ライフサイエンスコーパス: periventricular

1 dilated ventricle margin were classified as periventricular.

2 rojection pattern: superficial, central, and periventricular.

3 These results indicate that periventricular abnormalities arising from abnormal migr

4 rtal, sex differentiation in ventricular and periventricular anatomy and associated behavior, affecti

5 Immunoreactivity was seen in several periventricular and a few magnocellular neurons, and in

6 show that Kiss1 neurons in the anteroventral periventricular and anterior periventricular nuclei (AVP

7 These features closely mimic the periventricular and cortical pathology described in MS p

8 ies with multifocal small cavitations in the periventricular and deep cerebral white matter.

9 ion of white matter hyperintensities in both periventricular and deep white matter areas was found.

10 We measured the severity of periventricular and deep white matter hyperintensities a

11 We found that more severe periventricular and deep white matter hyperintensities c

12 Periventricular and deep WMH volumes were measured with

13 queductal gray, dorsal parabrachial nucleus, periventricular and rhomboid nuclei of the thalamus, and

14 ra of diseases that includes polymicrogyria, periventricular and subcortical heterotopia and lissence

15 conditions, and are located predominantly in periventricular and subpial regions.

16 ll infiltration and microglial activation in periventricular and superficial white matter structures

17 C-PK11195 was significantly increased in the periventricular and total NAWM (P = 0.016 and P < 0.001,

18 k was seen in the facial and vagal lobes and periventricular and ventral regions of the medulla oblon

19 including the medial preoptic, anteroventral periventricular, arcuate, and ventromedial nuclei.

20 and left medial prefrontal cortex and in the periventricular area in the patient group regardless of

21 urons were scattered throughout the preoptic periventricular areas (PV), but the vast majority of Kis

22 ghly vascularized compared to non-neurogenic periventricular areas, within which NSCs and precursors

23 CR-ir cell bodies were mainly distributed in periventricular areas.

24 oestradiol in the hypothalamic anteroventral periventricular (AVPV) and arcuate (ARC) nuclei, while t

25 ding kisspeptin neurons in the anteroventral periventricular (AVPV) and arcuate nuclei, providing hom

26 speptin neurons in arcuate and anteroventral periventricular (AVPV) nuclei, respectively.

27 ions of PR+ VMH neurons to the anteroventral periventricular (AVPV) nucleus change across the 5-day m

28 The first periventricular band was excluded from analysis to mitig

29 Here we show that periventricular blood vessels selectively influence neoc

30 The combination of microencephaly and periventricular calcifications was the most common neuro

31 aging abnormalities included microencephaly, periventricular calcifications, ventriculomegaly, pachyg

32 teral ventricular dilatation, necrotic foci, periventricular cysts and intraventricular hemorrhages w

33 cerebellar hypoplasia, porencephalic cysts, periventricular cysts, and hydrocephalus.

34 aired both the production and positioning of periventricular dopamine neurons.

35 , including any of the following: persistent periventricular echogenicity or echolucency on neuroimag

36 dymal cell microvilli and the development of periventricular edema.

37 Brain embryonic periventricular endothelial cells (PVEC) crosstalk with

38 Our data suggest that periventricular endothelial cells have intrinsic program

39 In addition, periventricular endothelial cells house a GABA signaling

40 Co-culture of human periventricular endothelial cells with human interneuron

41 We generated human periventricular endothelial cells, using human pluripote

42 urons occur in the white matter distant from periventricular foci, including the subplate region, in

43 ted in the subcortical frontal lobe, and the periventricular frontal and parietal caps of the brain.

44 fically in the subcortical frontal lobe, and periventricular frontal and parietal caps.

45 the generation of neural progenitors in the periventricular germinal zones, cell proliferation chara

46 ile AF64A damaged septal cholinergic fibers, periventricular GnRH-immunoreactive fibers remained inta

47 glutamatergic neurons were found within the periventricular gray layer throughout the brainstem, wit

48 cortex, medial thalamus, and periaqueductal/periventricular gray.

49 r each species, such as the preeminence of a periventricular group only in the rat, the lack of poste

50 VPT individuals with evidence of periventricular haemorrhage and ventricular dilatation o

51 Neonatal periventricular haemorrhage with ventricular dilatation

52 e onset of neuroblast migration give rise to periventricular heterotopia (clusters of neurons along t

53 Periventricular heterotopia (PH) is a disorder character

54 Periventricular heterotopia (PH) is a human malformation

55 ne filamin A (FlnA) cause the human disorder periventricular heterotopia (PH).

