ライフサイエンスコーパス: peripheral blood

1 anced egress of FA HSPCs from bone marrow to peripheral blood.

2 ell lines with immune components, often from peripheral blood.

3 relying instead on eosinophils isolated from peripheral blood.

4 tors in tumours, normal adjacent tissue, and peripheral blood.

5 ects >= 1 billion people worldwide, in human peripheral blood.

6 LC3s, and to a lesser extent ILC2s, in their peripheral blood.

7 tor cell colony-forming unit mobilization in peripheral blood.

8 er the period of active viral replication in peripheral blood.

9 le genetic loci from pure cell-free DNA from peripheral blood.

10 us resulting in reduced lymphocyte counts in peripheral blood.

11 eic acids and circulating tumor cells in the peripheral blood.

12 g the APOL1 protein, from DNA extracted from peripheral blood.

13 ansfer of CD115+-ACE10/GFP+ monocytes to the peripheral blood.

14 uccessful in more accessible samples such as peripheral blood.

15 t abundant TCR gammadelta cell population in peripheral blood.

16 also responsive to an acute GC challenge in peripheral blood.

17 he numbers of CD4(+) and CD8(+) cells in the peripheral blood.

18 In peripheral blood, a CCR6(+)T cell population with simila

19 1 macrophage differentiation, programs human peripheral blood adherent cells to form LGCs.

20 Edited T cells were detectable in peripheral blood after infusion.

21 negative, low (<200 copies per 105ABL in the peripheral blood and <1000 copies in the bone marrow asp

22 ar mtDNA content in PBMCs was higher than in peripheral blood and a surprisingly high level of cf mtD

23 T cells isolated from human peripheral blood and activated in culture using tetramer

24 To study this, matched peripheral blood and ascites fluid were collected from 3

25 ts with lung granulomatous diseases.Methods: Peripheral blood and BAL cells from patients with sarcoi

26 family expression is investigated in primary peripheral blood and bone marrow mononuclear cells from

27 Moreover, treatment of primary peripheral blood and bone marrow mononuclear cells from

28 CD4(+) T cells were isolated from the peripheral blood and dynamic adhesion to recombinant muc

29 n D5 (RvD5n-3 DPA) is diurnally regulated in peripheral blood and exerts tissue-protective actions du

30 derived from the brain can be isolated from peripheral blood and have been reported to contain highe

31 information from multiple cell types in the peripheral blood and identifies critical points in the t

32 nuation with near full-length viral DNA from peripheral blood and lymph node mononuclear cells (PBMC

33 g for 0.5% to 5% of all T lymphocytes in the peripheral blood and lymphoid tissues in mice and humans

34 ent succeed in eradicating HIV reservoirs in peripheral blood and lymphoid tissues, residual sources

35 xcess body mass index (BMI) might share both peripheral blood and placental gene signatures that link

36 nnectivity and betweenness centrality in the peripheral blood and placental modules.

37 tomic analyses of bronchoalveolar lavage and peripheral blood and proteomic analyses of serum.

38 as well as with monocyte activation in both peripheral blood and the draining axillary lymph node, i

39 uding male sex, history of hypertension, low peripheral blood, and elevated bronchoalveolar lavage ly

40 veral tissues including spleen, bone barrow, peripheral blood, and lung, in line with the diverse nat

41 ours) and serum cytokines, serum antibodies, peripheral blood antigen-specific T lymphocytes, and gen

42 CD14/CD16 characterized monocyte subsets in peripheral blood as well as their PD-L1 expression and c

43 demonstrate the origination of MA-TAMs from peripheral blood, as well as their potential association

44 and RT-PCR were performed in highly purified peripheral blood basophils and eosinophils of atopic and

45 -19 lung disease and highlights the need for peripheral blood biomarkers that inform about patient lu

46 llenge with A. phagocytophilum The bacterial peripheral blood burden was pronouncedly reduced in immu

47 Peripheral blood C-reactive protein (CRP) is a biomarker

48 ngitudinal accumulation of sTREM2 and higher peripheral blood C3 expression.

