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1 on velocities ranging from 7.5 to 27.0m/sec (peroneal).
2 82.0 +/- 2.1/78.3 +/- 1.7) versus MS (tibial/peroneal: 64.3 +/- 1.0/61.2 +/- 0.9, p < 0.0001).
3 was significantly higher in controls (tibial/peroneal: 82.0 +/- 2.1/78.3 +/- 1.7) versus MS (tibial/p
4 f paravertebral, vasti, sartorius, gracilis, peroneal and medial gastrocnemius muscles.
5 SA levels may slow demyelination of the deep peroneal and other peripheral nerves.
6 stimulation of individual fascicles (tibial, peroneal and sural).
7 , compound muscle action potentials ratio of peroneal and tibial nerves (split index, SI) and semi-qu
8 ves innervating both skin and muscle (common peroneal and tibial) or just muscle (lateral/medial gast
9 dies (NCSs) of peripheral motor (ulnar, deep peroneal) and sensory (median, sural) nerves.
10 lectrical stimulation of the femoral, common peroneal, and posterior tibial nerves (targeting the qua
11 otential (CMAP) amplitudes of median, ulnar, peroneal, and tibial nerves (P < 0.001), but was not rel
12  standardized sites adjacent to the sciatic, peroneal, and tibial nerves of one leg.
13                                              Peroneal artery hypoplasia occurred in the clubfoot limb
14 o a cutaneous nerve, usually the superficial peroneal close to the ankle, and intraneural microstimul
15 re subject to fasciotomy of the anterior and peroneal compartment using a dual-incision minimally inv
16 ve fasciotomy to decompress the anterior and peroneal compartments of the lower leg appears to be saf
17 ive technique to decompress the anterior and peroneal compartments of the lower leg.
18                  Release of the anterior and peroneal compartments was successful in all specimens.
19 jor arteries (anterior and posterior tibial, peroneal), demarcation of origin of major arteries, and
20 ressive skeletal muscle weakness in a humero-peroneal distribution, early contractures and prominent
21                                              Peroneal dNCV correlated negatively with long-term glyca
22 ts, eNG, and rNG all had similar patterns of peroneal functional index improvement after implantation
23 every 15 days after surgery to determine the peroneal functional index.
24 01) in femoral, popliteal, posterior tibial, peroneal, gastrocnemial, and soleal veins; reflux was di
25  vein (anterior tibial, posterior tibial, or peroneal) in 243 patients (63.2%) and a muscular branch
26                      MSNA was measured using peroneal microneurography and BRS was measured using the
27 y (MSNA) was measured during wakefulness via peroneal microneurography in seven patients with documen
28                                        SSNA (peroneal microneurography) and red cell flux (laser Dopp
29                                        SSNA (peroneal microneurography) and red cell flux (laser-Dopp
30                         In Protocol 1, SSNA (peroneal microneurography) and red cell flux in the affe
31 red muscle sympathetic nerve activity (MSNA, peroneal microneurography) in 5 healthy humans under con
32           Muscle sympathetic nerve activity (peroneal microneurography) was 74% higher in the older m
33 l of sympathetic activity by measuring MSNA (peroneal microneurography), arterial pressure (arterial
34                            We measured MSNA (peroneal microneurography), arterial pressure (brachial
35 n to sequences of SNA bursts and non-bursts (peroneal microneurography).
36 tested, elevated triglycerides and decreased peroneal motor NCV at baseline significantly correlated
37 001), IENFD (p = 0.04), sural (p = 0.02) and peroneal motor nerve conduction velocity (p = 0.03) dete
38                                              Peroneal motor nerve conduction velocity (p=0.03) and M-
39 nent of the plantar aponeurosis (PAL), short peroneal muscle (SPM) tendon, and third peroneal muscle
40 hort peroneal muscle (SPM) tendon, and third peroneal muscle (TPM) tendon was dissected.
41 uced by afferents of the quadriceps and deep peroneal muscle nerves (which discharged 70-80% of extra
42            Heart rate, arterial pressure and peroneal muscle sympathetic nerve activity (MSNA) were r
43                                  We recorded peroneal muscle sympathetic nerve activity in 9 POTS pat
44 seus muscles progressing to involve foot and peroneal muscles in most but not all cases.
45 s in the distribution of shoulder girdle and peroneal muscles.
