ライフサイエンスコーパス: peritoneal

1 We studied a reproducible model of intra-peritoneal adhesion formation in rats using laparotomy w

2 the adhesion score significantly and abolish peritoneal adhesions in 45% of animals in a rat model of

3 to the peritoneum or to the kidneys, whereas peritoneal administration of vancomycin (particularly li

4 entified increased Connexin-43 expression in peritoneal and hepatic macrophages.

5 -like B cells that predominantly inhabit the peritoneal and pleural cavities.

6 hours and allowing dissemination to various peritoneal and retroperitoneal organs including the kidn

7 use of V(H)11, V(H)12, and V(H)2 between the peritoneal and splenic PtC(+) populations with age.

8 -/-) mice exhibit reduced latency within the peritoneal B cell compartment and elevated latency withi

9 latent reservoir is predominantly hosted by peritoneal B cells.

10 ansitional-marginal zone precursor stage and peritoneal B-1 B cell development, the TI antibody respo

11 For example, peritoneal B-1 cells and splenic marginal zone B cells e

12 phosphatidylcholine (PtC)-specific (PtC(+)) peritoneal B-1a cell IgM does not change with age.

13 We have previously demonstrated peritoneal B-1a cell-derived phosphorylcholine-specific

14 ss to in vitro BCR stimulation compared with peritoneal B-2 cells and splenic follicular B cells, res

15 iota-independent expansion and activation of peritoneal B1b cells, which culminates in increased lami

16 f a previously uncharacterized population of peritoneal basophil-mast cell progenitors.

17 ithelial ovarian, primary fallopian tube, or peritoneal cancer in a phase 3 clinical trial.

18 duce the risk for breast, ovarian, tubal, or peritoneal cancer in women with potentially harmful BRCA

19 Peritoneal cancer index (PCI), as assessed by CT, is uti

20 the groups were similar, except that median peritoneal cancer index remained higher in the CRS-HIPEC

21 Tumor burden was assessed using the peritoneal cancer index.

22 family history of breast, ovarian, tubal, or peritoneal cancer or who have an ancestry associated wit

23 ategies to deliver emerging therapeutics for peritoneal cancer treatment using nanocarriers.

24 ithelial ovarian, fallopian tube, or primary peritoneal cancer who had been treated with three or mor

25 ious diagnosis of breast, ovarian, tubal, or peritoneal cancer who have completed treatment and are c

26 isk for breast, ovarian, fallopian tube, and peritoneal cancer.

27 iagnosed ovarian, fallopian tube, or primary peritoneal carcinoma.

28 of 103 patients, 18.4%) was associated with peritoneal carcinomatosis (RR: 5.9; 95% CI: 3.8, 9.2; P

29 d to the peritoneal cavity is referred to as peritoneal carcinomatosis and has a very poor prognosis.

30 ese tumors were invasive, and mice developed peritoneal carcinomatosis and lung metastases.

31 tein were correlated with cancer recurrence, peritoneal carcinomatosis and poor patient prognosis.

32 in a mouse xenograft model of ovarian cancer peritoneal carcinomatosis are provided: silencing the Re

33 is relatively contraindicated in those with peritoneal carcinomatosis due to theoretical risk and re

34 urgery, prior radiation therapy, evidence of peritoneal carcinomatosis, and complications.

35 thirty three (median age 55, range 4-88) had peritoneal carcinomatosis.

36 e model of aggressive mammary cancer-induced peritoneal carcinomatosis.

37 oration during implantation of an indwelling peritoneal catheter.

38 ice displayed an increase in B1 cells in the peritoneal cavity (PerC) and secondary lymphoid organs a

39 for innate-like B cells localized within the peritoneal cavity and demonstrates a novel strategy to a

40 and escaped across the mesothelium into the peritoneal cavity and immediately infected GATA-binding

41 infection, ST2 detection is abrogated in the peritoneal cavity and lung, consistent with systemic eff

42 cantly enhanced the bacterial burdens in the peritoneal cavity and lung.

