ライフサイエンスコーパス: peritoneal cavity

1 ng changes in the loading environment in the peritoneal cavity.

2 um along with 150 ml of hemorrhagic fluid in peritoneal cavity.

3 y cytokines and form implants throughout the peritoneal cavity.

4 in microglia and resident macrophages of the peritoneal cavity.

5 neal mucinous carcinomatosis confined to the peritoneal cavity.

6 productive antitumor immune response in the peritoneal cavity.

7 circumvented by T cell administration to the peritoneal cavity.

8 emically, highlighting the uniqueness of the peritoneal cavity.

9 ation of endometrial tissue, mostly into the peritoneal cavity.

10 ively activated macrophages (AAM) within the peritoneal cavity.

11 tial step of neutrophil recruitment into the peritoneal cavity.

12 n a few hours of IL-1beta injection into the peritoneal cavity.

13 was observed in CD8(+) memory T cells in the peritoneal cavity.

14 eby, promoting neutrophil migration into the peritoneal cavity.

15 production after LPS injection in the murine peritoneal cavity.

16 tress as biologic processes activated in the peritoneal cavity.

17 tation of human endometrial fragments in the peritoneal cavity.

18 on pathophysiological alterations within the peritoneal cavity.

19 nude mice had LS174T tumors implanted in the peritoneal cavity.

20 rate and metastasize over the surface of the peritoneal cavity.

21 cal attributes of S. aureus infection in the peritoneal cavity.

22 receptor (M-CSFR) in both the blood and the peritoneal cavity.

23 patients, the disease remains limited to the peritoneal cavity.

24 a integrins to interstitial collagens in the peritoneal cavity.

25 paracrine signaling from hepatocytes in the peritoneal cavity.

26 th dozens to hundreds of tumors studding the peritoneal cavity.

27 cells that reside in the normal, uninflamed peritoneal cavity.

28 spleens, peripheral blood, lungs, liver and peritoneal cavity.

29 and neutrophils in cremaster tissue and the peritoneal cavity.

30 IL-17-mediated monocyte recruitment into the peritoneal cavity.

31 ion of labeled donor cells into the mertk-/- peritoneal cavity.

32 robots that are inserted completely into the peritoneal cavity.

33 the fallopian tube, or the mesothelium-lined peritoneal cavity.

34 whole kidney marrow and abundant within the peritoneal cavity.

35 single layer of mesothelial cells lining the peritoneal cavity.

36 f Pseudomonas aeruginosa introduced into the peritoneal cavity.

37 cer, was labeled by virus injection into the peritoneal cavity.

38 host response to bacterial infection of the peritoneal cavity.

39 from lung containing metastases, spleen, and peritoneal cavity.

40 rol modestly, especially when given into the peritoneal cavity.

41 mically via an osmotic pump implanted in the peritoneal cavity.

42 number of neutrophils and macrophages in the peritoneal cavity.

43 andomized selection of the 9 segments of the peritoneal cavity.

44 lantation of autologous endometrium into the peritoneal cavity.

45 enhanced neutrophil recruitment to inflamed peritoneal cavity.

46 the macrophage disappearance reaction in the peritoneal cavity.

47 ns, than macrophages from lung tissue or the peritoneal cavity.

48 ictates the cellular tropism of MHV68 in the peritoneal cavity.

49 n a strong influx of the phagocytes into the peritoneal cavity.

50 nnate-like B-cell population enriched in the peritoneal cavity.

51 loid-derived suppressor cells (MDSCs) in the peritoneal cavity.

52 d if active thrombin was administered to the peritoneal cavity.

53 fluids reduce immunological defenses in the peritoneal cavity.

54 in mice when co-injected with CXCL6 into the peritoneal cavity.

55 , resolving, and chronic inflammation in the peritoneal cavity.

56 he cecum and placing it into the recipient's peritoneal cavity.

57 ntenance of IgM(+) B cells in the spleen and peritoneal cavity.

58 +)IgD(lo) B cells in both the spleen and the peritoneal cavity.

59 eater LPS-dependent production of TNF in the peritoneal cavity.

60 presence of small undetected nodules in the peritoneal cavity.

61 pus, bromodeoxyuridine was injected into the peritoneal cavity.

62 ld amount of free fluid was also seen in the peritoneal cavity.

63 Most of the BM-MSCs formed aggregates in the peritoneal cavity.

64 ltration of neutrophils and T cells into the peritoneal cavity.

65 ents of endometriotic lesion survival in the peritoneal cavity.

