ライフサイエンスコーパス: peritonitis

1 neutrophils at FALCs during zymosan-induced peritonitis.

2 ects and tissue remodeling during PD-related peritonitis.

3 n in perforated diverticulitis with purulent peritonitis.

4 thicillin-resistant S. aureus (MRSA) induced peritonitis.

5 beta and IL-18, and increased sensitivity to peritonitis.

6 n in a mouse model of Streptococcus pyogenes peritonitis.

7 tion is indicated to prevent perforation and peritonitis.

8 terations and transport defects during acute peritonitis.

9 ity rate comparable to spontaneous bacterial peritonitis.

10 therapeutic strategies for PD-related fungal peritonitis.

11 l response exacerbates septic progression of peritonitis.

12 substantially reduce the risk of PD-related peritonitis.

13 al variceal bleed, and spontaneous bacterial peritonitis.

14 cute perforated diverticulitis with purulent peritonitis.

15 parotomy for indication of acute generalized peritonitis.

16 soon after infection but not during sterile peritonitis.

17 the inflammatory site in an in vivo model of peritonitis.

18 he capture of peritoneal contaminants during peritonitis.

19 il transudation during colitis, skin rash or peritonitis.

20 lammatory syndrome and urate crystal-induced peritonitis.

21 P = .01) for patients who did not have fecal peritonitis.

22 ith adrenal insufficiency from polymicrobial peritonitis.

23 t sites of inflammation in a murine model of peritonitis.

24 l IL-1beta production following Alum-induced peritonitis.

25 gate the role of mast cells in severe septic peritonitis.

26 ammation and rescues mice from polymicrobial peritonitis.

27 cy in monosodium urate (MSU) crystal-induced peritonitis.

28 ntamoeba histolytica as well as to bacterial peritonitis.

29 nfection, and possibly spontaneous bacterial peritonitis.

30 model of sodium periodate (NaIO(4))-induced peritonitis.

31 s regulate neutrophil trafficking to control peritonitis.

32 and sepsis that are symptomatic of bacterial peritonitis.

33 in the macrophages and in an animal model of peritonitis.

34 t lymphangiogenesis in patients with UFF and peritonitis.

35 in patients undergoing PD with a history of peritonitis.

36 eritoneal membranes of patients with UFF and peritonitis.

37 yte infiltration in an experimental model of peritonitis.

38 ory exudates during the development of acute peritonitis.

39 indicating their potential as biomarkers of peritonitis.

40 ophils and macrophages in a model of sterile peritonitis.

41 ts EAF formation during OAT in patients with peritonitis.

42 tality in patients with pneumoperitoneum and peritonitis.

43 nterococcus faecium, using a murine model of peritonitis.

44 ing anastomotic leakage, pelvic abscess, and peritonitis.

45 ts were observed in a model of acute sterile peritonitis.

46 scites is a variant of spontaneous bacterial peritonitis.

47 hepatorenal syndrome (1.22 [1.21-1.22]), and peritonitis (1.18 [1.17-1.20]) had higher chances of hos

48 ma (2.7%), and intra-abdominal abscess/other peritonitis (1.9%).

49 All patients with fecal peritonitis (15 patients in the laparoscopic peritoneal

50 Deep SSIs included abscesses (58%), peritonitis (28%), deep incisional infections (8%), and

51 33 (1.06-1.65)], abscess [2.18 (1.60-2.97)], peritonitis [3.14 (1.99-4.97)], sepsis [1.88 (1.29-2.73)

52 proves survival during Klebsiella pneumoniae peritonitis (67 versus 23%, p < 0.005) in mice.

53 PF-562271 reduced monosodium urate-mediated peritonitis, a disease model used for studying the conse

54 delayed resolution of zymosan-induced murine peritonitis accompanied by decreased SPM levels without

55 nic inflammatory diseases in mouse models of peritonitis, acute lung injury, and atherosclerosis.

56 patients with proven diagnosis of secondary peritonitis admitted to the ICU were included in the stu

57 a PEG tube intra-abdominally with associated peritonitis after more than a month of PEG placement and

58 In Ag-induced peritonitis (AIP), thioglycolate-induced peritonitis, an

59 t in vivo models of disease (crystal-induced peritonitis, allergic airway inflammation and psoriasis)

60 up to 50, and impaired zymosan-induced mouse peritonitis along with reduced 5-LO product levels.

