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1  and flux, including puberty, pregnancy, and perimenopause.
2 en level occur with greater magnitude during perimenopause.
3 c used to relieve the negative components of perimenopause.
4 rement in SDMT learning observed during late perimenopause.
5 t of depression was temporally linked to the perimenopause.
6 rate enough by itself to rule in or rule out perimenopause.
7  such as hot flashes, certainly begin in the perimenopause.
8 osteoporosis, fractures and earlier onset of perimenopause.
9 c estradiol levels underpinned depression in perimenopause.
10 stage of ovarian failure comparable to human perimenopause.
11 0.12% [-.5 to .26]) and positive across late perimenopause (0.18% [-.10 to .45]) in WWH, and positive
12 rate of WC change were negative across early perimenopause (-0.21% [-.44 to .03]) and menopause (-0.1
13 ce rates of psychiatric disorders during the perimenopause (4 years surrounding the FMP) were compare
14 eases at the transition to menopause, termed perimenopause, a state characterized by erratic estrogen
15                                          The perimenopause, a time of changing ovarian function, prec
16 nea events per hour were 1.2 (0.7, 2.2) with perimenopause and 2.6 (1.4, 4.8) with postmenopause; odd
17 nea events per hour were 1.1 (0.5, 2.2) with perimenopause and 3.5 (1.4, 8.8) with postmenopause.
18            Although the relationship between perimenopause and changes in mood has been well establis
19             No association was found between perimenopause and first onsets of schizophrenia spectrum
20             No association was found between perimenopause and incidence rates of schizophrenia spect
21 tching to an INSTI-based regimen during late perimenopause and menopause is associated with faster in
22  failure (AOF) successfully replicates human perimenopause and postmenopause, including estrous acycl
23 in reproductive age, 1.0 (1.0) [0.0-10.0] in perimenopause, and 0.5 (1.0) [0.0-7.5] in post-menopause
24  BMI change across early perimenopause, late perimenopause, and menopause, respectively, compared to
25 es due to hormonal changes during pregnancy, perimenopause, and menopause, which affect fat distribut
26 , early puberty, late puberty, reproductive, perimenopause, and podtmenopausal) were approximated usi
27             The association of premenopause, perimenopause, and postmenopause with sleep-disordered b
28 63-0.96) but not in all other (premenopause, perimenopause, and unknown status) menopausal groups (n=
29 osis progression rate was accelerated during perimenopause, as shown using FIB-4 (0.12 units per year
30  1690 midlife women in premenopause or early perimenopause at study inception (who have since transit
31                   Clinicians should diagnose perimenopause based on menstrual history and age without
32 rual cycle markers to determine inception of perimenopause, defined as time from study enrollment to
33 SH, the levels of which are high during late perimenopause, directly stimulates bone resorption by os
34                             Importantly, the perimenopause emerges as a critical window of neural reo
35 he basis of symptom profile, duration of the perimenopause, endocrine measures, or past historical va
36   This issue provides a clinical overview of Perimenopause focusing on prevention, diagnosis, treatme
37 riods (i.e., a window of opportunity) during perimenopause for restoring ovarian hormones for the mos
38 ling in the emergence of hypertension during perimenopause has been hindered by animal models that ar
39               Accelerated fibrosis begins in perimenopause, highlighting a previously unrecognized gr
40 tility, iatrogenic adrenal insufficiency and perimenopause in patients with cystic fibrosis.
41 VMS was found to be higher during late-stage perimenopause in women with migraine.
42 0 person-years) and 1,133 (0.88%) during the perimenopause (incidence rate 2.33 per 1,000 person-year
43 disorders significantly increased during the perimenopause (incidence rate ratio (RR) of 1.52, 95% co
44                            Early puberty and perimenopause increase relapse risk; pediatric cases sho
45                  Compared to INSTI-, in late perimenopause, INSTI+ had 0.26 cm per 6 months (95% CI:
46                                      In late perimenopause, INSTI+ had faster increases in WC (0.37 c
47                                              Perimenopause is a period of high risk for mood disorder
48                                              Perimenopause is a time of transition for women at midli
49                                              Perimenopause is accompanied by an increased risk of new
50 ood disorders, and it has been proposed that perimenopause is also a window of risk for processes lin
51                     The prior probability of perimenopause is directly related to a woman's age.
52 se on annual rate of BMI change across early perimenopause, late perimenopause, and menopause, respec
53 data suggest that events related to the late perimenopause may be associated with an increased suscep
54               Hence, greater MAO-A VT during perimenopause may represent a new target for assessing n
55 related migraine, pregnancy, breast-feeding, perimenopause, menopause, nitric oxide, and estrogen rec
56 tory of depression had 1.2 times the rate of perimenopause of women with no such history (95% confide
57 y are responsible for the negative effect of perimenopause on cognitive processing speed.
58 he MT was defined as the first visit in late perimenopause (or first postmenopausal visit if particip
59                                              Perimenopause, or the menopausal transition, is the few-
60 dysphoric mood are associated with the early perimenopause, particularly among women with lower educa
61 ry high vulnerability within 10 years of the perimenopause period exhibited higher risk of earlier na
62                      Accelerated fibrosis in perimenopause persisted after adjustment for Hispanic et
63 en levels, and the ovariectomy model lacks a perimenopause phase.
64 l mood symptoms were evaluated in women with perimenopause-related depression.
65 nd 0.15% (-.42 to .71) across early and late perimenopause, respectively, and -0.40% (-1.24 to .45) a
66 .04), whereas women who transitioned to late perimenopause showed a 0.93-kg decline in grip strength
67 ning rate was 1.00 point smaller during late perimenopause than during premenopause (P = 0.04); furth
68              Here we investigate whether the perimenopause (that is, the years around the final menst
69                            However, in human perimenopause, the neurobiological changes implicated in
70  reducing the rise in LDL cholesterol during perimenopause to postmenopause but could not completely
71 sitioned directly from premenopause or early perimenopause to postmenopause).
72  cholesterol and weight gain in women during perimenopause to postmenopause.
73  the increase in weight from premenopause to perimenopause to postmenopause.
74                               In conclusion, perimenopause was associated with an increased risk of d
75          However, the largest effect size at perimenopause was observed for mania (RR of 2.12 (95% CI
76  did and did not become depressed during the perimenopause were determined by Student's t test, chi-s
77 that presented data relevant to diagnosis of perimenopause were identified in a MEDLINE search from 1
78    Women who developed depression during the perimenopause were not distinguished from those who rema
79  history of major depression who entered the perimenopause were twice as likely to develop significan
80  of Reproductive Aging Workshop criteria for perimenopause, which is mainly based on menstrual cycle