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1 alpha3 targeting domain are excluded and are perisynaptic.
2 lel fibers (PFs), indicating that NMDARs are perisynaptic.
3 em and prevented live-cell discrimination of perisynaptic actin filaments.
4                                              Perisynaptic actin forms a uniquely dynamic structure we
5 ly in the CNS, and mediate both synaptic and perisynaptic activities of endogenous cholinergic inputs
6 yrus granule cells, most likely by targeting perisynaptic alpha4betadelta receptors expressed at dist
7 ynaptic sites; synaptic glycine receptor and perisynaptic alpha7-nAChR clusters were not altered.
8 he neighboring synaptic glycine receptors or perisynaptic alpha7-nAChRs on chick ciliary ganglion (CG
9 mography revealed lower synaptic, but higher perisynaptic, AMPAR density in stg RTN.
10 ction leads to activity-dependent removal of perisynaptic AMPARs and suppresses the subsequent expres
11                         Here, we report that perisynaptic AMPARs are GluR2-lacking and they transloca
12                                   As long as perisynaptic AMPARs are present, both LTP and spine expa
13 ylation can alter the subunit composition of perisynaptic AMPARs by providing stability to GluR1 homo
14                                              Perisynaptic AMPARs do not normally detect synaptically
15 suggest that the delivery and maintenance of perisynaptic AMPARs may serve as a checkpoint in the exp
16                            The appearance of perisynaptic AMPARs requires postsynaptic exocytosis, PK
17  the baseline state together with removal of perisynaptic AMPARs, or they can enter a stabilized stat
18 ease together with synaptic incorporation of perisynaptic AMPARs.
19 nduces an increase in MMP-9 activity that is perisynaptic and enriched at thalamo-cortical synapses.
20 ld-type synapses, we found that nonsynaptic (perisynaptic and extrasynaptic) AChEs are mobile and gra
21  neurotrophin-3 (NT3), is expressed by mouse perisynaptic and myelinating Schwann cells from birth th
22  as well as regulate glutamate levels in the perisynaptic and presynaptic domains, the ultrastructura
23  we identified cisternae of the GA in distal perisynaptic and subependymal processes, in perivascular
24 nit, and subcellular localization (synaptic, perisynaptic, and dendritic) unique within this protein
25 ry method to simultaneously record synaptic, perisynaptic, and extrasynaptic dopaminergic transmissio
26 synaptic transmission; their localization in perisynaptic areas would appear to limit their activatio
27 ve been identified, the molecules underlying perisynaptic astroctye-neuron contacts are unknown.
28 t known contact-dependent mechanism by which perisynaptic astrocyte processes promote synaptogenesis.
29 novo protein synthesis occurs within distal, perisynaptic astrocyte processes.
30 es; and 3) Pcdh-gammaC5 is also localized in perisynaptic astrocyte processes.
31                                              Perisynaptic astrocytes express important glutamate tran
32 ensity of mitochondria in axon terminals and perisynaptic astrocytes was increased in the synGLT-1 KO
33 y brain, NPC1 was expressed predominantly in perisynaptic astrocytic glial processes.
34  EphA7, being preferentially in PSDs, and in perisynaptic astrocytic leaflets, provides morphologic e
35         The functional implications of GA in perisynaptic astrocytic processes and other processes ar
36                                              Perisynaptic astrocytic processes are an integral part o
37 ficacy that were coordinated with changes in perisynaptic astrocytic processes in the border region b
38 as used to investigate relationships between perisynaptic astroglia and dendritic spine synapses unde
39  other components of the neuropil, including perisynaptic astroglia and extracellular matrix proteins
40 sorder literature pointing to a key role for perisynaptic astroglia and signaling in the extracellula
41 while reducing the presence and proximity of perisynaptic astroglia near the axon-spine interface of
42   Under all conditions, >85% of synapses had perisynaptic astroglia processes within 120 nm of some p
43                                              Perisynaptic astroglia provide critical molecular and st
44                              Thus, access of perisynaptic astroglia to synapses is dynamically modula
45  to determine the extrinsic factors (such as perisynaptic astroglia) and the intrinsic factors (such
46  are characterized by unmyelinated axons and perisynaptic astroglial envelopes.