49 e of UROS-deficient erythroid cell lines and peripheral blood CD34+-derived erythroid cultures from a

50 cantly increased numbers of IL-17A-producing peripheral blood CD4(+) T cells compared to PBC patients

51 ading frames (ORFs), and can be found in the peripheral blood CD4(+) T cells of patients at all stage

52 The expression of alpha4beta1 integrin on peripheral blood CD4(+) T cells was quantified by flow c

53 ary endpoint was total HIV DNA isolated from peripheral blood CD4(+) T-cells at weeks 16 and 18 after

54 cantly increased numbers of IL-17A-producing peripheral blood CD4+ T cells compared to PBC patients a

55 dies, we prospectively and serially analyzed peripheral blood CD8 and CD4 T-cell subsets and monitore

56 study animals showed accelerated recovery in peripheral blood cell counts, bone marrow colony forming

57 Moreover, while changes in peripheral blood cells are becoming increasingly underst

58 d assessed sphingolipid de novo synthesis in peripheral blood cells by measuring the incorporation of

59 he presence of CMV and high-level EBV DNA in peripheral blood cells was associated with changes in HI

60 ased C9orf72 protein expression in brain and peripheral blood cells(4-6).

61 THC) is known to modulate immune response in peripheral blood cells.

62 Mutant peripheral-blood cells showed decreased ubiquitylation a

63 ts were prospectively enrolled, and repeated peripheral blood collections were performed.

64 utcompeted wild type cells and dominated the peripheral blood compartment over time.

65 in oligodendrocytes, and the OEMVs found in peripheral blood could be further explored for their pot

66 Furthermore, increased peripheral blood CRP in MDD has been associated with alt

67 ccelerated recovery from irradiation-induced peripheral blood cytopenia, bone marrow damage as well a

68 ct of CnCda4 deacetylation products on human peripheral blood-derived macrophages, leading to a model

69 Compared with unmodified iNK cells and peripheral blood-derived NK (PB-NK) cells, hnCD16-iNK ce

70 effect of RAP upon cardiovascular responses, peripheral blood displacement (i.e. central hypovolaemia

71 nes and then on circulating EV obtained from peripheral blood drawn from patients with IPMNs.

72 nistration, the asthma symptoms worsened and peripheral blood eosinophil count increased to 813/muL.

73 provement of asthma symptoms and reduced the peripheral blood eosinophil count to 0/muL.

74 A 65-year-old man with jaundice and peripheral blood eosinophilia.

75 ween IgG4 associated AIH and the presence of peripheral blood eosinophilia.

76 we identified subjects who demonstrated low peripheral blood eosinophils accompanied by increased ex

77 iveness, Ca(2+) -flux and apoptosis of human peripheral blood eosinophils were assessed in vitro.

78 irculating tumor cells (CTCs) from patients' peripheral blood facilitates on-demand monitoring of tum

79 e 11.6% [95% CI = 8.4-14.8; P < .0001]), and peripheral blood flow (mean increase 19.7% [95% CI = 10.

80 art rate, stroke volume, blood pressure, and peripheral blood flow) and electrocardiogram findings du

81 series gene expression profiles measured in peripheral blood from a group of subjects with respirato

82 -PCR, and Western blot, were performed using peripheral blood from healthy donors and patients with I

83 cells endogenously expressing high PD-L1 in peripheral blood from patients with head and neck cancer

84 Peripheral blood immune cell and plasma markers were ana

85 y extensive gene expression perturbations in peripheral blood immune cells.

