ライフサイエンスコーパス: peripheral resistance

1 dditive declines in blood pressure and total peripheral resistance.

2 pulsatile load and concurrently lowers total peripheral resistance.

3 sure, occurs as the consequence of increased peripheral resistance.

4 despite reduced diastolic pressure and total peripheral resistance.

5 ystem, and increased vascular tone and total peripheral resistance.

6 lity, systemic compliance, stroke volume and peripheral resistance.

7 ormal, where it is quickly reflected off the peripheral resistance.

8 rterial enlargement in response to increased peripheral resistance.

9 cant increases in SS RBC adhesion and in the peripheral resistance.

10 ke volume, possibly as a result of decreased peripheral resistance.

11 ut (5.5 vs. 4.9 l/min; P < 0.001), and lower peripheral resistance (1,487 vs. 1,666; P = 0.01), paral

12 15% vs. -5 +/- 10%), and a lower decrease in peripheral resistance (-17 +/- 12% vs. -26 +/- 12%) (p <

13 n increase in blood pressure (43%) and total peripheral resistance (65%) without any change in heart

14 cts peripheral resistance to blood flow, and peripheral resistance affects DBP.

15 anged significantly with a decrease in total peripheral resistance and an increase in common femoral

16 d by elevated heart rate and decreased total peripheral resistance and arterial blood pressure.

17 on were hypotensive, with decreases in total peripheral resistance and filtration fraction on day 1 i

18 rolled for, hypertensive subjects had higher peripheral resistance and lower arterial compliance than

19 late vasoconstriction, which increases total peripheral resistance and mean arterial pressure.

20 Peripheral resistance and peak filling rate were unchang

21 Total peripheral resistance and perivascular fibrosis in the h

22 during EFP than MLP (P = 0.030), while total peripheral resistance and plasma noradrenaline were not

23 nts present with low cardiac output and high peripheral resistance and that they respond poorly to fl

24 pressure and is a fundamental determinant of peripheral resistance and, hence, organ perfusion and sy

25 high rate of LV hypertrophy, in spite of low peripheral resistances and low-to-normal blood pressure,

26 Cardiac index, total peripheral resistance, and blood volume were not differe

27 volume, increased cardiac output, decreased peripheral resistance, and increased uteroplacental bloo

28 stemic oxygen delivery, stroke volume, total peripheral resistance, and organ blood flow in the liver

29 (P < 0.001), diastolic blood pressure, total peripheral resistance, and stroke volume compared with w

30 ate, preload, and cardiac output; decreasing peripheral resistance; and increasing ventricular compli

31 ecause endothelial dysfunction and increased peripheral resistance are hallmarks of hypertension, det

32 between central control of vascular flow and peripheral resistance are unclear.

33 ar volumes and end-systolic elastance (Ees), peripheral resistance, arterial elastance (Ea), arterial

34 Autonomic sympathetic nerves innervate peripheral resistance arteries, thereby regulating vascu

35 not Dex impaired NO-dependent relaxation of peripheral resistance arteries.

36 ationship between large artery stiffness and peripheral resistance artery function.

37 rs and identified the systemic and pulmonary peripheral resistance as being critical parameters for a

38 lood pressure is acutely controlled by total peripheral resistance as determined by the diameter of s

39 f altered arterial stiffness versus impaired peripheral resistance but is not superior to SBP+DBP in

40 ut and decreases in left atrial pressure and peripheral resistance but without eliciting a supplement

41 sure, portal pressure, cardiac output, total peripheral resistance, central blood volume, and extrace

42 al hemodynamics were determined by the total peripheral resistance, common femoral artery flow, and a

43 er (P = 0.09), heart rate was similar, total peripheral resistance decreased (2172 +/- 364 vs. 2543 +

44 nfusion before any symptoms developed, total peripheral resistance decreased 24% +/- 20% in group II

45 to tilt before any symptoms developed, total peripheral resistance decreased 9% +/- 14% in group I fr

46 Cardiac index and total peripheral resistance decreased in MSA patients by 33.4+

47 Total peripheral resistance decreased with rosuvastatin in bot

48 EP group and associated with increased total peripheral resistance (difference in means, 96.4 [95% co

49 s include increased cardiac work with normal peripheral resistance, diffuse slowing on electroencepha

50 a means for maintenance of vascular tone and peripheral resistance during hibernation.

51 Total peripheral resistance during tilt post flight was higher

52 tput, and calculated stroke volume and total peripheral resistance, during supine rest and 10 min of

53 ined by LV end-diastolic dimension and total peripheral resistance estimated by thoracic impedance.

54 to 86.9 +/- 21.7 ml (P = 0.06) and increased peripheral resistance from 1106 +/- 246 to 1246 +/- 222

55 autonomic nerves may impair control of total peripheral resistance giving rise to symptomatic orthost

56 EC size, occlusion of capillaries, elevated peripheral resistance, heart failure, and death.

57 sure (Normal: p < 0.001; HF: p < 0.001), and peripheral resistance (HF: p < 0.001), and transiently i

58 subjects, accompanied by a decrease in total peripheral resistance in 16 of them (64%, group A).