61 t were subjected to surgically induced fecal peritonitis and 22 pigs that were subjected to lipopolys

62 differentiating it from a teratoma, meconium peritonitis and abdominal ectopic pregnancy.

63 l responses but resulting in more persistent peritonitis and abscesses.

64 analysis in CD47(-/-) mice by inducing acute peritonitis and aggressive colitis observed consistent r

65 ression and function in experimental sterile peritonitis and autoimmune arthritis, respectively.

66 s are highly susceptible to Escherichia coli peritonitis and bacteremia.

67 wild-type adults are susceptible to E. coli peritonitis and bacteremia.

68 reduced both C. albicans burden during early peritonitis and C. albicans persistence within abscesses

69 the dissemination of B. fragilis after acute peritonitis and characterized the interactions of the in

70 tients on the day of presentation with acute peritonitis and discriminate between culture-negative, G

71 intestinal hollow-organ perforation leads to peritonitis and fulminant sepsis.

72 markers of bacterial infection in PD-related peritonitis and have the potential to contribute to dise

73 icrobiome alterations, spontaneous bacterial peritonitis and hepatic encephalopathy.

74 ality in patients with spontaneous bacterial peritonitis and improves outcome following large volume

75 recruitment following thioglycollate-induced peritonitis and in vitro chemotaxis were not affected by

76 n in 2 models: murine thioglycollate-induced peritonitis and in vitro macrophage migration.

77 Trained mice were efficiently protected from peritonitis and listeriosis for up to 5 weeks.

78 was initiated 6 hours after the induction of peritonitis and maintained throughout the experiment.

79 sis patients presenting with acute bacterial peritonitis and monitoring individuals before and during

80 ed by A. hydrophila NF strains in both mouse peritonitis and NF models in monomicrobial and polymicro

81 Moreover, in the mouse models of peritonitis and pneumonia, Neat1 deficiency significantl

82 inary biomarkers in an animal model of fecal peritonitis and recovery.

83 Using a self-resolving peritonitis and resolution indices coupled with lipid me

84 ted defects in solute transport during acute peritonitis and restored ultrafiltration.

85 Peritonitis and sepsis models were created using wild-ty

86 f patients presenting with symptoms of acute peritonitis and show that elevated peritoneal miR-223 an

87 an inflammatory to a reparative phenotype in peritonitis and skin wound healing models.

88 who presented with clinical signs of general peritonitis and suspected perforated diverticulitis were

89 cterized the inflammatory response in murine peritonitis and unexpectedly found the accumulation of a

90 8), treated with enalapril for 1 week before peritonitis and until study end, received fluids and nor

91 reduced in apo(a)tg and Lp(a)tg mice in both peritonitis and vascular injury inflammatory models, and

92 Feline infectious peritonitis and virulent, systemic calicivirus infection

93 MerTK is linked to the resolution of sterile peritonitis and, after ischemia-reperfusion (I/R) injury

94 AP1189 elicited anti-inflammatory actions in peritonitis and, upon administration at the peak of infl

95 "sigmoidectomy"), AND "Diverticulitis", AND "Peritonitis" AND "therapeutic irrigation" or "lavage" AN

96 mice, and in both in vivo (monosodium urate peritonitis) and in vitro models of inflammation.

97 , long-term (3 d) rat model of sepsis (fecal peritonitis) and recovery was used to understand the tem

98 g, serious infections, spontaneous bacterial peritonitis, and hepatorenal syndrome (RR = 0.42, 95% CI

99 ced peritonitis (AIP), thioglycolate-induced peritonitis, and LPS-induced lung inflammation, CSF-1 ne

100 i are a highly prominent infectious agent in peritonitis, and suggest caution against anticoagulants

101 ved in the repair process of zymosan-induced peritonitis, and thus, could be the basis of development

102 anscriptomic response to sepsis due to fecal peritonitis, and to compare these with the same paramete

103 se, but their expression and activity during peritonitis are poorly understood.

104 In this study, we use IL-1beta-induced peritonitis as a model for an acute immune response, whi

105 l of 120 peritonitis patients with secondary peritonitis as a result of a perforation of a hollow vis

106 NA intraperitoneal injection exhibited acute peritonitis as evidenced by marked neutrophil and monocy

107 response signature (SRS) subgroups in fecal peritonitis associated with early mortality (P = 0.01; h

108 a more effective treatment for patients with peritonitis-associated bacterial sepsis.