43 iated with a spread of infection both to the peritoneal cavity and retroperitoneum and result in a su

44 in mice promotes bacterial clearance in the peritoneal cavity and serves to facilitate the well-know

45 blishment of gammaherpesvirus latency in the peritoneal cavity and, to a lesser extent, viral reactiv

46 indicate that GATA6+ macrophages within the peritoneal cavity are a conduit of dissemination for i.v

47 and survival of ovarian cancer cells in the peritoneal cavity as nonadherent spheroids and their adh

48 cyte-derived antimicrobial protein, into the peritoneal cavity at a much higher level than did SCE.

49 determining region 3 sequences compared with peritoneal cavity B-1a cells.

50 lial migration or macrophage efflux from the peritoneal cavity but regulates macrophage migration thr

51 increased bacterial burden in the kidney and peritoneal cavity following GAS challenge.

52 icrobial RNases form a network to shield the peritoneal cavity from microbial invasion in patients un

53 er in the peritoneal pouch group than in the peritoneal cavity group.

54 upregulated on macrophages isolated from the peritoneal cavity in patients with peritonitis but not i

55 the sequential influx of immune cells in the peritoneal cavity in response to a bacterial stimulus th

56 Fibrin(ogen) deposition was noted in the peritoneal cavity in response to thioglycollate, with a

57 d SOD2 are necessary for colonization of the peritoneal cavity in vivo.

58 it routes, the movement of leukocytes in the peritoneal cavity is largely unknown.

59 Metastatic cancer involving spread to the peritoneal cavity is referred to as peritoneal carcinoma

60 e metastasis of their cancers throughout the peritoneal cavity leading to death.

61 cted GATA-binding factor 6-positive (GATA6+) peritoneal cavity macrophages.

62 The peritoneal cavity microenvironment is skewed toward immu

63 hitin-induced eosinophil accumulation in the peritoneal cavity occurs independent of GM-CSF, indicati

64 usly reported, macrophage migration into the peritoneal cavity of mice in response to thioglycollate

65 murine model, E. coli K1 grew rapidly in the peritoneal cavity of neonatal mice, causing fatal diseas

66 CM proteins to early metastatic onset in the peritoneal cavity remains unexplored.

67 of macrophage markers in spleen tissues and peritoneal cavity showed that the TRPV4 deficiency did n

68 ted latent gammaherpesvirus infection in the peritoneal cavity under conditions where the viral laten

69 ever, MAIT cell presence and function in the peritoneal cavity, a common anatomical site for infectio

70 ar aggregates (MCAs) from the tumor into the peritoneal cavity, adhesion to and retraction of periton

71 ed to defects in monocyte recruitment to the peritoneal cavity, and exogenous IL-12 restored monocyte

72 hal infection with high bacterial burdens in peritoneal cavity, blood and tissues and the infected mi

73 h in fat-associated lymphoid clusters in the peritoneal cavity, is associated with immune surveillanc

74 howed increased number of neutrophils in the peritoneal cavity, reduced bacterial load, and multiorga

75 is the passive dissemination of cells in the peritoneal cavity, remain largely unexplored.

76 after capsules were dispersed throughout the peritoneal cavity, the pO2 level was 61 +/- 11 mm Hg.

77 iding protection against tumor growth in the peritoneal cavity, thereby highlighting potential opport

78 ial amount of gas in the retroperitoneum and peritoneal cavity, which raised a suspicion of duodenal

79 ation of endometrial tissue, mostly into the peritoneal cavity.

80 number of neutrophils and macrophages in the peritoneal cavity.

81 the macrophage disappearance reaction in the peritoneal cavity.

82 ns, than macrophages from lung tissue or the peritoneal cavity.

83 ictates the cellular tropism of MHV68 in the peritoneal cavity.

84 n a strong influx of the phagocytes into the peritoneal cavity.