59 In vivo, human U-II markedly increases total peripheral resistance in anaesthetized non-human primate

60 aptation may contribute to the regulation of peripheral resistance in eNOS-KO mice.

61 t dilatation may contribute to the increased peripheral resistance in hypertension.

62 es to the elevation of wall shear stress and peripheral resistance in hypertension.

63 rdiac output (CO) but minor changes in total peripheral resistance in saline-treated rats.

64 ntry communication) and physiologic factors (peripheral resistance in the branch vessels, pump output

65 ge of factors: one key element of control is peripheral resistance in tissue capillary beds.

66 he true-lumen outflow caused by lowering the peripheral resistance in true-lumen branch vessels.

67 (P<0.05) compared with baseline, while total peripheral resistance increased (P<0.05).

68 Na+ intake, while baseline CO decreased and peripheral resistance increased in this group.

69 nd 1.15 versus 1.02 mm Hg/mL x m2) and total peripheral resistance index (3027 and 2805 versus 2566 d

70 n cardiac index, a 28% increase in the total peripheral resistance index (p < .01), and a 33% decreas

71 The total peripheral resistance index and stroke volume index tend

72 ed with increases in both cardiac output and peripheral resistance index.

73 This increased peripheral resistance is the result of an increased acti

74 intact, (ii) both calcification and enhanced peripheral resistance lead to reduced flow rates, reduce

75 Although total peripheral resistance markedly increased, mean arterial

76 mean arterial pressure of 18 mm Hg and total peripheral resistance of 665 AU and increases in heart r

77 roximately 20 mm Hg and an increase in total peripheral resistance of approximately 30%.

78 ressure (7 +/- 4 mmHg, P <= 0.001) and total peripheral resistance (P = 0.013) concomitantly with a r

79 01) while accentuating the increase in total peripheral resistance (P=0.012).

80 decreases in left atrial pressure (P<0.001), peripheral resistance (P=0.014), and hematocrit (P<0.001

81 on with a 94.5 dynes x sex/cm(5) lower total peripheral resistance (P=0.0368).

82 ries, but this drop can be offset by greater peripheral resistance, provided left ventricular functio

83 women relative wall thickness (r = 0.11) and peripheral resistance (r = -0.17).

84 Although total peripheral resistance remains decreased, Na+ retention c

85 duced microvascular blood flow and increased peripheral resistance, suggesting that vasodilatory decr

86 epsis), cardiac output, stroke volume, total peripheral resistance, systemic oxygen delivery, and org

87 oth muscle cells of a primary determinant of peripheral resistance - the small mesenteric artery.

88 Viscosity affects peripheral resistance to blood flow, and peripheral resi

89 cially the visceral type, is associated with peripheral resistance to insulin actions and hyperinsuli

90 modalities, such as exercise, may overcome a peripheral resistance to insulin, thus preventing GDM or

91 Peripheral resistance to leptin due to its impaired brai

92 ng leptin and have the potential to induce a peripheral resistance to leptin, similar to the central

93 roke volume (SV), cardiac output (CO), total peripheral resistance (TPR) and arterial compliance to a

94 tivity (MSNA) is positively related to total peripheral resistance (TPR) and inversely related to car

95 asures of preejection period (PEP) and total peripheral resistance (TPR) in healthy black (n=76) and

96 ed, whereas heart rate (HR), MSNA, and total peripheral resistance (TPR) increased during HUT (all P<

97 Total peripheral resistance (TPR) increased during LBNP in bot

98 Baseline total peripheral resistance (TPR) was not different between th

99 decrease in cardiac contractility and total peripheral resistance (TPR) were similar in TRPV(1)(+/+)

100 roke volume (SV), heart rate (HR), and total peripheral resistance (TPR), in 163 patients with tilt-i

101 tients with VAH show a greater rise in total peripheral resistance (TPR), suggesting a compensatory m

102 a sympathetically-mediated increase in total peripheral resistance (TPR).

103 ; n = 12) underwent assessments of BP, total peripheral resistance (TPR; Modelflow) and MSNA action p

104 Microvascular blockage resulted in increased peripheral resistance units (PRU).

105 d action of nitric oxide and endothelin-1 in peripheral resistance vessels of patients with syndrome

106 Total peripheral resistance was calculated from mean arterial

107 2 +/- 0.40 l min(-1), P = 0.01), while total peripheral resistance was greater (1327 +/- 117 vs. 903

108 Supine leg peripheral resistance was greater than control resistanc

109 ght with CFD than in those with PFD, whereas peripheral resistance was not significantly different.

110 volume (r=0.88+/-0.13, P<0.05), while total peripheral resistance was related to MSNA during 45 min

111 with control rats, but the increase in total peripheral resistance was significantly attenuated.

112 In the other 9 subjects, total peripheral resistance was well maintained even at presyn

113 eart rate, mean arterial pressure, and total peripheral resistance were greater, whereas cardiac outp

114 Upright heart rate and total peripheral resistance were greater, whereas stroke volum

115 Blood pressure and total peripheral resistance were higher during rest and tiltin

116 decrease in diastolic pressure and estimated peripheral resistance were observed in troglitazone-trea