56 ity gene lgl, exhibit MCCs resembling severe periventricular heterotopia (PH).

57 Periventricular heterotopia (PVH) is a congenital malfor

58 ng the perinatal period (GD19-PN2) induces a periventricular heterotopia in 100% of the offspring.

59 yonic mouse forebrain frequently resulted in periventricular heterotopia, developmental abnormalities

60 short stature, microcephaly, lissencephaly, periventricular heterotopia, polymicrogyria and other ma

61 he reeler mouse, double cortex syndrome, and periventricular heterotopia.

62 Periventricular heterotopias is a malformation of cortic

63 nd one child with cerebral ventriculomegaly, periventricular heterotopias, echogenic kidneys, and ren

64 tions in FLNA, the gene for filamin A, cause periventricular heterotopias.

65 cumulation of neural progenitor cells in the periventricular, hippocampal, cerebellar and olfactory b

66 0.24), and cholesterol (r = 0.20), and with periventricular hyperintensities for glycated hemoglobin

67 nts, showing ventricular enlargement in one, periventricular hyperintensities in another and frontal

68 factors and scores for deep white matter and periventricular hyperintensities, and stepwise multiple

69 interval, 1.64-3.79; P = 2 x 10(-5)) but not periventricular hyperintensities.

70 ic nucleus, accessory neurosecretory nuclei, periventricular hypothalamic nucleus, dorsomedial hypoth

71 ly to the periventricular hypothalamic zone, periventricular hypothalamic region, and lateral hypotha

72 ns, TH-ir neurons localized primarily to the periventricular hypothalamic zone, periventricular hypot

73 xpressing neurons are located in the ventral periventricular hypothalamus (the equivalent of the mamm

74 , an array of cells extending throughout the periventricular hypothalamus and ventral thalamus were e

75 essing neurons (located in the anteroventral periventricular hypothalamus, Kiss1(AVPV), and arcuate h

76 found in discrete populations in the ventral periventricular hypothalamus, the proposed arcuate homol

77 the preoptic area and in rostral and caudal periventricular hypothalamus.

78 ing lesions with macrophages and lymphocytic periventricular infiltrates, and chronic, inactive demye

79 While immunohistochemical analysis revealed periventricular inflammation and a loss of integrity of

80 least two of four locations (juxtacortical, periventricular, infratentorial, and spinal-cord) and DI

81 gene expression combinations were located in periventricular, intermediate and superficial strata.

82 y low-gestational-age infants with low-grade periventricular-intraventricular hemorrhage are not sign

83 igher rates of early-onset sepsis and severe periventricular-intraventricular hemorrhage as compared

84 Low-grade periventricular-intraventricular hemorrhage is a common

85 Low-grade periventricular-intraventricular hemorrhage.

86 suggest that needing three lesions to define periventricular involvement might slightly increase spec

87 asing the number of lesions needed to define periventricular involvement to three, combining cortical

88 The correlation between increased periventricular lesion burden and decreased CMT indicati

89 To quantify periventricular lesion load (PV-LL), we generated ventri

90 ular volume and total lesion load, increased periventricular lesion occupancy (percentage of PV-LL) s

91 ike phenotype that recapitulates the primary periventricular lesion, ventricular enlargement, and the

92 rise to abnormal astroglial cells and induce periventricular lesions and hemorrhage that leads to cer

93 Periventricular lesions developed within 10 d and evolve

94 Requirement of three periventricular lesions resulted in slightly lower sensi

95 scores for cortical, deep white matter, and periventricular lesions were 0.93 +/- 0.05, 0.97 +/- 0.0

96 f juxtacortical/cortical lesions, absence of periventricular lesions, absence of Dawson fingers, pres

97 es in compartments close to the CSF, such as periventricular lesions, might correlate with cortical p

98 ne-derived cells contribute to the repair of periventricular lesions.

99 g enterocolitis, bronchopulmonary dysplasia, periventricular leucomalacia, and retinopathy of prematu

100 epsis, intraventricular hemorrhage >grade 2, periventricular leukomalacia >grade 1, or necrotizing en

101 brain injuries, including the development of periventricular leukomalacia (PVL) and cerebral palsy (C

102 causes of perinatal brain injury leading to periventricular leukomalacia (PVL) and cerebral palsy.

