ライフサイエンスコーパス: peripheral

1 rds: Abdomen/GI, Cardiac, Infection, Nervous-Peripheral.

2 ed analyses of multiple cohorts, we identify peripheral and central adaptive immune changes in Alzhei

3 to reverse EAE by promoting the expansion of peripheral and CNS-infiltrating IL-10(+) T cells.

4 ell RNA-sequencing (scRNA-seq) data from 727 peripheral and nervous system cell types spanning 17 mou

5 its benefit for improving detection of both peripheral and transition zone lesions while reducing ad

6 reas subordinate males are passive, socially peripheral, and have little influence over normal moveme

7 .5%, 22.5%, and 7.5% (P = .003), and shallow peripheral anterior chamber in 65%, 60%, and 17.5% (P =

8 dial infarction, 1.78 (95% CI 1.53-2.07) for peripheral arterial disease, 1.32 (95% CI 1.15-1.50) for

9 Peripheral arterial narrowing is associated with increas

10 o the potential effect of arrhythmias on the peripheral arterial tonometry (PAT) amplitude and pulse

11 creased mortality, as has been suggested for peripheral arteries.

12 Patients with peripheral artery disease (PAD) are at heightened risk f

13 Patients with peripheral artery disease (PAD) have a higher risk of ma

14 Peripheral artery disease (PAD) is underrecognized, unde

15 gerated exercise pressor reflex in rats with peripheral artery disease (PAD).

16 l infarction (MI), ischemic stroke (IS), and peripheral artery disease (PAD).

17 was consistently associated with stroke and peripheral artery disease across the different analyses.

18 Rivaroxaban in peripheral artery disease after revascularization.

19 om classification over time in patients with peripheral artery disease and the association of changes

20 3.05 ([95% CI, 1.92-4.85] P=2.30x10(-6)) for peripheral artery disease in the inverse variance-weight

21 with established coronary artery disease or peripheral artery disease often have diabetes mellitus.

22 Revascularized peripheral artery disease patients face earlier limb and

23 d clinical trial that assigned patients with peripheral artery disease to clopidogrel or ticagrelor.

24 Patients with peripheral artery disease who have undergone lower-extre

25 EUCLID (Examining Use of Ticagrelor in Peripheral Artery Disease) was a randomized clinical tri

26 EUCLID trial (Examining Use of Ticagrelor in Peripheral Artery Disease), we examined the changes in R

27 lure (HF), atrial fibrillation (AF), stroke, peripheral artery disease, cancer, liver-, rheumatic-, a

28 low blood pressure targets in patients with peripheral artery disease.

29 stage of descending control of the mammalian peripheral auditory system through axon projections to t

30 atments have evolved from destruction of the peripheral avascular retina to inhibit angiogenic stimul

31 Methods: Ten patients with a peripheral AVM (mean age, 40 y; 4 men and 6 women) and s

32 rmine if Pou3f4 is normally required for SGN peripheral axon extension into the sensory domain, we us

33 ing and maintaining the myelin sheath around peripheral axons (Grove et al., 2017).

34 st of immature B cells, resulting in reduced peripheral B cell numbers.

35 15), and using mAbs (n = 14) generated from peripheral B cells of two patients.

36 ough CVID is thought to be a disorder of the peripheral B-cell compartment, in 25% of patients, early

37 owed for the first time that the increase in peripheral BACE1 activity is a common feature of LOAD an

38 Moreover, treatment of primary peripheral blood and bone marrow mononuclear cells from

39 information from multiple cell types in the peripheral blood and identifies critical points in the t

40 nuation with near full-length viral DNA from peripheral blood and lymph node mononuclear cells (PBMC

41 tomic analyses of bronchoalveolar lavage and peripheral blood and proteomic analyses of serum.

42 -19 lung disease and highlights the need for peripheral blood biomarkers that inform about patient lu

43 llenge with A. phagocytophilum The bacterial peripheral blood burden was pronouncedly reduced in immu

44 Peripheral blood C-reactive protein (CRP) is a biomarker

45 ading frames (ORFs), and can be found in the peripheral blood CD4(+) T cells of patients at all stage

46 ary endpoint was total HIV DNA isolated from peripheral blood CD4(+) T-cells at weeks 16 and 18 after

47 study animals showed accelerated recovery in peripheral blood cell counts, bone marrow colony forming

48 ts were prospectively enrolled, and repeated peripheral blood collections were performed.

