ライフサイエンスコーパス: periodontal pathogen

1 FimA, in Porphyromonas gingivalis, a primary periodontal pathogen.

2 luding Porphyromonas gingivalis, a potential periodontal pathogen.

3 hatase and reveal an invasin of an important periodontal pathogen.

4 volved in the production of fimbriae by this periodontal pathogen.

5 from Actinobacillus actinomycetemcomitans, a periodontal pathogen.

6 t not by that of Porphyromonas gingivalis, a periodontal pathogen.

7 valis is a peptide-fermenting asaccharolytic periodontal pathogen.

8 nce the inflammatory response induced by the periodontal pathogen.

9 tially a good choice for inhibiting invasive periodontal pathogens.

10 altered immune-inflammatory response against periodontal pathogens.

11 overall showed a stronger inhibition of the periodontal pathogens.

12 re- or PCR-positive results for the targeted periodontal pathogens.

13 de antibiotic that is active against several periodontal pathogens.

14 ogenicity and ability to interact with other periodontal pathogens.

15 e bacterial test (CST) for the most relevant periodontal pathogens.

16 y lower prevalence and abundance of putative periodontal pathogens.

17 wn to have anti-microbial properties against periodontal pathogens.

18 on may be related to an elevated exposure to periodontal pathogens.

19 ctiveness of clarithromycin against invasive periodontal pathogens.

20 cts who harbored two, or all three, of these periodontal pathogens.

21 plex interplay between the immune system and periodontal pathogens.

22 ntal therapy significantly reduced levels of periodontal pathogens.

23 performed to detect the presence of selected periodontal pathogens.

24 haps symptomatic of colonization by residual periodontal pathogens.

25 determined the occurrence of potential major periodontal pathogens.

26 outcomes in addition to previously reported periodontal pathogens.

27 lation of the innate immune host response to periodontal pathogens.

28 oss, and bone resorption that are induced by periodontal pathogens.

29 chment gain had a high prevalence of these 3 periodontal pathogens.

30 following interaction with several putative periodontal pathogens.

31 were positive for at least one of the target periodontal pathogens.

32 bgingival colonization and multiplication of periodontal pathogens.

33 y at high levels, is primarily restricted to periodontal pathogens.

34 allows more sensitive detection of all three periodontal pathogens.

35 ilable polymerase chain reaction test for 11 periodontal pathogens.

36 ely as a result of increased colonization of periodontal pathogens.

37 ated with peri-implantitis are recognized as periodontal pathogens.

38 nvironment and a distributional shift of key periodontal pathogens.

39 ymicrobial infection with well-characterized periodontal pathogens.

40 s been primarily focused on a small group of periodontal pathogens.

41 d by corrosion may enhance the attachment of periodontal pathogens.

42 to 71% increase), bacterial killing of these periodontal pathogens (22 to 38% reduction of bacterial

43 Oral injections of LPS derived from the periodontal pathogen A. actinomycetemcomitans can induce

44 The periodontal pathogen A. actinomycetemcomitans has been k

45 All targeted periodontal pathogens (A. actinomycetemcomitans, P. ging

46 bone loss in rats using LPS derived from the periodontal pathogen Actinobacillus actinomycetemcomitan

47 Fresh clinical isolates of the periodontal pathogen Actinobacillus actinomycetemcomitan

48 fresh clinical isolates of the gram-negative periodontal pathogen Actinobacillus actinomycetemcomitan

49 The periodontal pathogen Actinobacillus actinomycetemcomitan

50 Some clinical isolates of the gram-negative periodontal pathogen Actinobacillus actinomycetemcomitan

51 The Gram-negative periodontal pathogen Actinobacillus actinomycetemcomitan

52 Here, we report that the periodontal pathogen Actinobacillus actinomycetemcomitan

53 cts associated with a virulence locus of the periodontal pathogen Actinobacillus actinomycetemcomitan

54 Strains of the periodontal pathogen Actinobacillus actinomycetemcomitan

55 late three catalase-deficient mutants of the periodontal pathogen Actinobacillus actinomycetemcomitan

56 zation and confocal microscopy to detect the periodontal pathogens Actinobacillus actinomycetemcomita

57 d with intracellular bacteria, including the periodontal pathogens Actinobacillus actinomycetemcomita

