ライフサイエンスコーパス: periodontitis

1 riodontal treatment in patients with chronic periodontitis.

2 as a predictor for CAL loss in patients with periodontitis.

3 microbiome of young individuals with severe periodontitis.

4 epidemiological studies for the diagnosis of periodontitis.

5 ferent sets of criteria for the diagnosis of periodontitis.

6 ants were classified as non-severe or severe periodontitis.

7 cavity-related disorders mainly focusing on periodontitis.

8 in parallel with the severity and extent of periodontitis.

9 of alveolar bone lost owing to experimental periodontitis.

10 n summary, FLI was associated with prevalent periodontitis.

11 nforcing the genetic profile predisposing to periodontitis.

12 pe 2 diabetes mellitus with dyslipidemia and periodontitis.

13 developing prostatitis than patients without periodontitis.

14 inflammatory properties for the treatment of periodontitis.

15 e relationship between systemic diseases and periodontitis.

16 rized according to the new classification of periodontitis.

17 sociated with lower chance to develop severe periodontitis.

18 rial wall and aortic sinus induced by severe periodontitis.

19 four times greater than among those without periodontitis.

20 and alveolar bone resorption associated with periodontitis.

21 the minimal-set of questions associated with periodontitis.

22 dictive ability of the new classification of periodontitis.

23 ssociation between sleep duration and severe periodontitis.

24 sts regarding the role of IL-37 and IL-38 in periodontitis.

25 Therefore, we assessed such interactions in periodontitis.

26 gated the association between TBS and severe periodontitis.

27 arthritis pathogen and established agent of periodontitis.

28 ommended in the management of severe chronic periodontitis.

29 ridement (OFD) for the management of chronic periodontitis.

30 evaluated in a murine model of experimental periodontitis.

31 of chronic inflammatory diseases, including periodontitis.

32 -term follow-up (>10 years) in patients with periodontitis.

33 n medication consumption and the severity of periodontitis.

34 igher prevalence, severity, and extension of periodontitis.

35 and root planning (Q-SRP) in severe chronic periodontitis.

36 implications for prevention and treatment of periodontitis.

37 10, MMP1, and MMP3 in GFs from patients with periodontitis.

38 scular disease and diabetes in patients with periodontitis.

39 tory mediators in rats with ligature-induced periodontitis.

40 and lost teeth were valid to predict severe periodontitis.

41 ween periodontal health and Stage II and III periodontitis.

42 21 patients with chronic generalized severe periodontitis.

43 ally healthy, and Group II, Stage II and III periodontitis.

44 tes and at sites undergoing ligature-induced periodontitis.

45 d as a pivotal player in the defense against periodontitis.

46 ntribute to greater osteolysis in aggressive periodontitis.

47 sue homeostasis and inflammatory response in periodontitis.

48 g type 2 diabetes which is a risk factor for periodontitis.

49 ed on CBCT scans of 34 patients with grade C periodontitis.

50 he cause of extraction was persistent apical periodontitis (35.7%), but this increase didn't reach th

51 Non-treated periodontitis-affected mice and non-ligated mice were us

52 In periodontitis-affected tissues, the imbalance between T-

53 he Treg-lymphocyte detection and response in periodontitis-affected tissues.

54 hment loss in older age; presence of "frank" periodontitis affecting a single tooth; and assessment o

55 was strongly associated with moderate/severe periodontitis after adjusting by socio-demographic facto

56 , chronic periodontitis (CP), and aggressive periodontitis (AgP) based on clinical parameters.

57 Aggressive periodontitis (AgP), currently periodontitis grade C, pr

58 Treatment of periodontitis aims to halt progressive bone and attachme

59 gingivalis have been shown to play a role in periodontitis, although, to date, little is known about

60 Forty patients with periodontitis and 38 periodontally healthy individuals (

61 ently associated with localized Stage II/III periodontitis and a J-shaped association was suggestive

62 udies have identified an association between periodontitis and Alzheimer disease (AD); however, the n

63 periodontal tissue response to experimental periodontitis and appeared to be a protective factor aga

64 ppropriate for patients with Stage III or IV periodontitis and can be recommended in dental practice

65 liva and to evaluate suPAR as a biomarker of periodontitis and CHD.

