ライフサイエンスコーパス: peroxidation

1 glutathione peroxidase, lipidic and protein peroxidation).

2 s (ORAC, TRAP, HORAC and inhibition of lipid peroxidation).

3 roteinases (MMP)-3 and -9 and enhanced lipid peroxidation.

4 signaling pathway that is finalized by lipid peroxidation.

5 Spastin is required to relieve LDs of lipid peroxidation.

6 brogates the cell's protection towards lipid peroxidation.

7 adduct formation, redox activity, and lipid peroxidation.

8 ulation of reactive oxygen species and lipid peroxidation.

9 tochondrial potential and iron-induced lipid peroxidation.

10 etween the chemistry and physiology of lipid peroxidation.

11 e oxygen species levels and associated lipid peroxidation.

12 (Arabidopsis thaliana) leaves against lipid peroxidation.

13 varieties correlated with tolerance to lipid peroxidation.

14 rmed mutagenic DNA adduct derived from lipid peroxidation.

15 o-radicals (ABTS, DPPH) and to inhibit lipid peroxidation.

16 by an increase in oxidative stress and lipid peroxidation.

17 nsfer to the redox partner AhpC required for peroxidation.

18 ha-synuclein generated in vitro by enzymatic peroxidation.

19 PB extract/kg of TTB) displayed higher lipid peroxidation.

20 sis by removing fibrogenic products of lipid peroxidation.

21 changing the regio- and stereospecificity of peroxidation.

22 ess and cell membrane damage caused by lipid peroxidation.

23 f nitric oxide, protein nitration, and lipid peroxidation.

24 c aldehydes produced by metabolism and lipid peroxidation.

25 vitamin E-mediated protection against lipid peroxidation.

26 electivity of the organocatalytic asymmetric peroxidation.

27 resence of nitrotyrosine residues, and lipid peroxidation.

28 could initiate carcinogenesis through lipid peroxidation.

29 droplets, where they are less vulnerable to peroxidation.

30 g a role for myeloperoxidase-dependent lipid peroxidation.

31 on observed in tissue inflammation and lipid peroxidation.

32 reactions that are well precedented in lipid peroxidation.

33 ndogenous reactive oxygen species, and lipid peroxidation.

34 xidant activity by inhibiting membrane lipid peroxidation.

35 ty, decreased mitophagy, and increased lipid peroxidation.

36 ssociated with suppression of (phospho)lipid peroxidation.

37 tworks that induce and suppress lethal lipid peroxidation.

38 d cell death induced by iron-dependent lipid peroxidation.

39 which results in oxidative stress and lipid peroxidation.

40 ell death, inhibiting NO formation and lipid peroxidation.

41 rosis process driven by iron-dependent lipid peroxidation.

42 generated during inflammation-mediated lipid peroxidation.

43 omposition during initiation and propagation peroxidations.

44 mulation), oxidative/nitrative stress (lipid peroxidation, 3-nitrotyrosine formation, and expression

45 eutrophil infiltration (MPO activity), lipid peroxidation (4-HNE), and nitric oxide (iNOS) - were sig

46 oxidation (8-oxo-2'-desoxyguanosine), lipid peroxidation (4-hydroxy-2-nonenal, isoprostane), inflamm

47 lpha: 64% +/- 24% increase; P < 0.05), lipid peroxidation (4-hydroxynonenal, measured by ELISA: 0.30

48 ine (8-NO2Gua)) as well as products of lipid peroxidation (8-iso-prostaglandin F2alpha (8-isoPF2alpha

49 nontransferrin bound iron, markers of lipid peroxidation-8alpha-isoprostanes, protein oxidation-adva

50 of trained mice) and oxidative stress (lipid peroxidation, 9.1 +/- 1.4 vs. 5.2 +/- 0.9 mumol mg(-1) ;

51 ecent advances in our understanding of lipid peroxidation, a degenerative process that is believed to

52 Lipid peroxidation, a major consequence of oxidative stress, g

53 The role of lipid peroxidation, a potent form of oxidative stress, in medi

54 ging ability, linoleic acid and plasma lipid peroxidation ability.