86 Toward this end, we have identified a core peripheral blood immune signature across 63 hospital-tre

87 neration sequencing (NGS) of bone marrow and peripheral blood increasingly guides clinical care in he

88 By comparison, peripheral blood interferon gamma release assays in the

89 Peripheral blood involvement by cutaneous T-cell lymphom

90 Peripheral blood is a highly accessible biofluid providi

91 The presence of somatic mutations in the peripheral blood is termed clonal hematopoiesis of indet

92 idase inhibition, and stalk antibody titers; peripheral blood leukocyte host gene expression response

93 ere collected for hemogram determination and peripheral blood leukocytes (PBLs) isolation.

94 e had previously analysed gene expression in peripheral blood leukocytes for a subset of individuals

95 cluded any observational study comparing the peripheral blood levels of at least one KP metabolite be

96 s showed that individuals with BD have lower peripheral blood levels of tryptophan (SMD = -0.29), kyn

97 omain was sufficient to reduce the bacterial peripheral blood load in mice following challenge and el

98 erum immunoglobulins and complement factors, peripheral blood lymphocyte subpopulations, and whole bl

99 , we addressed whether antiviral response of peripheral blood lymphocytes differs between HG patients

100 ed significant with <0.05% memory B cells in peripheral blood lymphocytes.

101 nflammation have been largely examined using peripheral blood markers of inflammation, with few studi

102 ng predictive epigenomic marks of smokers in peripheral blood may allow for targeted risk stratificat

103 These data suggest that Th2 cells in peripheral blood may be a sensitive measure of increasin

104 PFS, overall survival (OS), peripheral-blood minimal residual disease (MRD) status,

105 use models, myeloid cells derived from human peripheral blood monocytes activate IGF1R and directly s

106 normal intracellular HA was also observed in peripheral blood monocytes derived from three different

107 compatibility complex class II expression on peripheral blood monocytes in patients.

108 n regimens, both in the MM6 cell line and of peripheral blood monocytes, inhibit an apparently consti

109 hosphorylation of Tyr(402)) in primary human peripheral blood monocytes.

110 e of cancer cells and induce cytotoxicity by peripheral blood mononuclear and engineered NK cells.

111 We performed peripheral blood mononuclear cell (PBMC) analysis on a s

112 a polysaccharide extracts were determined in peripheral blood mononuclear cell (PBMC) cultures.

113 genes demonstrated consistent modulation in peripheral blood mononuclear cell (PBMC) samples from HI

114 ecrosis factor alpha plasma levels and lower peripheral blood mononuclear cell activation versus both

115 The effect of peripheral blood mononuclear cell conditioned medium fro

116 Preweaning gene expression and peripheral blood mononuclear cell profiling may be usefu

117 more likely to be ever smokers, had shorter peripheral blood mononuclear cell telomeres, and were mo

118 KCNJ2 K(+) channel expression in peripheral blood mononuclear cell, which strongly correl

119 tor binding was studied with activated human peripheral blood mononuclear cells (hPBMCs).

120 uction capacity after ex vivo stimulation of peripheral blood mononuclear cells (MNCs) and bone marro

121 lysis of colonic mucosa samples (n = 87) and peripheral blood mononuclear cells (n = 85) from patient

122 In both peripheral blood mononuclear cells (PBMC) and macrophage

123 We performed microarray analyses of peripheral blood mononuclear cells (PBMC) RNA from subje

124 Parallel samples from plasma and peripheral blood mononuclear cells (PBMC) were also geno

125 d over 60 days, and TFV-DP concentrations in peripheral blood mononuclear cells (PBMC) were found to

126 cover novel early-pregnancy-induced genes in peripheral blood mononuclear cells (PBMC); and (2) chara

127 evels were quantified in allergen-stimulated peripheral blood mononuclear cells (PBMCs) and skin punc

128 livered R848 and PUUC were additive in human peripheral blood mononuclear cells (PBMCs) and synergist

129 of these proviruses detected in circulating peripheral blood mononuclear cells (PBMCs) are referred

130 granuloma model was developed by challenging peripheral blood mononuclear cells (PBMCs) derived from