58 Among putative periodontal pathogens, Actinobacillus actinomycetemcomit

59 ne whether Prevotella intermedia, a putative periodontal pathogen, activates populations of specific

60 The periodontal pathogen Aggregatibacter (Actinobacillus) ac

61 e cytolethal distending toxin (Cdt) from the periodontal pathogen Aggregatibacter actinomycetemcomita

62 cytolethal distending toxin (Cdt), from the periodontal pathogen Aggregatibacter actinomycetemcomita

63 crease in antibacterial activity against the periodontal pathogen Aggregatibacter actinomycetemcomita

64 hal distending toxin (Cdt), expressed by the periodontal pathogen Aggregatibacter actinomycetemcomita

65 Biofilm formation by the periodontal pathogen Aggregatibacter actinomycetemcomita

66 e major clonal lineages of the Gram-negative periodontal pathogen Aggregatibacter actinomycetemcomita

67 The periodontal pathogen Aggregatibacter actinomycetemcomita

68 The periodontal pathogen Aggregatibacter actinomycetemcomita

69 The periodontal pathogen Aggregatibacter actinomycetemcomita

70 sed a dual-species biofilm consisting of the periodontal pathogen Aggregatibacter actinomycetemcomita

71 The aim of this investigation is to quantify periodontal pathogens (Aggregatibacter actinomycetemcomi

72 sent investigation was to identify potential periodontal pathogens among these newly identified speci

73 Porphyromonas gingivalis is a keystone periodontal pathogen and its lipopolysaccharide (PgLPS)

74 hat patterns of high and low levels of eight periodontal pathogens and antibody levels against those

75 After the 2-week study period, putative periodontal pathogens and cariogenic bacteria were overa

76 maxillas and spleens from mice infected with periodontal pathogens and compared to those in the maxil

77 his may suggest possible association between periodontal pathogens and DG.

78 antibacterial activity against most putative periodontal pathogens and has been shown to kill bacteri

79 rivative possessing antimicrobial effects on periodontal pathogens and inhibitory properties on matri

80 Clarithromycin inhibits several periodontal pathogens and is concentrated inside gingiva

81 High levels of periodontal pathogens and low maternal IgG antibody resp

82 o 19 species, including established/putative periodontal pathogens and non-pathogenic bacteria, in 5,

83 reduced the relative abundance of classical periodontal pathogens and of Fretibacterium fastidiosum,

84 It increases the acquisition of periodontal pathogens and periodontal disease, colonizat

85 for the presence of familial aggregation of periodontal pathogens and periodontitis and have alluded

86 ciated with the subgingival presence of some periodontal pathogens and periodontitis.

87 hol consumption on the levels of subgingival periodontal pathogens and proinflammatory cytokines (int

88 In addition, putative periodontal pathogens and resistance of subgingival bact

89 MC-2 was correlated with traditional periodontal pathogens and several newly identified putat

90 ediates of microbial burden (levels of eight periodontal pathogens) and local inflammatory response (

91 e we report that Porphyromonas gingivalis, a periodontal pathogen, and Escherichia coli LPS induce os

92 anaerobe, Porphyromonas gingivalis, is a key periodontal pathogen, and several lines of evidence link

93 ship of gingivitis with salivary biomarkers, periodontal pathogens, and interleukin (IL)-1 polymorphi

94 king, age, IgG levels against other selected periodontal pathogens, and race.

95 of host cells by a limited number of 'model' periodontal pathogens, and therefore may not adequately

96 The wide variability found in periodontal pathogen antibiotic-resistance patterns shou

97 the hypothesis that oral bacteria other than periodontal pathogens are also associated with pregnancy

98 ntal disease, high levels of colonization of periodontal pathogens are associated with an increased r

99 Periodontal pathogens are detrimental to periodontal hea

100 While traditional putative periodontal pathogens are implicated, research involving

101 Periodontal pathogens are present in atherosclerotic pla

102 e by impairing the innate immune response to periodontal pathogens as well as by increasing free radi

103 Furthermore, in response to these periodontal pathogens (as mono- and polymicrobial heat-k

104 Persistence of putative and novel periodontal pathogens, as well as low prevalence of bene

105 The oral spirochete Treponema denticola, a periodontal pathogen associated with human periodontitis

106 f this study was to test the hypothesis that periodontal pathogens associated with aggressive periodo

107 nema denticola, and Tannerella forsythia are periodontal pathogens associated with the etiology of ad

108 ophaga spp. have been implicated as putative periodontal pathogens associated with various periodonta

109 Periodontal pathogens belonging to the genera Fusobacter

110 iodontal parameters and serum IgG levels for periodontal pathogens between PLBW and healthy delivery

111 ission is not limited to the well-recognized periodontal pathogens, but instead may also involve the

112 ached to gingival cells in vitro, inhibiting periodontal pathogens by competition, adherence, and dis

113 Periodontal pathogens/byproducts may reach the placenta

114 tainment proofs 1 to 6 provides support that periodontal pathogens can contribute to atherosclerosis.

