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1 f mitochondria (mitophagy), and peroxisomes (pexophagy).
2 ance is degradation via selective autophagy (pexophagy).
3 bers is through an autophagic process called pexophagy.
4  (PEX2) is the causative agent for mammalian pexophagy.
5 s sequestered within vacuoles as a result of pexophagy.
6 peroxisomal and is required specifically for pexophagy.
7 ks pexophagy, and its overexpression induces pexophagy.
8 e is necessary but not sufficient to trigger pexophagy.
9 heir delivery to the autophagy machinery for pexophagy.
10 pAtg30, mediates peroxisome selection during pexophagy.
11 tions, at least one of which is required for pexophagy.
12 pecies-induced pejvakin-LC3B interaction and pexophagy.
13  Cvt pathway and efficient autophagy but not pexophagy.
14 oxisomes in the process of being engulfed by pexophagy.
15 d the targeted degradation of peroxisomes by pexophagy.
16 storis, Gsa9 is required for glucose-induced pexophagy.
17 appears to be required for an early event in pexophagy.
18 de proteins that might provide signal(s) for pexophagy.
19  with OPTN is a key feature of OPTN-mediated pexophagy.
20 ated peroxisome membrane proteins to prevent pexophagy.
21 f the autophagic machinery for OPTN-mediated pexophagy.
22 as growth and division, rather than impaired pexophagy.
23 o types of selective autophagy, ER-phagy and pexophagy.
24 endently localises to peroxisomes and drives pexophagy.
25 (TbHK1) protein levels are maintained during pexophagy, acidification inactivates the activity.
26 -resolved mapping of peroxisome turnover and pexophagy across development, metabolism, and disease.
27 e shown that these proteins are required for pexophagy and autophagy in P. pastoris and the Cvt pathw
28                                     Although pexophagy and mitophagy also require actin assembly, del
29 y, including cytoplasm-to-vacuole targeting, pexophagy and mitophagy, and mammalian genetic screens h
30 of cellular recycling mechanisms (autophagy, pexophagy and ribosome breakdown) in maintaining cell vi
31 verlap of macroautophagy with the process of pexophagy and with the biosynthetic cytoplasm-to-vacuole
32 ame this protein Atg36 as its absence blocks pexophagy, and its overexpression induces pexophagy.
33                                   Autophagy, pexophagy, and the Cvt pathway are processes that delive
34 oxisome proliferation and identify defective pexophagy as a cause of noise-induced hearing loss.
35 l membrane protein required specifically for pexophagy at the stage of phagophore formation.
36                                       During pexophagy, Atg30 undergoes phosphorylation, a prerequisi
37 ignal transduction pathway are necessary for pexophagy but not for pexophagosome formation or other n
38                          It is necessary for pexophagy, but not for other selective and nonselective
39           We demonstrate that USP30 prevents pexophagy by counteracting the action of the peroxisomal
40  study, we investigated the role of Sar1p in pexophagy by expressing dominant-negative mutant forms o
41 suggesting that the mTORC1 pathway regulates pexophagy by regulating PEX2 expression levels.
42 n Atg11/FIP200 interaction motif to modulate pexophagy by virtue of its conserved phospho-metabolite
43  of Atg26 and Vac8 functions under different pexophagy conditions demonstrates that not only pexophag
44 ains also displayed defects in autophagy and pexophagy, degradative pathways that share protein machi
45   We find that overexpressing USP30 prevents pexophagy during amino acid starvation, and its depletio
46 g Atg11 phosphorylation by Atg1 critical for pexophagy during phosphate starvation.
47            PpAtg30 overexpression stimulates pexophagy even under peroxisome-induction conditions, im
48 Lc3b cDNAs completely restored sound-induced pexophagy, fully prevented the development of oxidative
49                                              Pexophagy (fusion of glycosomes with acidic lysosomes) h
50 ion of peroxisomal membrane proteins signals pexophagy; however, the E3 ligase responsible for mediat
51 iron chelator DFP induces both mitophagy and pexophagy in a BNIP3/NIX-dependent manner.
52 , and xenophagy, has also been implicated in pexophagy in HEK-293 cells, the underlying mechanisms re
53         However, the role of this protein in pexophagy in other yeast remained unclear.
54 eling, provide evidence for the existence of pexophagy in plants, and indicate that peroxisome destru
55 peroxisome pool, and establishes its role in pexophagy in S. cerevisiae.