109 In a mouse model of enterococcus peritonitis, BALB-C mice were challenged with a high dos

110 In septic peritonitis, blood neutrophils and monocytes are rapidly

111 In murine peritonitis, both pre- and posttreatment with CO inhalat

112 from the peritoneal cavity in patients with peritonitis but not in healthy controls.

113 mice were also more susceptible to abdominal peritonitis, but this was associated with no significant

114 Although radiation-induced mesenteritis or peritonitis can potentially exacerbate the risk of bowel

115 f tumor recurrence and metastases, including peritonitis carcinomatosa.

116 16.7% (9/54) for "nonclassical" indications (peritonitis, carcinomatosis, and so on) (P < 0.0001).

117 Peritonitis caused by staphylococci species is a complic

118 cardiac myocyte level, colon ascendens stent peritonitis cells showed reduced cell shortening, Ca tra

119 age over fibrinogen-deficient mice following peritonitis challenge.

120 thin 6 hours after infection, mice developed peritonitis, characterized by high yeast burdens, neutro

121 ASSO algorithm identified bowel gangrene and peritonitis, coagulopathy, age, the use of stoma, and ch

122 In a murine model of LPS-induced peritonitis, cold exposure potentiated hypothermia and d

123 6 month mortality from sepsis due to CAP or peritonitis combined, 28-day mortality from CAP sepsis,

124 nfection, and possibly spontaneous bacterial peritonitis, community-acquired pneumonia, and infection

125 ng neutrophil influx promoting resolution of peritonitis compared with Ntn1(+/+).

126 codextrin use (R = 0.52; 95% CI, 0.20-0.84), peritonitis count (R = 0.16; 95% CI, 0.03-0.29), and dia

127 th resolvin D1 (RvD1) enhanced resolution of peritonitis, decreased accumulation of apoptotic thymocy

128 loid cells (IMCs) with thioglycolate-induced peritonitis, DSS-induced colitis, and H. felis-induced g

129 rained and subjected to systemic infections, peritonitis, enteritis and pneumonia induced by Staphylo

130 rained and subjected to systemic infections, peritonitis, enteritis, and pneumonia induced by Staphyl

131 tored peritoneal function in mouse models of peritonitis, even in mice treated with standard-of-care

132 AIM (-/-) mice subjected to zymosan-induced peritonitis exhibited progressive inflammation and susta

133 formation is also detected in vivo in murine peritonitis exudates.

134 anulomatous disease called feline infectious peritonitis (FIP), which is one of the most important fa

135 andomized to receive after 12 hours of fecal peritonitis fluid resuscitation and either norepinephrin

136 evealed impaired monocyte chemotaxis towards peritonitis following high fat diet due to retention of

137 vivo role of YB-1 in murine models of acute peritonitis following LPS injection, in sterile renal in

138 Notably, there was no pneumoperitoneum and peritonitis had not yet set in.

139 Perforated diverticulitis with purulent peritonitis has traditionally been treated with open col

140 beta in the peritoneal membrane during acute peritonitis have not been investigated.

141 Colon ascendens stent peritonitis hearts showed a significant increase in oxid

142 Exclusion criteria included patients with peritonitis, hemodynamic instability, unreliable physica

143 Moreover, in animal models of peritonitis, hepatic ischemia-reperfusion injury, Salmon

144 rated diverticulitis with purulent or faecal peritonitis (Hinchey III or IV disease) in a randomised

145 Perforated diverticulitis with purulent peritonitis (Hinchey III) has traditionally been treated

146 During zymosan-induced peritonitis, hPR3Tg displayed an increased accumulation

147 esults demonstrate CTR biosynthesis in mouse peritonitis, human spleens, and human macrophages, as we

148 Rates of complicated appendicitis with peritonitis identified at the time of surgical operation

149 patorenal syndrome, or spontaneous bacterial peritonitis identified by ICD-9/CPT codes.

150 leading to laparotomy was the development of peritonitis in 70%.

151 Fungal peritonitis in a patient on peritoneal dialysis (PD) is

152 reated case of catheter-related C. difficile peritonitis in a patient undergoing peritoneal dialysis.