47 d by high-affinity transporters expressed by perisynaptic astroglial processes (PAPs): this helps mai
48 o alpha-bungarotoxin, though concentrated in perisynaptic clusters on neurons, can generate a large a
49  of Fabp7 mRNA and protein in the astrocytic perisynaptic compartment, and observed a diurnal change
50 ytes control synaptic activity by modulating perisynaptic concentrations of ions and neurotransmitter
51 pressed in cortical astrocytes, localizes to perisynaptic contacts and is required to restrict neurop
52 ion (LTD) was associated with a reduction in perisynaptic CP-AMPAR levels.
53                   This restrictive action of perisynaptic CSPGs in mature neural tissue may account f
54 robe how signaling between cell types within perisynaptic ecosystems creates the synaptic plasticity
55 ) synaptic signaling significantly decreases perisynaptic enshealthing of astroglial processes on syn
56 ently, the released histamine is taken up by perisynaptic epithelial glia and converted into inactive
57                            Specifically, the perisynaptic expression of GAT1 enables it to regulate G
58 lutamate transport was inhibited, indicating perisynaptic expression of NR2B NMDARs.
59 g spillover from synapses due to their dense perisynaptic expression primarily on astroglia.
60 e postsynaptic NMDAR current is reliant on a perisynaptic extracellular alkaline shift generated by t
61 ell as adjacent astroglial processes and the perisynaptic extracellular matrix.
62 -labeled D1Rs were distinctly distributed on perisynaptic/extrasynaptic membranes and the axoplasm of
63           Instead, it reduces recruitment of perisynaptic/extrasynaptic NR2B-containing NMDARs, there
64 ge in the molecular composition of NMDARs at perisynaptic/extrasynaptic sites.
65 olume, as well as dysregulated expression of perisynaptic genes associated with mature astrocyte func
66 nsports neurotransmitter metabolites between perisynaptic glia and neuronal cell bodies to mediate lo
67 ork transports histamine metabolites between perisynaptic glia and photoreceptor cell bodies to media
68 membranes were targeted, acute disruption of perisynaptic glia and SC membranes at nodes of Ranvier (
69   In HD mice, the decay time constant of the perisynaptic Glu concentration (TauD), as an indicator o
70 es, we can now quantify the time constant of perisynaptic [Glu] decay (as an indicator of uptake) and
71 urons that can be attributed to a release of perisynaptic GluA1-containing AMPA receptors into the po
72    Immunogold staining revealed synaptic and perisynaptic GluD1 labeling at putative axo-dendritic an
73                                        Thus, perisynaptic GluR2-lacking AMPARs play a critical role i
74 er as well as alterations in the kinetics of perisynaptic glutamate buffering and uptake contributing
75 uggesting that astrocyte-mediated removal of perisynaptic glutamate is important in limiting NSV-indu
76        This retardation boosts activation of perisynaptic group I metabotropic glutamate receptors (m
77 aptic or postsynaptic cell, suggesting local perisynaptic influences on the development and/or state
78 ceptors, thought to mediate tonic (extra- or perisynaptic) inhibition, are unknown.
79 smaller synapses and with slower kinetics of perisynaptic ion transients.
80 s either were nonimmunoreactive or displayed perisynaptic labeling.
81                    Our results demonstrate a perisynaptic localization of delta subunit-containing GA
82                Functional correlates of this perisynaptic localization were obtained by comparing inh
83 pes exhibiting substantial extrasynaptic and perisynaptic localization.
