ライフサイエンスコーパス: perivascular

1 Perivascular access and distribution of full-length IgG

2 Osmolyte co-infusion significantly enhanced perivascular access of the larger antibody from the CSF,

3 Perivascular access to smooth muscle basement membrane c

4 Collectively, our findings identify the perivascular accumulation of BM-derived macrophages as p

5 Perivascular accumulation of lymphocytes can be a promin

6 Adrb1 activation stimulates WAT resident perivascular (Acta2+) cells to form cold-induced beige a

7 manifesting as lymphocytic inflammation in a perivascular (acute perivascular rejection [AR]) or peri

8 ACT: Previous studies have demonstrated that perivascular adipose tissue (PVAT) causes vasoconstricti

9 We have previously proposed that perivascular adipose tissue (PVAT) controls vascular fun

10 Perivascular adipose tissue (PVAT) has been shown to med

11 In vivo, uterine perivascular adipose tissue (PVAT) potentiates uterine a

12 vascular and circulating T cells, but not in perivascular adipose tissue adipocytes.

13 ic microenvironment in which adventitial and perivascular adipose tissue cells initiate and regulate

14 Y POINTS: The fat surrounding blood vessels (perivascular adipose tissue or PVAT) releases vasoactive

15 Vascular injury and dysfunction of the perivascular adipose tissue promote expansion of the vas

16 Ang II-induced recruitment of T cells into perivascular adipose tissue was abolished in miR-214(-/-

17 -induced T-cell activation in the spleen and perivascular adipose tissue was blunted in Tcrdelta(-/-)

18 ed in an organ bath after minimal removal of perivascular adipose tissue.

19 tissue surrounding uterine arteries (uterine perivascular adipose tissue; PVAT) is a novel local medi

20 We propose that mammalian perivascular adventitial 'cuffs' are conserved sites in

21 The altered perivascular adventitial compartment and its associated

22 In contrast, perivascular adventitial fibroblast expression of VCAM1

23 oal of the current study was to characterize perivascular adventitial fibroblast states in inflammato

24 ence may hinder the dissection of aspects of perivascular adventitial pathology.

25 pathologies, such as arterial stiffening and perivascular alterations, may impede the inflow.

26 ubtypes include border-associated meningeal, perivascular and choroid plexus macrophages.

27 MiR-214 induction was pronounced in perivascular and circulating T cells, but not in perivas

28 We identified a niche of CXCL13(+) perivascular and CXCL12(+)LTB(+) and PD-L1(+) epithelial

29 l organogenesis, most kidney macrophages are perivascular and express F4/80 and CD206.

30 (CNS)-associated macrophages (CAMs), such as perivascular and meningeal macrophages, are implicated i

31 They are also enriched in perivascular and necrotic regions.

32 nal landscape of mouse bone marrow vascular, perivascular and osteoblast cell populations at single-c

33 lation of versican and HA, especially in the perivascular and peribronchial regions, which were enric

34 n multiple brain regions, with enrichment in perivascular and perimeningeal tissues.

35 Staining clustered in perivascular and perinecrotic tumor regions.

36 NOTCH3 signalling in the differentiation of perivascular and sublining fibroblasts that express CD90

37 simultaneously limit parallel routes of both perivascular and televascular invasion through both gray

38 both regions, the rates of migration for the perivascular and televascular routes of invasion were in

39 oreactivity was abnormal because the typical perivascular ANXA1 immunoreactivity was reduced.

40 er alterations in AQP4 expression or loss of perivascular AQP4 localization are features of the aging

41 of AQP4 protein, AQP4 immunoreactivity, and perivascular AQP4 localization in the frontal cortex wer

42 Loss of perivascular AQP4 localization may be a factor that rend

43 When controlling for age, loss of perivascular AQP4 localization was associated with incre

44 Perivascular AQP4 localization was significantly associa

45 activated mast cells and neutrophils in the perivascular area of atherosclerotic plaques.

46 , CCR2 was required for iMO trafficking from perivascular areas to sites of virus infection within th

47 cord parenchyma, meaningfully re-established perivascular astrocyte end-feet, and enhanced spinal cor

48 Here, we show that AQP4 polarization in the perivascular astrocytic end feet was impaired after TBI,

49 phosphorylated tau in neuronal perikarya and perivascular astroglial processes.

50 yte process detachment and disruption of the perivascular basement membrane surrounding the VECs.