49 provement of asthma symptoms and reduced the peripheral blood eosinophil count to 0/muL.

50 ween IgG4 associated AIH and the presence of peripheral blood eosinophilia.

51 y extensive gene expression perturbations in peripheral blood immune cells.

52 By comparison, peripheral blood interferon gamma release assays in the

53 Peripheral blood is a highly accessible biofluid providi

54 The presence of somatic mutations in the peripheral blood is termed clonal hematopoiesis of indet

55 ere collected for hemogram determination and peripheral blood leukocytes (PBLs) isolation.

56 cluded any observational study comparing the peripheral blood levels of at least one KP metabolite be

57 s showed that individuals with BD have lower peripheral blood levels of tryptophan (SMD = -0.29), kyn

58 omain was sufficient to reduce the bacterial peripheral blood load in mice following challenge and el

59 , we addressed whether antiviral response of peripheral blood lymphocytes differs between HG patients

60 ng predictive epigenomic marks of smokers in peripheral blood may allow for targeted risk stratificat

61 We performed peripheral blood mononuclear cell (PBMC) analysis on a s

62 KCNJ2 K(+) channel expression in peripheral blood mononuclear cell, which strongly correl

63 We performed microarray analyses of peripheral blood mononuclear cells (PBMC) RNA from subje

64 evels were quantified in allergen-stimulated peripheral blood mononuclear cells (PBMCs) and skin punc

65 (NANPs) using a validated preclinical model, peripheral blood mononuclear cells (PBMCs), that is high

66 n 1187 spot-forming cells [SFCs] per million peripheral blood mononuclear cells [IQR 841-2428], n=24;

67 High levels of IL-8 in plasma, peripheral blood mononuclear cells and tumors were assoc

68 inase-mediated phosphorylation events within peripheral blood mononuclear cells collected prior to va

69 olyclonal MBCs at the single-cell level from peripheral blood mononuclear cells collected ~2 weeks or

70 Profiling of ~82,000 single peripheral blood mononuclear cells from adults with SLE

71 eceptor (IL7R) were significantly reduced in peripheral blood mononuclear cells from patients with CO

72 Peripheral blood mononuclear cells from premalignant cas

73 Likewise, their peripheral blood mononuclear cells mounted a negligible

74 eover, TLR2 block prior to DENV infection of peripheral blood mononuclear cells prevents activation o

75 Peripheral blood mononuclear cells were collected from d

76 Peripheral blood mononuclear cells were quantified for T

77 cing robust viral reactivation in plasma and peripheral blood mononuclear cells.

78 tive site geometries in three cell lines and peripheral blood mononuclear cells.

79 The three mature peripheral blood neutrophil subsets arise from distinct

80 to delete CD38 (CD38KO) in ex vivo expanded peripheral blood NK cells.

81 expression was examined in ILC2 sorted from peripheral blood of healthy controls and asthma patients

82 NK cells freshly isolated from peripheral blood of healthy donors were stimulated with

83 HA in MiHApos donors and TAAs are present in peripheral blood of healthy individuals.

84 mphocyte count, monocyte count, and ratio of peripheral blood oxygen saturation to fraction of inspir

85 llograft rejection and treatment resistance, peripheral blood samples and intestinal allograft biopsi

86 a to support its implementation, we analyzed peripheral blood samples from 400 HIV-1(+) adults on ART

87 disease (GVHD), a common complication after peripheral blood stem cell or bone marrow transplantatio

88 Monocytes were isolated from peripheral blood to determine immune functionality, meta

89 on comparing eosinophils isolated from human peripheral blood vs human adipose tissue.

90 ed, placebo-controlled, crossover study, and peripheral blood was collected at baseline, 2, 4, 6, and

91 ansfer of CD115+-ACE10/GFP+ monocytes to the peripheral blood.

92 er the period of active viral replication in peripheral blood.

93 Mutant peripheral-blood cells showed decreased ubiquitylation a

94 es throughout the protein originating in the peripheral C-terminal ligand binding site and culminatin

95 These data provide a link between sensing of peripheral cancer initiation by the bone marrow and hema

96 oupled with metabolic changes due to loss of peripheral catecholamine production.

97 Our data indicate that peripheral cells may be a useful source to measure CAG e

98 euroimaging, and neuropsychology to test how peripheral changes in a key marker for oxidative stress,

99 ained release of platelets directly into the peripheral circulation during both fetal and adult life

100 xpression and proportions between foveal and peripheral cohorts of shared types.