84 shift of the gamma2 subunit from synaptic to perisynaptic locations and potential partnership of the
85 rmed a decrease in delta subunit labeling at perisynaptic locations in the molecular layer of the den
86 R1-S845A mutants, CP-AMPARs were absent from perisynaptic locations mainly due to lysosomal degradati
87 mma2 subunit labeling was also found at many perisynaptic locations on granule cell dendrites, consis
88 ed mice, alpha4 subunit labeling remained at perisynaptic locations, but increased gamma2 subunit lab
89  Chmp2b was shown to concentrate beneath the perisynaptic membrane of dendritic spines.
90 ted from the postsynaptic density toward the perisynaptic membrane within the spine, leading to synap
91  57% particles were evenly distributed along perisynaptic membranes and the remaining 43% of particle
92 C1 indicate an extrasynaptic localization on perisynaptic membranes that are near release sites.
93 onal glutamate transporter, localized on the perisynaptic membranes that are near release sites.
94 must be overwhelmed by glutamate to activate perisynaptic metabotropic glutamate receptors.
95 st that Car14 regulates pH transients in the perisynaptic microenvironment and govern their impact on
96 , our study indicates that the activation of perisynaptic MMP-3 supports L-type channel-dependent LTP
97 how that LRx to adult amblyopic mice induces perisynaptic MMP2/9 activity and extracellular matrix (E
98 strocytes, which leads to hyperactivation of perisynaptic N-methyl-D-aspartate receptors and tagging
99  in neuron-astrocyte interactions, including perisynaptic neuron-astrocyte interactions.
100 wn about how different cell types within the perisynaptic neuropil regulate synaptic functions and dy
101 even though key neuropathology exists in the perisynaptic neuropil that homeostatically regulates syn
102 ent of glutamate spillover and activation of perisynaptic NMDA receptors at ON GCs.
103  This morphological evidence for exclusively perisynaptic NMDARs localizations suggests a distinct ro
104 on generated by the persistent activation of perisynaptic or extrasynaptic GABA(A)Rs, which can detec
105      Although most GIRK subunit labeling was perisynaptic or extrasynaptic, GIRK2 was found occasiona
106 ptophysin, synaptotagmin, or proteins of the perisynaptic plasma membrane such as GABA transporter 1
107 tivity stems from long-lasting pre, post and perisynaptic plasticity, including insertion of Ca(2+)-p
108 s in expression and trafficking Fabp7 to the perisynaptic process are not known.
109 he ephrin-A3 ligand, which is located in the perisynaptic processes of astrocytes, is essential for m
110 n pre- and postsynaptic profiles and also in perisynaptic processes.
111 rain astrocytes and radial glia that exhibit perisynaptic processes.
112 lecule autotaxin (ATX) present in astrocytic perisynaptic processes.
113 xpressed by astrocytes and localize to their perisynaptic processes.
114                    Previously we showed that perisynaptic proteolysis by MMP9 mediates the enhancemen
115 from the amblyopic pathway to trigger robust perisynaptic proteolysis.
116 C amplitude, which is primarily dependent on perisynaptic receptor occupancy.
117 lel fibre synapses can lead to activation of perisynaptic receptors that mediate short- and long-term
118  Because group I mGluRs are localized to the perisynaptic region in juxtaposition to synaptic NMDARs
119 otransmitter receptors that are found in the perisynaptic region of the postsynaptic membrane.
120 -term potentiation (LTP) but are absent from perisynaptic regions after the full expression of LTP.
121 hat AMPA receptors (AMPARs) are delivered to perisynaptic regions after the induction of long-term po
122                        TRPC1 is expressed in perisynaptic regions of the cerebellar parallel fibre-Pu
123 t undergo CEDE are in somata, dendrites, and perisynaptic regions, identified by using immunocytochem
124  amino acid carrier 1 (EAAC1) is enriched in perisynaptic regions, where it may regulate synaptic spi
125 c inhibition of PKC activity holds AMPARs at perisynaptic regions.