66 and tumor metastasis within the pleural and peritoneal cavities.

67 mbrane chambers that were implanted into rat peritoneal cavities.

68 In mast cells harvested from peritoneal cavities, 2 cytokine signals are required for

69 es (4/5), spleens (4/10), livers (6/10), and peritoneal cavities (8/10).

70 ynecological malignancies disseminate in the peritoneal cavity - a condition known as peritoneal carc

71 ever, MAIT cell presence and function in the peritoneal cavity, a common anatomical site for infectio

72 In the peritoneal cavity, a further loss was demonstrated early

73 r CLP with a concomitant increase within the peritoneal cavity, a pattern that was ablated in CXCR3-d

74 ar aggregates (MCAs) from the tumor into the peritoneal cavity, adhesion to and retraction of periton

75 rotein 10, IP-10) increase in plasma and the peritoneal cavity after CLP, peak at 8 hours after infec

76 Migration of inflammatory monocytes into the peritoneal cavity after CLP, which is dependent on VLA-4

77 However, the peritoneal cavity also contains relatively high frequenc

78 crophage population within bacteria-infected peritoneal cavities and increased the systemic level of

79 septic shock, in part, by migration into the peritoneal cavity and amplification of the proinflammato

80 ancer cells primarily disseminate within the peritoneal cavity and are only superficially invasive.

81 for innate-like B cells localized within the peritoneal cavity and demonstrates a novel strategy to a

82 esiding in nonlymphoid organs, including the peritoneal cavity and fat pads, are more resistant to ap

83 Mesothelial cells (MCs) line the peritoneal cavity and help define peritoneal response to

84 and escaped across the mesothelium into the peritoneal cavity and immediately infected GATA-binding

85 In the peritoneal cavity and liver, C1q enhancement of type 2 m

86 infection, ST2 detection is abrogated in the peritoneal cavity and lung, consistent with systemic eff

87 cantly enhanced the bacterial burdens in the peritoneal cavity and lung.

88 eated transfused neutrophils to the inflamed peritoneal cavity and lungs was significantly elevated.

89 re, neutrophil recruitment into the inflamed peritoneal cavity and lymphocyte homing to secondary lym

90 veloped increased neutrophil influx into the peritoneal cavity and more efficient bacterial clearance

91 cells (IgM>>IgG) that migrate rapidly to the peritoneal cavity and persist there indefinitely, ready

92 ary, they disseminate into the organs of the peritoneal cavity and produce ascites, typical of ovaria

93 iated with a spread of infection both to the peritoneal cavity and retroperitoneum and result in a su

94 n of several chemokines and cytokines in the peritoneal cavity and serum (CCL2, CCL3, CCL4, CCL11, CX

95 in mice promotes bacterial clearance in the peritoneal cavity and serves to facilitate the well-know

96 mentum collected antigens and cells from the peritoneal cavity and supported T cell-dependent B cell

97 red cytokine and chemokine production in the peritoneal cavity and suppressed neutrophil recruitment.

98 inhibits infiltration of macrophages to the peritoneal cavity and suppresses the activity of TNF-alp

99 d on B1 cells outside of the bone marrow and peritoneal cavity and that pathogenic natural IgM titers

100 B-1a cells reside in both the peritoneal cavity and the spleen.

101 tigens, particulates, and pathogens from the peritoneal cavity and, depending on the stimuli, promoti

102 blishment of gammaherpesvirus latency in the peritoneal cavity and, to a lesser extent, viral reactiv

103 sent <1% of total B cells, accumulate in the peritoneal cavity, and account for near-normal levels of

104 om the colon, blood, thymus, spleen, uterus, peritoneal cavity, and allergen-challenged lung.

105 tion of IFN-regulated gene expression in the peritoneal cavity, and an increased production of myeloi

106 bdominal aorta) and catheters (jugular vein, peritoneal cavity, and distal abdominal aorta).

107 ed to defects in monocyte recruitment to the peritoneal cavity, and exogenous IL-12 restored monocyte

108 e bone marrow, peripheral blood, spleen, and peritoneal cavity, and improved responses to T-independe

109 se model, augmented cfB levels in the serum, peritoneal cavity, and major organs including the kidney

110 GM3 BLT mice and populate the immune system, peritoneal cavity, and peripheral tissues.

111 spontaneous metastasis to the lungs, to the peritoneal cavity, and resulted in 90% lethality within

112 , the persistence of kappa+ B-1 cells in the peritoneal cavity, and significant levels of serum IgM,k

113 lorohydrins were also instilled in the rats' peritoneal cavity, and their effects on peritoneal macro

114 indicate that GATA6+ macrophages within the peritoneal cavity are a conduit of dissemination for i.v

115 opped gallstones in the peritoneal and extra-peritoneal cavity are usually asymptomatic.