115 Porphyromonas gingivalis, a periodontal pathogen, can efficiently invade human gingi

116 sent an overreaction of the host response to periodontal pathogens caused by excessive production of

117 We used GWAS data of CP (n = 4,504) and periodontal pathogen colonization (n = 1,020) from a coh

118 6); AP3B2, p = 2.2 x 10(-6)) and 2 with high periodontal pathogen colonization (red complex-KCNK1, p

119 ce on host genetic risk loci associated with periodontal pathogen colonization.

120 Periodontal pathogens colonized implants symptomatic thr

121 Periodontal pathogens commonly associated with chronic a

122 Exposures consisted of 19 periodontal pathogens, constructed factors and clusters,

123 est positive for antiCl, likely because some periodontal pathogens contain antigens homologous to the

124 periodontitis patients contain aCl, and some periodontal pathogens contain antigens with peptide sequ

125 The study aim was to determine whether periodontal pathogens could be enriched from pooled sali

126 ew findings provide mechanistic support that periodontal pathogens create a unique microenvironment i

127 Risk for harboring 2 or more periodontal pathogens decreased with the years the paren

128 Rats infected with the periodontal pathogens displayed a five-fold increase in

129 Increased levels of periodontal pathogens disrupt the homeostasis between th

130 ne the role of saliva-derived biomarkers and periodontal pathogens during periodontal disease progres

131 ues, we tested the ability of three putative periodontal pathogens-Eikenella corrodens, Porphyromonas

132 It is known that periodontal pathogens elicit host-derived immune respons

133 hogens and several newly identified putative periodontal pathogens: Fretibacterium fastidiosum, Freti

134 d with increased odds of detection of common periodontal pathogens from individuals with aggressive p

135 culated that periodontal probes can transmit periodontal pathogens from site to site.

136 support the hypothesis of a translocation of periodontal pathogens from subgingival microbiota to the

137 patients with and without DM, and 2) isolate periodontal pathogens from these patients' blood.

138 A defining characteristic of the suspected periodontal pathogen Fusobacterium nucleatum is its abil

139 s among the presence of three orange-complex periodontal pathogens (Fusobacterium nucleatum, Prevotel

140 that growth of the PFOR-containing anaerobic periodontal pathogens, grown in both monospecies as well

141 Periodontal pathogens have been detected from atheromato

142 Since a few periodontal pathogens have been reported to invade oral

143 However, some periodontal pathogens have developed strategies to evade

144 ampylobacter rectus has been implicated as a periodontal pathogen; however, association with periodon

145 Filifactor alocis is a recently recognized periodontal pathogen; however, little is known regarding

146 Porphyromonas gingivalis is a periodontal pathogen implicated in a range of pregnancy

147 ent reports focusing on virulence factors of periodontal pathogens implicated proteinases as major de

148 ion was conducted to confirm the presence of periodontal pathogens in bone particles harvested intrao

149 gingivalis is recognized as one of the major periodontal pathogens in chronic periodontitis, a common

150 Importantly, the levels of IL-17 induced by periodontal pathogens in CII-specific T cells directly c

151 Detection of periodontal pathogens in early periodontitis suggests an

152 ot an adequate method for identifying DNA of periodontal pathogens in low quantities because of the h

153 biotic resistance among selected subgingival periodontal pathogens in patients with CP.

154 a key role in the innate immune responses to periodontal pathogens in periodontal disease.

155 tion of S. dentisani supernatant against the periodontal pathogens in pure culture.

156 ere used to determine the IgG titres against periodontal pathogens in serum samples.

157 nested multiplex PCR method to detect three periodontal pathogens in subgingival plaque collected be

158 and their association with the levels of key periodontal pathogens in subgingival plaque.

