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1 rization, angina, stroke, heart failure, and peripheral arterial disease).
2 riables (C) (smoking, diabetes mellitus, and peripheral arterial disease).
3 tection of disease and monitoring therapy in peripheral arterial disease.
4 ients with stable coronary artery disease or peripheral arterial disease.
5 tients, women, and patients with diabetes or peripheral arterial disease.
6 ld yield the most efficacious treatments for peripheral arterial disease.
7 t-reported walking performance developed for peripheral arterial disease.
8 t disease (CHD), cerebrovascular disease and peripheral arterial disease.
9 in ischemia-induced angiogenesis, such as in peripheral arterial disease.
10 bility could be extended to patients without peripheral arterial disease.
11 ther the WIQ can be used in patients without peripheral arterial disease.
12 ith diabetes, tobacco use, heart failure, or peripheral arterial disease.
13 lar morbidity and mortality in patients with peripheral arterial disease.
14 ent-resistant hypertension, preeclampsia, or peripheral arterial disease.
15 oke, nephropathy, ischemic heart disease and peripheral arterial disease.
16 t limb salvage rates in patients with severe peripheral arterial disease.
17 ial femoral artery of diabetic patients with peripheral arterial disease.
18 tudied and validated therapy for symptomatic peripheral arterial disease.
19 art disease, coronary revascularisation, and peripheral arterial disease.
20 ative to PTA for treatment of infrapopliteal peripheral arterial disease.
21 ry heart disease, heart failure, stroke, and peripheral arterial disease.
22 heart disease, cerebrovascular disease, and peripheral arterial disease.
23 schaemic heart disease, ischaemic stroke, or peripheral arterial disease.
24 with less functional decline in persons with peripheral arterial disease.
25 th less functional decline among people with peripheral arterial disease.
26 ations for clinical imaging in patients with peripheral arterial disease.
27 aemorrhage, carotid dissection, and, rarely, peripheral arterial disease.
28 onal decline in persons with lower-extremity peripheral arterial disease.
29 ans taking care of patients with symptomatic peripheral arterial disease.
30 , and depression worsens vascular outcome in peripheral arterial disease.
31 ponses are impaired in elderly patients with peripheral arterial disease.
32 utive patients underwent PER for symptomatic peripheral arterial disease.
33 schemia, such as coronary artery disease and peripheral arterial disease.
34 ed as coronary heart disease, stroke, and/or peripheral arterial disease.
35 CVD included CHD, stroke, heart failure, and peripheral arterial disease.
36 diseases such as coronary artery disease and peripheral arterial disease.
37 ) is a widely utilized measure for detecting peripheral arterial disease.
38 o 2.69 for stroke, and from 1.66 to 4.28 for peripheral arterial disease.
39 ovascular and surgical revascularization for peripheral arterial disease.
40 o 1.53 for stroke, and from 0.61 to 1.58 for peripheral arterial disease.
41 chemia is a life-threatening complication of peripheral arterial disease.
42 interventions in patients with asymptomatic peripheral arterial disease.
43 f progression of kidney disease, stroke, and peripheral arterial disease.
44 ischemic heart disease, ischemic stroke, and peripheral arterial disease.
45 patients with chronic coronary syndromes or peripheral arterial disease.
46 al limb ischemia represents the end stage of peripheral arterial disease.
47 reperfuse the limbs of certain patients with peripheral arterial disease.
48 s (46% of possible matches), 95% of whom had peripheral arterial disease.
49 s in patients with stable coronary artery or peripheral arterial disease.
50 ipheral blood flow in selected patients with peripheral arterial diseases.
52 dial infarction, 1.78 (95% CI 1.53-2.07) for peripheral arterial disease, 1.32 (95% CI 1.15-1.50) for
53 presentation, of which the most common were peripheral arterial disease (16.2%, n=992) and heart fai
54 e patients with stent occlusion or stenoses, peripheral arterial disease (ABI <1.0), symptomatic card
55 etes was strongly positively associated with peripheral arterial disease (adjusted cause-specific haz
56 pe 2 diabetes was positively associated with peripheral arterial disease (adjusted HR 2.98 [95% CI 2.
57 ovascular disease mortality and one study on peripheral arterial disease).All but one study reported
58 ion of serum selenium with the prevalence of peripheral arterial disease among 2,062 US men and women
62 ng is critical for blood flow restoration in peripheral arterial disease and is triggered by increasi
63 Ischemia (CLI) is the most advanced stage of peripheral arterial disease and is usually treated with
65 ncreased in a range of conditions, including peripheral arterial disease and myocardial infarction, w
66 in the response to ischemia associated with peripheral arterial disease and myocardial infarction.