51 -transmembrane protein normally expressed in perivascular brain astrocyte end feet that is essential

52 EdU pulse-chase experiments demonstrated a perivascular cancer stem cell population in Pten/Trp53 d

53 oglial activation, reduced neuronal density, perivascular CD3-positive T-lymphocyte clustering, and f

54 pairs perivascular cell migration, increases perivascular cell death, delays endothelial cell migrati

55 us remodeling, arterial differentiation, and perivascular cell maturation.

56 (SMC-P) knockout of Oct4 that Oct4 regulates perivascular cell migration and recruitment during angio

57 in perivascular cells significantly impairs perivascular cell migration, increases perivascular cell

58 We used perivascular-cell-specific and pericyte-specific lineage

59 Knockout of Oct4 in perivascular cells also impairs perfusion recovery and d

60 Genetic inactivation of Klf4 in perivascular cells decreased formation of a pre-metastat

61 The origin of these scars is thought to be perivascular cells entering lesions on ingrowing blood v

62 nce that Oct4 plays an essential role within perivascular cells in injury- and hypoxia-induced angiog

63 Oct4 in cultured SMCs, and in Oct4-deficient perivascular cells in ischemic hindlimb muscle.

64 in capillary and artery numbers, but not of perivascular cells in pancreas, testis and thyroid gland

65 revealed a previously unidentified role for perivascular cells in pre-metastatic niche formation and

66 arization of neural crest cell (NCC)-derived perivascular cells in the brain, autophagy in the retina

67 lineage-tracing models to trace the fate of perivascular cells in the pre-metastatic and metastatic

68 We show that perivascular cells lose the expression of traditional vS

69 y gene Klf4 in these phenotypically switched perivascular cells promoted a less differentiated state,

70 Knockout of Oct4 in perivascular cells significantly impairs perivascular ce

71 of perivascular cells, we hypothesized that perivascular cells similarly regulate tumor cell fate at

72 his study was to examine the contribution of perivascular cells to odontoblasts during the developmen

73 Sox10(+) stem cells could differentiate into perivascular cells to stabilize newly formed microvessel

74 losa cells, immune cells, endothelial cells, perivascular cells, and stromal cells.

75 niche, including its sinusoidal vessels and perivascular cells, contributing to delayed hematopoieti

76 Cells captured by DDX4 antibody are perivascular cells, not oogonial stem cells.

77 The contractile perivascular cells, pericytes (PC), are hijacked by glio

78 Given the well-described plasticity of perivascular cells, we hypothesized that perivascular ce

79 in the kidney detected evident expression in perivascular cells, with negligible expression in the en

80 nditions, including an adipocytic skewing of perivascular cells.

81 criteria, abundant alveolar cellularity and perivascular cellularity (PVC), were assessed by 4 inves

82 Perivascular cellularity agreement among investigators s

83 th multifocal immune cell-mediated injury at perivascular cerebrospinal fluid (CSF)-brain barriers, a

84 ognized role for Th1 cells as integrators of perivascular CF and cardiac dysfunction in nonischemic H

85 ables RGS protein binding accumulated in the perivascular channels of thymic corticomedullary venules

86 aste removal during sleep via glia-supported perivascular channels.

87 pithelium, the presence of a thin choroid, a perivascular choroidal inflammatory infiltrate, and atro

88 g cycle of increased vascular Abeta, reduced perivascular clearance and further CAA and AD progressio

89 between CAA and AD, representing overload of perivascular clearance pathways and the effects of remov

90 We find perivascular clustering of oligodendrocyte precursor cel

91 on human third molar pulp sections showed a perivascular co-localization of the mesenchymal stem cel

92 as a combination of interstitial microscars, perivascular collagen fiber deposition, and increased th

93 side in a vascular niche, located within the perivascular compartment of adipose tissue blood vessels

94 mber that was restricted to the interstitial/perivascular compartment, without recruitment of macroph

95 Ciliated preadipocytes abundantly populate perivascular compartments in fat and are activated by a

96 dic channels, mimicking aspects of the tight perivascular conduits and white-matter tracts in brain p

97 Importantly, the perivascular cuff lymphocyte numbers correlate to the qu

98 S, we determined that macrophages within the perivascular cuff of post-capillary venules are highly g

99 ty to spread systemically, PA14::hepP formed perivascular cuffs around the blood vessels within the s

