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1 ses, and unaffected by oxidation with sodium periodate.
2 ished by treatment of the glycopeptides with periodate.
3 ith hydrogen peroxide, air oxygen, or dilute periodate.
4 to o-aminophenols in the presence of sodium periodate.
5 s secondary alcohols, at specified levels of periodate.
6 hloride and subsequent oxidation with sodium periodate.
7 were subjected to oxidation by tyrosinase or periodate.
8 atment of T. vaginalis with metronidazole or periodate abolished the adhesion of parasites to cell mo
9 of binding to MAb 1B5 whereas treatment with periodate abolished this binding, suggesting the presenc
10 oelectric point of 4.2-4.6, and was shown by periodate acid Schiff's staining to be glycosylated.
13 elective interaction of peanut proteins with periodate amongst chloride, sulphate, iodide, phosphate,
14 s abolished by pretreatment of sections with periodate and in the presence of excess sialic acid or l
15 etected by oxidization of carbohydrates with periodate and labeling with hydrazide-conjugated digoxig
17 chment of terminal glycerol for oxidation by periodate and reaction of the resulting aldehyde with am
18 reated Arabidopsis thaliana RNA samples with periodate and then performed tRNA-seq to distinguish bet
19 harides were isolated, oxidized using sodium periodate, and conjugated to CRM197 by reductive aminati
20 mmediately fixed in paraformaldehyde, sodium periodate, and lysine (PLP); (2) some were stored at 4 d
24 M 3 binding when specimens were treated with periodate at high pH, which specifically destroys NeuPSA
27 nnan-dependent resistance can be overcome by periodate-borohydride conversion of mannose polysacchari
28 charides to polyalcohols; cells treated with periodate-borohydride initiate the alternative pathway w
30 posure of C. pneumoniae elementary bodies to periodate, but not elevated temperatures, inhibited chol
31 d be demonstrated that iodate is oxidized to periodate by a CuO-activated hypohalous acid, which is a
32 amine oxidized with one equivalent of sodium periodate causes a concentration-dependent inactivation
34 oretic methods and by controlled cleavage at periodate-cleavable moieties incorporated during synthes
35 oretic methods and by controlled cleavage at periodate-cleavable moieties incorporated during synthes
40 oprotein P by N-glycosidase assay and by the periodate-dansylhydrazine test, which indicated no detec
43 d oxidation with hydrogen peroxide or sodium periodate (i.e., the phenylselenocysteine side chain is
46 bilizes developing charge on both sulfur and periodate in the transition state via cation-pi and elec
48 ut were susceptible to treatment with sodium periodate, indicating that the antigen inducing opsonic
49 Using mildly basic reaction conditions, the periodate-induced diol cleavage of meso-tetraphenyl-2,3-
53 ced by 3,4-dihydroxyphenylalanine (DOPA) for periodate-mediated chemical cross-linking, and biotin wa
58 s employed for this derivatization is sodium periodate (NaIO(4)) oxidation of vicinal diols found wit
61 xyfunctionalization of steroidal ethers with periodate or bromate as terminal oxidants in phosphate b
62 chains of cell surface Sia residues by mild periodate oxidation (known to abrogate Sia recognition b
63 ion with ethylthioacetyl chloride and sodium periodate oxidation afforded a E/Z mixture of alpha-sulf
66 ribe a technique termed GLIB (glucosylation, periodate oxidation and biotinylation), which combines s
67 emical modification of a standard heparin by periodate oxidation and borohydride reduction enhanced i
68 ivatives of digoxin have been synthesized by periodate oxidation and reductive amination using a vari
69 ide (2AB) labeling derivatization, including periodate oxidation and reductive amination, took only ~
70 after modification of the GalNAc residues by periodate oxidation and sodium borohydride reduction, in
71 ides were monitored for activation by sodium periodate oxidation and subsequent analysis by gas chrom
72 bstituted, peptide-linked GalNAc residues by periodate oxidation and subsequent trimming of the remai
75 etal-dependent hydroxyquinone formation from periodate oxidation of catechol 1, suggested a rate-limi