103 Periventricular leukomalacia (PVL) is a structural loss

104 nitive abnormalities in preterm infants with periventricular leukomalacia (PVL) is uncertain.

105 te of cerebral palsy in premature infants is periventricular leukomalacia (PVL), a disorder of the im

106 is generally thought to consist primarily of periventricular leukomalacia (PVL), a distinctive form o

107 lying cerebral palsy in premature infants is periventricular leukomalacia (PVL), a lesion of the imma

108 ellular models of Huntington's disease (HD), periventricular leukomalacia (PVL), and kidney dysfuncti

109 e infant leads to white matter injury termed periventricular leukomalacia (PVL), the leading cause of

110 e matter is important in the pathogenesis of periventricular leukomalacia (PVL), the major pathologic

111 white-matter damage of immaturity, including periventricular leukomalacia (PVL), was the most common

112 ctor for multiple brain pathologies, notably periventricular leukomalacia (PVL), which is distinguish

113 lnerability for white matter injury, such as periventricular leukomalacia (PVL).

114 7.5 percent vs. 23.9 percent, P=0.03) and of periventricular leukomalacia alone (5.2 percent vs. 9.0

115 tial failure of recovery in insults, such as periventricular leukomalacia and multiple sclerosis.

116 n lesions of white matter disorders, such as periventricular leukomalacia and multiple sclerosis.

117 associated with BAS; however, neonates with periventricular leukomalacia had lower preoperative oxyg

118 plications with respect to the mechanisms of periventricular leukomalacia in infants and of persisten

119 severe intracranial hemorrhage and/or cystic periventricular leukomalacia in the neonatal period.

120 ith clinical potential for disorders such as periventricular leukomalacia in the preterm and neonatal

121 Periventricular leukomalacia is the predominant injury i

122 mes of myelination or remyelination, such as periventricular leukomalacia leading to cerebral palsy,

123 elination is also noted in children with the periventricular leukomalacia of cerebral palsy, another

124 Periventricular leukomalacia should be added to the diff

125 In preterm infants (n = 44), periventricular leukomalacia was associated with larger

126 Periventricular leukomalacia was not associated with BAS

127 ncluding multiple sclerosis, cerebral palsy (periventricular leukomalacia), and spinal cord injury.

128 ffuse white matter injury, previously called periventricular leukomalacia, a major form of brain inju

129 lasia, sepsis, intraventricular haemorrhage, periventricular leukomalacia, and necrotising enterocoli

130 mes included severe intracranial hemorrhage, periventricular leukomalacia, and ventriculomegaly.

131 asia, severe intracranial hemorrhage, cystic periventricular leukomalacia, and/or severe retinopathy

132 on, length of stay, intracranial hemorrhage, periventricular leukomalacia, chronic lung disease, pate

133 ntraventricular hemorrhage grade 3-4, cystic periventricular leukomalacia, necrotizing enterocolitis,

134 mbined end point of intracranial hemorrhage, periventricular leukomalacia, or ventriculomegaly (17.5

135 II or IV intraventricular hemorrhage, cystic periventricular leukomalacia, severe bronchopulmonary dy

136 lar hemorrhage grade of 3 or greater, cystic periventricular leukomalacia, surgical necrotizing enter

137 ing diseases, such as multiple sclerosis and periventricular leukomalacia.

138 prematurity, necrotizing enterocolitis, and periventricular leukomalacia.

139 to have hypoxic brain injury in the form of periventricular leukomalacia.

140 time to surgery (P=0.028) than those without periventricular leukomalacia.

141 e rates of severe intracranial hemorrhage or periventricular leukomalacia.

142 r hemorrhage grade of 3 or greater or cystic periventricular leukomalacia.

143 severe intracranial hemorrhage and/or cystic periventricular leukomalacia.

144 eath; death, intraventricular hemorrhage, or periventricular leukomalacia; and death or necrotizing e

145 le sclerosis lesions (e.g. juxtacortical and periventricular location, cortical involvement).

146 An abnormal periventricular magnetization transfer ratio gradient oc

147 nal migration, resulting in the formation of periventricular masses.