116 imarily produced by B1a cells outside of the peritoneal cavity, are integrally involved.

117 and B-2 cell numbers increased in lungs and peritoneal cavity as early as day 1 postinfection, but l

118 and survival of ovarian cancer cells in the peritoneal cavity as nonadherent spheroids and their adh

119 ring nude mice, PTX was best retained in the peritoneal cavity as PTX-gel (microparticulate PTX entra

120 ion of polymorphonuclear leukocytes into the peritoneal cavity, as well as abdominal writhings.

121 ave fewer cells than controls present in the peritoneal cavity, as well as fewer leukocytes leaving t

122 ger RNA for 145 C. albicans genes within the peritoneal cavity at 48 hours.

123 cyte-derived antimicrobial protein, into the peritoneal cavity at a much higher level than did SCE.

124 Importantly, IL-10 production by peritoneal cavity B cells significantly reduced disease

125 Bone marrow and spleen mature B cell and peritoneal cavity B-1 cell numbers were also halved, whe

126 Peritoneal cavity B-1a B cells exhibited the most acapsu

127 Indeed, mature peritoneal cavity B-1a cells migrated to BM in a CXCR4-d

128 determining region 3 sequences compared with peritoneal cavity B-1a cells.

129 Innate-like splenic marginal zone (MZ) and peritoneal cavity B1 B lymphocytes share critical respon

130 In this study, peritoneal cavity B10 cells shared similar cell surface

131 The BCR repertoire of peritoneal cavity B10 cells was diverse, as occurs in th

132 However, peritoneal cavity B10 cells were 10-fold more frequent a

133 Cdkn2c expression in the splenic B cells and peritoneal cavity B1a cells from Sle2c1-carrying mice, w

134 lenic B cells and increased proliferation of peritoneal cavity B1a cells.

135 ing rate and cumulative dose released in the peritoneal cavity based on the in vitro release data.

136 d in the spleen but remained elevated in the peritoneal cavity beyond 40 d postimmunization.

137 hal infection with high bacterial burdens in peritoneal cavity, blood and tissues and the infected mi

138 strointestinal system as well as pleural and peritoneal cavities but not the brain.

139 lial migration or macrophage efflux from the peritoneal cavity but regulates macrophage migration thr

140 e expression in macrophages infiltrating the peritoneal cavity but was not observed in the resident p

141 n be used to visualize ovarian tumors in the peritoneal cavity by multimodal MR and near infra-red im

142 al pathogens and affect the cells lining the peritoneal cavity by triggering local inflammation and i

143 Cytofluorimetry of splenocytes and peritoneal cavity cells of MDA-LDL- or heat shock protei

144 rticles, which distribute more evenly in the peritoneal cavity compared to the large microparticles,

145 ited invasion of inflammatory cells into the peritoneal cavity compared with mice deficient in CD40 a

146 eased accumulation of neutrophils within the peritoneal cavity compared with wild-type control mice,

147 phaMbeta2 suppressed Mvarphi egress from the peritoneal cavity, decreased the production of anti-infl

148 igen receptor stimulation that is blunted in peritoneal cavity-derived B1 B cells.

149 8(+) splenocytes from dnTGF-betaRII mice and peritoneal cavity-derived, but not spleen-derived, CD19(

150 ts with PCI, the presence of free air in the peritoneal cavity does not confirm an intestinal perfora

151 the accumulation of lipid-rich lymph in the peritoneal cavity due to disruption of the lymphatic sys

152 The efficacy of irrigating the peritoneal cavity during appendectomy for perforated app

153 hermore, addition of exogenous PGE2 into the peritoneal cavity during infection overrode the protecti

154 t natural killer (NK) cells migrate into the peritoneal cavity during intraabdominal sepsis, but the

155 ion of hepatocytes with microcarriers in the peritoneal cavity efficiently rescued animals with liver

156 -reduction by gaining direct access into the peritoneal cavity, enabling elevated drug levels versus

157 and antigen presentation occurred within the peritoneal cavity, even in the presence of highly activa

158 Similarly, iNKT cells from the human peritoneal cavity expressed lower transcription factor l

159 increased bacterial burden in the kidney and peritoneal cavity following GAS challenge.