101 most frequent noise level and that auditory peripheral compression, rather than the medial olivococh

102 nd microglial ablation implicate central and peripheral contributions to K2KO-induced metabolic dysfu

103 ehydrogenase-1 (3beta-HSD1), which catalyzes peripheral conversion from adrenal dehydroepiandrosteron

104 Subjects with a peripheral corneal apex had stronger correlations with v

105 valuated the central corneal ECD (CCECD) and peripheral corneal ECD (PCECD) in the area of the tube;

106 ed DSA had significantly lower quantities of peripheral CXCR5IFN-gammaCD8 T cells (P = 0.01) and sign

107 sition within sparse tumors (p = 0.045), and peripheral deposition (p < 0.0001) of Lipiodol showed im

108 s accompanied by an extra core twist, as the peripheral dihedral angle increases from 16.5 degrees in

109 Peripheral distribution of opacities was more common in

110 ipulated gastric rhythm using domperidone, a peripheral dopamine D2/D3 antagonist and common anti-eme

111 + 2 microtubule structure consisting of nine peripheral doublets surrounding a central pair apparatus

112 neurons and macrophages, revealing potential peripheral DRG targets for neuropathic pain management.

113 ng evidence supporting an important role for peripheral dysfunction, particularly within skeletal mus

114 Moreover, central, paracentral, and peripheral En/DMT correlated significantly with graft re

115 We compared the relationships between peripheral endocrine, metabolic, and immune signaling an

116 Peripheral eosinophilia predicted resolution.

117 Here, we report that Drp1 directly shapes peripheral ER tubules in human and mouse cells.

118 skeletal muscle sensory feedback related to peripheral fatigue development are thought to restrict m

119 NF phase locking by highlighting the role of peripheral filtering mechanisms in shaping responses of

120 findings highlight the significant impact of peripheral filtering mechanisms on phase locking.

121 Signals from peripheral glutamate receptors converge onto TRPV1, lead

122 bility of patients to better identify moving peripheral hazardous objects.

123 ere used to determine increased awareness of peripheral hazards while wearing the DSpecs.

124 also found that PRMT5 controlled thymic and peripheral homeostasis in the CD4+ Th cell life cycle an

125 other neurological manifestations of central/peripheral hyperexcitabability including seizures, encep

126 f neuroinflammation, rather than focusing on peripheral immune activation.

127 disease has been associated with changes in peripheral immune activity, including increased levels o

128 Peripheral immune cells, such as monocytes and lymphocyt

129 present study aimed to characterize whether peripheral immune factors measured in blood samples coll

130 Collectively, we provide a cell atlas of the peripheral immune response to severe COVID-19.

131 eased neuronal complexity three months after peripheral immune stimulation is accompanied by impairme

132 In sum, the peripheral immune system can be conceptualized as a dyna

133 holestatic autoimmune liver disease, and the peripheral immune system remains to be fully understood.

134 ance and function of Treg cells in the adult peripheral immune system remains unclear.

135 Identification of this unique peripheral immunophenotype in DLB could guide developmen

136 ve of this study was to provide evidence for peripheral inflammation by examining levels of APPs, and

137 To date there have been no studies examining peripheral inflammation in DLB using multiplex immunoass

138 s initiates a positive feedback loop between peripheral inflammatory cells and networked brain region

139 d imaging, we show preliminary evidence that peripheral injection of alpha-syn fibrils induces unique

140 Our results suggest that peripheral injection of alpha-syn has acute systemic eff

141 rocessing.SIGNIFICANCE STATEMENT Amputation, peripheral injury, and stroke patients experience widesp

142 ss acidosis associated with both central and peripheral innate immune activation.

143 ure in the AL that develops independently of peripheral input, suggesting that the AL is initially pr

144 tes-like phenotypes without markedly causing peripheral insulin resistance.

145 ions and thereby counteracts inflammation of peripheral insulin-sensitive tissues and, thus, obesity-

146 s for realizing viable devices for long-term peripheral interfacing.

147 the angle and a concave configuration of the peripheral iris in both eyes.

148 ly of inflammatory diseases of the spine and peripheral joints.

149 Predominantly peripheral lesions (PPL) and DR severity were graded fro

150 ysis of this large cohort of eyes identified peripheral lesions and intraocular inflammation as disti

151 s from PIC to MFC because of newly developed peripheral lesions.