148 d the expression and function of OCTs in the periventricular medial hypothalamus of male Sprague Dawl

149 Periventricular microglia exhibit a set of similar chara

150 Periventricular microglia exhibit notable differences ac

151 stinctions suggest differential functions of periventricular microglia in rat and rhesus monkey, yet

152 ct subset of microglial cells, which we term periventricular microglia, that are located near the lat

153 up showed prolonged mean transit time in the periventricular NAWM, as compared with the control group

154 of ganglionic eminence vessels and resultant periventricular neural apoptosis resulted in a PVL-like

155 differentiation and overexpression produces periventricular neuronal masses, demonstrating its funct

156 lamin function, leading to the generation of periventricular neurons independent of normal neocortica

157 a model for how temporal dynamics in tectal periventricular neurons might arise from computations be

158 we found transgene expression in subsets of periventricular neurons of the hypothalamus, Purkinje ce

159 and their subventricular dispersion from the periventricular niche during neocortical development.

160 s), polymicrogyria with megalencephaly (20), periventricular nodular heterotopia (61), and pachygyria

161 und mutations in DCX and LIS1), persons with periventricular nodular heterotopia (FLNA), and persons

162 ear their site of origin and form persistent periventricular nodular heterotopia (PH).

163 Neurodevelopmental disorders with periventricular nodular heterotopia (PNH) are etiologica

164 Polymicrogyria (PMG) and periventricular nodular heterotopia (PNH) are two develo

165 gate whether it is possible in patients with periventricular nodular heterotopia (PVNH) to detect abn

166 eral cortical malformations, polymicrogyria, periventricular nodular heterotopia and diffuse megalenc

167 and Melnick-Needles syndrome, with X-linked periventricular nodular heterotopia and FG syndrome (Omi

168 ) recording in 8 patients with epilepsy from periventricular nodular heterotopia and matched healthy

169 zygous loss of function of human FLNA causes periventricular nodular heterotopia in females and is ge

170 inct neuronal migration disorders, including periventricular nodular heterotopia, subcortical band he

171 ree cases of schizencephaly, and 15 cases of periventricular nodular heterotopia.

172 indirect interactor with FlnA, also lead to periventricular nodule formation in mice.

173 enudation of the neuroepithelium, leading to periventricular nodule formation.

174 migrated appropriately into the cortex, that periventricular nodules were primarily composed of later

175 but an investigation of the relationship of periventricular normal-appearing white matter abnormalit

176 e anteroventral periventricular and anterior periventricular nuclei (AVPV/PeN) of males and females e

177 roducing oxytocin in the paraventricular and periventricular nuclei (PVN and PeVN, respectively) are

178 tin neurons in the arcuate and anteroventral periventricular nuclei are postulated to mediate negativ

179 lei of the thalamus, and paraventricular and periventricular nuclei of the hypothalamus.

180 ventricular nucleus (PVH), the anteroventral periventricular nucleus (AVPe), and the central nucleus

181 vation of Kiss1 neurons in the anteroventral periventricular nucleus (AVPV) is linked to the inductio

182 In rodents, the hypothalamic anteroventral periventricular nucleus (AVPV) is sexually differentiate

183 The expression of Kiss1 in the anteroventral periventricular nucleus (AVPV) is sexually dimorphic, an

184 ese studies, we focused on the anteroventral periventricular nucleus (AVPV), a nucleus that is larger

185 pha, ERbeta, and Kiss1 in the anterioventral periventricular nucleus (AVPV), medial preoptic area (MP

186 creased NOP mRNA expression in anteroventral periventricular nucleus (AVPV), median preoptic nucleus,

187 erifornical area (LH/PFA), and anteroventral periventricular nucleus (AVPV).

188 the expression of Kiss1 in the anteroventral periventricular nucleus (AVPV).

189 rominent in the female rostral anteroventral periventricular nucleus (AVPV/PeN), but largely absent i

190 ll bodies were localized in the intermediate periventricular nucleus (IPe) in the hypothalamus, as de

191 i of male Sprague-Dawley rats (anteroventral periventricular nucleus [AVPV], median preoptic area [Me

192 mRNA-containing neurons in the anteroventral periventricular nucleus and likewise possessed fewer mot

193 ex difference was found in the anteroventral periventricular nucleus of P21, but not P0 or P4, mice,

194 thalamic and hypothalamic nuclei, including periventricular nucleus of posterior tuberculum, lateral

195 Neurons in the anteroventral periventricular nucleus of the hypothalamus (AVPV) media

196 n was increased in the subfornical organ and periventricular nucleus of the hypothalamus, but not in

197 ion of dopamine neurons in the anteroventral periventricular nucleus of the preoptic region of the hy

198 ut3 and Nmur2 genes, and placed these in the periventricular nucleus with many synaptic afferents ari

199 area, median preoptic nucleus, anteroventral periventricular nucleus, and bed nucleus of the stria te

200 was detected in the retrochiasmatic nucleus, periventricular nucleus, arcuate nucleus and restricted

201 hypothalamus, arcuate nucleus, anteroventral periventricular nucleus, medial preoptic nucleus, parave

202 n the medial preoptic area and anteroventral periventricular nucleus, regions important for generatio

203 ales' medial preoptic nucleus, anteroventral periventricular nucleus, ventromedial hypothalamus, and

204 matter tracts in the corpus callosum and the periventricular optic radiations.