85 d if active thrombin was administered to the peritoneal cavity.

86 ng changes in the loading environment in the peritoneal cavity.

87 olves changes in the tropism of MHV68 in the peritoneal cavity.IMPORTANCE Liver X receptors (LXRs) ar

88 ost cell attachment, although the numbers of peritoneal CD20 B cells, CD4 and CD8 T cells, and CD14 m

89 l composition, ex vivo cytokine secretion by peritoneal cells or bone marrow derived macrophages.

90 and functional profiling of bone marrow and peritoneal cells provided a detailed road map of basophi

91 ing attenuated expression in IRF-7-deficient peritoneal cells.

92 reactivation of latent gammaherpesvirus from peritoneal cells.

93 Additionally, peritoneal collagen fibers adopted a more linear anisotr

94 ated lymphoid clusters (FALCs) that collects peritoneal contaminants and provides a first layer of im

95 ed neutrophil aggregation and the capture of peritoneal contaminants by omental FALCs.

96 d the mechanisms that mediate the capture of peritoneal contaminants during peritonitis.

97 nt and aggregation of neutrophils to capture peritoneal contaminants.

98 h immune surveillance and protection against peritoneal contaminants.

99 tional status in children undergoing chronic peritoneal dialysis (CPD) around the globe.

100 Liposome-supported peritoneal dialysis (LSPD) with transmembrane pH-gradien

101 ed by treatment modality-hemodialysis versus peritoneal dialysis (P<0.001 for interaction)-and was st

102 Acute peritoneal dialysis (PD) is an important low-cost treatm

103 ucosone-3-ene (3,4-DGE), which is present in peritoneal dialysis (PD) solutions after heat sterilizat

104 participants with end-stage renal disease on peritoneal dialysis (PD) underwent randomization and cro

105 complication in patients undergoing chronic peritoneal dialysis (PD), limiting the duration of PD as

106 ts with end stage kidney failure who receive peritoneal dialysis (PD).

107 Patients on haemodialysis or peritoneal dialysis are likely to be at increased risk o

108 e kidney was transplanted into a 61-year-old peritoneal dialysis dependent without complication.

109 Data from 14 patients undergoing peritoneal dialysis for end-stage renal disease but with

110 A total of 1,730 peritoneal dialysis patients in the CRC for ESRD prospec

111 e aim to assess mortality risk prediction in peritoneal dialysis patients using machine-learning algo

112 If peritoneal dialysis patients with high mCCI (>4) were ag

113 notable risk factors for mortality in Korean peritoneal dialysis patients.

114 elihood of pregnancy was seen among women on peritoneal dialysis than on hemodialysis (HR, 0.47; 95%

115 ic or uremic patients and of those receiving peritoneal dialysis treatment have increased levels of t

116 were on dialysis (73.6% hemodialysis; 26.4% peritoneal dialysis).

117 Two of the study's 59 patients were on peritoneal dialysis, and 57 were on hemodialysis.

118 om CKD rabbits, patients on hemodialysis and peritoneal dialysis, and HNE-modified HDL.

119 hemodialysis (either in-center or at home), peritoneal dialysis, and kidney transplant.

120 Removers remove excess fluid through peritoneal dialysis, aquaphoresis, or hemodialysis.

121 idney replacement therapies (KRTs, including peritoneal dialysis, continuous KRT, haemodialysis and h

122 eers, 25 patients on hemodialysis, and 20 on peritoneal dialysis.

123 rrhosis, indwelling catheters, or undergoing peritoneal dialysis.

124 ients treated, 81 had baseline mesenteric or peritoneal disease, among whom 5 (6%) experienced at lea

125 l obstruction in patients with mesenteric or peritoneal disease, likely by inducing inflammation.

126 patients eventually succumbed to progressive peritoneal disease.

127 cological malignancy and is characterized by peritoneal disseminated metastasis.

