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1 g adjunct to inhaled NO for the treatment of persistent pulmonary hypertension.
2 e decreases severe hypoxemia in infants with persistent pulmonary hypertension.
3 of BMPR2 in SMC, decreasing ARRB2 prevented persistent pulmonary hypertension.
4 ecific deletion of Bmpr2 (EC-Bmpr2(-/-)) and persistent pulmonary hypertension.
5 ate or severe right ventricular hypokinesis, persistent pulmonary hypertension, a patent foramen oval
6 oembolic pulmonary hypertension (CTEPH), but persistent pulmonary hypertension after PTE, as a result
7 tion-based cohort study examines the risk of persistent pulmonary hypertension among newborns in Denm
8 mproves systemic oxygenation in infants with persistent pulmonary hypertension and may reduce the nee
9 ysis may identify CTEPH patients at risk for persistent pulmonary hypertension and poor outcome after
10 ed to the underlying disease associated with persistent pulmonary hypertension and that lung recruitm
11 borns with hypoxemic respiratory failure and persistent pulmonary hypertension, and is now a standard
12 ates with hypoxaemic respiratory failure and persistent pulmonary hypertension, but potential adverse
13 neonates transferred for rescue therapy for persistent pulmonary hypertension during the study perio
14 reduced hypoxia-induced vasoconstriction and persistent pulmonary hypertension following recovery fro
16 rginine would correlate with the presence of persistent pulmonary hypertension in newborns and that t
17 psis, septicemia, septic shock, endotoxemia, persistent pulmonary hypertension, nitric oxide, and ext
18 psis, septicemia, septic shock, endotoxemia, persistent pulmonary hypertension, nitric oxide, extraco
21 idepressant use during pregnancy and risk of persistent pulmonary hypertension of the newborn (PPHN)
26 nary vascular resistance after birth, and in persistent pulmonary hypertension of the newborn (PPHN),
27 e serotonin reuptake inhibitors (SSRIs) with persistent pulmonary hypertension of the newborn (PPHN),
28 nation and clinical outcomes in infants with persistent pulmonary hypertension of the newborn and ass
29 ness of inhaled NO treatment in infants with persistent pulmonary hypertension of the newborn and ass
32 ression may increase levels of superoxide in persistent pulmonary hypertension of the newborn lung ti
34 ation (ECMO) use or death among infants with persistent pulmonary hypertension of the newborn who rec
35 al unit admission for greater than 48 hours, persistent pulmonary hypertension of the newborn, or mec
39 reliable in identifying patients at risk for persistent pulmonary hypertension or predicting postoper
41 ation fetal lambs, and in newborn lambs with persistent pulmonary hypertension (PPHN) after prenatal
42 led nitric oxide (iNO) in newborn lambs with persistent pulmonary hypertension (PPHN) following prena
45 full-term infants with severe hypoxemia and persistent pulmonary hypertension were randomly assigned