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1 ne fever virus (CSFV), a devastating porcine pestivirus.
2  diarrhea virus (BVDV), a positive-sense RNA pestivirus.
3 , but did not amplify viral RNA from related pestiviruses.
4 f this family, such as orthoflaviviruses and pestiviruses.
5 the 5' limit of the IRES in both the HCV and pestivirus 5'NTRs.
6                      Notably, replication of pestiviruses, a major threat to milk and meat industries
7 nti-PrP(Sc) activity was independent of anti-pestivirus activity.
8        Altogether, our findings suggest that pestiviruses, although capable of miRNA-independent repl
9            In 2013, envelope proteins from a pestivirus and hepatitis C virus were found to have two
10 rane fusion machinery of the closely related pestiviruses and hepatitis C virus defines a new structu
11              Y2441 is highly conserved among pestiviruses and is located in a region of NS4B predicte
12 he interface explains the acid resistance of pestiviruses and their requirement for a redox activatio
13 bosome entry site-containing picornaviruses, pestiviruses, and hepatitis C viruses.
14  Thus, the zinc-binding ability of N(pro) in pestiviruses appears to be essential for the virus-media
15 ivirus K, commonly known as atypical porcine pestivirus (APPV), is the most common cause of congenita
16 oteins of the hepaciviruses, pegiviruses and pestiviruses are structurally distinct, may represent a
17 e viral diarrhea virus (BVDV), the prototype pestivirus, are divided into cytopathic (cp) and noncyto
18  of Neospora caninum, Toxoplasma gondii, and pestiviruses associated with livestock abortion in Tanza
19 port, we show that the NS3 proteinase of the pestivirus bovine viral diarrhea virus (BVDV) (NADL stra
20  the hypothesis that the NS5A protein of the pestivirus bovine viral diarrhea virus (BVDV) is a zinc-
21 ted the mechanism of miRNA dependency of the pestivirus bovine viral diarrhea virus (BVDV).
22 rallels the observation made for the related pestivirus bovine viral diarrhea virus.
23 activity, while the IIId2 sub-domains of two pestiviruses, classical swine fever virus (CSFV) and bor
24                      miR-17 sequestration by pestiviruses conferred reduced AGO binding and functiona
25      The viral N-terminal protease N(pro) of pestiviruses counteracts cellular antiviral defenses thr
26 947 and similar compounds for the control of pestivirus diseases, and for hepatitis C virus drug disc
27       Despite increasing characterization of pestivirus-encoded proteins, functions for nonstructural
28                                              Pestiviruses form a genus in the Flaviviridae family of
29                                We expect the pestivirus fusion apparatus to be conserved in hepatitis
30 nomes of Flaviviridae of the Hepacivirus and Pestivirus genera contain internal ribosomal entry sites
31 esidues, conserved among the Hepacivirus and Pestivirus genera, fitting the formula of CX17CXCX20C.
32 l species in the Hepacivirus, Pegivirus, and Pestivirus genera.
33 ement in the NS5A proteins of members of the Pestivirus genus has led to the hypothesis that the NS5A
34  calves persistently infected with HoBi-like pestivirus (HoBi-like PI calves) were generated and samp
35 ism of translation initiation on hepacivirus/pestivirus (HP) IRESs, which involves factor-independent
36 e-strand RNA virus and a member of the genus Pestivirus in the family Flaviviridae.
37 st effective measures for controlling bovine pestiviruses, including bovine viral diarrhea virus (BVD
38                                              Pestiviruses, including bovine viral diarrhea virus (BVD
39                                              Pestiviruses, including bovine viral diarrhea virus, are
40 7 inhibits both cytopathic and noncytopathic pestiviruses, including isolates of BVDV-1, BVDV-2, bord
41 a novel antiviral host factor that restricts pestivirus infection.
42                                     Although pestivirus infections impose an important toll on the li
43 mmunosuppression is a feature of a number of pestivirus infections; our studies suggest type III IFN
44  sheds light on the molecular basis by which pestiviruses interplay with the host cell.
45                                              Pestivirus K, commonly known as atypical porcine pestivi
46 unction of this unique viral protease in the pestivirus life cycle remains to be elucidated.
47   This suggests that the Hepaci-, Pegi-, and Pestiviruses may possess a common and novel membrane fus
48                                 Although the pestivirus N-terminal protease, N(pro), has been shown t
49                                          The pestivirus N-terminal protease, N(pro), is essential for
50                                          The pestivirus noncytopathic bovine viral diarrhea virus (BV
51 ongly conserved among related Flavivirus and Pestivirus nontranslated regions.
52    We report here the X-ray structure of the pestivirus NS3 helicase domain (pNS3h) at a 2.5-A resolu
53 e site preference, a structural model of the pestivirus NS3 proteinase predicts a highly hydrophobic
54 the similarities between the Hepacivirus and Pestivirus NS5A proteins and suggest that both proteins
55  classical swine fever virus (CSFV), a fatal pestivirus of pigs, remain unknown.
56 n of bovine viral diarrhea virus, a ruminant pestivirus of the family Flaviviridae, but has no apprec
57                                              Pestivirus particles are composed of an RNA genome, a ho
58 y the single large open reading frame of the pestivirus positive-sense RNA genome and has an autoprot
59 ES domains of several viral RNA genomes: two pestiviruses related to HCV, classical swine fever virus
60 up new questions about pNS3h function during pestivirus replication.
61 e discovery of a small molecule inhibitor of pestivirus replication.
62 ctive NS3 serine proteinase is essential for pestivirus replication.
63   We generated a self-replicating cytopathic pestivirus RNA to enhance production and presentation of
64 ortant for optimal translation initiation of pestivirus RNAs.
65 ing two stem-loops not present in the HCV or pestivirus structures.
66                                              Pestiviruses, such as bovine viral diarrhea virus and cl
67                                              Pestiviruses, such as classical swine fever virus (CSFV)
68    These findings suggest dual mechanisms of pestivirus superinfection exclusion, one at the level of
69                                    Unique to pestiviruses, the N-terminal protein encoded by the bovi
70 lation was multiple logs more resistant than pestivirus to DB772, suggesting that the anti-PrP(Sc) ac
71 eractions, miR-17 and let-7 binding enhanced pestivirus translation and RNA stability.
72    Bovine viral diarrhea virus (BVDV) (genus Pestivirus) was reported to trigger interferon productio
73 (HCV) and was also described for the related pestiviruses, which are important livestock pathogens.