128 There is an anecdotal association between peritoneal dissemination and bile spillage during the in

129 EACAM1 is associated with poor prognosis and peritoneal dissemination of patients with gastric cancer

130 dent prognostic factor and a risk factor for peritoneal dissemination.

131 On the basis of peritoneal dose and feasibility for outpatient administr

132 Tunneled peritoneal drainage catheters are described as an effect

133 ng colon perforation after implantation of a peritoneal drainage in a patient with refractory ascites

134 apy included all interventions (antibiotics, peritoneal drains, resuscitation) excluding surgery.

135 D (hazard ratio [HR], 0.7; 95% CI, 0.3-1.4), peritoneal EHD (HR, 2.2; 95% CI, 1.1-4.2) and RAS/TP53 c

136 hat neutrophils are rapidly recruited to the peritoneal environment early after endometriotic lesion

137 in the characteristically immune-suppressive peritoneal environment presents a potential strategy to

138 Here, we report that mouse survival of peritoneal Escherichia coli sepsis is compromised by lon

139 creasing the range of drainage sites for the peritoneal expansion of lymphoid malignancies.

140 acity of EOC single cells and MCAs to murine peritoneal explants and impaired MCA survival and mesoth

141 ion and invasion, leading to colonization of peritoneal explants.

142 ) CTR (PCTR), and resolvin CTR in infectious peritoneal exudates and distal spleens, as well as inves

143 ere identified to be temporally regulated in peritoneal exudates and spleens.

144 ty acids (FFAs) and decrease in subcutaneous/peritoneal fat depots compared to non-tumor bearing cont

145 In the chlorhexidine gluconate-induced peritoneal fibrosis model, AAM s worsened the fibrosis,

146 uclease (RNase) A Superfamily are present in peritoneal fluid and increase during peritonitis in pati

147 SARS-CoV-2 was detected in peritoneal fluid at a higher concentration than in respi

148 Furthermore, the liposome-containing peritoneal fluid contained significantly higher ammonia

149 sult describing the presence of the virus in peritoneal fluid during an emergency surgical procedure

150 hat resident macrophages were nonadherent in peritoneal fluid during homeostasis.

151 Interestingly, peritoneal fluid from neonatal mice contained significan

152 Concentrations of IGF-1 were elevated in peritoneal fluid from women with endometriosis and posit

153 Detection of SARS-CoV-2 in peritoneal fluid has never been reported.

154 Oral DCA reduced peritoneal fluid lactate concentrations and endometriosi

155 supernatants and viable cells obtained from peritoneal fluid of chronic PD patients.

156 are elevated in the systemic circulation and peritoneal fluid of endometriosis patients; however, whe

157 Here, we show the following: 1) in peritoneal immune cells, particularly macrophages, from

158 eutrophil depletion altered the systemic and peritoneal immune microenvironment of mice with endometr

159 "castling" of metastatic tumor cells in the peritoneal immunoprivileged site.See related article by

160 in vivo studies were performed upon surgical peritoneal implantation of nanotextile implants in ortho

161 for malignancy were enlarged lymph nodes or peritoneal implants, high DWI signal greater than that i

162 In a murine model of peritoneal infection, the particulate form of the staphy

163 anticoagulants in individuals susceptible to peritoneal infections.

164 and alpha(D) (-/-) monocytes in the model of peritoneal inflammation and in vitro two-dimensional and

165 Tyrphostin AG825 decreased peritoneal inflammation in zymosan-treated mice, and inc

166 In the model of thioglycollate-induced peritoneal inflammation, the injection of cyclic P5 pept

167 Using an in vivo model of peritoneal inflammation, we compared the phenotype of ce

168 Peritoneal involvement in cancer is the harbinger of a p

169 ced mild iron deficiency decreased offspring peritoneal iron, decreased bacterial growth, and conferr

170 Additionally, VEGF levels measured in peritoneal lavage fluid were 300-fold lower compared to

171 e and lower IL-1beta levels were detected in peritoneal lavages from Cd44(-/-) mice (p < 0.01).