67 l arterial disease, the use of paclitaxel in peripheral arterial disease and other conditions, the ha
68 er peripheral arterial revascularization for peripheral arterial disease and to assess whether readmi
69 VD, defined as coronary, cerebrovascular, or peripheral arterial disease, and (2) incident heart fail
70 0.722 for ADA HbA1c clinical categories for peripheral arterial disease, and 0.683 for ADA fasting g
71 l duration, the absence of beta-blocker use, peripheral arterial disease, and a deeper prosthesis ins
72 rt failure with preserved ejection fraction, peripheral arterial disease, and abdominal aortic aneury
73 nary heart disease, cerebrovascular disease, peripheral arterial disease, and all-cause cardiovascula
74 onic kidney disease, cardiovascular disease, peripheral arterial disease, and all-cause mortality tha
75 are performed in patients with diabetes and peripheral arterial disease, and at least 25% require su
76 r disease, including coronary heart disease, peripheral arterial disease, and cerebrovascular disease
78 CAC] scores, carotid intima-media thickness, peripheral arterial disease, and pulse wave velocity).
80 eria included diabetes mellitus, symptomatic peripheral arterial disease, and superficial femoral art
81 hypertension, previous heart failure, MI, or peripheral arterial disease; and who received coronary a
82 patients more often had a history of stroke, peripheral arterial disease, angina, heart failure, diab
83 eart failure after myocardial infarction and peripheral arterial disease are predominant, devastating
85 options for the treatment of lower extremity peripheral arterial disease are typified with diminished
87 ongly associated with coronary, cerebral and peripheral arterial disease, as well as with microangiop
89 roke, coronary heart disease, heart failure, peripheral arterial disease, asthma, chronic kidney dise
90 roke, coronary heart disease, heart failure, peripheral arterial disease, asthma, chronic kidney dise
91 erosclerosis (ie, risk factors, coronary and peripheral arterial disease, asymptomatic carotid stenos
92 r the treatment of patients with symptomatic peripheral arterial disease because they are associated
94 rization, angina, stroke, heart failure, and peripheral arterial disease), but less effective in prev
95 e and blood flow in an experimental model of peripheral arterial disease, by exploiting fluorescence
98 ction, pre-procedural myocardial infarction, peripheral arterial disease, congestive heart disease, r
99 ary artery disease, cerebrovascular disease, peripheral arterial disease, congestive heart failure, m
101 y of autologous cell therapy for intractable peripheral arterial disease/critical limb ischemia.
102 age-, sex-, and race-adjusted prevalence of peripheral arterial disease decreased with increasing se
104 r, rheumatic heart disease, aortic aneurysm, peripheral arterial disease, endocarditis, and all other
106 monary disease, acute myocardial infarction, peripheral arterial disease, epilepsy, substance abuse,
107 ace, renal disease, diabetes mellitus, known peripheral arterial disease, evaluation by a vascular sp
109 1 years) and 13 patients suspected of having peripheral arterial disease (five men; mean age, 67 year
110 vascular disease (coronary artery disease or peripheral arterial disease) followed from 1997 to 2009,
111 Isner, the field of cell-based therapies for peripheral arterial disease has been in a state of conti
112 shed CV disease, including HTN, HF, CHD, and peripheral arterial disease, have a better prognosis com
113 1.5-2.0) for stable angina, ischemic stroke, peripheral arterial disease, heart failure, and cardiac
114 risk factors include advanced age, smoking, peripheral arterial disease, high blood pressure, corona
115 tio [HR], 0.77; 95% CI, 0.62-0.94; P = .01), peripheral arterial disease (HR, 0.65; 95% CI, 0.49-0.87
117 hiectomy was noted for fractures (HR, 1.80), peripheral arterial disease (HR, 2.25), venous thromboem
118 95% confidence interval [CI]: 1.82 to 2.29), peripheral arterial disease (HR: 1.95; 95% CI: 1.72 to 2
120 pulmonary disease, coronary artery disease, peripheral arterial disease, hypertension, pulmonary hyp
121 ischemia may contribute to worse outcomes of peripheral arterial disease in patients with diabetes me
124 open AAA repair, bypass for lower extremity peripheral arterial disease - in Ontario, Canada, betwee
129 hospitalization, cardiac revascularization, peripheral arterial disease intervention, or cardiovascu
139 self as acute coronary syndrome, stroke, and peripheral arterial diseases, is a chronic progressive i
141 cerebrovascular accident, heart failure, and peripheral arterial disease), kidney disease (a composit
147 pes) (effective sample size [n(e)] = 9,396); peripheral arterial disease (n(e) = 5,215); abdominal ao
148 40,258; cerebrovascular disease, n = 18,843; peripheral arterial disease, n = 8273) or 3 or more risk
150 ferumoxytol-enhanced MR angiography revealed peripheral arterial disease not recognized with duplex U
151 ce interval [CI], 0.31-0.45; P=5.5*10(-26)], peripheral arterial disease (odds ratio 0.42; 95% CI, 0.