55 key hallmarks of ferroptosis including lipid peroxidation, abnormal iron metabolism, and hypersensiti

56 y identified the novel end-products of lipid peroxidation, accumulating in circulation in hyperlipide

57 increased the tissue iron content and lipid peroxidation accumulation, along with key protein (GPX4

58 rization, production of free radicals, lipid peroxidation, activation of phospholipase C, IP3 recepto

59 a significant reducing power and anti-lipid peroxidation activities.

60 teine, a potent antioxidant, abolished lipid peroxidation activity and ameliorated EAE in IFN-gamma-s

61 Importantly, "free" myelin debris and lipid peroxidation activity at CNS lesions was increased in mi

62 ossess heightened levels of markers of lipid peroxidation after bacterial infection.

63 ee iron, mitochondrial superoxide, and lipid peroxidation, all of which are important hallmarks of fe

64 (~100-nm diameter) form rapidly due to lipid peroxidation, allowing calcium entry to initiate lysosom

65 eral, AntiOxCINs derivatives prevented lipid peroxidation and acted as inhibitors of the mitochondria

66 In addition, antioxidant, anti-lipid peroxidation and antibacterial activities were improved.

67 alkylation by the aldehyde products of lipid peroxidation and by the metabolic byproducts of vinyl ch

68 found high consistency in measures of lipid peroxidation and circulating non-enzymatic antioxidants

69 to chronic exposure was found through lipid peroxidation and DNA damage assessments of liver, gill,

70 FA-AKI in mice associates with lipid peroxidation and downregulation of glutathione metabolis

71 d by a marked reduction in VPA-induced lipid peroxidation and endoplasmic reticulum stress.

72 , as indicated by reduced ROS, lowered lipid peroxidation and enhanced photosynthesis.

73 as a consequence, promotes tumour cell lipid peroxidation and ferroptosis.

74 PX4 and glutathione to suppress phospholipid peroxidation and ferroptosis.

75 Less lipid peroxidation and higher alpha-amylase activity, higher a

76 in IR-induced lung injury by reducing lipid peroxidation and increasing the glutathione and GPX4 lev

77 ane area increase corresponds to the lipids' peroxidation and is initiated by the delocalization of t

78 d Gpx4 expression as well as increased lipid peroxidation and is likewise suppressed by Fer-1 treatme

79 Ferroptosis is triggered by lipid peroxidation and is tightly regulated by SLC7A11, a key

80 ralleled with increased levels of ROS, lipid peroxidation and lactate, depletion in glutathione (GSH)

81 dialdehyde (MDA) epitopes, products of lipid peroxidation and markers for enhanced oxidative stress,

82 Our observations suggest that lipid peroxidation and mitochondrial biogenesis are the key in

83 Our observations suggest that lipid peroxidation and mitochondrial biogenesis are the key in

84 Also, lipid peroxidation and mitochondrial dysfunction appeared to b

85 Lipid peroxidation and mitochondrial function and structure we

86 seen including elevated mitochondrial lipid peroxidation and mitochondrial membrane defects, as well

87 MJ33 increased the levels of lipid peroxidation and mitochondrial O2(* horizontal line ) pr

88 Levels of lipid peroxidation and of superoxide anion (O2(* horizontal li

89 identify 5-HT as a potent inhibitor of lipid peroxidation and offer a different perspective on the ro

90 protein), that triggers iron-mediated lipid peroxidation and oligodendrocyte loss (via ferroptosis).

91 A and proteins, amino acid metabolism, lipid peroxidation and one carbon metabolism (1-C).

92 nt defence, subjugation of TBI-induced lipid peroxidation and phenotypic polarization of intestinal m

93 Lipid peroxidation and polar compounds formation in sunflower

94 Rice seedlings also exhibited severe lipid peroxidation and protein carbonylation, for oxidative st

95 ytoprotective activity on lymphocytes, lipid peroxidation and protein degradation.

