodontic micro-surgery proced

163 e that AAV-mediated Atp6i/TIRC7 knockdown in periapical tissues can inhibit endodontic disease develo

164 to evaluate whether evoked bleeding from the periapical tissues elicits the influx of MSCs into the r

165 showed that local delivery of AAV-sh-Ac45 in periapical tissues in bacterium-induced inflammatory les

166 IL-17 was strongly induced in periapical tissues in wild-type (WT) mice by 7 d after t

167 ntic treatment (ET) and the condition of the periapical tissues of permanent teeth based on cone-beam

168 al debridement, intracanal bleeding from the periapical tissues was achieved, and intracanal blood sa

169 , and after 0, 7, or 14 days, total RNA from periapical tissues was submitted for sequencing and bioi

170 acterized by inflammation and destruction of periapical tissues, leading to severe bone resorption an

171 nguish between hypoxic and normoxic pulp and periapical tissues.

172 ated clinically and radiographically through periapical x-rays after one week, three, and six months.

173 Radiographic assessment was performed using periapical X-rays.