152 These findings suggest peripheral lipid variation may not have a prominent role

153 The peripheral lung showed reduced ventilation and a greater

154 proximal lung was better ventilated than the peripheral lung.

155 Peripheral lymphatic vessels in patients with HFpEF exhi

156 tted to reduce the coding biases inherent in peripheral mechanics, and wind cues are brought into the

157 One of the primary peripheral mechanisms suggested to underlie exercise int

158 rolling an interaction of the channel with a peripheral membrane-associated Ca(2+)-binding protein, l

159 Although this mechanism contributes to peripheral metabolic dysfunction, effects on the brain r

160 Kynurenic acid (KynA) levels link peripheral metabolic status to neural functions includin

161 cruitment of microvessels of the central and peripheral microcirculation.

162 The results imply that a peripheral modification at the NMJ contributes to the ma

163 Importantly, we found that peripheral monocytes and lymphocytes do not express subs

164 Moreover, after the RMs niche is emptied, peripheral monocytes rapidly differentiate into BMRMs, w

165 (firing rate, variability) and biomarkers of peripheral MU remodelling (MUP size, complexity, stabili

166 ustic qualities that are directly coupled to peripheral musculoskeletal tensioning of the body, such

167 nalogous approaches to peripheral nerves and peripheral myelin have fallen behind evolving technical

168 antitatively show that the helical tetraspan peripheral myelin protein 22 (PMP22) exhibits a pronounc

169 In rodent models overexpressing the PMP22 (peripheral myelin protein 22) protein and in dermal fibr

170 sease development, however, its functions in peripheral myelination and myelin repair remain elusive.

171 r2, the central transcriptional regulator of peripheral myelination, to its target genes.

172 Further, we show that microglia, not peripheral myeloid cells, release IL-1alpha ex vivo.

173 I3Kdelta in Treg cells developed spontaneous peripheral nerve inflammation.

174 Neuropathic pain caused by peripheral nerve injuries significantly affects sensory

175 animals with spared nerve injury, a model of peripheral nerve injury (PNI)-induced neuropathic pain.

176 as a comprehensive, in-depth perspective on peripheral nerve regeneration, particularly nerve guidan

177 coma (LMS), synovial sarcoma (SS), malignant peripheral nerve sheath tumor (MPNST), and undifferentia

178 as a central oncogenic driver for malignant peripheral nerve sheath tumors (MPNSTs), which are highl

179 ub-populations of sensory neurons, and their peripheral nerve terminal endings, interact with bone.

180 Peripheral nerve transection is associated with permanen

181 Recent studies have shown that peripheral nerves (sympathetic, parasympathetic and sens

182 motor capabilities, analogous approaches to peripheral nerves and peripheral myelin have fallen behi

183 r, these studies highlight the importance of peripheral nerves as a new therapeutic target for metabo

184 tail tip to the origin of the most posterior peripheral nerves from the dorsal nerve cord.

185 Imaging studies of peripheral nerves have increased considerably in the las

186 n this review, we summarize the functions of peripheral nerves in glucose regulation and metabolism.

187 As peripheral nerves ramify within the dermis, all the nucl

188 Better understanding myelination of peripheral nerves would benefit patients affected by per

189 of a spectrum of autoimmune diseases of the peripheral nerves, causing weakness and sensory symptoms

190 al depigmentation and absence of myelin from peripheral nerves, resulting from alterations in melanoc

191 t target muscle, the neuromuscular junction, peripheral nerves, the spinal cord or the brain and disc

192 e in Nox4 expression and activity in SCs and peripheral nerves, which were further verified in skin b

193 of psychosine results in diffuse central and peripheral nervous system (CNS, PNS) demyelination.

194 nisms of how cholesterol/lipid metabolism in peripheral nervous system (PNS) contributes to the patho

195 tent and -incompetent sensory neurons in the peripheral nervous system but also allowed temporal tuni

196 wing somatosensory neurons in the Drosophila peripheral nervous system exhibit organ sparing at the l

197 Despite the relevance of the peripheral nervous system for normal sensory and motor c

198 affect multiple organs, with the central and peripheral nervous system often affected.

199 Cholinergic inputs originating from the peripheral nervous system regulate the inflammatory immu

200 e-Tooth disease (CMT) is a neuropathy of the peripheral nervous system that afflicts ~1:2500 people.