100 olism of macrophages for transmigration from perivascular cuffs into the CNS parenchyma and identifie

101 yperintense, T1-hypointense, and appeared as perivascular demyelinated lesions with dystrophic neuron

102 irculation within the interstitial fluid and perivascular drainage pathways and its brain clearance,

103 he absence of CLU, Abeta clearance shifts to perivascular drainage pathways, resulting in fewer paren

104 d capillary permeability, and re-established perivascular end-feet astrocytes in symptomatic ALS mice

105 ced astrogliosis, microgliosis, and enhanced perivascular end-feet astrocytes were also determined in

106 the water channel aquaporin-4 at astrocytic perivascular endfeet of the BBB.

107 CHEST cells are a primary cell line with perivascular endothelial properties that expand hematopo

108 an in vitro platform to study the biology of perivascular-endothelial interactions.

109 hronic lymphocytic inflammation with pontine perivascular enhancement responsive to steroids (CLIPPER

110 induced a lung distal neomuscularization and perivascular extracellular matrix activation.

111 Renal sinus fat (RSF) is a perivascular fat compartment located around renal arteri

112 gluteus maximus; and pericardial and aortic perivascular fat mainly in women.

113 na also showed increased vascularization and perivascular fibers containing NPY.

114 High levels of perivascular fibrillar collagen and pulmonary interleuki

115 nic mice develop interstitial myocardial and perivascular fibrosis and left ventricular hypertrophy a

116 ells into RAG1(-/-) mice resulted in reduced perivascular fibrosis compared with the effect of WT T c

117 T-cell-derived miR-214 controls pathological perivascular fibrosis in hypertension mediated by T cell

118 importance of vascular stiffening linked to perivascular fibrosis in hypertension, the molecular and

119 a model revealed that cachectic mice develop perivascular fibrosis in major metabolic organs, includi

120 nase kinase 6-p38 developed interstitial and perivascular fibrosis in the heart, lung, and kidney as

121 ce, -1.3 [95% CI, -2.5 to -0.2]; P=0.016 for perivascular fibrosis), worse cardiac dysfunction (mean

122 xpression of fetal genes and coronary artery perivascular fibrosis, with ischaemia indicated by enhan

123 e within minutes of an ischemic insult along perivascular flow channels.

124 The perivascular flow model is solved numerically, discoveri

125 We report for the first time in HFpEF perivascular fluid cuff formation around extra-alveolar

126 try into the glymphatic system, a network of perivascular fluid transport that clears metabolic waste

127 ma led to profound structural alterations in perivascular FRCs and associated high endothelial venule

128 CLIPPERS patients had brainstem predominant perivascular gadolinium enhancing lesions on magnetic re

129 ce VEGF as a new player in Epo induction and perivascular Gli1(+)SMA(+)PDGFRbeta(+) cells as a previo

130 Coalescing and mostly perivascular granuloma-like accumulations of storage-lad

131 Proneural, perivascular GSCs activated EZH2, whereas mesenchymal GS

132 0L-immunized mice exhibited marked pulmonary perivascular hemorrhage post-MERS-CoV challenge despite

133 ing LSEC fenestration and protecting against perivascular hepatic fibrosis.

134 nses modified the chemotactic cues along the perivascular homing paths, leading to rapid block of ent

135 ologically, it is defined by the presence of perivascular hyperphosphorylated tau aggregates in corti

136 brain there was evidence of vasculitis, with perivascular infiltrates of inflammatory cells and rare

137 Expanded adventitial compartments with perivascular infiltrates similar to the human settings w

138 dermal dysmaturation, neutrophil exocytosis, perivascular infiltration of lymphocytes and neutrophils

139 patient samples revealed that a significant perivascular infiltration of M1, but not M2, macrophages

140 Peribronchial and perivascular inflammation and mucus production were larg

141 fficked into the lung parenchyma and ignited perivascular inflammation to promote eosinophil and CD4+

142 is) exert protective effects in DN improving perivascular inflammation.

143 omosis within the bronchial circulation, and perivascular inflammation.

144 71% of biopsies demonstrated epineurial perivascular inflammation.

145 axis resulting in attenuation of profibrotic perivascular inflammation.

146 entiated nuclear layers of the retina, and a perivascular inflammatory infiltrate within the choroid.

147 ype I interferon-stimulated genes and causes perivascular inflammatory lung disease in mice.