172 ways relating to oxidative stress in ectopic peritoneal lesions.

173 We also find that peritoneal LPMs infiltrate early ovarian tumours and tha

174 he serous macrophage pool and that targeting peritoneal LPMs may improve ovarian cancer outcomes.

175 y and have limited effects on neutrophil and peritoneal macrophage activity.

176 ins, but in vitro complement killing assays, peritoneal macrophage and whole blood stimulations, phag

177 or the self-renewal and maintenance of large peritoneal macrophages (LPMs), but not that of other tis

178 Peritoneal macrophages (PMs) regulate inflammation and c

179 nous transcriptional response of single-cell peritoneal macrophages after exposure to apoptotic cells

180 ed a significant but comparable reduction of peritoneal macrophages and lymphocytes, accompanied by a

181 Furthermore, murine peritoneal macrophages expressed an M2 marker arginase-1

182 ROS generation in RAW 264.7 macrophages and peritoneal macrophages from BALB/c mice.

183 and HFE:TFR1 complex (nonfunctional TFR1) in peritoneal macrophages from C57BL/6 mice, resulting in i

184 In vitro, peritoneal macrophages from IL-37Tg mice reduced LPS-ind

185 monocyte-derived macrophage (MDM) and mouse peritoneal macrophages has been shown to be strongly ass

186 a IL-1beta as well as IL-1beta production by peritoneal macrophages in a model of LPS-induced sepsis.

187 hat IL-4 activation of different lineages of peritoneal macrophages in mice is accompanied by lineage

188 ion of coagulation factor V (FV) by resident peritoneal macrophages in mice promotes bacterial cleara

189 ss I Ag presentation suppressed by ESAT-6 in peritoneal macrophages isolated from C57BL/6 mice.

190 Depletion of peritoneal macrophages or neutralization of endogenous I

191 Cultured mouse peritoneal macrophages release large numbers of ~30-nm c

192 those in the infected mice, and depletion of peritoneal macrophages rendered the mice significantly m

193 Enano had a higher binding affinity to mouse peritoneal macrophages than Enano.

194 consistent with that of GPCR, allowing mouse peritoneal macrophages to migrate toward its ligand CCL5

195 genes were specifically modified by exposing peritoneal macrophages to PS or PC liposomes in vivo.

196 Ex vivo stimulation studies using murine peritoneal macrophages were also used to elucidate the p

197 yl-beta-cyclodextrin (MbetaCD) to load mouse peritoneal macrophages with [(13)C]cholesterol.

198 stimulation of murine bone marrow-derived or peritoneal macrophages with IL-33 failed to promote argi

199 n this model in that in vitro stimulation of peritoneal macrophages with killed LAC-4 induced a simil

200 changes in circulating Ly6C(+) monocytes and peritoneal macrophages, along with increased CD36 expres

201 M1-polarized murine macrophages, as well as peritoneal macrophages, and was associated with increase

202 s or knockdown of the CD36 receptor in mouse peritoneal macrophages, confirming the specific binding

203 In mice deficient in GATA6+ peritoneal macrophages, neutrophils infiltrated more rob

204 d a significant reduction in survival within peritoneal macrophages.

205 lity of 0.776 in bovine MDM and 0.8 in mouse peritoneal macrophages.

206 urvived significantly less in BALB/c-derived peritoneal macrophages.

207 gulate MEK-1/2 and ERK-1/2 in BALB/c-derived peritoneal macrophages.

208 hages and in vivo by thioglycolate-recruited peritoneal macrophages.

209 During SBP, peritoneal MAIT cell frequencies increased most among al

210 This suggests that peritoneal MAIT cells could be of interest for immune-in

211 Peritoneal MAIT cells displayed an activated tissue-resi

212 mbers and impaired phenotype in circulation, peritoneal MAIT cells remain abundant, activated, and hi

213 However, peritoneal mast cell numbers were significantly lower in

214 RIalpha mAbs safely removed >98% of IgE from peritoneal mast cells and completely suppressed IgE-medi

215 erived mast cells and cytokine production in peritoneal mast cells.