159 his study is to detect and quantify the main periodontal pathogens in the oral microbiota of postmeno

160 ion is to compare the presence and number of periodontal pathogens in the subgingival microbiota of s

161 ted with decreased levels of IgG antibody to periodontal pathogens in women with periodontitis when a

162 logical testing showed the presence of known periodontal pathogens including Actinobacillus actinomyc

163 y 40% compared to that of HC, and killing of periodontal pathogens, including Porphyromonas gingivali

164 loading time increased, but colonization by periodontal pathogens, including red complex species, wa

165 their precise roles are not well understood, periodontal pathogens, including Treponema denticola, ar

166 Porphyromonas gingivalis, an important periodontal pathogen, infects primary gingival epithelia

167 However the mechanisms by which periodontal pathogens influence the development of predi

168 One of the features of this periodontal pathogen is its ability to attach to a varie

169 The simultaneous detection of three periodontal pathogens is an advantage of this technique

170 The inflammatory response to periodontal pathogens is dynamically controlled by the c

171 ibroblasts that occurs during infection with periodontal pathogens is, in part, mediated by TNF.

172 Porphyromonas gingivalis, an important periodontal pathogen, is an effective colonizer of oral

173 Porphyromonas gingivalis, a key periodontal pathogen, is capable of invading a variety o

174 Treponema denticola, a periodontal pathogen, is relatively resistant to human b

175 the rate of preterm delivery, a decrease in periodontal pathogen load, and a decrease in both GCF IL

176 tween Helicobacter pylori (Hp) and groups of periodontal pathogens may alter the onset of Alzheimer's

177 tudy was devised to test the hypothesis that periodontal pathogens may be present and affect human pl

178 This study indicates that periodontal pathogens may play important roles in the sh

179 in the field and addressed the link between periodontal pathogens measured with the benzoyl-DL-argin

180 The major fimbriae of this periodontal pathogen mediate binding to host gingival ep

181 In the surgical specimens positive for periodontal pathogens, more than 1 species was most ofte

182 Thus, in order to colonize, successful periodontal pathogens must devise means to interfere wit

183 t group also demonstrated significantly less periodontal pathogens of red and orange complexes and a

184 g effects of co-eradication of Hp and select periodontal pathogens on neurodegenerative disease.

185 th Porphyromonas gingivalis (Pg), a putative periodontal pathogen, on the progression of atherosclero

186 Likewise, periodontal pathogens, opportunistic pathogens, or norma

187 Baseline levels of selected periodontal pathogens or changes in these bacteria resul

188 lis (P gingivalis) (10(7) CFU), an important periodontal pathogen, or vehicle once per week for 14 or

189 The presence of periodontal pathogens P.g., P.i., C.r., and B.f. in subg

190 serum levels of IgG antibody to the panel of periodontal pathogens (P = 0.0018), to P. gingivalis (P

191 Thus, S. mutans and the periodontal pathogens, P. gingivalis and B. forsythus, w

192 The periodontal pathogens Pg, Pi, Tf, and Fn are associated

193 te genome representing a novel strain of the periodontal pathogen Porphyromonas gingivalis (P. gingiv

194 The interaction of the periodontal pathogen Porphyromonas gingivalis (Pg) with

195 tide (VIP) modulates immune responses to the periodontal pathogen Porphyromonas gingivalis (Pg).

196 The periodontal pathogen Porphyromonas gingivalis adheres to

197 ibute to osteolytic lesions, we injected the periodontal pathogen Porphyromonas gingivalis adjacent t

198 mine the antibacterial effects of EMD on the periodontal pathogen Porphyromonas gingivalis and 2) to

199 lipid 430 and lipid 654, are produced by the periodontal pathogen Porphyromonas gingivalis and can be

200 The periodontal pathogen Porphyromonas gingivalis and the en

201 Two types of fimbriae, FimA and Mfa1, of the periodontal pathogen Porphyromonas gingivalis are respon

202 However, the periodontal pathogen Porphyromonas gingivalis can contro

203 The periodontal pathogen Porphyromonas gingivalis employs a

204 Here, we demonstrate that infection with the periodontal pathogen Porphyromonas gingivalis enhances t

205 Lipopolysaccharide (LPS) derived from the periodontal pathogen Porphyromonas gingivalis has been r

206 The capsular polysaccharide (CPS) of the periodontal pathogen Porphyromonas gingivalis is an impo

207 Previously, we have established that the periodontal pathogen Porphyromonas gingivalis is capable

208 The periodontal pathogen Porphyromonas gingivalis is implica

209 One of the features of the periodontal pathogen Porphyromonas gingivalis is the pre

210 work suggests that brain colonization by the periodontal pathogen Porphyromonas gingivalis may link t