96 ars, sucrose, ethylene, ascorbic acid, lipid peroxidation and reactive oxygen species.

97 of piperine combined with different in vitro peroxidation and reducing assays: (i) 1,1-diphenyl-2-pic

98 activation of SAT1 expression induces lipid peroxidation and sensitizes cells to undergo ferroptosis

99 ated cell death caused by unrestricted lipid peroxidation and subsequent membrane damage.

100 m mounting an adequate defense against lipid peroxidation and thereby promote ferroptosis.

101 GPX4 deficiency enhanced cellular lipid peroxidation and thus specifically inhibited the cGAS-ST

102 Leaf proline content, lipid peroxidation, and activities of antioxidant enzymes (CAT

103 stress parameters such as glutathione, lipid peroxidation, and calcium levels along with the glutathi

104 respiratory deficiency, sensitivity to lipid peroxidation, and decreased Q(6) biosynthesis of the coq

105 n in senescence markers in older mice, lipid peroxidation, and fibrosis.

106 ced mitochondrial oxidative stress and lipid peroxidation, and induced ferroptosis.

107 sible symptoms (i.e. cell death), less lipid peroxidation, and lower NADPH oxidase activity, indicati

108 hetic pigment contents, plant biomass, lipid peroxidation, and membrane permeability) were not affect

109 g ROS production and scavenging, cardiolipin peroxidation, and mitochondrial protein import.

110 positively with EVOO to inhibit phospholipid peroxidation, and thus, McPC-EEVOO could be a potential

111 ndicate that lipoxygenase activity and lipid peroxidation are increased in those with colon polyps.

112 s cellular membrane damage, mainly via lipid peroxidation as a result of reactive oxygen species (ROS

113 inhibition of TMEM16A or inhibition of lipid peroxidation as potentially powerful therapeutic approac

114 yl radicals), copper-induced LDL-cholesterol peroxidation, as well as alpha-glucosidase and lipase ac

115 sruption of the antioxidant system and lipid peroxidation, as well as alterations in lysosomal membra

116 most effective for protection against lipid peroxidation, as: P#3 (VHHA) > P#5 (LHALLL) > P#7 (LLPHH

117 malondialdehyde (MDA) as a measure of lipid peroxidation, ascorbate, total phenolic concentration (T

118 and in L-alpha-phosphatidylcholine liposome peroxidation assay measured following formation of conju

119 of phenolic compounds in non-enzymatic lipid peroxidation assays.

120 nging, beta-carotene-linoleic acid and lipid peroxidation assays; the antibacterial activity was eval

121 insights into asthma mechanisms once lipidic peroxidation assessed by urinary metabolomics is related

122 st 52.68% of inhibition of the linoleic acid peroxidation at 10 uL/mL and 76.34% of inhibition of the

123 (TUNEL and chromomycin A3 assay), and lipid peroxidation (Bodipy probe) in 18 infertile men with gra

124 5-HT contributes to the termination of lipid peroxidation by direct interaction with active groups of

125 These lipids are the substrates for lipid peroxidation by lipoxygenase enzymes.

126 pids, the rate-limiting substrates for lipid peroxidation, by activating the expression of hypoxia-in

127 lated significantly with (1) increased lipid peroxidation byproducts and endoplasmic reticulum (ER) s

128 XBP1 activation, fueled by lipid peroxidation byproducts, induced a triglyceride biosynth

129 cal targeting of increased endothelial lipid peroxidation can attenuate diabetes-induced comorbiditie

130 scale of inhibition properties of the lipid peroxidation can be devised.

131 found that ferroptotic cell death and lipid peroxidation can be inhibited by treatments that induce

132 Increases in lipid peroxidation can cause ferroptosis, a form of cell death

133 plets protect glia and also neuroblasts from peroxidation chain reactions that can damage many types

134 e control, and decreased the degree of lipid peroxidation compared with the HF diet.