205 erebral hemorrhage, seizure, cardiomyopathy, periventricular or intraventricular hemorrhage, necrotiz

206 Open-ring lesions were periventricular or juxtacortical and enhanced centripeta

207 particular, the dopaminergic nucleus of the periventricular organ was evidenced with dopamine antibo

208 clei, the hypothalamus including supraoptic, periventricular, paraventricular (PVN), arcuate nuclei a

209 50% and 90% of TH-expressing neurons in the periventricular, paraventricular, and arcuate hypothalam

210 Minocycline treatment decreases cortical and periventricular pathology in the chronic phase of EAE, i

211 uromyelitis optica patients (18.1% brainstem periventricular/periaqueductal, 32.7% periependymal alon

212 f EC stability, ependymal cell function, and periventricular permeability.

213 Diencephalic TH-ir neurons in the periventricular posterior tuberculum, known to be dopami

214 tments like the fetal cortex, but low in the periventricular progenitor cell regions.

215 We found that periventricular PV-immunofluorescence showed positive co

216 e third ventricle (RP3V) and in the anterior periventricular (PVa), arcuate, and dorsomedial hypothal

217 ons of the arcuate nucleus and anteroventral-periventricular region (AVPV) may differentially regulat

218 eased number and morphologic changes) in the periventricular region and hippocampus of the brain of n

219 e suprachiasmatic nucleus terminating in the periventricular region immediately dorsal to the nucleus

220 d in medial HVC and pvMSt, a newly described periventricular region in the medial striatum.

221 es of immunoreactive fibers were seen in the periventricular region in the thalamus, hypothalamus, an

222 n-ir cell bodies are detected in the rostral periventricular region of the third ventricle (RP3V) and

223 of kisspeptin neurons located in the rostral periventricular region of the third ventricle (RP3V) of

224 and white matter hyperintensities, including periventricular regions and both frontal and temporal su

225 over, recombinant GALC was found not only in periventricular regions but also at sites distant to the

226 n in association with ependymal surfaces and periventricular regions of formalin-fixed brain tissue,

227 xpression in the ventromedial telencephalon, periventricular regions of the thalamus and anterior hyp

228 Brain (18)F-FAC accumulation localizes to periventricular regions with significant leukocyte infil

229 sion recovery sequences predominantly in the periventricular regions, the posterior limb of the inter

230 rpus callosum, olfactory bulbs, subpial, and periventricular regions.

231 layer separating the ependymal lining from a periventricular ribbon of astrocytes.

232 The early targeting of visual and periventricular structures followed by more widespread C

233 Here we describe cortical, periventricular subcortical lesions and callosal demyeli

234 bes and were mostly confluent, affecting the periventricular subcortical white matter and U-fibers.

235 Neuronal specification occurs at the periventricular surface of the embryonic central nervous

236 ce of brain lesions [T2 lesions (P = 0.918), periventricular T2 lesions (P = 0.580) or gadolinium-enh

237 Central neurocytoma (CN) is a rare periventricular tumor, whose derivation, lineage potenti

238 eviously shown that endothelial cells of the periventricular vascular network are the natural substra

239 Endothelial cells of the periventricular vascular network have molecular identiti

240 perinatal ischemic stroke (APPIS), or fetal periventricular venous infarction (PVI) were recruited.

241 ridization was observed in the anteroventral periventricular, ventromedial preoptic, suprachiasmatic,

242 r and exit from a VEGF-dependent phase, with periventricular vessels being the last to mature.

243 cate that damage to VEGF-dependent, immature periventricular vessels contributes to PVL development.

244 ght a prominent interaction between RGPs and periventricular vessels important for proper production

245 ntal program permitted selective ablation of periventricular vessels via episodic VEGF blockade withi

246 ssively grow radial glial fibers anchored to periventricular vessels.

247 xhibited meningeal, subpial neocortical, and periventricular virus.