148 e sclerosis includes both focal inflammatory perivascular injury and injury to superficial structures

149 astrocytes and in situ in astrocytes at the perivascular interface with endothelial cells.

150 Rapid growth and perivascular invasion are hallmarks of glioblastoma (GBM

151 First, perivascular invasiveness along remaining blood vessels,

152 We report perivascular iron deposition in multiple sclerosis lesio

153 o cancer-associated fibroblast (CAF) and two perivascular-like (PVL) subpopulations.

154 Perivascular-like cells as an eventual target in NSCLC w

155 Together, our data show that perivascular-like cells present in NSCLC retain function

156 ger and distributed in both interstitial and perivascular locations.

157 man skin diseases and relate these states to perivascular lymphocyte accumulation.

158 anthosis and focal lymphocytes with moderate perivascular lymphocyte infiltration.

159 We report multifocal perivascular lymphocytic cuffs contain increased numbers

160 distinguished by CD90 expression, and dense perivascular lymphocytic infiltrates are uncommon.

161 nsplant recipients requires demonstration of perivascular lymphocytic infiltration in alveolar tissue

162 Circulating autoantibodies, lung perivascular lymphoid tissue, and elevated cytokines hav

163 Instead, perivascular macrophage-like myeloid cells populate the

164 Brain perivascular macrophages (PVM) located in the perivascul

165 While the brain parenchyma is patrolled by perivascular macrophages and microglia, the meningeal sp

166 tion.IMPORTANCE Brain-resident microglia and perivascular macrophages are important HIV reservoirs in

167 In dysbiosis, gaps exist between the perivascular macrophages correlating with increased bact

168 ong expression of MCT-4, EMMPRIN and LDHA in perivascular macrophages in MS brains.

169 robiome and the maturation of lamina propria perivascular macrophages into a tight anatomical barrier

170 CD163+ perivascular macrophages were identified by immunohistoc

171 Brain-resident microglia and myeloid cells (perivascular macrophages) are important HIV reservoirs i

172 ment that includes parenchymal microglia and perivascular macrophages, as well as choroid plexus and

173 Upon depletion of circulating and perivascular memory T cells, this brain signature was en

174 ling significant changes in the abundance of perivascular mesenchymal stromal cells (MSCs)/osteoproge

175 interaction in MS suggests that aberrant OPC perivascular migration not only impairs their lesion rec

176 ty to properly detach from vessels following perivascular migration.

177 formation in microenvironments enriched for perivascular MSC/osteoprogenitors and high osteogenic po

178 Pericytes are perivascular mural cells of brain capillaries.

179 l hypoperfusion on cerebral hemodynamics and perivascular nerve density in a rat model.

180 es with remarkable changes in morphology and perivascular nerve density, suggesting a functional role

181 evidence indicating that in vivo electrical perivascular nerve stimulation in rat mesenteric small a

182 Most studies on perivascular nerve-mediated vasodilatation are made in v

183 ve density, suggesting a functional role for perivascular nerves in cerebral autoregulation.

184 Immunohistochemical analysis revealed perivascular, neuronal, and glial cells immunoreactive t

185 We show that chemoresistant DTCs occupy the perivascular niche (PVN) of distant tissues, where they

186 sed in the tumor cell niches compared to the perivascular niche across multiple regions in GBM patien

187 s within skin injuries, where they home to a perivascular niche and generate alternatively activated,

188 ling axis as crucial for exploitation of the perivascular niche and identify potential therapeutic ta

189 that of maintaining the integrity of the BM perivascular niche and improving BM niche recovery after

190 elial cell branching and inflammation in the perivascular niche by upregulating the pro-inflammatory

191 d, tumor-initiating stem cells housed in the perivascular niche close to remaining blood vessels were

192 To simulate perivascular niche conditions and analyze consequential

193 Collectively, perivascular niche conditions promote GBM growth and inv

194 oietic stem cells (HSCs) are maintained in a perivascular niche in bone marrow, in which leptin recep

195 endothelial cells and stem cells within the perivascular niche in dental pulps is unclear.