160 provide effective drug concentrations in the peritoneal cavity for an extended period of time.

161 nesis and proinflammatory environment in the peritoneal cavity for the establishment and maintenance

162 When injected into the peritoneal cavity, free and liposomal alendronic acid we

163 icrobial RNases form a network to shield the peritoneal cavity from microbial invasion in patients un

164 er in the peritoneal pouch group than in the peritoneal cavity group.

165 ice with inflammation of the airways, or the peritoneal cavity had poor priming ability resulting in

166 ollectively, these data demonstrate that the peritoneal cavity has a unique environment capable of el

167 numerically small B10 cell subset within the peritoneal cavity has regulatory function and is importa

168 s in large microcapsules transplanted in the peritoneal cavity have failed to reverse diabetes in hum

169 owed decreased macrophage migration into the peritoneal cavity; however, MP inhibited this effect.

170 olves changes in the tropism of MHV68 in the peritoneal cavity.IMPORTANCE Liver X receptors (LXRs) ar

171 multiple tissues, including the pleural and peritoneal cavities in mice.

172 distributed solid tumors and ascites in the peritoneal cavity in 100% of animals.

173 , Ly6C(+) monocytes constitutively enter the peritoneal cavity in a CCR2-dependent manner, where they

174 promote the migration of neutrophils to the peritoneal cavity in a CXCR2-dependent manner.

175 eased the levels of several cytokines in the peritoneal cavity in Cot/tpl2 KO mice compared with Wt c

176 howed a diminished ability to migrate to the peritoneal cavity in cox-2(-/-) mice.

177 upregulated on macrophages isolated from the peritoneal cavity in patients with peritonitis but not i

178 the sequential influx of immune cells in the peritoneal cavity in response to a bacterial stimulus th

179 ion of elicited monocytes/macrophages in the peritoneal cavity in response to inflammatory thioglycol

180 nocytes were preferentially recruited to the peritoneal cavity in response to pristane.

181 Fibrin(ogen) deposition was noted in the peritoneal cavity in response to thioglycollate, with a

182 gammadelta T cells accumulated in the peritoneal cavity in response to tumor challenge and cou

183 d SOD2 are necessary for colonization of the peritoneal cavity in vivo.

184 rcentage of Dectin-1-expressing cells in the peritoneal cavity increased by 774% with cecal ligation

185 W/W(v) mice had neutrophil migration to the peritoneal cavity, increased CXCR2 expression, and reduc

186 ired for survival and abscess formation in a peritoneal cavity infection model.

187 lation of the footpad, or inoculation of the peritoneal cavity (intraperitoneal [i.p.] inoculation).

188 retention of surgical foreign bodies in the peritoneal cavity is estimated to occur once in every 10

189 it routes, the movement of leukocytes in the peritoneal cavity is largely unknown.

190 Tumor cell metastasis to the peritoneal cavity is observed in patients with tumors of

191 The peritoneal cavity is recognized as an important site for

192 Metastatic cancer involving spread to the peritoneal cavity is referred to as peritoneal carcinoma

193 though colonization of Peyer patches and the peritoneal cavity is significantly impaired.

194 h in fat-associated lymphoid clusters in the peritoneal cavity, is associated with immune surveillanc

195 When injected into the mouse peritoneal cavity, lactoferrin also caused a marked recr

196 e metastasis of their cancers throughout the peritoneal cavity leading to death.

197 flammation in the mouse cremaster muscle and peritoneal cavity led to ICAM-1 expression on intravascu

198 The simulated drug release in the peritoneal cavity linearly correlated with treatment eff

199 In contrast, peritoneal cavity macrophages basally express NAIP1 and

200 cted GATA-binding factor 6-positive (GATA6+) peritoneal cavity macrophages.

201 These findings suggest that spread to the peritoneal cavity may require no or very little further

202 The peritoneal cavity microenvironment is skewed toward immu

203 m (n = 4), portal venous system (n = 1), and peritoneal cavity (n = 1).

204 hitin-induced eosinophil accumulation in the peritoneal cavity occurs independent of GM-CSF, indicati

205 this subset, macrophages were collected from peritoneal cavities of mice injected with saline, thiogl

206 on macrophage foam cell formation within the peritoneal cavities of mice.