216 nrenal removal of sodium directly across the peritoneal membrane (direct sodium removal [DSR]) with a

217 , compressive loads stimulated remodeling of peritoneal mesothelial cell surface ultrastructure via i

218 We have shown that human peritoneal mesothelial cells (HPMCs) recovered from the

219 establishes host-derived Wnt5a, expressed by peritoneal mesothelial cells and adipocytes, as a primar

220 TNT-mediated interaction between peritoneal mesothelial cells and OvCa cells was enhanced

221 toneal cavity, adhesion to and retraction of peritoneal mesothelial cells and subsequent anchoring.

222 the ovarian tumor microenvironment, notably peritoneal mesothelial cells and visceral adipose, secre

223 nt5a promoted ovarian tumor cell adhesion to peritoneal mesothelial cells as well as migration and in

224 .4 y, P = 0.0012), but not for patients with peritoneal mesothelioma (median survival 8.2 y versus 5.

225 Malignant peritoneal mesothelioma (MPeM) is a rare cancer of the m

226 nd 67-75% (median survival of 27 months) for peritoneal mesothelioma.

227 Colorectal Peritoneal metastases (CPM) develop in 15% of colorectal

228 Gastric cancer (GC) with peritoneal metastases (PMs) is a poor prognostic evoluti

229 Peritoneal metastases are the leading cause of morbidity

230 tern recognition exerts antitumor effects on peritoneal metastases by inducing classical complement c

231 roduced as a new treatment for patients with peritoneal metastases in November, 2011.

232 wever, multiclonal mixtures form large solid peritoneal metastases, populated almost entirely by CL31

233 rategies for the prevention and treatment of peritoneal metastases.

234 xtra-osseous (hepatic, pulmonary, nodal, and peritoneal) metastases.

235 ases (kappa = 0.856, agreement = 94.9%), and peritoneal metastasis (kappa = 0.772, agreement = 94.9%)

236 ritoneal administration for the treatment of peritoneal metastasis due to their physical stability, t

237 NK1 in OCCC cells also profoundly suppresses peritoneal metastasis in mouse implantation models of hu

238 oikis resistance, mesothelial clearance, and peritoneal metastasis in vitro and in vivo.

239 om patients during cytoreductive surgery for peritoneal metastasis of colon cancer with an aqueous so

240 a treatment option for refractory, isolated peritoneal metastasis of various origins.

241 Risk factors for overall survival and peritoneal metastasis were calculated based on CEACAM1 e

242 malignant ascites accompanied by widespread peritoneal metastasis.

243 knockout of host WNT5A significantly reduced peritoneal metastatic tumor burden.

244 on ovarian tumor cells and components of the peritoneal microenvironment using a panel of in vitro, e

245 of acute peritonitis and show that elevated peritoneal miR-223 and reduced miR-31 levels were useful

246 n in RAW264.7 cells, bone marrow-derived and peritoneal mouse macrophages, as well as human monocyte-

247 Peritoneal neutrophils from mHO-1 KO mice exhibited high

248 he total number of patients with preexisting peritoneal or mesenteric disease.

249 ipants with stage III or IV ovarian, primary peritoneal, or fallopian tube cancer (newly diagnosed, c

250 stage III to IV epithelial ovarian, primary peritoneal, or fallopian tube cancers, and an ECOG perfo

251 rade serous or endometrioid ovarian, primary peritoneal, or fallopian tube carcinoma and an Eastern C

252 ically confirmed epithelial ovarian, primary peritoneal, or fallopian tube carcinoma with first disea

253 gy results do not show any toxicity to major peritoneal organs.

254 the frequency of GATA6(+) macrophages in the peritoneal, pleural and pericardial cavities.