201 wann cells, the principal glial cells of the peripheral nervous system, are now considered to be impo

202 d widespread gene expression in, central and peripheral nervous system, liver, kidney and skeletal mu

203 egulate synaptic activity in the central and peripheral nervous system, presynaptically and postsynap

204 k between macrophages and nociceptors in the peripheral nervous system, which may contribute to the s

205 y inside the tissues of both the central and peripheral nervous systems was normalized, and the neuro

206 their proper targets in both the central and peripheral nervous systems.

207 nificant gadolinium retention in central and peripheral nervous tissues (1.8-333.2 nmol Gd/g tissue).

208 that male mosquitoes possess the central or peripheral neural circuits required to host-seek and tha

209 ary thromboembolism), one of whom died; (iv) peripheral neurological disorders (n = 8), seven with Gu

210 e brain, nAChRs are also highly expressed in peripheral neurons, epithelia and immune cells, where th

211 r some types of colorectal carcinoma, causes peripheral neuropathic pain in patients.

212 chwann cells or their myelin sheaths lead to peripheral neuropathies such as Charcot-Marie-Tooth dise

213 al nerves would benefit patients affected by peripheral neuropathies, including Charcot-Marie-Tooth d

214 complement-mediated myopathies, myasthenia, peripheral neuropathies, neuromyelitis and other CNS dis

215 Diabetic Peripheral Neuropathy (DPN) typically is accompanied by

216 Growing evidence indicates that peripheral neuropathy (PN) is common even in the absence

217 inal findings identify novel contributors to peripheral neuropathy and emphasize the fundamental depe

218 Paclitaxel induces peripheral neuropathy as a side effect of cancer treatme

219 Peripheral neuropathy is a debilitating and painful side

220 Peripheral neuropathy of grade 2 or above was less frequ

221 ogic disorders known as chemotherapy-induced peripheral neuropathy, mechanistic understanding and tre

222 nduced high response rates with low rates of peripheral neuropathy, the main dose-limiting toxicity o

223 ant dose-limiting neurotoxicity resulting in peripheral neuropathy.

224 ld be developed to prevent cisplatin induced peripheral neuropathy.

225 In contrast to studies with peripheral NKT cells, the proliferation of thymic NKT ce

226 These novel spectacles may enhance peripheral objects awareness by enlarging the functional

227 Patients' scores in identifying and counting peripheral objects with the remapped images were the mai

228 rved between RA and measures of co-localised peripheral ocular length.

229 tributing effects of low SMN in one relevant peripheral organ - skeletal muscle - by selectively depl

230 The sympathetic nervous system innervates peripheral organs to regulate their function and maintai

231 ue features of CNS vasculature absent in the peripheral organs.

232 ration of viremia and viral dissemination to peripheral organs.

233 To examine the role of peripheral ORs in triggering brain hypoxia, we used oxyg

234 ipheral polysynovitis in RA versus axial and peripheral osteoproliferative inflammation in SpA.

235 model resolves a sensory ambiguity where the peripheral otolith organs in the inner ear sense both he

236 We found that peripheral OXTR ligand binding in the head is mostly int

237 outcome was severe desaturation (defined as peripheral oxygen saturation reading < 80% during intuba

238 stress response are associated with reduced peripheral parasympathetic activity during negative emot

239 Mounting evidence has demonstrated that peripheral pathological conditions induce physiological

240 to the guest packing in and the shape of the peripheral pockets, to afford large changes in the globa

241 a one-dimensional channel connected to three peripheral pockets.

242 erent disease phenotypes such as destructive peripheral polysynovitis in RA versus axial and peripher

243 We show that a peripheral portion of the collar is diminished in the De

244 We found that p18/LAMTOR1-mediated peripheral positioning of late endosomes allows delivery

245 lass of possible interactions that result in peripheral positioning to include boundary interactions

246 ere associated with cortical regions showing peripheral preference (e.g. parahippocampal cortex).

247 oup of MIA offspring that displayed elevated peripheral production of innate inflammatory cytokines,

248 weaning, significantly suppressed postnatal peripheral proinflammatory insult-induced systemic infla

249 hat membrane curvature qualitatively affects peripheral protein-lipid interactions and also highlight

250 CC6803 to elucidate the action of Ca(2+) and peripheral proteins.

251 venting RAB13 RNA localization, we show that peripheral RAB13 translation is not important for the ov

252 eased serum interleukin (IL)-6 and IL-2, but peripheral regulatory T cell (Treg) numbers did not incr

253 24 h after successful extinction, outlasting peripheral responding.