46 -Marie-Tooth disease (CMT) due to atrophy of peroneal muscles.
47 rized by weakness in the shoulder-girdle and peroneal muscles.
48     Because height has a retarding effect on peroneal NCV, NCV was adjusted for height (dNCV).
49 ex was conditioned by stimulating the common peroneal nerve (CPN) at short (2, 3, and 4 ms) and long
50                                       Common peroneal nerve (CPN) stimulation paired with transcrania
51 sion of the soleus H-reflex evoked by common peroneal nerve (CPN) stimulation, D1 inhibition] and het
52 rupt cutaneous feedback from the superficial peroneal nerve (foot dorsum) and medial plantar nerve (f
53 ded muscle sympathetic nerve activity in the peroneal nerve (intraneural electrodes) and the ECG (sur
54  amplitude of the ulnar nerve (p=0.0103) and peroneal nerve (p<0.0001), compared with baseline, were
55 olus to identify and protect the superficial peroneal nerve (SPN).
56  neuropathy symptom profile, lower sural and peroneal nerve amplitudes, abnormal thermal thresholds,
57          Morphometric analysis of the distal peroneal nerve and extensor digitorum muscle weight were
58  rat dorsiflexors (n = 46) by activating the peroneal nerve and plantarflexing the foot ~40 deg, corr
59 We assessed SSNA (microneurography) from the peroneal nerve and skin blood flow (forearm laser Dopple
60 y 150 msec), and combined stimulation of the peroneal nerve and the motor cortex with transcranial ma
61 th stimulating electrodes on the left common peroneal nerve and with electromyographic (EMG) electrod
62 unilaterally stimulated via the right common peroneal nerve at 10 Hz and supramaximal voltage for 8 h
63 15+ cells correlated with NCVs of motor deep peroneal nerve at 2 years post-gene therapy, and ARSA le
64 eolus, just before stimulation of the common peroneal nerve at the head of the fibula, decreased the
65  by electrically stimulating the superficial peroneal nerve bilaterally, before and after staggered l
66 mpathetic nerve activity was measured in the peroneal nerve by microneurography, and the slope of the
67           People with severe PAD have poorer peroneal nerve conduction velocity compared with people
68 mporally dependent PAS applied to the common peroneal nerve during the swing phase of walking would i
69 n lower limb motor cortex paired with common peroneal nerve electrical stimulation produces a lasting
70 chronically and to stimulate the superficial peroneal nerve electrically to evoke cutaneous reflexes.
71 lied at ST36-37 acupoints overlying the deep peroneal nerve for 30 min twice weekly for five weeks wh
72                                              Peroneal nerve function was present in half the rats at
73 oots were cut flush to the spinal cord and a peroneal nerve graft was inserted into the lateral spina
74     Continuous unilateral stimulation of the peroneal nerve in rats for 8 h per day for 2 or 7 days c
75                                 Crush of the peroneal nerve induced increased mitochondrial peroxide
76  sympathetic nerve fibres of the superficial peroneal nerve innervating the dorsum of the foot were r
77       The primary endpoint was the change in peroneal nerve motor conduction velocity.
78 toplethysmographic finger arterial pressure, peroneal nerve muscle sympathetic activity and plasma no
79 inger arterial pressures and in 15 patients, peroneal nerve muscle sympathetic activity before and du
80 mographic arterial pressure, respiration and peroneal nerve muscle sympathetic activity in four healt
81 mographic arterial pressure, respiration and peroneal nerve muscle sympathetic activity in nine healt
82 rbon dioxide concentrations and volumes, and peroneal nerve muscle sympathetic activity on Earth (in
83 espiratory carbon dioxide concentrations and peroneal nerve muscle sympathetic activity.
84 tory carbon dioxide levels, tidal volume and peroneal nerve muscle sympathetic activity.
85 erve activity to muscle circulation from the peroneal nerve of 12 chronic heart failure patients whil
86                     Motoneuron perikarya and peroneal nerve of diabetic rats showed no evidence of in
87                                   The common peroneal nerve of Sprague-Dawley rats was transected and
88 on of cutaneous afferents in the superficial peroneal nerve on the locomotor discharges of single med
89                                       Common peroneal nerve palsy was present in two patients.
90                        Measurements included peroneal nerve skin and tibial nerve muscle sympathetic
91 cles was assessed before and after 30 min of peroneal nerve stimulation at motor threshold intensity.