66 gingival fibroblasts (GF) from patients with periodontitis and controls.

67 studies indicate a close association between periodontitis and diabetes.

68 Degraded TBS is associated with severe periodontitis and has a synergistic effect with poor ora

69 neralized severe periodontitis with moderate periodontitis and healthy group: oral hypoglycemic agent

70 ore the relationship between the severity of periodontitis and of abdominal aortic aneurysm (AAA).

71 oss occurs in inflammatory disorders such as periodontitis and osteoporosis.

72 The present results suggest that periodontitis and P. intermedia are associated with seve

73 5,510 patients with newly diagnosed chronic periodontitis and participated in therapies were selecte

74 resent study demonstrated that patients with periodontitis and periodontitis + CHD presented higher s

75 l examination to determine the occurrence of periodontitis and PISA.

76 Association between periodontitis and prostate diseases of benign prostatic

77 bacteria may mediate the association between periodontitis and RA, and monitoring the bacterial compo

78 Using a mouse model of ligature-induced periodontitis and resolution, we tested the ability of a

79 ant positive associations were found between periodontitis and severe asthma, (odds ratio [OR](adjust

80 ssociated with chronic conditions, for which periodontitis and sleep are established risk factors.

81 nificantly contributes to the development of periodontitis and the anaerobic bacterium Porphyromonas

82 gs taken by elderly patients with or without periodontitis and the possible association between medic

83 Twenty-five Stage 3 periodontitis and twenty-five periodontally healthy indi

84 bstances-and oral health outcomes, including periodontitis and untreated caries, among US adults.

85 V significantly inhibited the development of periodontitis and vascular inflammation and remarkably b

86 as to analyze the impact of gingival health, periodontitis, and CHD on suPAR levels in plasma and sal

87 cto measure of healthy behaviors)," outcome "periodontitis," and confounders "age, sex, household inc

88 n the microflora in disease-either caries or periodontitis-and only recently have they considered fac

89 pe 2 diabetes mellitus with dyslipidemia and periodontitis; and (T2DMpoorly-DL-P) poorly-controlled t

90 included 130 individuals: 40 with aggressive periodontitis (AP group), 40 with chronic periodontitis

91 Obesity and periodontitis are conditions with high burden and cost.

92 2 diabetes mellitus (T2DM), dyslipidemia and periodontitis are frequently associated pathologies; how

93 ve capability for detecting individuals with periodontitis (area under the curve [AUC] = 0.85 95% CI

94 more in participants with mild and moderate periodontitis as compared to those with gingivitis in mi

95 Women were classified for periodontitis, as follows: Center for Disease Control an

96 Taken together, unculturable periodontitis-associated bacteria may play an important

97 ted various formulations to grow health- and periodontitis-associated subgingival microbiomes in para

98 Patients presented with periodontitis at the University of Michigan between Janu

99 chronic periodontitis (stage 3/4 generalized periodontitis) at 6 months in the French population.

100 factor (TNF)-alpha in patients with Stage 3 periodontitis before and after non-surgical periodontal

101 owed a clear association between low BMD and periodontitis, but only in women above 58 years old and

102 eriodontal healthy (control group); (P) with periodontitis, but systemically healthy; (DL-P) with dys

103 the exacerbation of atherosclerosis induced periodontitis by inhibiting local, systemic and vascular

104 The inflammatory burden imposed by periodontitis can increase serum hsCRP levels in adults

105 s of oral diseases such as dental caries and periodontitis, can be potentially achieved by detecting

106 tients with periodontitis (P <.001) and with periodontitis + CHD (P <.001) presented higher median pl

107 strated that patients with periodontitis and periodontitis + CHD presented higher suPAR levels in bot

108 With stratification, risks between two periodontitis cohorts exhibited no difference.

109 In two periodontitis cohorts, 636 and 638 participants compared

110 n patients with stage III and IV generalized periodontitis compared with no/localized/generalized sta