152 ression analysis revealed that patients with peripheral arterial disease of the lower extremities are
154 artery disease, cerebrovascular disease, or peripheral arterial disease or with multiple risk factor
155 hrombotic (coronary, cerebrovascular, and/or peripheral arterial) disease or multiple atherothromboti
156 pertension (OR, 0.58; 95% CI, 0.43-0.78) and peripheral arterial disease (OR, 0.43; 95% CI, 0.22-0.85
157 a-blocker use (OR, 0.12; 95% CI, 0.03-0.45), peripheral arterial disease (OR, 6.36; 95% CI, 1.56-25.8
158 ificant associations between the CAD-GRS and peripheral arterial disease (OR: 1.28; 95% CI: 1.23 to 1
160 option for patients with renal dysfunction, peripheral arterial disease, or following a brief P2Y12-
164 diabetes (P<0.0001), hypertension (P<0.001), peripheral arterial disease (P=0.0002), smoking (P<0.000
165 ves (adjusted chi-square=38.3, p <0.001) and peripheral arterial disease (PAD) (adjusted chi-square=1
167 ardiovascular disease (CVD) risk factors and peripheral arterial disease (PAD) after adjustment for t
168 risk factors explain the high prevalence of peripheral arterial disease (PAD) among African American
169 2 diabetes respond poorly to treatments for peripheral arterial disease (PAD) and are more likely to
170 lthough exertional leg pain is a hallmark of peripheral arterial disease (PAD) and can occur in perso
171 els, inorganic arsenic exposure is linked to peripheral arterial disease (PAD) and cardiovascular dis
172 eadmill walking performance in patients with peripheral arterial disease (PAD) and intermittent claud
173 o determine differences in the prevalence of peripheral arterial disease (PAD) and its associations w
174 ent study were to describe the prevalence of peripheral arterial disease (PAD) and its effects on pro
177 rs that regulate angiogenesis are altered in peripheral arterial disease (PAD) and whether these fact
178 m a vascular cohort with a history of either peripheral arterial disease (PAD) and/or other cardiovas
182 aimed to investigate the pathophysiology of peripheral arterial disease (PAD) by examining magnetic
185 HF), myocardial infarction (MI), stroke, and peripheral arterial disease (PAD) during the subsequent
191 vious studies have indicated higher rates of peripheral arterial disease (PAD) in blacks than in non-
194 Homocysteine levels are associated with peripheral arterial disease (PAD) in observational studi
195 index (BMI; weight (kg)/height (m)(2)) with peripheral arterial disease (PAD) in prior studies may r
196 scular reactivity and functional capacity in peripheral arterial disease (PAD) in small, short-term s
197 europathy, chronic kidney disease (CKD), and peripheral arterial disease (PAD) in the general populat
211 le of inflammation in the pathophysiology of peripheral arterial disease (PAD) is well established, t
212 ides an advantageous approach to symptomatic peripheral arterial disease (PAD) over the longer term.
213 in patients with and without lower-extremity peripheral arterial disease (PAD) over three-year follow
214 etics plays an important role in determining peripheral arterial disease (PAD) pathology, which cause
215 I] 1.0 +/- 0.1, mean age 30 +/- 7 years); 2) peripheral arterial disease (PAD) patients (n = 12; mean
216 gnetic resonance spectroscopy in symptomatic peripheral arterial disease (PAD) patients compared with
217 in gastrocnemius biopsies from patients with peripheral arterial disease (PAD) predict mortality rate
219 rmining the amputation risk in patients with peripheral arterial disease (PAD) remains difficult.