196 endothelial cells and stem cells within the perivascular niche is required for the maintenance of st

197 However, the mechanisms by which the perivascular niche regulates GBM invasion and CSCs remai

198 (MSCs) are recruited from the endosteal and perivascular niche to become fibrosis-driving myofibrobl

199 st that preservation of the integrity of the perivascular niche via VEGF-C signaling could be exploit

200 pts this positive feedback loop in the tumor perivascular niche, which eventually lessens tumor initi

201 netrant medulloblastoma originating from the perivascular niche, which exhibited abnormal blood vesse

202 cells (CSCs) and their association with the perivascular niche.

203 (LepR+) cells led to a disruption of the BM perivascular niche.

204 vironment modulate the fate of stem cells in perivascular niches in tissues (e.g., bone) and organs (

205 Some adult animals presented perivascular niches outside the V-SVZ.

206 ming abilities, with the majority located in perivascular niches where GSCs are found.

207 rounded by ALDH1(high) or Bmi-1(high) cells (perivascular niches) compared to tissues formed upon tra

208 cancer cells (GSCs) that home to specialized perivascular niches.

209 Perivascular OPCs can themselves disrupt the BBB, interf

210 with high bone turnover and an abundance of perivascular osteoprogenitors, ZOL significantly (P < 0.

211 This arises from the differentiation of perivascular osterix-positive MSC/osteoprogenitors into

212 tly detected in paratrabecular (P < .05) and perivascular (P < .01) locations, and associated with bo

213 d cells from an intravascular (P = 0.004) to perivascular (P < 0.0001) distribution.CONCLUSIONWithin

214 rospinal fluid along a brain-wide network of perivascular pathways recently termed the glymphatic sys

215 MV reactivation was observed in intratumoral perivascular pericytes and tumor cells in mouse and huma

216 Funduscopic examination revealed perivascular pigmentary clumping and atrophic changes ra

217 The perivascular polarization of AQP4 is highest during the

218 eration, are recruited to a Nestin-GFP(high) perivascular population, and contribute to cartilage rep

219 te and moderate macrophage infiltrates, with perivascular predominance as well as diffuse parenchymal

220 cal 0.75 m mannitol increasing the number of perivascular profiles per slice area accessed by IgG by

221 Pten inactivation created an abnormal perivascular proliferative niche in the cerebellum that

222 Among patients, striking perivascular radial enhancement was found on brain magne

223 lic wastes and amyloid-beta (Abeta) plaques, perivascular reactive astrogliosis, and mislocalization

224 idney revealed foci of EBV-infected cells in perivascular regions in close association with programme

225 ation allowed immuno-NPs to deposit into the perivascular regions of the tumor, which coincided with

226 that the human islet contains macrophages in perivascular regions that are the main local source of t

227 noparticle distributions in the vascular and perivascular regions.

228 ing collectively invading tumor cells and in perivascular regions.

229 ocytic inflammation in a perivascular (acute perivascular rejection [AR]) or peribronchiolar (lymphoc

230 Perivascular sensory-motor nerves have been shown to cau

231 enlargement and eccentric thickening of the perivascular space (fibrillar collagen type I deposition

232 The flow inside the perivascular space (PVS) is modeled using a first-princi

233 ubset of white matter vessels have increased perivascular space (with jagged contours) and collagen i

234 Here, we review perivascular space anatomy, physiology and pathology, pa

235 ich are immune cells that are present in the perivascular space and are a major peripheral source of

236 HSCs moved in the perivascular space and showed intermittent close contact

237 with the flow in the thin annular gap of the perivascular space between an impermeable artery and the

238 brainwide CSF transport system that uses the perivascular space for fast inflow of CSF.

239 advances have facilitated in vivo studies of perivascular space function in intact rodent models duri

240 remain, but what is now clear is that normal perivascular space function is important for maintaining

241 Using MRI-visible perivascular space location and severity together with o

242 ess and sleep, and MRI in humans has enabled perivascular space morphology to be related to cognitive

243 scence revealed prelabeled MAPC cells in the perivascular space of kidneys during NMP.

244 MRI-visible perivascular space severity in either location did not p

245 appeared to be iron-laden macrophages in the perivascular space tracking a network of injured vessels

246 erivascular macrophages (PVM) located in the perivascular space, a major site of brain Abeta collecti

247 ibe patterns of contrast distribution in the perivascular space, subarachnoid space, and space surrou

248 tivation, and T-cell transmigration into the perivascular space, where (ii) blinatumomab induced B-ce

249 ron-laden macrophages, predominately seen in perivascular space.

250 allowing substances in the CSF to enter the perivascular space.