135 he pex11a line showed higher levels of lipid peroxidation content and lower expression of genes invol

136 lve ROS generation, carnosol inhibited lipid peroxidation, contrary to carnosic acid.

137 Lipid peroxidation correlated negatively with ascorbate, TPC,

138 Lipid peroxidation correlated with increased cyst volume as sh

139 Strategies to decrease lipid peroxidation could improve mitochondrial energy generati

140 ochondrial function by measuring H2O2, lipid peroxidation, cytochrome c oxidase activity and mitochon

141 that protects RPE cells in vitro from lipid peroxidation cytotoxicity mediated by 4-hydroxynonenal (

142 vy hydrogen isotope deuterium at the site of peroxidation (D-PUFA) prevented PUFA oxidation and block

143 e aldehydes, like those generated from lipid peroxidation damage, the contributions of these enzymes

144 ted with a decrease in age-exacerbated lipid peroxidation, demyelination and axon loss.

145 levels of reactive oxygen species and lipid peroxidation, depleting and oxidizing glutathione and ni

146 del of assessment (HOMA), and systemic lipid peroxidation determined by plasma F2-isoprostane levels.

147 indicate that activation of TMEM16A by lipid peroxidation drives growth of renal cysts.

148 We report here the mechanism of lipid peroxidation during ferroptosis, which involves phosphor

149 uglandins (isoLGs), are generated from lipid peroxidation during the inflammatory response and form c

150 underpinning the cascade reactions of lipid peroxidation (enzymatic or free radical), the reactive n

151 mary of fundamental concepts regarding lipid peroxidation, experimental tools for the study of such p

152 Antioxidant 11 inhibited peroxidation for 7-fold longer than that recorded with a

153 Our data suggest that lipid peroxidation functions as a spatial redox relay that ena

154 Lipid peroxidation generates reactive dicarbonyls including is

155 ran) and have the potential to inhibit lipid peroxidation given their phenolic structure.

156 te pathway (G6PD), and defense against lipid peroxidation (GPX4) scored high as synthetic sick/lethal

157 acid converged on half-millimetre-long lipid peroxidation gradients that promoted leukocyte attractio

158 cellular metabolism and iron-dependent lipid peroxidation, has been implicated in diseases such as is

159 eath elicited by iron-dependent phospholipid peroxidation, has been implicated in ischemic events.

160 ell death that is induced by excessive lipid peroxidation, has been recently identified as a new tumo

161 risingly, the root and consequences of lipid peroxidation have garnered increasing attention from mul

162 Lipid peroxidation have not changed significantly however, the

163 ted increased reactive oxygen species, lipid peroxidation, histological evidence of balloon degenerat

164 6.51 +/- 3.6 mug/mL) and inhibition of lipid peroxidation (IC50=12.34 +/- 2.3 mug/mL) as compared to

165 ability of these compounds to inhibit lipid peroxidation in a liposome membrane system was examined

166 Additionally, NCX inhibited lipid peroxidation in an emulsified system of Sacha inchi oil

167 Our understanding of lipid peroxidation in biology and medicine is rapidly evolving

168 reases in hepatic weight, lipid content, and peroxidation in C57BL/6J mice.

169 es promise to help clarify the role of lipid peroxidation in cell death and human disease.

170 utathione concentrations and decreased lipid peroxidation in cultured hepatocytes.

171 in / iron overload, ROS production and lipid peroxidation in ectopic lesions.

172 itochondrial biogenesis, and increased lipid peroxidation in female mouse offspring exposed to an obe

173 tration was negatively correlated with lipid peroxidation in foliar tissue under ozone stress and zin

174 prevents anemia, ROS accumulation and lipid peroxidation in Gpx4-deficient cells remain high.

175 ntional red grape juice consumption on lipid peroxidation in healthy individuals.