92 ls received either hypothalamic stimulation, peroneal nerve stimulation, or both.
93                These effects were reduced by peroneal nerve stimulation.
94 l patients underwent microneurography of the peroneal nerve to compare the sympathomimetic effects du
95 ted in an surgically created gap in the host peroneal nerve to evaluate their regeneration supporting
96 anglionic section of dorsal roots L4-L6, the peroneal nerve was stimulated (10 Hz, 8 h day(-1)) for 2
97 d muscle sympathetic nerve activity from the peroneal nerve were recorded continuously.
98        Two nerve injuries of the superficial peroneal nerve were reported.
99 ty (MSNA) with intraneural electrodes in the peroneal nerve while the subject inspired (primarily wit
100    We measured MSNA (microneurography of the peroneal nerve) and forearm blood flow (FBF, Doppler ult
101 sculature using intraneural microelectrodes (peroneal nerve) during intranasal cocaine (2 mg/kg, n =
102                MSNA (microneurography of the peroneal nerve), continuous arterial pressure (photoplet
103 l sweat rate and SSNA (microneurography from peroneal nerve).
104 MSNA was measured by microneurography at the peroneal nerve, and arterial blood pressure, electrocard
105 e sciatic nerve and its branches such as the peroneal nerve, the tibial nerve, and the sural nerve.
106 imulation of group I afferents in the common peroneal nerve, was assessed from changes in the H refle
107 etrodotoxin (TTX)-administered to the common peroneal nerve-resulted in reductions in muscle mass of
108 ficantly slower NCV, most pronounced for the peroneal nerve.
109 leus H-reflex with stimulation of the common peroneal nerve.
110 ltaneous stimulation of the hypothalamus and peroneal nerve.
111 in the hypothalamus and in the isolated left peroneal nerve.
112  (conditioned stimulus applied to the common peroneal nerve; test reflex elicited by posterior tibial
113 cally stimulating left and right superficial peroneal nerves before and after two thoracic lateral he
114    Group II afferents of quadriceps and deep peroneal nerves evoked potentials mainly at the rostral
115 isografts (15 mm long) were implanted in the peroneal nerves of F-344 rats.
116 nduction velocity for the median, ulnar, and peroneal nerves was decreased in patients with high vers
117 (brachial plexus, ulnar, femoral, and common peroneal nerves) for biceps brachii, first dorsal intero
118 , deep peroneal, sciatic, tibial, and common peroneal nerves-for up to 12 weeks.
119 hies, particularly of the median, ulnar, and peroneal nerves.
120  truncations and deletions of the tibial and peroneal nerves.
121 pared to muscle of primarily type II fibres (peroneal, Per, 84 % type II).
122 rse peripheral nerves-occipital, vagus, deep peroneal, sciatic, tibial, and common peroneal nerves-fo
123                         The frequently cited peroneal spastic flatfoot is an uncommon means of identi
124 ould contribute to a better understanding of peroneal tendon disorders.
125 mputed tomographic data for the diagnosis of peroneal tendon subluxation or dislocation by using the
126 s who underwent surgery because of suspected peroneal tendon tear (14 tendons).
127 mpingement, osteochondral lesion, or partial peroneal tendon tear).
128 d consider all soft tissue structures (i.e., peroneal tendons, ligaments of the ankle, subtalar joint
129 n unit descended medial and posterior to the peroneal tendons.
130 ctional nerve conduction study analyzing the peroneal, tibial, median motor, and median sensory nerve
131 size of the muscle and the dimensions of the peroneal tubercle and retrotrochlear eminence were recor
132 easured the width, length, and height of the peroneal tubercle in 100 asymptomatic patients, comparin
133 ngs show that ultrasound measurements of the peroneal tubercle were consistent with values reported i
134 d imaging as a promising tool to measure the peroneal tubercle, and it could contribute to a better u
135 otrochlear eminence but not with an enlarged peroneal tubercle.
136 and accurate tool to measure the size of the peroneal tubercle.
137 ar eminence of the calcaneus rather than the peroneal tubercle.
138 ase or a slight increase in NCVs of the deep peroneal, ulnar and medial nerves afterwards.
139 ts (age, 32+/-2 years; mean+/-s.e.m.), MSNA (peroneal) was assessed using standard microneurographic

 
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