220 STF) recently released an update to its 1996 Peripheral Arterial Disease (PAD) Screening Recommendati
221 CKD) substantially increases the severity of peripheral arterial disease (PAD) symptomology, however,
223 blood and urine cadmium concentrations with peripheral arterial disease (PAD) were evaluated by usin
224 rmined whether patients with lower-extremity peripheral arterial disease (PAD) who are more physicall
225 the association of progressive versus stable peripheral arterial disease (PAD) with the risk of futur
226 cise nor strength training for patients with peripheral arterial disease (PAD) without intermittent c
227 hypothesized that women with lower extremity peripheral arterial disease (PAD) would have greater mob
228 ABI categories were defined: <0.90 (definite peripheral arterial disease (PAD)), 0.90-0.99 (borderlin
229 l nervous system, but their association with peripheral arterial disease (PAD), a high-prevalence vas
230 CRP (P< or =0.05) and increasing severity of peripheral arterial disease (PAD), after adjustment for
231 sessed plasma-based factors as predictors of peripheral arterial disease (PAD), and comparative data
233 Several risk factors for atherosclerotic peripheral arterial disease (PAD), such as dyslipidemia,
234 the management of patients with established peripheral arterial disease (PAD), the prevalence of PAD
236 R angiography for suspected or known chronic peripheral arterial disease (PAD), with contrast materia
253 promote physiological angiogenesis to treat peripheral arterial diseases (PAD) by increasing the vas
255 ETHODS AND The study population included 203 peripheral arterial disease participants who underwent v
257 2 improves treadmill exercise performance in peripheral arterial disease patients with claudication-l
258 rticularly important for the large number of peripheral arterial disease persons without access to su
259 oronary artery disease, limb ischemia due to peripheral arterial disease, pressure-overload heart fai
262 orbidities including hypertension, diabetes, peripheral arterial disease, previous myocardial infarct
263 e, diabetes mellitus, hypertension, smoking, peripheral arterial disease, previous stroke, previous c
264 s without clinical CVD but with asymptomatic peripheral arterial disease, regardless of its low CVD r
265 3 US Medicare patients who underwent a major peripheral arterial disease-related amputation during th
267 ulmonary disease, calcified ascending aorta, peripheral arterial disease, renal failure, and previous
268 presentation, the most common of which were peripheral arterial disease (reported in 992 [16.2%] of
271 myocardium, atrial fibrillation, aortic and peripheral arterial disease, rheumatic heart disease, an
272 at interventions to prevent mobility loss in peripheral arterial disease should focus on reversing pa
273 roke, chronic obstructive pulmonary disease, peripheral arterial disease, smoking, diabetes, renal fa
274 pecificity, 96%; LR, 3.1 [95% CI, 2.0-4.7]), peripheral arterial disease (specificity, 97%; LR, 2.7 [
275 usion in "no-option" patients suffering from peripheral arterial diseases, such as critical limb isch
276 tients with prior coronary artery disease or peripheral arterial disease the COMPASS (Cardiovascular
277 ional, and surgical therapy for coronary and peripheral arterial disease, the burden of these illness
278 eaders Forum reviewed the natural history of peripheral arterial disease, the use of paclitaxel in pe
279 are recommended for secondary prevention in peripheral arterial disease, their effectiveness in CLI
280 a risk equivalent condition for coronary and peripheral arterial diseases, these findings create a pa
281 ion procedures is increased in patients with peripheral arterial disease, thus increasing the inciden
282 f myocardial infarction, ischemic stroke, or peripheral arterial disease to receive vorapaxar (2.5 mg
283 AND Three hundred seventy participants with peripheral arterial disease underwent baseline measureme
288 serum selenium levels and the prevalence of peripheral arterial disease was not statistically signif
289 icenter study, 141 patients with symptomatic peripheral arterial disease were assigned to treatment w
290 na, recent coronary artery bypass graft, and peripheral arterial disease were associated with prescri
291 , chronic obstructive pulmonary disease, and peripheral arterial disease were less likely to improve
292 ntrolled trial, 76 patients with symptomatic peripheral arterial disease were randomized 2:1 to recei
293 left ventricular ejection fraction <20%, and peripheral arterial disease were significant predictors
294 Treatment options for diabetic patients with peripheral arterial disease when revascularization is no
295 effect on angina, myocardial infarction, and peripheral arterial disease, whereas raised diastolic bl
296 igger of chronic coronary, intracranial, and peripheral arterial diseases, which together account for
297 unweighted hospitalizations of patients with peripheral arterial disease who had peripheral arterial
299 andomized trial in symptomatic patients with peripheral arterial disease who underwent endovascular t
300 ong 61 969 hospitalizations of patients with peripheral arterial disease who were discharged alive af