153 Herein, with the use of microbial-induced peritonitis in mice and liquid chromatography-tandem mas

154 Using Staphylococcus aureus peritonitis in mice, we found significant Sod2 gene indu

155 Here, using self-limited Escherichia coli peritonitis in mice, we identified endogenous maresin (M

156 ned (75%) during the resolution of zymosan A peritonitis in mice.

157 Finally, models of peritonitis in p53(-/-) and pifithrin-alpha-treated mice

158 ammasome is activated during acute bacterial peritonitis in patients on PD, and this activation assoc

159 sent in peritoneal fluid and increase during peritonitis in patients undergoing chronic PD.

160 nd is the result of infection and subsequent peritonitis in the majority of cases.

161 ell-characterized model of sterile resolving peritonitis in the mouse.

162 /liposome-induced interleukin-1beta-mediated peritonitis in vivo.

163 cites, encephalopathy, spontaneous bacterial peritonitis) increased with severity of AKI.

164 The Mannheim Peritonitis Index and Acute Physiology and Chronic Healt

165 A mouse model of peritonitis indicated that monocyte/macrophage recruitme

166 molol on myocardial and vascular function in peritonitis-induced septic rats and to explore the infla

167 ells were collected at baseline (just before peritonitis induction) and at the end of the in vivo exp

168 e end of the in vivo experiment (24 hr after peritonitis induction).

169 de (LPS); underwent induction E. coli 018:K1 peritonitis induction, followed by treatment with AB103;

170 during endotoxemia and severe polymicrobial peritonitis, integrin VLA-3 (CD49c/CD29) is specifically

171 Peritonitis is a common and serious complication in indi

172 Infectious peritonitis is a common complication in patients undergo

173 Acute peritonitis is a frequent medical condition that can tri

174 Spontaneous bacterial peritonitis is an acute bacterial infection of ascitic f

175 Here, we report that resolution of acute peritonitis is delayed in obese diabetic (db/db) mice.

176 t was previously thought, the risk of septic peritonitis is reduced and not increased with each recur

177 Peritonitis is the major disease problem of laying hens

178 uals with a history of spontaneous bacterial peritonitis, known to have defective cellular immunity.

179 ed during polymicrobial versus monomicrobial peritonitis, leading to increased inflammatory infiltrat

180 opolysaccharide- or Escherichia coli-induced peritonitis led to IL-1beta release in the peritoneal me

181 a role for G. anatis in the pathogenesis of peritonitis, little is known about the organism's virule

182 by macrophages derived from zymosan-induced peritonitis, M1- and M2a-like bone marrow derived macrop

183 cell infiltration in a zymosan-induced mouse peritonitis model accompanied by impaired levels of cyst

184 fects of hydrogen sulfide in vivo, we used a peritonitis model by way of intraperitoneal injection of

185 Using a sterile peritonitis model in mice, we identified interleukin (IL

186 In a mouse peritonitis model of gout, using monosodium urate crysta

187 y, pharmacokinetics, and efficacy in a mouse peritonitis model of infection, which led to the discove

188 vivo experimental data obtained from a mouse peritonitis model of inflammation, which is widely used

189 d in inflamed cremaster muscle venules, in a peritonitis model, and in an in vitro chemotaxis assay.

190 In a murine peritonitis model, HlgAB contributes to S. aureus bacter

191 In the mouse peritonitis model, LNZ (at doses that mimic human pharma

192 In an in vivo peritonitis model, significantly less Ly6C(high) monocyt

193 Employing a mouse peritonitis model, we also determined that cells collect

194 Using an in vivo peritonitis model, we have validated that the IL-1beta i

195 +) and 3-Cl-Tyr were detected readily in the peritonitis model, whereas in the arterial inflammation

196 psis in mice using the colon ascendens stent peritonitis model.

197 ued by prior treatment with 12(S)-HETE, in a peritonitis model.

198 to adoptive transfer into an on-going murine peritonitis model.

199 inflammatory activity in a SEB-induced mouse peritonitis model.

200 showed impaired neutrophil recruitment in a peritonitis model.

201 reduce RIPK2-mediated effects in an in vivo peritonitis model.

202 lular recruitment is inhibited in an in vivo peritonitis model.

203 P) monocytes in vivo using a sterile zymosan peritonitis model.

204 el and the cecal ligation and puncture (CLP) peritonitis model.

205 monosodium urate crystal inflammatory murine peritonitis model.

206 nt of the inflammatory response in the mouse peritonitis model.

207 lammatory/proresolving phenotype in a murine peritonitis model.