111 History of periodontitis confirmed its well-known role as a risk fa

112 e often been interpreted as evidence against periodontitis contribution to the etiology of systemic h

113 eening for serum PCT levels in patients with periodontitis could act not only as a guide to assess th

114 thway of causal relationship for obesity and periodontitis could help in developing management strate

115 ve periodontitis (AP group), 40 with chronic periodontitis (CP group), and 50 periodontally healthy c

116 e gingival crevicular fluid (GCF) of chronic periodontitis (CP) patients with type-2 diabetes mellitu

117 iodontal health (H), gingivitis (G), chronic periodontitis (CP), and aggressive periodontitis (AgP) b

118 mokers (S) and non-smokers (NS) with chronic periodontitis (CP), and compare them with those of perio

119 users and non-users [controls] with chronic periodontitis [CP]) remains uninvestigated.

120 Regardless of medium type, the periodontitis-derived microbiomes showed higher species

121 and significantly worsened dysbiosis in the periodontitis-derived microbiomes.

122 ranged from 1.02 to 6.19), and AMI, for both periodontitis diagnostic criteria.

123 In GF from a non-periodontitis donor, stimulation with either TNF-alpha,

124 ded into three groups: control, experimental periodontitis (Ep), and Ep-melatonin (Ep-Mel).

125 Thus, oral inflammation, such as periodontitis, exacerbates gut inflammation by supplying

126 patterns identified were not associated with periodontitis extent.

127 graphy (CBCT) scans of patients with grade C periodontitis for detection of non-visible changes in th

128 d TNF-alpha levels in generalized aggressive periodontitis (G-AgP).

129 Smoking is considered a risk factor for periodontitis genesis and progression.

130 gorized into a Healthy group (H, n = 20) and periodontitis/gingivitis group (PG, n = 70) according to

131 Aggressive periodontitis (AgP), currently periodontitis grade C, presents early onset, rapid progr

132 oup were considerably lower than that in the periodontitis group (saliva = 0.22 ng/mL and serum = 1.8

133 statistically higher intake frequency in the periodontitis group relative to healthy controls (21.5%

134 d diuretics were significantly higher in the periodontitis group with OR = 2.49, 2.32, 2.08 and 1.79,

135 0.001) and IL-10 (P <0.01) compared with the periodontitis group.

136 the number of osteoclasts compared with the periodontitis group.

137 Despite treatment, the patients with periodontitis had higher risk of developing prostatitis

138 ionship of body mass index (BMI) with severe periodontitis has been reported.

139 association between metabolic disorders and periodontitis has been widely studied and recognized.

140 f boldine on alveolar bone resorption during periodontitis has not been elucidated yet.

141 c blockade of select IL-1 family members for periodontitis has only been partially investigated in pr

142 Porphyromonas gingivalis, a known driver of periodontitis, has been proposed as the microbial link u

143 The exact mechanisms of bone resorption in periodontitis have not been fully elucidated.

144 a high probability of suffering from severe periodontitis (high-mPESS).

145 We examined the relationships between periodontitis, hours of sleep and white blood cell (WBC)

146 e gold-standard approach for surveillance of periodontitis; however, it requires large resources.

147 Gingivitis was found in 68.2%, periodontitis in 21.2%, and retinopathy in 64.7%, GADA (

148 SNPs as genetic markers of susceptibility to periodontitis in a Brazilian population.

149 alidity of a self-reported questionnaire for periodontitis in a Spanish population from Barcelona dur

150 ntifies individuals who are most affected by periodontitis in different ages.

151 leotide polymorphisms (SNPs) associated with periodontitis in diverse populations.

152 in osteocytes appeared to be reduced during periodontitis in knock-in mice.

153 d to be associated with lower risk to severe periodontitis in men.

154 ct of Msx2-null mutation during experimental periodontitis in mice.

155 ome and gingival transcriptome in health and periodontitis in nonhuman primates (Macaca mulatta).

156 Sites with periodontitis in obese individuals showed higher levels

157 periodontal debridement for the treatment of periodontitis in patients type 2 diabetes.