254 y found in the posterior pole and around the peripheral retina in AHT.

255 riptomes of ~85,000 cells from the fovea and peripheral retina of seven adult human donors.

256 line, consistent with largely nonprogressive peripheral retinal dysfunction.

257 erval: 1.28 to 1.40) and major amputation or peripheral revascularization (hazard ratio: 8.13; 95% co

258 This study sought to describe outcomes after peripheral revascularization and assess relationships be

259 r adverse cardiovascular events, while prior peripheral revascularization or amputation is associated

260 packed conformation, with ordered inner and peripheral rings, that prevents dimerization and activat

261 Regulatory T cells (T(regs)) maintain peripheral self-tolerance and limit immune mediated path

262 are essential for providing and maintaining peripheral self-tolerance of potentially autoreactive ce

263 Peripheral sensory neurons regenerate their axon after n

264 tigue, nausea, diarrhea, anorexia, vomiting, peripheral sensory neuropathy, and keratitis/keratopathy

265 context of electrophysiological findings of peripheral sensory polyneuropathy.

266 (Tph1), the rate limiting enzyme regulating peripheral serotonin synthesis.

267 We are able to identify concordant peripheral signatures associated with prevalent AD arisi

268 s mediate immunosurveillance through diverse peripheral sites.

269 beginning to understand the diversity of the peripheral T cell compartment, which arises from the spe

270 cell tolerance by promoting antigen-induced peripheral T cell deletion.

271 tains a proportional reserve of NF-kappaB in peripheral T cells poised for responding to agonistic an

272 e report that thymic involution, declines in peripheral T-cell counts, and reduced major histocompati

273 ally for the broader category of the disease-peripheral T-cell lymphoma-let alone any of the specific

274 a number of T-cell diseases, including some peripheral T-cell lymphomas, hemophagocytic lymphohistio

275 , DRG organoid sensory neurons contact their peripheral targets and reconstitute the muscle spindle p

276 y 3DVUS, and 11.5% by CACS), particularly in peripheral territories examined by vascular ultrasound.

277 ecular interactions, which exist between the peripheral thiophene of the IDT unit and the benzothiadi

278 ggered by alloreactive T cells, which damage peripheral tissues and lymphoid organs.

279 ave identified altered metabolic profiles in peripheral tissues associated with MDD.

280 d lipolysis, increases lipid accumulation in peripheral tissues, and exacerbates tissue-specific and

281 Notably, T reg cells in peripheral tissues, including tumors, are more sensitive

282 However, within the germinal center, peripheral tolerance was still enforced, and there was s

283 ond to defined neoantigens in the context of peripheral tolerance, transplantation, autoimmune diseas

284 Our data show that peripheral treatment using TCMCB07 with intraperitoneal,

285 mediated conversion in the periphery (called peripheral Treg [pTreg] cells).

286 but its use has not been reported in case of peripheral ulcerative keratitis (PUK).

287 r dual antiplatelet therapy (DAPT) following peripheral vascular intervention (PVI), there are limite

288 UWFFA; 13 eyes demonstrated the presence of peripheral vascular leakage, 5 eyes showed optic disc le

289 s induces perturbations in the heart and the peripheral vasculature that differ in physiology and cli

290 beled antibodies injected intravenously in a peripheral vessel in the left arm.

291 d intravenously with unlabeled antibody in a peripheral vessel in the right arm (10 mg/kg, providing

292 Monocular peripheral VF defects were measured and defined with a h

293 esults therefore highlight the importance of peripheral vision during trans-saccadic processing in bu

294 unction, photopic peripheral vision, mesopic peripheral vision, and photosensitivity.

295 owhide" stimuli were presented 15 degrees in peripheral vision, either in isolation or surrounded by

296 ast sensitivity, scotopic function, photopic peripheral vision, mesopic peripheral vision, and photos

297 al vision, whereas resolution is poor in our peripheral visual field, and this loss of resolution fol

298 d covert spatial selection are influenced by peripheral visual forms.

299 rofiles of protein-coding genes expressed in peripheral white blood cells (PWBCs), and circulating mi

300 mised, such that they resemble extrastriolar/peripheral zones in multiple features.