211 Although the periodontal pathogen Porphyromonas gingivalis must withs

212 f lipopolysaccharide (LPS) purified from the periodontal pathogen Porphyromonas gingivalis on human m

213 re, we investigate the direct effects of the periodontal pathogen Porphyromonas gingivalis on osteocl

214 el to study the effect of infection with the periodontal pathogen Porphyromonas gingivalis on pregnan

215 Finally, the abundance of the periodontal pathogen Porphyromonas gingivalis trended wi

216 lutinin B (rHagB), a virulence factor of the periodontal pathogen Porphyromonas gingivalis, has been

217 In the periodontal pathogen Porphyromonas gingivalis, the CTD i

218 eated diabetic mice, infected with the human periodontal pathogen Porphyromonas gingivalis, with solu

219 LR4(-/-)) mice were orally infected with the periodontal pathogen Porphyromonas gingivalis.

220 presence or absence of LPS purified from the periodontal pathogen Porphyromonas gingivalis.

221 The presence of periodontal pathogens Porphyromonas gingivalis (P.g.), P

222 activity of major virulence factors from the periodontal pathogens Porphyromonas gingivalis and Actin

223 his probiotic by measuring inhibition of the periodontal pathogens Porphyromonas gingivalis and Fusob

224 Repeated oral inoculations of periodontal pathogens Porphyromonas gingivalis and Prevo

225 For example, the periodontal pathogens Porphyromonas gingivalis and Tanne

226 Growth inhibition of the periodontal pathogens Porphyromonas gingivalis, Prevotel

227 , and Campylobacter rectus), two red-complex periodontal pathogens (Porphyromonas gingivalis and Tann

228 The presence of periodontal pathogens (Porphyromonas gingivalis, Aggrega

229 ously reported that oral administration of a periodontal pathogen, Porphyromonas gingivalis (Pg) to W

230 We postulated that the periodontal pathogen, Porphyromonas gingivalis may suppr

231 colonization with a well-characterized human periodontal pathogen, Porphyromonas gingivalis We found

232 es and subsequent proteomic responses to the periodontal pathogen, Porphyromonas gingivalis, donor-ma

233 ar premolars followed by an application of a periodontal pathogen, Porphyromonas gingivalis, to induc

234 the present study, LPS derived from the oral periodontal pathogen, Porphyromonas gingivalis, was comp

235 Deoxyribonucleic acids (DNA) of periodontal pathogens, Porphyromonas gingivalis (Pg) and

236 o characterize host responses of interest to periodontal pathogens, Porphyromonas gingivalis was intr

237 antibacterial activity against two anaerobic periodontal pathogens: Porphyromonas gingivalis and Acti

238 Thirteen (59%) of the 22 periodontal pathogen-positive surgical specimens were po

239 Recent evidence suggests that certain periodontal pathogens preferentially stimulate T cells e

240 This study evaluated the levels of periodontal pathogens Prevotella intermedia, Porphyromon

241 as well as a cysteine proteinase of another periodontal pathogen, Prevotella intermedia, resulted in

242 ormerly Bacteroides forsythus) is one of the periodontal pathogens recently implicated in the develop

243 is report was to compare the distribution of periodontal pathogens recovered from failing implants an

244 hat activation of the acquired immunity by a periodontal pathogen reduces the coupling of bone format

245 United States frequently yielded subgingival periodontal pathogens resistant in vitro to therapeutic

246 of the patients with CP revealed subgingival periodontal pathogens resistant to at least one of the t

247 een percent of patients harbored subgingival periodontal pathogens resistant to both amoxicillin and

248 parental and feoB1-deficient strains of the periodontal pathogen revealed that the feoB1-deficient m

249 Porphyromonas gingivalis, an invasive periodontal pathogen, secretes the HAD family phosphoser

250 pants assembled into three clusters based on periodontal pathogens, serum and salivary biomarkers.