158 d immunological benefits to the treatment of periodontitis in patients with type 2 diabetes.

159 n between low bone mineral density (BMD) and periodontitis in perimenopausal women is controversial.

160 sed in a variety of epidemiologic studies of periodontitis in pregnant females.

161 variables associated with the occurrence of periodontitis in the final logistic model were: MetS (od

162 Clinical examinations showed a prevalence of periodontitis in the sample (n = 585) of 99.4% and 86.3%

163 etween habitual flavonoid intake and risk of periodontitis in the study population.

164 to identify people who are most affected by periodontitis in their age group is currently unavailabl

165 Melatonin restricts the periodontitis-induced inflammatory stress, apoptosis, an

166 ct of physical inactivity induced obesity to periodontitis is 14%.

167 Periodontitis is a chronic inflammatory disease characte

168 Periodontitis is a chronic inflammatory disease triggere

169 Periodontitis is a complex, multifactorial chronic disea

170 Periodontitis is a suspected environmental risk factor f

171 Periodontitis is an inflammatory disease that can lead t

172 vertheless, common chronic diseases to which periodontitis is associated have multifactorial etiologi

173 It has been suggested that periodontitis is associated with metabolic abnormalities

174 ts and were consistent with the concept that periodontitis is associated with perturbations in the re

175 Considering that periodontitis is epidemiologically linked to liver disea

176 l dissemination of the new classification of periodontitis is facilitated by emphasis on the basic gr

177 Periodontitis is increasingly associated with increased

178 Periodontitis is positively linked to cardiovascular dis

179 ating between Stage I/II versus Stage III/IV periodontitis is relatively uncomplicated; further disti

180 Periodontitis leads to expansion of oral pathobionts, in

181 The probability of severe periodontitis (main dependent variable) was assessed by

182 Using a rodent periodontitis model, we assessed the ability of P. gingi

183 eriodontal status classified as healthy/mild periodontitis (n = 356) and moderate/severe periodontiti

184 periodontitis (n = 356) and moderate/severe periodontitis (n = 358).

185 Individuals with gingivitis or mild-moderate periodontitis (n=36) were included and randomly assigned

186 ber of chronic inflammatory diseases such as periodontitis, neurogenic inflammation, and inflammatory

187 I was associated with a higher prevalence of periodontitis (Odds ratio (OR) (highest vs. lowest quart

188 tigate the potential inflammatory effects of periodontitis on cardiac left ventricular tissue and the

189 tudy were to inspect the possible effects of periodontitis on the structure and functions of the kidn

190 of risk factors, and the two-way effects of periodontitis or its treatment on general health; (v) sh

191 PY gene) presented increased risk for severe periodontitis (OR = 2.34; 95% CI = 1.19-4.59).

192 ne) demonstrated increased risk for moderate periodontitis (OR = 2.58; 95% CI = 1.28-5.18).

193 % CI ranged from 1.03 to 10.05) and moderate periodontitis (OR(adjusted) ranged from 1.96 to 2.51; 95

194 During experimental periodontitis, osteonecrosis area and osteoclast number

195 Patients with periodontitis (P <.001) and with periodontitis + CHD (P

196 antly higher in the kidneys of the rats with periodontitis (P <0.05).

197 l treatment compared with baseline values of periodontitis patients (P < .001).

198 y from GF, with comparable levels in GF from periodontitis patients and controls.

199 One hundred samples were collected from 10 periodontitis patients and seven healthy individuals.

200 CSF-1 was increased in gingival tissue from periodontitis patients compared with controls (P < 0.05)

201 ical periodontal measurements were recorded; periodontitis patients received non-surgical periodontal

202 individuals and shallow and deep sites from periodontitis patients were individually collected at ba

203 Periodontitis patients were more likely to develop prost

204 ree parallel arms (#NCT04038801), 60 chronic periodontitis patients were randomly assigned to three s

205 bone changes between healthy individuals and periodontitis patients with fractal dimension analysis o

206 ts diagnosed with severe generalized chronic periodontitis (periodontitis stage 3/4) were included in