208 ore important for NF2 than NF1 in the murine peritonitis model.

209 dies using small animals (p < 0.0001) and in peritonitis models (p < 0.0001).

210 hloride-induced fibrosis and zymosan-induced peritonitis models, MVs ameliorated inflammation.

211 zymosan air pouch and thioglycolate-induced peritonitis models, the i.m. treatment with xylazine or

212 f AIM in prevention of progression of fungal peritonitis models.

213 peptide (AC3-I) and in colon ascendens stent peritonitis myocytes isolated from mutant mice that have

214 was also preserved in colon ascendens stent peritonitis myocytes isolated from transgenic mice expre

215 eticulum Ca content in colon ascendens stent peritonitis myocytes.

216 The sheep were randomized to fecal peritonitis (n = 10) or a sham procedure (n = 5), and cr

217 d to intensive care with sepsis due to fecal peritonitis (n = 117) or community-acquired pneumonia (n

218 = 2), infected joint prosthesis (n = 2), and peritonitis (n = 2) being the most common, thus expandin

219 In zymosan-initiated peritonitis, neutrophil polymorphonuclear leukocyte infi

220 associated with necrosis in zymosan-induced peritonitis of AIM (-/-) mice.

221 ions produced in living macrophage cells and peritonitis of living mice with high contrast.

222 In the absence of peritonitis, operative treatment is associated with incr

223 Among patients with clinical peritonitis, operative treatment was associated with red

224 ammation, induced by thioglychollate-induced peritonitis or following infection withSalmonella enteri

225 e presence of clinical symptoms and signs of peritonitis or intra-abdominal abscess and isolation of

226 O-deficient (Mpo(-/-)) mice with established peritonitis or localized arterial inflammation, and tiss

227 of neutrophils during thioglycolate-induced peritonitis or MIP2-induced ear pouch inflammation.

228 diagnoses of ascites, spontaneous bacterial peritonitis, or esophageal variceal hemorrhage.

229 diagnoses for ascites, spontaneous bacterial peritonitis, or esophageal variceal hemorrhage.

230 asome-dependent inflammation in the model of peritonitis (P<0.05) in wild-type but not in miR-21 knoc

231 PAF exacerbates peritonitis partly through inflammasome activation, but

232 A total of 120 peritonitis patients with secondary peritonitis as a res

233 functionally stabilised in PD effluent from peritonitis patients, with a proportion likely to be inc

234 ction in bacterial burden in mouse models of peritonitis, pneumonia and urinary tract infection.

235 injury, cardiac arrest, gastric haemorrhage, peritonitis, pneumothorax, septic shock, and sudden deat

236 l replacement therapy, spontaneous bacterial peritonitis, positive blood culture, and infection by fu

237 Animals with zymosan peritonitis reached a clinical and biochemical nadir on

238 Peritonitis remains a major cause of morbidity and morta

239 nt therapies, fungal-bacterial polymicrobial peritonitis remains a serious complication for surgery p

240 Bacterial peritonitis remains the main cause of technique failure

241 date macrophages was impaired during sterile peritonitis, resulting in enhanced and prolonged inflamm

242 During peritonitis, RNase 3 increased 55-fold and RNase 7 level

243 Furthermore, colon ascendens stent peritonitis S2814A mice showed preserved ejection fracti

244 ion (UTI) (28.5%), and spontaneous bacterial peritonitis (SBP) (22.5%) were the most prevalent infect

245 ver 20 points and with spontaneous bacterial peritonitis (SBP) as a precipitating event was almost si

246 prophylaxis to prevent spontaneous bacterial peritonitis (SBP) in patients colonized with multidrug-r

247 owever, development of spontaneous bacterial peritonitis (SBP) in these patients could preclude treat

248 Spontaneous bacterial peritonitis (SBP) is a common, life-threatening complica

249 sponse syndrome (SIRS), spontaneous bacteria peritonitis (SBP), and pneumonia; and O: the CLIF consor

250 which can also lead to spontaneous bacterial peritonitis (SBP).

251 e in case of suspected spontaneous bacterial peritonitis (SBP).

252 comes of patients with spontaneous bacterial peritonitis (SBP).

253 hosis, with or without spontaneous bacterial peritonitis (SBP).

254 oteinase-8 is a critical component of septic peritonitis secondary to intestinal compromise.