251 ject-based morphologic estimates of enlarged perivascular spaces (ePVSs) in clinical-field-strength (

252 However, whether large perivascular spaces (L-PVSs) (>3 mm in diameter) visible

253 ssel disease (cSVD), solute transport in the perivascular spaces (PVS) and PVS-to-tissue transfer was

254 Imaging the perivascular spaces (PVS), also known as Virchow-Robin s

255 infarcts, cerebral microbleeds, and enlarged perivascular spaces (PVS), as well as PET-based biomarke

256 spaces around cerebral blood vessels, called perivascular spaces (PVS), through which cerebrospinal f

257 Perivascular spaces (PVSs) in brain have a close relatio

258 th advancing age, an increased visibility of perivascular spaces (PVSs) on magnetic resonance imaging

259 acytic inflammation, with a predilection for perivascular spaces and collagenous tissues, was observe

260 vasoconstriction, which in turn enlarged the perivascular spaces and doubled glymphatic inflow speeds

261 lation of mature thymocytes within medullary perivascular spaces and reduced numbers of recent thymic

262 odies exhibited size-dependent access to the perivascular spaces and tunica media basement membranes

263 pace may provide unique entry sites into the perivascular spaces from the CSF.

264 grade), whereas the severity of MRI-visible perivascular spaces in the basal ganglia was associated

265 justed analyses, the severity of MRI-visible perivascular spaces in the centrum semi-ovale was indepe

266 We also hypothesized that MRI-visible perivascular spaces in the centrum semi-ovale would be a

267 's disease, we hypothesized that MRI-visible perivascular spaces in the centrum semi-ovale would be a

268 Perivascular spaces include a variety of passageways aro

269 en cerebrospinal fluid (CSF) influx from the perivascular spaces into the interstitial fluid space ra

270 t the anatomical distribution of MRI-visible perivascular spaces may reflect the underlying cerebral

271 distribution to deep brain regions along the perivascular spaces of all vessel types, with sdAb acces

272 he brain surface and convective transport in perivascular spaces of cerebral blood vessels.

273 The glymphatic system, a network of perivascular spaces promoting fluid exchange between CSF

274 Many questions about perivascular spaces remain, but what is now clear is tha

275 Perivascular spaces that are visible on magnetic resonan

276 The glymphatic system is a network of perivascular spaces that promotes movement of cerebrospi

277 Although perivascular spaces were first identified over 150 years

278 MRI-visible perivascular spaces were rated using a validated 4-point

279 method for multimodal autoidentification of perivascular spaces yields individual whole-brain morpho

280 ensity - WMH, microbleeds, lacunes, enlarged perivascular spaces, brain atrophy) as seen on structura

281 hite matter hyperintensities (WMH), enlarged perivascular spaces, cerebral microbleeds and lacunes.

282 matrix and reduces NP confinement within the perivascular spaces.

283 t, Tregs were restricted to the meninges and perivascular spaces.

284 on structural MRI, visual scores and volume; perivascular spaces; lacunes and microbleeds), and vascu

285 PAH.Measurements and Main Results: Pulmonary perivascular-specific activation of the complement casca

286 single-cell RNA sequencing data and confirm perivascular staining at the protein level.

287 Multipotent human adipose-derived perivascular stem cells (hAd-PSCs) represent an attracti

288 o the red pulp sinuses, T cells latched onto perivascular stromal cells in a manner that was independ

289 nly sustained by a subpopulation of ICAM1(+) perivascular stromal cells.

290 ermittent close contacts with SCF-expressing perivascular stromal cells.

291 ATP, released from perivascular sympathetic nerves, causes vasoconstriction

292 tis and lupus and is associated with a dense perivascular T cell infiltrate.

293 pheral lung was diffuse alveolar damage with perivascular T-cell infiltration.

294 isorder histopathologically characterized by perivascular tangles of hyperphosphorylated tau at the d

295 owed hyaluronan in intact alveolar walls and perivascular tissue.

296 Out of 381 miRs screened in the perivascular tissues in response to Ang II (angiotensin

297 kinetics of PGZ from fat depots transplanted perivascular to jugular vein were assessed by HPLC/MS/MS

298 Fat explants placed perivascular to the external jugular vein were retained,

299 Perivascular transport involved blood vessels of all cal

300 evelopment of fluid dynamics including flow, perivascular transport, drainage, and barriers.