131 e; and immune dysregulation/inflammation, in peripheral blood mononuclear cells (PBMCs) from healthy

132 e-cell RNA sequencing (scRNA-seq) to profile peripheral blood mononuclear cells (PBMCs) from seven pa

133 in blood and semen, and serologic testing of peripheral blood mononuclear cells (PBMCs) in a subset o

134 ied genome-wide levels of DNA methylation in peripheral blood mononuclear cells (PBMCs) longitudinall

135 intracellular cytokine staining (ICS) using peripheral blood mononuclear cells (PBMCs) of individual

136 l whole blood was incubated with third-party peripheral blood mononuclear cells (PBMCs) pretreated wi

137 , while pre-enrichment of B cells from whole peripheral blood mononuclear cells (PBMCs) results in th

138 ed that PINK1 modulates the bioenergetics of peripheral blood mononuclear cells (PBMCs) under basal c

139 ransferase gene-knockout (GTKO), and TKO pig peripheral blood mononuclear cells (PBMCs) using sera fr

140 ore the detected epidemiologic associations, peripheral blood mononuclear cells (PBMCs) were collecte

141 Furthermore, circulating, activated human peripheral blood mononuclear cells (PBMCs) were suppress

142 (NANPs) using a validated preclinical model, peripheral blood mononuclear cells (PBMCs), that is high

143 glycolysis and mitochondrial respiration) of peripheral blood mononuclear cells (PBMCs), which was no

144 myosarcoma (RD), neuroblastoma (SH-SY5Y) and peripheral blood mononuclear cells (PBMCs).

145 alpha EC(50) > 50 muM) was observed in human peripheral blood mononuclear cells (PBMCs).

146 transcripts exposed on the surface of human peripheral blood mononuclear cells (PBMCs).

147 We profiled total peripheral blood mononuclear cells (PBMCs, 106,545 cells

148 n 1187 spot-forming cells [SFCs] per million peripheral blood mononuclear cells [IQR 841-2428], n=24;

149 amma ELISPOT responses (median 521 SFU/10(6) peripheral blood mononuclear cells [PBMCs] in the tetrav

150 tructural VZV proteins in both BM and blood (peripheral blood mononuclear cells [PBMCs]).

151 pro-Th17 cytokine profile from primary human peripheral blood mononuclear cells and demonstrated effi

152 First, we performed mass cytometry of peripheral blood mononuclear cells and discovered an imm

153 = 102 healthy mother-newborn dyads, maternal peripheral blood mononuclear cells and neonatal umbilica

154 d activation/exhaustion immunophenotyping on peripheral blood mononuclear cells and quantified interf

155 Using flow cytometry on peripheral blood mononuclear cells and serum immunoassay

156 n K(+) channel mRNA levels in ventricles and peripheral blood mononuclear cells and their changes in

157 circulating pIL-8 and IL8 gene expression in peripheral blood mononuclear cells and tumors of patient

158 High levels of IL-8 in plasma, peripheral blood mononuclear cells and tumors were assoc

159 tion on arginine methylation in normal human peripheral blood mononuclear cells and utilize a broad p

160 RNA expression levels of SQSTM1 and INSR in peripheral blood mononuclear cells are related to the se

161 Third, peripheral blood mononuclear cells are used to explore h

162 nificant increase in IFN-gamma production by peripheral blood mononuclear cells at day 42 in the adju

163 We collected peripheral blood mononuclear cells at influenza diagnosi

164 inase-mediated phosphorylation events within peripheral blood mononuclear cells collected prior to va

165 olyclonal MBCs at the single-cell level from peripheral blood mononuclear cells collected ~2 weeks or

166 The OLA-guided therapy group had pre-ART peripheral blood mononuclear cells evaluated for drug re

167 s, and CD3 + T cells were sorted from frozen peripheral blood mononuclear cells from 11 RV-infected h