207 patients presenting with moderate to severe periodontitis (PPD = 5 to 8 mm) in at least 2 pockets pe

208 ients with generalized stage III-IV, grade B periodontitis presenting with a 6- to 9-mm interproximal

209 a Brazilian population of genetic markers of periodontitis previously investigated by GWAS and bioinf

210 bone resorption is a distinctive feature of periodontitis progression and determinant for tooth loss

211 ttern [treelet component (TC)] and extent of periodontitis [proportion of sites with clinical attachm

212 d sex- and smoking-habit-specific effects on periodontitis; reinforcing the genetic profile predispos

213 cardiac left ventricular tissues provoked by periodontitis-related bacteria and/or periodontal inflam

214 In addition, periodontitis-related histological damages and apoptotic

215 ses were performed to quantify the levels of periodontitis-related unculturable bacteria (Eubacterium

216 onship between metabolic syndrome (MetS) and periodontitis remains controversial.

217 ealing in preclinical studies and in chronic periodontitis, represents a potential treatment option.

218 ously with 1,4-DPCA/hydrogel at the onset of periodontitis resolution displayed significantly increas

219 In parallel, periodontitis results in generation of oral pathobiont-r

220 specific nutrients has been associated with periodontitis risk but there has been little research in

221 proach was used to determine obesity-related periodontitis risk using the Australian National Survey

222 oral hygiene procedures on the recurrence of periodontitis (RP) during periodontal maintenance therap

223 ion of adenosine receptors, CD39 and CD73 of periodontitis samples were retrieved from a public datab

224 , six men) aged between 21 and 75 years with periodontitis scheduled for periodontal surgery were inc

225 e performance of self-reported questions for periodontitis screening in a representative sample of a

226 vestigated the association between levels of periodontitis severity (exposure) and AMI (outcome).

227 dy evidenced potential relationships between periodontitis severity and size of AAA.

228 a possible relationship among the levels of periodontitis severity and the cardiovascular condition.

229 Levels of periodontitis severity followed two criteria: (1) Center

230 The ability of FTND and NCC to predict periodontitis severity was assessed and compared.

231 taneously affected by T2DM, dyslipidemia and periodontitis, showed an altered molecular profile mainl

232 At the multilevel analysis, history of periodontitis, sinus elevation with lateral approach, an

233 th severe generalized chronic periodontitis (periodontitis stage 3/4) were included in this study.

234 h periodontitis stage III and IV compared to periodontitis stage I was observed (99.31 ng/mL [IQR: 85

235 concentration of MMP-8 and -9 in women with periodontitis stage III and IV compared to periodontitis

236 t findings suggest that smoker patients with periodontitis Stage III and IV, Grade C respond well to

237 trabony defects in subjects with generalized periodontitis stage III, grade B.

238 RP) outcomes in patients with severe chronic periodontitis (stage 3/4 generalized periodontitis) at 6

239 Severe and progressive periodontitis (stage IV and grade C) tended to be more f

240 esidual periodontal pockets in patients with periodontitis (Stages III and IV) after non-surgical per

241 y, we observed that IgG anticardiolipin from periodontitis subjects also causes fetal loss in mice.

242 We observed that anticardiolipin from periodontitis subjects competes for annexin V on an arti

243 vascular inflammation, and IgG purified from periodontitis subjects with elevated anticardiolipin sti

244 ses such as EBV and CMV in CAD patients with periodontitis suggesting it as one of the risk factors f

245 eficiency is considered as a risk factor for periodontitis, supplementation during periodontal treatm

246 ically healthy; (DL-P) with dyslipidemia and periodontitis; (T2DMwell-DL-P) well-controlled type 2 di

247 d with higher risk of localized Stage II/III periodontitis than their counterparts (24.0 to 26.9 kg/m

248 cteroidetes, is a keystone pathogen in human periodontitis that may also contribute to the developmen

249 es illustrated that the effect of obesity on periodontitis that was not mediated through poor dental

250 ed that among those with moderate and severe periodontitis, the chance of having AMI was approximatel

251 whether FLI itself would be associated with periodontitis through a secondary analysis of previously

252 igher than the indirect effect of obesity on periodontitis through dental visiting behavior.