176 racts of P. trunciflora fruits prevent lipid peroxidation in HepG2 cells with higher efficacy than ot

177 d with significantly increased cardiac lipid peroxidation in HFD-fed WT mice relative to GCN5L1 cKO a

178 and correlated with the inhibition of lipid peroxidation in human erythrocytes (LP) and total conten

179 ng the mechanisms of inhibition of the lipid peroxidation in micelles, in view of bibliographic data,

180 nt properties, preventing iron-induced lipid peroxidation in mitochondria.

181 n, mitochondrial hyperpolarization and lipid peroxidation in neuronal cells, but they do so by distin

182 E-42 has an enhanced capacity to cause lipid peroxidation in primary cortical mouse neurons compared

183 We used it to probe the importance of lipid peroxidation in progression of NASH beyond simple steato

184 MERTK expression and lipid peroxidation in synaptoneurosomes also increased to a si

185 Basil addition inhibited fat peroxidation in the cakes, measured as the malondialdehy

186 8 quadruple mutants prevents increased lipid peroxidation in the vte2 background and restores pathoge

187 a main endogenous product of cellular lipid peroxidation in tissues and is reported to play pathogen

188 ) T cells enhance ferroptosis-specific lipid peroxidation in tumour cells, and that increased ferropt

189 ydroxy-2,6-alkadienals), biomarkers of lipid peroxidation, in exhaled breath condensate of three heal

190 hile, reducing power and inhibition of lipid peroxidation increased.

191 ne-to-glutathione disulfide ratio, and lipid peroxidation indicated that HFD-induced oxidative stress

192 cies production and hence the level of lipid peroxidation, indicating a role of TAG in protection aga

193 f glutathione reductase, catalase, and lipid peroxidation, indicating increased antioxidant defences

194 d fractions of sea buckthorn inhibited lipid peroxidation induced by H2O2, however, the non-polar fra

195 formation of ethylene as a product of lipid peroxidation induced by the respiratory burst.

196 adicals, inhibit non-enzymatic linoleic acid peroxidation, inhibit human serum oxidation in the prese

197 ces produced in Southern Brazil showed lipid peroxidation inhibition abilities in healthy subjects, r

198 e antiradical activity and significant lipid peroxidation inhibition activities, with their IC50 resu

199 free radical scavenging activities and lipid peroxidation inhibition activities.

200 dicaffeoylquinic acid; whereas higher lipid peroxidation inhibition was attributed to the presence o

201 properties (mainly reducing power and lipid peroxidation inhibition), antibacterial activity against

202 Lipid peroxidation is a major consequence of oxidative stress

203 Lipid peroxidation is a salient feature of ferroptosis, an iro

204 Lipid peroxidation is connected to increases in mitochondrial

205 Substrate peroxidation is measured by microamperage-level current

206 How BD affects lipid peroxidation is not known.

207 rate-determining step in free radical lipid peroxidation is the propagation of the peroxyl radical,

208 In cellular membranes, lipid autoxidation (peroxidation) is linked with oxidative stress, age-relat

209 and is photoprotective, as it reduces lipid peroxidation levels.

210 is strongly associated with increased lipid peroxidation levels.

211 by sensing a wide range of endogenous lipid peroxidation ligands and activating innate immune signal

212 e peroxidase 4 (GPX4) protects against lipid peroxidation (LPO) and cell death termed ferroptosis.

213 ads to production of ROS, resulting in lipid peroxidation (LPO) and steatosis in the absence of iron

214 , PUFAs are susceptible to the noxious lipid peroxidation (LPO) chain reaction, which is a common fea

215 oduced endogenously via reactions with lipid peroxidation (LPO) products.

216 50 +/- 360%, respectively, and urinary lipid peroxidation marker malondialdehyde was decreased by 32

217 levels of malondialdehyde (MDA), as a lipid peroxidation marker, and 8-hydroxydeoxyguanosine (8-OHdG

218 Brain death (BD)-related lipid peroxidation, measured as serum malondialdehyde (MDA) le

219 , indicating the occurrence of cell-membrane peroxidation mediated by ROS.