168 in children with and without asthma.Methods: Peripheral blood mononuclear cells from 17 children with

169 Peripheral blood mononuclear cells from 23 participants

170 le-cell RNA sequencing, we profiled ~276,000 peripheral blood mononuclear cells from 33 children with

171 Profiling of ~82,000 single peripheral blood mononuclear cells from adults with SLE

172 Stimulating peripheral blood mononuclear cells from COVID-19 convale

173 By performing scRNA-seq on peripheral blood mononuclear cells from four untreated i

174 estigated the immunomodulatory effects using peripheral blood mononuclear cells from healthy donors e

175 We evaluated peripheral blood mononuclear cells from MOG-AAD patients

176 rm single-cell RNA sequencing (scRNA-seq) of peripheral blood mononuclear cells from opioid-dependent

177 Peripheral blood mononuclear cells from patients with ac

178 differed from those of active UC in that the peripheral blood mononuclear cells from patients with CD

179 eceptor (IL7R) were significantly reduced in peripheral blood mononuclear cells from patients with CO

180 Peripheral blood mononuclear cells from premalignant cas

181 In primary peripheral blood mononuclear cells from the patients, do

182 el temporal dynamics of the transcriptome of peripheral blood mononuclear cells in a two-dimensional

183 gitudinal multi-omic profiling of plasma and peripheral blood mononuclear cells including metabolome,

184 Likewise, their peripheral blood mononuclear cells mounted a negligible

185 Integrated HIV-1 DNA was quantified in peripheral blood mononuclear cells obtained from acutely

186 Because NAS also increased IDO1 activity in peripheral blood mononuclear cells of a significant prop

187 eover, TLR2 block prior to DENV infection of peripheral blood mononuclear cells prevents activation o

188 Immunophenotyping of peripheral blood mononuclear cells revealed an increase

189 4 variants by caspase-8 sensitized patients' peripheral blood mononuclear cells to RIPK1 activation,

190 rkers and enhanced proliferative response of peripheral blood mononuclear cells to WHV peptides.

191 ecific CD8+ T-cell and B-cell responses from peripheral blood mononuclear cells using flow cytometry

192 Peripheral blood mononuclear cells were collected from 3

193 Peripheral blood mononuclear cells were collected from d

194 Peripheral blood mononuclear cells were cultured with at

195 Peripheral blood mononuclear cells were infected with re

196 Peripheral blood mononuclear cells were quantified for T

197 Peripheral blood mononuclear cells were stained and anal

198 Peripheral blood mononuclear cells were stimulated with

199 genes in 2 or more studies of whole blood or peripheral blood mononuclear cells with concordant direc

200 osetting model by coculturing HLA-II-matched peripheral blood mononuclear cells with HL cell lines an

201 tosis and increased inflammatory response in peripheral blood mononuclear cells, as well as a compens

202 4 (median 856 spot-forming cells per million peripheral blood mononuclear cells, IQR 493-1802; n=43).

203 scription results in reduced DAP1 protein in peripheral blood mononuclear cells, monocytes, and lymph

204 Human peripheral blood mononuclear cells, or immature dendriti

205 In human peripheral blood mononuclear cells, these molecules supp

206 anine dinucleotide (CpG) sites measured from peripheral blood mononuclear cells, we identified 15 sit

207 immature dendritic cells derived from human peripheral blood mononuclear cells, were cultured in med

208 nces despite analysing more than 1.5 billion peripheral blood mononuclear cells, which raises the pos

209 227 IgG antibody-secreting cells per million peripheral blood mononuclear cells.

210 n-3 in live human hepatic stellate cells and peripheral blood mononuclear cells.

211 can patients living with HIV and produced in peripheral blood mononuclear cells.

212 NA sequencing measurements on whole blood or peripheral blood mononuclear cells.

213 s assessed using the Seahorse XFe96 in fresh peripheral blood mononuclear cells.

214 cing robust viral reactivation in plasma and peripheral blood mononuclear cells.