81 lboronate was achieved in part by controlled periodate oxidation of the 9,21-diol to the 21-nor-9-oxo
83 3) of triacylglycerol was measured following periodate oxidation of the glycerol isolated from hydrol
92 l nucleic acid ribosides, such as ATP, using periodate oxidation simplifies the chromatogram and mini
94 ethylated and 2'-hydroxylated nucleosides to periodate oxidation to develop Nm-seq, a sensitive metho
97 by labeling with biotin hydrazide following periodate oxidation, a specific and well established met
98 first approach, termed GLIB (glucosylation, periodate oxidation, biotinylation) uses a combination o
100 l N-BocGlu, further Boc N-protection, OsO(4)-periodate oxidation, O-Me oxime formation, and RaNi-cata
101 four monokines were found to be sensitive to periodate oxidation, the TNF-alpha-stimulating activity
102 on of FT-IR, methylation and GC-MS analysis, periodate oxidation-Smith degradation, partial acid hydr
108 Antigenicity was abolished by chemical (periodate) oxidation of gTSSA-I glycosylation but retain
110 ed tetrahydro-beta-carbolines undergo sodium periodate oxidative ring expansion in the presence of fo
111 blocked by P2X7RS-specific concentrations of periodate oxidised ATP (OxATP: 100 microM) and brilliant
112 ophages with a P2Z/P2X7 receptor antagonist, periodate oxidized adenosine 5'-triphosphate (o-ATP), su
113 receptor antagonists, including suramin and periodate oxidized ATP (oATP), resulted in a significant
114 -2',4'-disulphonic acid, Brilliant Blue G or periodate oxidized ATP dialdehyde to the site of ATP rel
115 ct of the cross-linking reaction between the periodate-oxidized AMP moiety of the tRNA and Lys207 is
116 etween aldehyde groups on the Fc moieties of periodate-oxidized antibodies and hydrazide groups on fu
117 tropic nucleotide (P2X) receptors, including periodate-oxidized ATP (oATP), attenuate a subset of end
118 In vivo short-term P2X7R targeting (using periodate-oxidized ATP [oATP]) delays islet allograft re
119 y the P2X(7) purinergic receptor antagonists periodate-oxidized ATP and pyridoxal-phosphate-6-azophen
120 ular ATP/P2X purinergic signaling pathway by periodate-oxidized ATP delayed the progression of the dy
128 ted enhanced macrophage and mast cell death; periodate-oxidized-ATP (oATP)-treated macrophage and mas
130 vivo perfusion with paraformaldehyde-lysine-periodate (PLP) and processed for light and electron mic
132 n good agreement with those from the current periodate-resorcinol method (P>0.05) thus indicating tha
133 iol (1) to Cu(II) and subsequent addition of periodate resulted in rapid formation of the TPQ-like co
134 dation but rather were obtained using sodium periodate, resulting in variable NDA yields (13-51%) fro
137 ng a battery of monoclonal antibodies (MAbs) periodate-sensitive epitopes were identified on Tf190, s
140 Using both thin-layer chromatography and the periodate-thiobarbituric acid reaction, we found that th
143 ting can be miniaturized by treating it with periodate to produce higher-resolution, high-fidelity mi
148 hand, lymphocytes from mice sensitized with periodate-treated SEA did not produce any of these same
150 Furthermore, sensitization of mice with periodate-treated SEA significantly reduced levels of Ag
151 ice sensitized with native SEA, but not with periodate-treated SEA, produced IL-4, IL-5, and IL-10 wh
153 those containing a cP, are cleaved through a periodate treatment after phosphatase treatment; hence,
154 ective cleavage at the core mannitol by mild periodate treatment and analysis of the reaction product
155 region, which were missing from the mutants; periodate treatment and carbohydrate staining confirmed
157 t treatment of E. chaffeensis had no effect, periodate treatment completely abolished the ability of
162 Binding of MAb 4A4-2 was inhibited by mild periodate treatment of N. caninum antigen, demonstrating
164 the synthetic (nonglycosylated) peptide, and periodate treatment of the recombinant glycopeptide epit
165 nsitive to proteinase K treatment but not to periodate treatment, indicating that the cognate epitope
173 eness of modification of sialic acid by mild periodate was verified with monoclonal antibody to sialy