220 The mechanism of the effect of lipid peroxidation, mediated by 4-hydroxynonenal ([4HNE] a byp

221 gen species (ROS) generation, membrane lipid peroxidation, membrane fluidity, intracellular calcium r

222 Increased lipid peroxidation, mitochondrial calcium overload, and mitoch

223 f normal PLA2G6 gene activity leads to lipid peroxidation, mitochondrial dysfunction and subsequent m

224 A detailed mechanism for peroxidation, molecular rearrangement, and deperoxidatio

225 les to implement a UV-induced photocatalytic/peroxidation nanoparticle/DNAzyme reaction cascade that

226 lyphenols and were more active against lipid peroxidation, NO production, and tumour cells growth.

227 Lower lipid peroxidation occurred in the meat of animals that consum

228 No sulfur peroxidation occurs, and the system remains stable.

229 Highly efficient, selective, and direct C-H peroxidation of 9-substituted fluorenes has been achieve

230 ivo from free-radical-catalyzed nonenzymatic peroxidation of alpha-linolenic acid (1).

231 recedented highly enantioselective catalytic peroxidation of enals.

232 )/Ascorbic acid (Fe(+3)/AsA) system mediated peroxidation of l-alpha-phosphatidylcholine aqueous disp

233 f a defined pathway for O2 in regulating the peroxidation of linoleic acid by soybean lipoxygenase 1.

234 d the inhibition of the metmyoglobin-induced peroxidation of linoleic acid.

235 l death that is caused by the iron-dependent peroxidation of lipids(1,2).

236 hotosensitizer; irradiation at 730 nm led to peroxidation of liposomal lipids, allowing drug release.

237 process of intrinsic apoptosis relies on the peroxidation of mitochondrial lipids as a critical molec

238 ly reactive protein crosslinker derived from peroxidation of n-6 polyunsaturated fatty acids and gene

239 Peroxidation of phospholipids in human and mouse kidneys

240 is, a form of regulated cell death driven by peroxidation of phospholipids.

241 and an MDCK in vitro cyst model, we assessed peroxidation of plasma membrane phospholipids in human a

242 mes, which in turn drive ferroptosis through peroxidation of polyunsaturated fatty acids (PUFAs) at t

243 -hydroxynonenal ([4HNE] a byproduct of lipid peroxidation) on mitochondrial function and structure wa

244 g ferroptosis, either by inhibition of lipid peroxidation or by limiting iron retention, mitigates ag

245 radicals by mitochondria thus causing lipid peroxidation, oxidative and acidic stress, which can lea

246 001), blood pressure (P < 0.0001), and lipid peroxidation (P = 0.001) were also observed for the HF a

247 We here identified that targeting the lipid peroxidation product 12(S)-hydroxyeicosatetraenoic acid-

248 CaARP-expressing plants showed lower lipid peroxidation product content in presence or absence of s

249 Lipid peroxidation product-mediated crosslinking of proteins i

250 notoxic N-nitroso compounds (NOCs) and lipid peroxidation products (LPOs) in the gut.

251 ctional role of aS in sequestering the early peroxidation products of fatty acids, thereby reducing t

252 These data suggest that lipid peroxidation products play a role in progression of live

253 iet and traditional medicines and from lipid peroxidation products, in human prostate and renal speci

254 from 2,3-epoxyaldehydes of endogenous lipid peroxidation products, were present in all subjects (16.

255 lable for the generation of neurotoxic lipid peroxidation products.

256 (P < 0.01), and reduced intracellular lipid peroxidation, reactive oxygen species (ROS), and cytokin

257 ayed an increased membrane leakage and lipid peroxidation relative to Cu-GGH and OV-3 alone.

258 ric acid (TBA) number, an indicator of lipid peroxidation responsible for off-flavour generation.