126 ontaining beta-isoform primarily to non-PSD, perisynaptic regions.
127 emonstrate the presence of dynamic D1R/NMDAR perisynaptic reservoirs favoring a rapid and bidirection
128 -deficient mdx mice, but was retained on the perisynaptic sarcolemma even in mice lacking both utroph
129  spine structure and function, probably as a perisynaptic scaffold and barrier within the spine neck.
130 uscular junction (NMJ), early alterations in perisynaptic Schwann cell (PSC), glial cells at this syn
131 nt, including presynaptic, postsynaptic, and perisynaptic Schwann cell defects in these diseases.
132  occurs in this model, concomitant injury to perisynaptic Schwann cells (pSC) could indirectly contri
133                                              Perisynaptic Schwann cells (PSCs) are specialized, non-m
134 sing a novel technique to selectively ablate perisynaptic Schwann cells (PSCs) at the neuromuscular j
135 mmunofluorescence, COX-2 was detected in the perisynaptic Schwann cells (PSCs) surrounding the NMJ.
136 rvation occurs early in the process and that perisynaptic Schwann cells (PSCs), glial cells at the NM
137  developed an approach to selectively ablate perisynaptic Schwann cells (PSCs), the glial cells at th
138            Emerging studies demonstrate that perisynaptic Schwann cells (PSCs), which are the glia ce
139 ese new insights, this article suggests that perisynaptic Schwann cells and synaptically associated a
140 tter release, postsynaptic excitability, and perisynaptic Schwann cells are discussed as avenues to i
141 ated that presynaptic neuronal membranes and perisynaptic Schwann cells are targets for anti-GQ1b ant
142 velopment resulted in an increased number of perisynaptic Schwann cells at neuromuscular synapses, wi
143 T3 is not required for normal development of perisynaptic Schwann cells or synapses.
144 ng the nuclei of the motor end plate, and in perisynaptic Schwann cells, and localized close to nicot
145 nduction is required to retain AMPARs at the perisynaptic site after their appearance.
146               AMPARs can be detected at this perisynaptic site before, but not after, the full expres
147  from the NMDARs and diffuse to a presumably perisynaptic site, where they become available to an end
148 ols causes the rapid delivery of AMPARs to a perisynaptic site.
149 being expressed largely at extrasynaptic and perisynaptic sites in neuronal cell bodies, dendrites, a
150 he PSD, but were reduced near the PSD and at perisynaptic sites of dendritic spines in extinction-res
151 strate that AMPARs are rapidly trafficked to perisynaptic sites shortly after LTP induction and sugge
152 ning CP-AMPARs on the surface, especially at perisynaptic sites, and suggest that the regulation of t
153 ransporters effectively reuptake dopamine at perisynaptic sites, confining dopamine within synaptic c
154 ering is likely to take place at synaptic or perisynaptic sites.
155 urocan), brevican is mainly expressed in the perisynaptic space closely associated with both the pre-
156 esicular structures in dendrites, and in the perisynaptic space encircling presynaptic terminals of t
157 ineuronal nets and is highly enriched in the perisynaptic space suggesting a role for synaptic transm
158 ic vGlut1 and ultrastructural changes in the perisynaptic space.
159  pre- and postsynaptic as well as extra- and perisynaptic structures of the primate prefrontal cortex
160 lation and molecular characterization of the perisynaptic subset of astrocytic Golgi may be feasible,
161 d glial processes were often perivascular or perisynaptic, surrounding asymmetric excitatory-type axo
162 nctions requires appropriate channel gating, perisynaptic targeting, and linkage to intracellular sig
163 d type A (GABAA) receptors (GABAR) increases perisynaptic to excitatory synapses in CA1 hippocampus.
164 pic glutamate receptors (mGluRs) are located perisynaptic to the postsynaptic specializations of asym
165 ic shafts and spines, ERbeta-ir was near the perisynaptic zone adjacent to synapses formed by unlabel

 
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