248 Volumetric WML measures (deep and periventricular) were obtained with 1.5T magnetic resona

249 rvival of oligodendrocytes in intragyral and periventricular white matter (p < 0.05) and increased br

250 Basal flow in the periventricular white matter (PVWM) was significantly lo

251 mine cerebral hypoxemia in specific regions (periventricular white matter and both hippocampi).

252 he whole brain, and in the internal capsule, periventricular white matter and sensorimotor cortex.

253 and NAA/Cho ratios measured in the posterior periventricular white matter at term-equivalent age is p

254 analysis in neonatal rodent optic nerve and periventricular white matter axons studied under modeled

255 tes similar to the astrocyte ribbon in human periventricular white matter biopsies that is reported t

256 ignificant cerebral white matter hypoplasia, periventricular white matter gliosis, and axonal and epe

257 iated white matter hyperintensities included periventricular white matter hyperintensities (frontal a

258 More severe periventricular white matter hyperintensities also corre

259 revealed: (i) a significant total effect of periventricular white matter hyperintensities on WAB-AQ

260 08); (ii) a non-significant direct effect of periventricular white matter hyperintensities on WAB-AQ

261 significant indirect effects of more severe periventricular white matter hyperintensities on worse a

262 cerebral leukoencephalopathy that harboured periventricular white matter hyperintensities were selec

263 Periventricular white matter injury (PVWMI) in premature

264 Periventricular white matter injury (PWMI) is the leadin

265 Although periventricular white matter injury (PWMI) is the leadin

266 Periventricular white matter injury (PWMI) is the leadin

267 Periventricular white matter injury (PWMI) is the major

268 in premature infants frequently arises from periventricular white matter injury (PWMI), a condition

269 form of injury in the preterm population is periventricular white matter injury (PWMI), a pathology

270 n is a major pathological sequela of chronic periventricular white matter injury in survivors of prem

271 failure in chronic hypoxia-ischemia-induced periventricular white matter injury is related to persis

272 motor functional connectivity in 26 uCP with periventricular white matter lesions (mean age (standard

273 /88]; adjusted RR, 0.20; 95% CI, 0.05-0.90), periventricular white matter loss (18% [14/77] vs 33% [2

274 ing corpus callosum atrophy (7/7 [100%]) and periventricular white matter loss (6/7 [85%]).

275 ther posthemorrhagic ventricular dilation or periventricular white matter loss.

276 Additionally, Cho/Cr ratio in the periventricular white matter region of OSA group was hig

277 ic blood pressure was associated with WMH in periventricular white matter regions.

278 elopment of IVH leads to inflammation of the periventricular white matter, apoptosis and arrested mat

279 ns in the ventricular/subventricular region, periventricular white matter, central white matter, and

280 In the periventricular white matter, NAA/Cho ratio in OSA patie

281 The onset of IVH induces inflammation of the periventricular white matter, which results in arrested

282 om a 2-cm(3) voxel centered in the posterior periventricular white matter.

283 anatomical changes, including damage to the periventricular white matter.

284 ory lesions that have a predilection for the periventricular white matter.

285 ulomegaly (65%) often with colpocephaly, and periventricular white-matter signal abnormalities (68%).

286 higher BMI contributed to increased deep-to-periventricular WMH ratio through elevated IL6.

287 had positive correlations with the total and periventricular WMH volume (r = 0.55 and 0.59, P < 0.01)

288 d with an increasing load of subcortical and periventricular WMHs.

289 The posterior periventricular zone contained short vimentin-immunoposi

290 rosphere morphology of NPCs derived from the periventricular zone of mice brain.

291 arily innervate neuroendocrine nuclei in the periventricular zone of the hypothalamus, including the

292 ng to the median eminence and throughout the periventricular zone of the hypothalamus.

293 The lateral periventricular zone received forceps-like process syste

294 feration of neuroprogenitors in the germinal periventricular zone.

295 ivation cells, preferentially lie within the periventricular zone; the other cell types are distribut

296 rative study of six (D, Vd, Vv, Dm, Dl, Ppa) periventricular zones (PVZs) harboring proliferative cel

297 amine (DA) and serotonin (5-HT) occur in the periventricular zones of the hypothalamic region of most

298 is and stroke, T-cell infiltration occurs in periventricular zones where NPCs are located and is asso

299 and located preferentially in white matter, periventricular zones, and meninges.

300 nhanced radial migration of NSPCs out of the periventricular zones, possibly by epithelial-mesenchyma