207 ng freshly isolated human PEL cells into the peritoneal cavities of NOD/SCID mice without in vitro ce

208 L cells were detected in both the spleen and peritoneal cavity of animals for up to 75 days.

209 , respectively, n = 9) was injected into the peritoneal cavity of female Lewis rats 30 min before int

210 lation was found in spleen, bone marrow, and peritoneal cavity of humanized mice and included distinc

211 e bone marrow, spleen, peripheral blood, and peritoneal cavity of JAK2V617F transgenic mice than from

212 lasmacytoid DCs, T cells, and B cells in the peritoneal cavity of mertk-/- mice when compared with wi

213 6(-)PD-L2(-)MHCII(-)UCP1(+) phenotype in the peritoneal cavity of mice and during the formation of li

214 directly to s.c. xenograft tumors and to the peritoneal cavity of mice bearing primary and metastatic

215 capable of imaging hydrogen peroxide in the peritoneal cavity of mice during a lipopolysaccharide-in

216 usly reported, macrophage migration into the peritoneal cavity of mice in response to thioglycollate

217 ure from a flow stream in vitro and from the peritoneal cavity of mice in vivo.

218 nclude fabrication and implantation into the peritoneal cavity of mice, incubation, retrieval via per

219 yi nematodes are implanted surgically in the peritoneal cavity of mice, we identified differential ex

220 ith these tachyzoites once inoculated in the peritoneal cavity of mice.

221 measure the amount of ovarian tumors in the peritoneal cavity of mice.

222 oliferation was drastically decreased in the peritoneal cavity of Nbs1(B/B) mice.

223 murine model, E. coli K1 grew rapidly in the peritoneal cavity of neonatal mice, causing fatal diseas

224 rowing subcutaneously or multifocally in the peritoneal cavity of nonobese diabetic/severe combined i

225 capsulated canine pancreatic islets into the peritoneal cavity of pancreatectomized canines.

226 unting long-term recall responses enrich the peritoneal cavity of PD patients.

227 The IL-6-rich peritoneal cavity of SHIP(-/-) mice shows more than 700-

228 ters were subsequently transplanted into the peritoneal cavity of streptozotocin-induced diabetic sev

229 CD8+ T cells in the peritoneal cavity of TCF-1-deficient mice had decreased

230 trophils compared with WT neutrophils in the peritoneal cavity of TG-exposed WT mice.

231 attenuated recruitment of neutrophils in the peritoneal cavity of TRPC6(-/-) mice.

232 Fewer DCs also accumulated in the peritoneal cavity of UV-chimeric mice (ie, mice transpla

233 both in a Transwell chamber assay and in the peritoneal cavity of wild-type (WT) mice.

234 l as the recruitment of neutrophils into the peritoneal cavity of wild-type mice, but not RICK-defici

235 Within the peritoneal cavity, omental tissue is a common site for m

236 effect, myeloid cells were isolated from the peritoneal cavity or bone marrow.

237 ad extravasation of contrast medium into the peritoneal cavity or the presacral space on a postoperat

238 clusters into the mesothelial cell lining of peritoneal cavity organs; however, the tumor-specific fa

239 There is no advantage to irrigation of the peritoneal cavity over suction alone during laparoscopic

240 ansplanted into the hepatic sinus (H group), peritoneal cavity (P group), omentum (O group), and kidn

241 d that the presence of a foreign body in the peritoneal cavity (PC) might alter the inflammatory resp

242 ice displayed an increase in B1 cells in the peritoneal cavity (PerC) and secondary lymphoid organs a

243 CDR-3 of the Ig H chain (CDR-H3) content in peritoneal cavity (PerC) B cells, we analyzed the compos

244 Peritoneal cavity (PerC) B-1 cells have long been known

245 The peritoneal cavity (PerC) is a unique compartment within

246 In vivo, the introduction of sHz into the peritoneal cavity produces an inflammatory response char

247 vehicle for cancer cell dissemination in the peritoneal cavity, protecting cells from environmental s

248 howed increased number of neutrophils in the peritoneal cavity, reduced bacterial load, and multiorga

249 is the passive dissemination of cells in the peritoneal cavity, remain largely unexplored.

250 Fibrosis of the peritoneal cavity remains a serious, life-threatening pr

251 CM proteins to early metastatic onset in the peritoneal cavity remains unexplored.

252 hese Ab-secreting cells (ASC) originate from peritoneal cavity-resident cells, because transfer of pe

253 he homeostasis, function and localization of peritoneal cavity-resident macrophages.

