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1 ic regimen targeting DC-STAMP could suppress periodontal bone loss.
2 hat resembles pulpitis while also displaying periodontal bone loss.
3 al link between neuronal TRPV1 signaling and periodontal bone loss.
4 hat TLR9 signaling mediates the induction of periodontal bone loss.
5 periodontitis and contribute to induction of periodontal bone loss.
6 eir growth and differentiation contribute to periodontal bone loss.
7 ignificant reduction in number of teeth with periodontal bone loss.
8 ienced >60% more naturally occurring chronic periodontal bone loss.
9 ture delineation of the precise mechanism of periodontal bone loss.
10 een recognized as a key factor implicated in periodontal bone loss.
11 ntitis, as evidenced by reduced induction of periodontal bone loss.
12  cannot be complacent about the diagnosis of periodontal bone loss.
13 axillary jaws were removed for assessment of periodontal bone loss.
14 ce of factors that may be involved in active periodontal bone loss.
15 he possible role of PLAP-1 and sclerostin in periodontal bone loss.
16 ion of Pg-LPS-induced osteoclastogenesis and periodontal bone loss.
17  central proinflammatory cytokine related to periodontal bone loss.
18 teria are associated with the progression of periodontal bone loss.
19 mic PTB administration significantly reduced periodontal bone loss, AGE deposition, and expressions o
20 lla forsythia is also a vital contributor to periodontal bone loss, almost nothing is known about imm
21                                              Periodontal bone loss (an index of PD severity) was eval
22  a relationship between poor oral hygiene or periodontal bone loss and chronic obstructive pulmonary
23 ingiva of mandibular first molars to measure periodontal bone loss and collagen content.
24 cco use is generally linked with accelerated periodontal bone loss and diminished post-surgical wound
25           This effect might be important for periodontal bone loss and for the enhanced bone loss see
26 , in part, explain the increased rapidity of periodontal bone loss and refractory disease incidence i
27                                              Periodontal bone loss and tooth mobility were also incre
28 thod was evaluated for its ability to detect periodontal bone loss and was compared with that of conv
29 mmatory processes affecting atherosclerosis, periodontal bone loss ,and possibly, diet-induced weight
30  treatment of estrogen deficiency-aggravated periodontal bone loss, and berberine represents a promis
31  is critical for macrophage polarization and periodontal bone loss, and for the first time, to our kn
32                                              Periodontal bone loss appears to be a major factor in th
33 g to this definition, the patients exhibited periodontal bone loss at a much higher prevalence (60%)
34 dal anti-inflammatory drugs (NSAIDs) inhibit periodontal bone loss, but little is known about the mec
35 e, we hypothesize that berberine ameliorates periodontal bone loss by improving the intestinal barrie
36  CD5(+) B cell transfer demonstrated reduced periodontal bone loss compared to the no-transfer group
37  significant in multivariable models), while periodontal bone loss did not.
38                                   AG reduced periodontal bone loss during the induction of experiment
39 th rHag B on protection against experimental periodontal bone loss following infection with P. gingiv
40 sion) in which there is a furcal lesion with periodontal bone loss; Group I (intermediate) in which t
41 vD1) confers protection against IL-17-driven periodontal bone loss in a Del-1-dependent manner, indic
42 Coffee consumption may be protective against periodontal bone loss in adult males.
43                       Dry mouth may indicate periodontal bone loss in children with diabetes.
44 ne the effects of TLR-activated B10 cells on periodontal bone loss in experimental periodontitis.
45 ic animals revealed higher susceptibility to periodontal bone loss in Mc3r(-/-) compared with wild-ty
46 aboratory revealed that T. forsythia induces periodontal bone loss in mice and that this bone loss de
47 ined the extent and severity of radiographic periodontal bone loss in patients with different stages
48 ntal contribution of periodontal bacteria to periodontal bone loss in patients with MetS remains uncl
49 icroinjection of anti-MFG-E8 mAb exacerbated periodontal bone loss in wild-type mice.
50 r TLR2-deficient mice were both resistant to periodontal bone loss, in stark contrast with wild-type
51        Finally, radiographic measurements of periodontal bone loss indicated that rats immunized with
52 ge-associated disorder clinically defined by periodontal bone loss, inflammation of the specialized t
53  vivo studies revealed that ligature-induced periodontal bone loss is significantly greater in Akita
54 ion, using a P. gingivalis infection-induced periodontal bone loss model, we found that SGK1 inhibiti
55 fic Stat3-deficient mice exhibited increased periodontal bone loss (p < 0.001), whereas T cell- and B
56 in significantly restricted ligature-induced periodontal bone loss (P <0 .01) and suppressed the leve
57 in significantly restricted ligature-induced periodontal bone loss (P <0 .01) and suppressed the leve
58  hydrogel sites showed significantly reduced periodontal bone loss (P <0.05) and inflammatory infiltr
59 ings of the upper posterior teeth, including periodontal bone loss, periapical lesions, and root cana
60 lysis was used to determine the influence of periodontal bone loss, periapical lesions, and root cana
61 h hypophosphatemic rickets are more prone to periodontal bone loss than the general population and ma
62  Th17-to-Tfh plasticity resulted in elevated periodontal bone loss that was not simply due to increas
63 in 2 (NOD2) signaling in atherosclerosis and periodontal bone loss using an Apolipoprotein E(-/-) (Ap
64 ligand (RANKL), which, in turn, promotes the periodontal bone loss via upregulation of osteoclastogen
65 ctices were determined by questionnaire, and periodontal bone loss was defined as alveolar bone loss
66                                              Periodontal bone loss was induced in 16-wk-old myeloid-s
67                                              Periodontal bone loss was measured via histological and
68 with experimental periodontitis, significant periodontal bone loss was noted in animals both with and
69                                       Severe periodontal bone loss was significantly associated with
70                                       Severe periodontal bone loss was significantly associated with
71                                              Periodontal bone loss was significantly associated with
72                                              Periodontal bone loss was significantly decreased and th
73                      In the induction sites, periodontal bone loss was significantly reduced (P <0.05
74               The CsA-induced attenuation of periodontal bone loss was strongly correlated positively
75                                              Periodontal bone loss was three times more common among
76                          Sinuses with severe periodontal bone loss were three times more likely to ha
77 reased inflammation, osteoclastogenesis, and periodontal bone loss when transferred to normal germ-fr
78 e correlations of CsA-induced attenuation of periodontal bone loss with the expressions of gelatinase