255 Peritonitis secondary to perforated diverticulitis has c

256 opean community-acquired pneumonia (CAP) and peritonitis sepsis cases, and 477 controls from the Unit

257 combined, 28-day mortality from CAP sepsis, peritonitis sepsis, pneumococcal sepsis or sepsis in you

258 erapeutic efficacies, using either the mouse peritonitis-sepsis model or the thigh infection model.

259 resistant Acinetobacter baumannii in a mouse peritonitis-sepsis model, without observed hemolysis or

260 Using a murine peritonitis/sepsis model, we identified increased Connex

261 rization, major bleeding, sepsis, pneumonia, peritonitis, severe arrhythmia, or renal failure.

262 n alphaCD40-induced acute colitis and during peritonitis, suggesting an altered monocyte migration.

263 wenty-four hours after colon ascendens stent peritonitis surgery, we observed that wild type mice had

264 o caecal ligation and puncture (CLP)-induced peritonitis than wild-type (WT) mice.

265 ar recruitment site for neutrophils in acute peritonitis that is indispensable for host defence again

266 a of BG2 (1 x 10(8) or 1 x 10(7) CFU) caused peritonitis that progressed to abscesses.

267 o study resolution of thioglycollate-induced peritonitis, the model in which earlier work indicated t

268 During peritonitis, the peritoneal membrane undergoes structura

269 o patients presented with signs of bacterial peritonitis; the third had pyomyositis of the thigh.

270 antilever sensor for detecting gentamicin, a peritonitis therapeutic small-molecule drug.

271 aride or cecal ligation and puncture-induced peritonitis, these marginated cells are rapidly released

272 TDM) is especially crucial for patients with peritonitis to avoid adverse reactions from a high conce

273 A final diagnosis of CNS, complicated with peritonitis tracking into the scrotal sacs was arrived a

274 111 patients with acute peritonitis underwent emergency laparotomy: number of pe

275 tion: prior episode of spontaneous bacterial peritonitis, upper gastrointestinal bleeding, and low-pr

276 Results were compared to those in mouse peritonitis, using LNZ via oral gavage at 80 and 120 mg/

277 Feline infectious peritonitis virus (FIPV) belongs to the genus Alphacoron

278 Feline infectious peritonitis virus (FIPV) is an alphacoronavirus that cau

279 Feline infectious peritonitis virus (FIPV) is the leading cause of death i

280 ain protease (M(pro)) from feline infectious peritonitis virus (FIPV), which leads to lethal systemic

281 tients with sepsis, hepatorenal syndrome, or peritonitis warrants further investigation, and the low

282 ere pre-injected (ip.) with sCD48, and then, peritonitis was induced by SEB injection; peritoneal lav

283 and autoinflammation in MSU crystal-induced peritonitis was not reduced in GSDMD-deficient mice.

284 ll, a mortality rate of 22.1% for OAT due to peritonitis was observed.

285 10,14-tetramethylpentadecane (TMPD)-mediated peritonitis was similarly aggravated in STING-deficient

286 Using a murine model of peritonitis, we also demonstrate that these agents are h

287 Using a mouse model of peritonitis, we demonstrate that neutrophils elicited in

288 In the absence of peritonitis, we detected RNase 3, RNase 6, and RNase 7 i

289 Using a murine model of peritonitis, we have demonstrated that monomicrobial inf

290 ow-derived macrophages and a murine model of peritonitis, we show in this study that receptor-interac

291 Using a mouse model of GBS-induced peritonitis, we show in this study that the chemokines C

292 variceal bleeding, and spontaneous bacterial peritonitis were associated with a higher mortality rate

293 protected 20% of mice (1 of 5) from E. coli peritonitis, whereas 100% (15 of 15) survived when AB103

294 flux and inflammatory markers in MSU-induced peritonitis, whereas equimolar anakinra administered 24

295 in the pathogenesis of spontaneous bacterial peritonitis, which is a common complication of liver cir

296 ment and IL-1beta generation in alum-induced peritonitis, which is a typical IL-1 signaling-dependent

297 refractory ascites or spontaneous bacterial peritonitis while other data suggests a survival benefit

298 cecal ligation and puncture (CLP) to induce peritonitis, while control groups had a sham operation.

299 ns of cecal contents to induce polymicrobial peritonitis without tissue compromise in the recipient.

300 lammatory response using the zymosan-induced peritonitis (ZIP) model.