254 l-free viral transmission of HHV-6A into the peritoneal cavity resulted in detectable viral DNA in at

255 oss of vascular fluid into the intestine and peritoneal cavity, resulting in rapid (less than 30 min)

256 responses at the site of initial infection (peritoneal cavity) revealed that CLP TRPV1KO mice exhibi

257 ently clear Klebsiella pneumoniae from their peritoneal cavities reveals an essential role for this s

258 of macrophage markers in spleen tissues and peritoneal cavity showed that the TRPV4 deficiency did n

259 II, CD80, and CD86) become detectable in the peritoneal cavity, skin, lung, and lymph nodes under inf

260 y imaged in most mouse organs, including the peritoneal cavity, stomach, small intestine, and colon.

261 al and regional malignancies confined in the peritoneal cavity such as ovarian cancer.

262 ized to nearby and distant organs within the peritoneal cavity, such as abdominal lymph nodes, mesent

263 on of cells and cellular aggregates into the peritoneal cavity, survival of matrix-detached cells in

264 hours after infection, and are higher in the peritoneal cavity than in plasma.

265 ontrast to recent studies in the pleural and peritoneal cavities, the proliferation of resident and a

266 after capsules were dispersed throughout the peritoneal cavity, the pO2 level was 61 +/- 11 mm Hg.

267 iding protection against tumor growth in the peritoneal cavity, thereby highlighting potential opport

268 ributed to the retention of B-1 cells in the peritoneal cavity through high expression of the chemoki

269 d redistribution of radioactivity out of the peritoneal cavity to circulating blood, which cleared vi

270 and monocytes are rapidly recruited into the peritoneal cavity to control infection, but the role of

271 migration of cox-2(-/-) macrophages from the peritoneal cavity to lymph nodes, as well as cell adhesi

272 fluids continuously circulating through the peritoneal cavity to rapidly induce and maintain hypothe

273 pairs dendritic cell (DC) migration from the peritoneal cavity to the draining lymph node in mice imm

274 that microbes disseminated rapidly from the peritoneal cavity to the lung and spleen, where they rep

275 that CD11c(+)Gr-1(+) cells migrate from the peritoneal cavity to the spleen.

276 Following release into the peritoneal cavity, tumor cells rapidly attach to the ome

277 ted latent gammaherpesvirus infection in the peritoneal cavity under conditions where the viral laten

278 induce the recruitment of neutrophils in the peritoneal cavity via CXCR2.

279 The detection of tumors in the peritoneal cavity was exclusively possible with sequenti

280 Access to the peritoneal cavity was gained using a modified blunt dila

281 107 colony-forming units); 24 hrs later, the peritoneal cavity was lavaged and the major organs were

282 ment or localization of B10 cells within the peritoneal cavity was not dependent on the presence of c

283 utrophil and monocyte extravasation into the peritoneal cavity was severely reduced in St3Gal4(-/-) m

284 ent neutrophil recruitment into the inflamed peritoneal cavity was sharply suppressed in Tyrobp(-/-)

285 o recruitment of these cells to the inflamed peritoneal cavity was significantly enhanced.

286 However, using the peritoneal cavity, we found that although innate immune-

287 rophages in the spleen, lymph nodes, and the peritoneal cavity were reduced.

288 ells from blood and spleen into the inflamed peritoneal cavity where they appear to facilitate the ac

289 m circulating monocytes and recruited to the peritoneal cavity where they differentiate into macropha

290 s the main site of neutrophil entry into the peritoneal cavity, where MMP-2 and MMP-9 act synergistic

291 t arise in spleen but rapidly migrate to the peritoneal cavity, where they persist indefinitely but d

292 rmed metastasis in mice when injected in the peritoneal cavity, whereas aggressive ovarian cancer cel

293 e content of the resident macrophages in the peritoneal cavity, whereas it reduced the content of the

294 RF2 normally populated the marginal zone and peritoneal cavity, whereas mice using inverted RF1 had i

295 CCR3 and CCR6, but not CXCR3, to home to the peritoneal cavity, whereas Th9 homing to the CNS during

296 ortion of the autoislets placed within their peritoneal cavities, which appeared to be functioning no

297 red for Ly6C(hi) monocyte trafficking to the peritoneal cavity, which is thought to be one of the ini

298 cancer cells disseminate readily within the peritoneal cavity, which promotes metastasis, and are of

299 ial amount of gas in the retroperitoneum and peritoneal cavity, which raised a suspicion of duodenal

300 multicellular aggregates or spheroids in the peritoneal cavity, which seed abdominal surfaces and org