259 tegrity by inducing ROS generation and lipid peroxidation, resulting in decreased membrane fluidity,

260 teracts the respiratory deficiency and lipid peroxidation sensitivity phenotypes of the coq10Delta mu

261 that photoexcited SWCNTs can catalyze lipid peroxidation similarly to lipoxygenases.

262 Thus, cellular stress-induced lipid peroxidation specifically attenuates the STING DNA-sensi

263 operties of a number of antioxidants against peroxidation, started by a 2,2'-azobis[2-(2-imidazolin-2

264 respiratory-incompetent, sensitive to lipid peroxidation stress, and unable to synthesize Q(6) The y

265 Reactive oxygen species and lipid peroxidation strongly activated TMEM16A, leading to depl

266 e predominantly associated with higher lipid peroxidation (TBARS) [exp(beta) = 1.09-1.78, p < 0.01-0.

267 dant activity (FRAP, ABTS), as well as lipid peroxidation (TBARS) were determined at the end of the e

268 UFA-ePLs), which act as substrates for lipid peroxidation that, in turn, results in the induction of

269 lite, and crotonaldehyde, a product of lipid peroxidation, these findings further implicate infiltrat

270 PSEE exhibited a protection of lipid peroxidation threefold higher than a positive control.

271 s, which paralleled with reductions in lipid peroxidation, thus suggesting plants from the highest al

272 d in TTB as a protective agent against lipid peroxidation to extend its shelf-life up to two months.

273 e fused bicyclic core and a cobalt-catalyzed peroxidation to install the peroxide functional group.

274 Lipid peroxidation took place after 3 weeks of storage in dark

275 s, iron-mediated free radicals trigger lipid peroxidation under conditions of glutathione insufficien

276 Increased lipid peroxidation via oxidative stress was also detected by t

277 Reducing LD accumulation in glia and lipid peroxidation via targeted lipase overexpression and/or l

278 Hepatic lipid peroxidation was also elevated in the nose-only group.

279 ant status (SOD, CAT, GPX and GSH) and lipid peroxidation was also studied.

280 Quality of olive oils was improved, since peroxidation was inhibited.

281 were captured by a digital camera, and lipid peroxidation was monitored using colorimetric determinat

282 , egg quality, blood chemistry and egg lipid peroxidation was studied.

283 the antioxidant role of Vitamin E, as lipid peroxidation was suppressed in HeLa cells both under bas

284 Muscle lipid peroxidation was unaffected by the dietary treatments, a

285 ary malondialdehyde (MDA), a marker of lipid peroxidation, was measured in 24 hour urine collections.

286 etermine possible underlying causes of lipid peroxidation, we investigated the renal redox balance by

287 ccumulation, apoptosis, and changes in lipid peroxidation were attenuated.

288 non-treated samples, and H(2)O(2) and lipid peroxidation were concomitantly increased.

289 agmentation, protamine deficiency, and lipid peroxidation were significantly higher in infertile men

290 ion, amount of glutathione stores, and lipid peroxidation were similar irrespective of the insult to

291 unsaturated fatty acids, which inhibit lipid peroxidation, were able to partially rescue the locomoto

292 sity lipoprotein, serum amyloid A, and lipid peroxidation, were significantly altered by polybacteria

293 ps lipid peroxyl radicals that mediate lipid peroxidation, whereas FSP1 catalyses the regeneration of

294 4 inactivation increased production of lipid peroxidation, which led to STING carbonylation at C88 an

295 at inhibits cellular NO production and lipid peroxidation, which set the stage for further exploratio

296 fies Nrf2-driven transcription to fuel lipid peroxidation while inactivating Nrf2-mediated antioxidan

297 werful effect in non-enzymatic linoleic acid peroxidation with IC50 values 2.4 mM +/- 0.21 and 0.055

298 arly and integral component of in vivo lipid peroxidation with important clinical implications as a b

299 polyunsaturated FA and the highest levels of peroxidation without exceeding critical limits.

300 respiratory-deficient and sensitive to lipid peroxidation, yet it continues to produce Q(6) at an imp