215 tive site geometries in three cell lines and peripheral blood mononuclear cells.

216 activity in SupT1 T cells and donor-derived peripheral blood mononuclear cells.

217 95% confidence interval: 1.47-2.04) and log peripheral blood neutrophil lymphocyte ratio (odds ratio

218 The three mature peripheral blood neutrophil subsets arise from distinct

219 eflect the effects of smoking and disease on peripheral blood NK cell phenotypes, provide insight int

220 to delete CD38 (CD38KO) in ex vivo expanded peripheral blood NK cells.

221 sation and content of granules compared with peripheral blood NK cells.

222 r conditions and gene expression patterns in peripheral blood obtained at early pregnancy.

223 rWGS was conducted on peripheral blood obtained from ill pediatric patients (a

224 Peripheral blood of 1298 individuals was analyzed for ac

225 resence of autoreactive IgE were assessed in peripheral blood of a cohort of patients with MCTD and i

226 Gene expression in peripheral blood of AKI/DCD recipients offers a novel pl

227 Gag-specific CD8(+) T cell responses in the peripheral blood of all nine study participants who were

228 rt is HIV-1 reservoirs and correlates in the peripheral blood of children who achieved sustained viro

229 al indirect measure of reservoir size in the peripheral blood of children with perinatally acquired H

230 served higher frequencies of NK cells in the peripheral blood of CL patients compared with healthy su

231 mice and did not significantly change in the peripheral blood of CNV mice.

232 expression was examined in ILC2 sorted from peripheral blood of healthy controls and asthma patients

233 rovirus-specific T cells were generated from peripheral blood of healthy donors by stimulation with o

234 NK cells freshly isolated from peripheral blood of healthy donors were stimulated with

235 HA in MiHApos donors and TAAs are present in peripheral blood of healthy individuals.

236 We found that in the peripheral blood of normal individuals, plasmablasts wer

237 tected in the tumor tissue as well as in the peripheral blood of patients with bladder cancer.

238 ls and time, with consistent findings in the peripheral blood of patients with breast cancer.

239 the spike glycoprotein of SARS-CoV-2 in the peripheral blood of patients with COVID-19 and SARS-CoV-

240 els were significantly decreased both in the peripheral blood of patients with MDD and in the two dep

241 In peripheral blood of patients with rheumatological manife

242 variants, and it can be observed in matched peripheral blood of patients with tumor hypermethylation

243 3(+), CD206(+), and CD80(+) monocytes in the peripheral blood of patients with wet AMD, patients with

244 t recognize these HIPs are detectable in the peripheral blood of subjects with T1D and exhibit an eff

245 gher expression of CD163(+) monocytes in the peripheral blood on day 7 post injury in mice.

246 e staining of B cell subpopulations in human peripheral blood or various anatomical compartments in t

247 mphocyte count, monocyte count, and ratio of peripheral blood oxygen saturation to fraction of inspir

248 causes severe clinical syndromes despite low peripheral blood parasitemia.

249 e cases, MDS/AML patients showed a recurrent peripheral blood pattern of acquired, granulocyte-specif

250 successfully for all patients from the donor peripheral blood (PB) and consisted mostly of CD3+ lymph

251 CLL) cells cycle between lymph node (LN) and peripheral blood (PB) and display major shifts in Bcl-2

252 Peripheral blood (PB) Foxp3+ Treg was collected from RA

253 represented in the bone marrow compared with peripheral blood (PB), and in PB CLL subsets expressing

254 DNA from CD4(+) T cell subsets derived from peripheral blood (PB), lymph node (LN), and gut tissues

255 Peripheral blood plasmablasts are normally highly clonal

256 We isolated single peripheral blood plasmablasts from children with KD 1-3

257 een changes in plasma SPM concentrations and peripheral blood platelet and leukocyte responses.

258 ed transcription of select genes in PBMC and peripheral blood polymorphonuclear cells (PMN).