56 pastoris and the Cvt pathway, autophagy, and pexophagy in S. cerevisiae.
57 endently localized to peroxisomes to promote pexophagy in several physiological conditions, illustrat
58 nd that its interaction with Atg30 regulates pexophagy in the yeast P. pastoris.
59 s, determine the requirements for subsequent pexophagy in yeast.
60 ctive autophagic degradation of peroxisomes (pexophagy) in auditory hair cells from wild-type, but no
61 nases in selective autophagy of peroxisomes (pexophagy) in Saccharomyces cerevisiae.
62 ophagy conditions demonstrates that not only pexophagy inducers, such as glucose or ethanol, but also
63 cid starvation, and its depletion results in pexophagy induction under basal conditions.
64 shes a versatile platform for dissecting how pexophagy integrates with mitochondrial quality control
65         PpAtg30 is required for formation of pexophagy intermediates, such as the micropexophagy appa
66                                              Pexophagy is a process that selectively degrades peroxis
67                                 For example, pexophagy is a selective process for the regulated degra
68 hat the function of sterol glucoside (SG) in pexophagy is both species and peroxisome inducer specifi
69                         We further show that pexophagy is stimulated under the same physiological con
70 ne via interaction with peroxins, and during pexophagy it colocalizes transiently at the preautophago
71 terminal half of overexpressed OPTN triggers pexophagy, likely by oligomerizing with endogenous OPTN.
72 , regulation of division and protein import, pexophagy, matrix protein degradation, solute transport,
73 r hydrolase, aminopeptidase I (API), whereas pexophagy mediates the delivery of excess peroxisomes fo
74 n iron chelation and show that mitophagy and pexophagy occur in a BNIP3L/NIX-dependent manner.
75 ls that activate this pathway do not trigger pexophagy on their own, suggesting that this MAPK cascad
76 ential for Cvt vesicle formation but not for pexophagy or induction of autophagy.
77 stinct from endoplasmic reticulum-autophagy, pexophagy, or mitophagy, despite the close association b
78                             Both the Cvt and pexophagy pathways are selective processes that specific
79        Overall, our results demonstrate that pexophagy plays a key role in noise-induced peroxisome p
80                                       During pexophagy, PpAtg30 undergoes multiple phosphorylations,
81                          Autophagy (Apg) and pexophagy, processes that use the majority of the same p
82                          The Pichia pastoris pexophagy receptor Atg30 is recruited to peroxisomes und
83  We find that latent activation of the yeast pexophagy receptor Atg36 by the casein kinase Hrr25 in r
84           We conclude that Pex3 recruits the pexophagy receptor Atg36.
85 gest that pejvakin acts as a redox-activated pexophagy receptor/adaptor, thereby identifying a previo
86           However, the mechanisms regulating pexophagy remain poorly understood in mammalian cells.
87                              We propose that pexophagy requires the simultaneous activation of this M
88  identified as an activator and inhibitor of pexophagy, respectively.
89 autophagy, in processes termed mitophagy and pexophagy, respectively.
90                            Additionally, the pexophagy-specific phagophore assembly site, organized b
91 ome turnover by autophagy-related processes (pexophagy), termed micropexophagy and macropexophagy, is
92 solated pex3 alleles blocked specifically in pexophagy that cannot recruit Atg36 to peroxisomes.
93  regulator, is also necessary for regulating pexophagy, the selective autophagic degradation of perox
94 size of peroxisomes before their turnover by pexophagy, the selective autophagy of peroxisomes, and f
95                                              Pexophagy, the selective degradation of peroxisomes, is
96 rols the selective autophagy of peroxisomes (pexophagy) through the assembly of a receptor protein co
97 e for NIX, not only in mitophagy but also in pexophagy, thus illustrating the interconnection between
98 rgo rapid, selective autophagic degradation (pexophagy) when the metabolic pathways they contain are
99 on of both mitochondria and peroxisomes (via pexophagy), whereas Hog1 functions specifically in mitop
100 ked autophagic flux and decreased mitophagy, pexophagy, xenophagy, and ribophagy.