255 compare Mtb growth in mouse alveolar (AMs), peritoneal (PMs), and liver (Kupffer cells; KCs) macroph

256 Peritoneal pO2 levels were measured on days 0 and 7 usin

257 The peritoneal pO2 of normal NHPs is relatively low and we p

258 A peritoneal pouch graft with high tissue volume (1000 isl

259 Serum miR-375 was significantly lower in the peritoneal pouch group than in the peritoneal cavity gro

260 Peritoneal pouch islet grafts showed high neovasculariza

261 BL/6 donor islets were transplanted into the peritoneal pouch of diabetic mouse recipients.

262 Human islets transplanted into the peritoneal pouch of diabetic nude mice also reversed hyp

263 splantation of 300 syngeneic islets into the peritoneal pouch of recipients reversed hyperglycemia fo

264 Islets transplanted into a dissected peritoneal pouch show high efficiency to reverse diabete

265 A peritoneal pouch was formed by dissecting the parietal p

266 oth the young and aged mice as compared with peritoneal PtC(+) B-1a cells.

267 Whereas the peritoneal PtC(+) population increased V(H)12 use with a

268 The infection induces acute peritoneal recruitment of neutrophils and other innate i

269 Mean (+/- SD) projected peritoneal residence time was 22.4 +/- 7.9 hours.

270 ction with the collagen matrix, and extended peritoneal residence time.

271 two patients (48, three, and one with DSRCT, peritoneal rhabdomyosarcoma, and Ewing sarcoma, respecti

272 l-type clusters, promoting a miliary mode of peritoneal seeding that complicates surgical removal and

273 In a peritoneal sepsis murine model, the minimum lethal dose

274 Surgical treatment (peritoneal shunt) was required for 2 patients (1 with VS

275 termine the locations of capsules within the peritoneal space on days 0 and 7.

276 dose-dependent ammonia sequestration in the peritoneal space provide a strong basis for the clinical

277 ating efficient ammonia sequestration in the peritoneal space.

278 ints of vulnerability for therapy to disrupt peritoneal spread and adhesion of ovarian cancer cells.

279 t cell transition to nonadherent form during peritoneal spread and metastasis.

280 Peritoneal spread is the primary mechanism of metastasis

281 trepton (FOXM1 inhibitor) reduced growth and peritoneal spread of ovarian cancer cells more effective

282 and CD133+ stem cells in the injury sites of peritoneal surface at day 5 post-operation.

283 therapeutic strategy commonly used to treat peritoneal surface malignancies.

284 carcinomas typically expand and spread over peritoneal surfaces.

285 alterations were associated with restricted peritoneal susceptibility to metastatic colonization by

286 mation in rats using laparotomy with several peritoneal sutures to produce the adhesions.

287 Here we demonstrate that peritoneal tissue-resident macrophages use an active inh

288 Peritoneal tissues from healthy mice injected with LPA e

289 floating cells/MCAs and the response of host peritoneal tissues to LPA remain unclear.

290 ecific characteristics that led to increased peritoneal tumor accumulation using MCM-41 type mesoporo

291 ound significant deposition of IgM and C3 on peritoneal tumor cells as early as 5 days post-treatment

292 of Zic1 suppressed both lung metastasis and peritoneal tumor dissemination of GC in mice.

293 We also investigated the components of the peritoneal tumor stroma that facilitated nanoparticle-tu

294 odel which forms malignant ascites and solid peritoneal tumors upon intraperitoneal transplantation i

295 ing nanoparticle concentrations in avascular peritoneal tumors, little is known about the mechanism o

296 oncentrations of nanoparticles in metastatic peritoneal tumors.

297 vaginal microenvironment after laparoscopic peritoneal vaginoplasty might play an important role in

298 ts with artificial vagina after laparoscopic peritoneal vaginoplasty were included in this study.

299 tures in patients who underwent laparoscopic peritoneal vaginoplasty.

300 ginal dysbiosis is common after laparoscopic peritoneal vaginoplasty.