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2 wall appearance, distention, bile color, and pericholecystic fluid were not individually significant
3 udge and/or stones, (c) wall appearance, (d) pericholecystic fluid, and (e) common bile duct size and
4 Sensitivities of increased wall thickness, pericholecystic fluid, and adjacent fat signal intensity
7 enhancement (P<.001) and increased transient pericholecystic hepatic enhancement (P=.003) were the mo
8 der wall enhancement and increased transient pericholecystic hepatic enhancement had the highest comb
9 der wall enhancement and increased transient pericholecystic hepatic enhancement were 74% (14 of 19 p
11 nstrated gallbladder nonvisualization with a pericholecystic rim sign at 1 hr, a cholescintigraphic p
12 from 72% (gallbladder nonvisualization with pericholecystic rim sign before morphine) to 86% (gallbl
13 tion at 1 hr (before morphine); those with a pericholecystic rim sign were further evaluated for pers
14 ntigrams were analyzed for the presence of a pericholecystic rim sign, marked or mild, associated wit
15 1 hr of gallbladder nonvisualization with a pericholecystic rim sign, regardless of its intensity.
19 ered present if there was enlargement of the pericholecystic space (i.e., gallbladder fossa) and the
21 tudied - wall thickness, intramural nodules, pericholecystic stranding, wall thickness, THAD, fat in