253 uciniphila or Amuc_1100 in the management of periodontitis through their anti-inflammatory properties

254 eoclastogenic properties during experimental periodontitis; thus, further promoting the Th17-driven b

255 During periodontitis, tooth-supporting alveolar bone is resorbe

256 e alveolar bone in the jaws can occur due to periodontitis, trauma or following tumor resection.

257 ubgingival microbiome of young patients with periodontitis treated by means of mechanical and antibio

258 re + desipramine: rats with ligature-induced periodontitis treated with desipramine (20 mg/kg/d in ve

259 al composition of young patients with severe periodontitis treated with scaling and root planing and

260 ne); 2) ligature: rats with ligature-induced periodontitis treated with vehicle; 3) ligature + desipr

261 it can be used as an excellent candidate for periodontitis treatment.

262 A total of 73 individuals with chronic periodontitis underwent scaling and root planing (SRP).

263 s, flavones, and flavonols) and incidence of periodontitis using Cox proportional hazard models.

264 gate potential associations between diet and periodontitis using novel statistical techniques for die

265 llitus (DM) in patients with Stage III or IV periodontitis using the FINDRISC questionnaire.

266 .4%) with OR of 2.43, 1.99, and 2.28 (severe periodontitis versus healthy group), respectively.

267 The mean FD of individuals with periodontitis was 0.97, whereas it was 1.04 for the heal

268 In this study, experimental periodontitis was associated with the progressive and in

269 Case definition for severe periodontitis was defined according to World Workshop (2

270 The severity of localized periodontitis was defined as healthy (n = 42), Stage I (

271 Periodontitis was defined by the Periodontal Profile Cla

272 By contrast, periodontitis was directly related to WBC count and %neu

273 Higher risk for moderate periodontitis was found in male with rs7762544-AG close

274 In the highest FLI quartile, prevalence of periodontitis was higher in individuals with diabetes (O

275 ct of physical inactivity induced obesity on periodontitis was higher than the indirect effect of obe

276 Periodontitis was highly prevalent in both stable and un

277 Periodontitis was induced by ligatures around the lower

278 Periodontitis was induced by placing 3.0-silk sutures su

279 Experimental periodontitis was induced for 30 days in wild-type and M

280 stronger association with self-report severe periodontitis was noted when alcohol consumption exceedi

281 A variation in the occurrence of periodontitis was observed.

282 Moderate or severe periodontitis was present in 52.6% of the women.

283 The presence or absence of periodontitis was recorded from each subject with full m

284 important risk association between MetS and periodontitis was reported, being that individuals with

285 ssociation between risk variables, MetS, and periodontitis was tested through univariate analysis and

286 multivariate regression analysis showed that periodontitis was the only significant predictor of plas

287 Data from 35 healthy and 35 individuals with periodontitis were confirmed from the database of our fa

288 f co-used substances, the odds of having any periodontitis were higher among cocaine users who consum

289 Odds of any periodontitis were higher among those reported ancillary

290 2DM subjects with generalized severe chronic periodontitis were recruited along with 20 periodontally

291 lth characteristics, neither WBC markers nor periodontitis were related to hours of sleep.

292 tions, and sex or smoking effects of SNPs on periodontitis were tested using multiple logistic regres

293 Subjects with periodontitis were treated with scaling and root planing

294 ed with susceptibility to moderate or severe periodontitis; whereas the TLR9 marker was associated wi

295 However, the relationship of periodontitis with %neutrophils and %lymphocytes may be

296 ent association comparing generalized severe periodontitis with moderate periodontitis and healthy gr

297 which makes them more useful for studies of periodontitis with the aim of using diagnosis for confir

298 o a breakdown in host-bacterial mutualism in periodontitis, with interbacterial interactions rather t

299 A total of 98.9% of the participants had periodontitis, with stages III and IV found in 26.1% and

300 tus (T2DM) is an established risk factor for periodontitis, yet its contribution to creating host-bac