ライフサイエンスコーパス: periarticular

1 In this study, we demonstrate that periarticular administration of exosomes purified from e

2 Similarly, local, periarticular administration of lactoferrin into S aureu

3 These individuals lacking ENT1 exhibit periarticular and ectopic mineralization, which confirms

4 Interestingly, ASCs protected periarticular and systemic bone loss in CIA mice by main

5 aracterized by joint inflammation as well as periarticular and systemic bone loss.

6 joints were examined for marrow, articular, periarticular, and soft-tissue findings.

7 I can be used for the diagnosis of intra- or periarticular blood metabolites because their potential

8 Because progressive destruction of the periarticular bone contributes significantly to joint dy

9 e osteoclast activity is responsible for the periarticular bone destruction that characteristically o

10 Periarticular bone erosion and bone edema were scored ac

11 ith devastating joint tissue destruction and periarticular bone erosion.

12 Finally, osteoclastogenesis and periarticular bone erosions are markedly increased in SH

13 DF8 was predictive of atrophy, weakness, and periarticular bone loss 6 months following surgical ACL

14 and attenuated PTOA severity and deficits in periarticular bone microarchitecture.

15 rder that involves cartilage degradation and periarticular bone response.

16 d schedule-dependent preservation of BMD and periarticular bone while essentially eliminating intrale

17 oarthritis (OA), a disorder of cartilage and periarticular bone, is a public health problem without e

18 might have adverse effects on cartilage and periarticular bone.

19 with erosion of the articular cartilage and periarticular bone.

20 NTPPPH PC-1 caused infantile wrist and ankle periarticular calcification and vascular calcification.

21 Elbow and MCP joint periarticular calcifications were observed in 35 and 5 p

22 the elongation of columns and stimulation of periarticular chondrocyte differentiation in these model

23 n of periarticular to columnar chondrocytes (periarticular chondrocyte differentiation) and thereby r

24 action, PTHrP upregulation, acceleration of periarticular chondrocyte differentiation, and elongatio

25 , elongation of columns, and acceleration of periarticular chondrocyte differentiation.

26 t Ihh positively controls differentiation of periarticular chondrocytes independently of PTHrP.

27 In the developing growth plate, periarticular chondrocytes proliferate, differentiate in

28 was also noted in this mouse, while most of periarticular chondrocytes retained PPR signaling.

29 These results demonstrate that Ihh acts on periarticular chondrocytes to stimulate their differenti

30 of columnar chondrocytes, Ihh action in the periarticular chondrocytes was upregulated because of ec

31 es, chondrocytes near the articular surface (periarticular chondrocytes) proliferate, differentiate i

32 tflow buffering facilitates fluid escape and periarticular edema.

33 Periarticular erosion formation may not necessarily depe

34 degree of flexor tenosynovitis (P = 0.532), periarticular erosions (P = 0.579), or degree of bone ed

35 reased osteoclastic bone resorption leads to periarticular erosions and systemic osteoporosis in RA p

36 acroscopic evidence of CIA first appeared as periarticular erythema and edema in the hind paws by day

37 ilage, bone, synovium, ligament, muscle, and periarticular fat, leading to joint dysfunction, pain, s

38 traindications to MRI or in the setting of a periarticular ferromagnetic foreign body obscuring the f

39 rtrophic differentiation by signaling to the periarticular growth plate and also determines the site

40 -related peptide (PTHrP), synthesized in the periarticular growth plate, regulates the site at which

41 patients (88.9%) demonstrated a concomitant periarticular haemorrhage.

42 but is less important in the pathogenesis of periarticular inflammation in this disease.

43 tivity after both peripheral soft tissue and periarticular inflammation was abolished.

44 between days 10 and 18, was characterized by periarticular inflammation with marked synovitis, synovi

45 aw swelling and deformity, less synovial and periarticular inflammation, and markedly decreased bone

46 Alternatively, in states of severe periarticular inflammation, TNF-alpha may fully exert it

47 ogical evidence of focal erosive lesions and periarticular inflammation.

48 raphy and MRI typically show a predominantly periarticular inflammatory process.

49 ddition of steroids to a multimodal cocktail periarticular injection (MCPI) in patients undergoing kn

50 ydrochloride alone (control) administered by periarticular injection at the time of surgery.

51 Periarticular injection of 125I-lactoferrin confirmed th

52 A total of 18 patients suffered from a periarticular injury, and 16 patients (88.9%) demonstrat

53 profoundly inhibited joint inflammation and periarticular joint destruction in a dose-dependent mann

54 knee pain and those without knee pain, other periarticular lesions (including bursitis and iliotibial

55 However, other periarticular lesions were present in 14.9% of patients

56 infrapatellar, deep infrapatellar) or "other periarticular lesions" (semimembranosus-tibial collatera

57 MRIs were read for the presence of periarticular lesions, which were categorized (according

58 recovery or pain associated with the use of periarticular liposomal bupivacaine compared with bupiva

59 lux, joint levels of prostaglandin E(2), and periarticular osteoclast formation were inhibited by tur

60 rom ovariectomy-induced osteoporosis and the periarticular osteolysis attending inflammatory arthriti

61 therefore, IL-1 plays a role in TNF-induced periarticular osteolysis.

62 lar joint tissue associated with progressive periarticular osteolytic lesions.

63 are frequently unremarkable, but may reveal periarticular osteopenia 3-6 weeks after the onset of cl

64 Mice in both age groups developed periarticular osteophytes at the tibial plateau in respo

65 roid hormone-related protein (PTHrP), in the periarticular perichondrium.

66 se models, upregulation of Ihh action in the periarticular region was also observed.

67 dependence due to pathologic accumulation of periarticular scar tissue.

68 In the marrow, periarticular signal intensity abnormality was common in

69 localized to one painful, tender and swollen periarticular site 86% of the time.

70 oint space including synovia, ligaments, and periarticular soft tissue.

71 identifying uric acid deposits in joints and periarticular soft tissues in patients suspected of havi

72 The surrounding periarticular soft tissues, bone structures, joint space

73 In the periarticular soft tissues, edema, enhancement, and ulce

74 ealed only scattered rare lymphocytes in the periarticular soft tissues, without joint destruction.

75 y disease that mainly affects the joints and periarticular soft tissues.

76 aterally to rapidly stress the knee's medial periarticular structures and create a potentially destab

77 The MR appearances of all articular and periarticular structures were analyzed and compared with

78 The MR appearances of all intraarticular and periarticular structures were analyzed and correlated wi

79 yloid infiltration into soft tissue, joints, periarticular structures, and bones can bring patients w

80 roved the visualization of all articular and periarticular structures.

81 ient serum concentrations, the levels in the periarticular tissue during TKA may be insufficient to p

82 PTX3 was induced in the periarticular tissue of mice postinjection of MSU crysta

83 ods of time when injected locally within the periarticular tissue surrounding the ankle joints of mic

84 ity over TIMP action in the invading pannus, periarticular tissue, or SF.

85 onosodium urate (MSU) crystals in joints and periarticular tissues.

86 inflammation, both in synovium and in other periarticular tissues.

87 show that Ihh stimulates differentiation of periarticular to columnar chondrocytes (periarticular ch

88 Acceleration of periarticular to columnar differentiation was also noted

89 e terminal step but also at an earlier step: periarticular to columnar differentiation.