251 In response to endotoxins derived from periodontal pathogens, several osteoclast-related mediat

252 selected controls from the above cohort, the periodontal pathogen-specific maternal serum IgG levels

253 provide mechanistic support that SCFAs from periodontal pathogens stimulate KSHV replication and inf

254 onizing the colonization and accumulation of periodontal pathogens such as P. gingivalis.

255 Periodontal pathogens such as Porphyromonas gingivalis a

256 Known putative periodontal pathogens such as Porphyromonas gingivalis,

257 al has strong antibacterial activity against periodontal pathogens such as Streptococcus mutans, Porp

258 tegerin) combined with red-complex anaerobic periodontal pathogens (such as Porphyromonas gingivalis

259 cells of the periodontium to encounter known periodontal pathogens, such as Actinobacillus actinomyce

260 was associated with elevated levels of known periodontal pathogens, such as P. nigrescens, T. forsyth

261 Virulence factors from periodontal pathogens, such as Porphyromonas gingivalis

262 The virulence of periodontal pathogens, such as Porphyromonas gingivalis,

263 crobial co-occurrence analysis revealed that periodontal pathogens, such as Porphyromonas gingivalis,

264 Moreover, traditional periodontal pathogens, such as Porphyromonas gingivalis,

265 ssociation of miR-146a expression with these periodontal pathogens, suggesting that miR-146a may dire

266 Aggregatibacter actinomycetemcomitans, a periodontal pathogen, synthesizes leukotoxin (LtxA), a p

267 athogens by reconstructing the genome of the periodontal pathogen Tannerella forsythia and also ident

268 We found the periodontal pathogen Tannerella forsythia to be associat

269 e genes, (iv) a genome reconstruction of the periodontal pathogen Tannerella forsythia, (v) 239 bacte

270 Antepartum, the levels of periodontal pathogens tended to be higher in the preterm

271 tis have higher levels of GCF biomarkers and periodontal pathogens than clinically healthy sites from

272 Porphyromonas gingivalis is a periodontal pathogen that also localizes to atherosclero

273 Porphyromonas gingivalis (Pg) is a major periodontal pathogen that contains immunostimulatory com

274 Porphyromonas gingivalis is a consensus periodontal pathogen that has been implicated in adult f

275 Treponema denticola is a consensus periodontal pathogen that has recently been associated w

276 d to RA through the production of enzymes by periodontal pathogens that citrullinate proteins.

277 obacillus actinomycetemcomitans are putative periodontal pathogens that may be harbored in subgingiva

278 addition, in comparison with other anaerobic periodontal pathogens, the removal of 8-oxoG was unique

279 gingival epithelium forms a barrier against periodontal pathogens, this study was undertaken to dete

280 response to systemic inflammation induced by periodontal pathogens through mechanisms involving downr

281 Selected periodontal pathogen titers, factors, and clusters inter

282 parameters and the salivary presence of six periodontal pathogens to age-related macular degeneratio

283 The periodontal pathogen Treponema denticola produces dentil

284 The metabolism of glutathione by the periodontal pathogen Treponema denticola produces hydrog

285 ue samples were assessed for the presence of periodontal pathogens using indirect immunofluorescence.

286 nd assessed their response to infection with periodontal pathogens using microarray analysis, quantit

287 ontal pockets and the prevalence of selected periodontal pathogens was assessed in 10 patients with a

288 d" DNA-DNA hybridization quantification of 8 periodontal pathogens was performed.

289 Detection of known periodontal pathogens was rare.

290 minutes after membrane placement, suspected periodontal pathogens were detected in several ePTFE mem

291 cocci, was measured in 78.2% of thrombi, and periodontal pathogens were measured in 34.7%.

292 , whereas the carriage rates of the examined periodontal pathogens were not.

293 Microbiologic assessments of eight putative periodontal pathogens were performed using the checkerbo

294 The samples were cultured, and selected periodontal pathogens were tested in vitro for susceptib

295 ar lipids of Porphyromas gingivalis, a known periodontal pathogen, were previously shown to promote d

296 Porphyromonas gingivalis is a periodontal pathogen whose primary niche is the anaerobi

297 y worsen oral health, favoring the growth of periodontal pathogens, whose detection could improve the

298 Campylobacter rectus is a periodontal pathogen with a 150-kDa protein on its cell

299 newly developed CST can detect five typical periodontal pathogens with a somewhat lower sensitivity

300 ence intervals (CIs) for the associations of periodontal pathogens with total cancer and site-specifi