259 Integration of bone marrow and peripheral blood precursor datasets identified two putat

260 /s/cm(-5)) and elevation in central, but not peripheral, blood pressure.

261 xtent to which immune cell phenotypes in the peripheral blood reflect within-tumor immune activity pr

262 tmental changes in protein levels of CSF and peripheral blood, respectively, including many disease-a

263 Immunophenotyping of peripheral blood revealed reductions of non-classical mo

264 ell RNA sequencing in mouse spleen and human peripheral blood revealed that only mouse neutrophils an

265 approach performed on maternal DNA from both peripheral blood sample and urine-derived podocyte-linea

266 tation had not been previously detected on a peripheral blood sample of maternal DNA.

267 llograft rejection and treatment resistance, peripheral blood samples and intestinal allograft biopsi

268 ed to reconstruct absorbed radiation dose in peripheral blood samples collected from potentially expo

269 a to support its implementation, we analyzed peripheral blood samples from 400 HIV-1(+) adults on ART

270 he very low number of CTCs in standard 10-mL peripheral blood samples limits their clinical utility.

271 e we performed single-cell RNA sequencing in peripheral blood samples of 5 healthy donors and 13 pati

272 r cells (M-MDSCs), and Lox-1(+) PMN-MDSCs in peripheral blood samples of 62 NSCLC patients before and

273 Using multiple pre-disease peripheral blood samples on the Illumina 450 K and EPIC

274 al and should be complementary to the use of peripheral blood samples to allow for a comprehensive un

275 -enhanced CT, (18)F-FDG PET/CT, and complete peripheral blood sampling at baseline before ICI treatme

276 ptional activity before detection by routine peripheral blood sampling.

277 D4 T cells led to increased myeloid cells in peripheral blood, spleen, and bone marrow, as well as ex

278 disease (GVHD), a common complication after peripheral blood stem cell or bone marrow transplantatio

279 by 3 courses of high-dose chemotherapy with peripheral blood stem cell rescue and involved-field rad

280 posttransplant cyclophosphamide (PTCY) using peripheral blood stem cells (PBSC) as a source of graft.

281 Peripheral blood stem cells were the stem cell source in

282 of cigarette smoking have been identified in peripheral blood studies, but because of tissue specific

283 n (TRG) and delta-chain (TRD) repertoires of peripheral blood T cells in newborns, infants, and young

284 Multiparametric flow cytometry on peripheral blood T cells was performed on a daily basis.

285 We found that human peripheral blood Th cells in resting state do not show s

286 Monocytes were isolated from peripheral blood to determine immune functionality, meta

287 s; however, there are no studies showing the peripheral blood transcript profile of these combined di

288 compared with haploidentical bone marrow and peripheral-blood transplantation (HR, 1.91; P = .0001; a

289 marrow transplantation and 59% and 52% after peripheral-blood transplantation, respectively.

290 Compared with haploidentical bone marrow and peripheral-blood transplantations and adjusted for age,

291 Tregs, and innate lymphoid cells (ILC) from peripheral blood using flow cytometry.

292 on comparing eosinophils isolated from human peripheral blood vs human adipose tissue.

293 ed, placebo-controlled, crossover study, and peripheral blood was collected at baseline, 2, 4, 6, and

294 ytokine profile in seminal fluid, but not in peripheral blood, was significantly different between me

295 sults revealed that metabolic changes in the peripheral blood were associated with MDD, particularly

296 GI.1-specific cellular responses in peripheral blood were observed 9 days postchallenge with

297 For this, isolated neutrophils from human peripheral blood were stimulated in an in vitro infectio

298 ent tissue can also typically be detected in peripheral blood, which suggests a convenient approach t

299 We sequenced DNA from peripheral blood with a customized 21-gene panel at a me

300 al trials have measured HIV induction in the peripheral blood with minimal focus on tissue reservoirs