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1 gp91(phox) and p67(phox) (in which "phox" is phagocyte oxidase).
2 e reactive oxygen species generated by NADPH phagocyte oxidase.
3 g is lacking in the absence of a functioning phagocyte oxidase.
4 ofound block in superoxide production by the phagocyte oxidase.
5 etely reversed in mice lacking both iNOS and phagocyte oxidase.
6 gamma-regulated macrophage mechanism-iNOS or phagocyte oxidase.
7 ve oxygen species but was independent of the phagocyte oxidase.
8 antly corrected the CGD functional defect in phagocyte oxidase activity in vitro.
9 d CGD, in which high-level reconstitution of phagocyte oxidase activity may be important for full cor
10                   p40(phox) contains SH3 and phagocyte oxidase and Bem1p (PB1) domains that can media
11 munity help explain an apparent paradox: the phagocyte oxidase and inducible nitric oxide synthase ar
12 n activated primary macrophages lacking both phagocyte oxidase and inducible nitric oxide synthase.
13 eactive oxygen intermediate (ROI)-generating phagocyte oxidase and the reactive nitrogen intermediate
14 regulatory proteins during activation of the phagocyte oxidase, and its translocation to the membrane
15 licited host defense proteins, including the phagocyte oxidase, antimicrobial peptides, and autophagy
16 emented with lipA ROS derived from the NADPH phagocyte oxidase complex and RNS derived from the induc
17 lmonella to inhibit the recruitment of NADPH phagocyte oxidase-containing vesicles to SCVs.
18  (iNOS) knockout (KO) and respiratory burst (phagocyte oxidase)-deficient chronic granulomatous disea
19 scherichia coli cells ingested by normal and phagocyte oxidase-deficient human neutrophils.
20 itrite and F(2)-isoprostanes was observed in phagocyte oxidase-deficient mice.
21                             We conclude that phagocyte oxidase-derived reactive oxygen species (ROS)
22       We further conclude that while neither phagocyte oxidase-derived ROS nor iNOS-derived RNS are e
23 ella appears to be linked to an active NADPH phagocyte oxidase enzymatic complex, since the flavoprot
24 homologues of the catalytic component of the phagocyte oxidase, gp91phox, were identified in various
25 b1, YOTB/ZK632.12, Vac1, and EEA1) and Vam7p phagocyte oxidase homology domains, which are revealed t
26 exposed to oxyradicals produced by the NADPH phagocyte oxidase in an acute model of infection.
27 is required for the bacterium to resist host phagocyte oxidase in vivo.
28 stimulated macrophages from mice lacking the phagocyte oxidase inhibited escape from vacuoles by the
29 ls and indicate that a system related to the phagocyte oxidase is active in these cells.
30            They also suggest that neutrophil phagocyte oxidase is not critical for defense against E.
31 ctive oxygen species (ROS) produced by NADPH phagocyte oxidase isoform (NOX2) are critical for the el
32 earance of H. pylori from interleukin-10 and phagocyte oxidase mice, provide evidence that severe inf
33 VSMCs) derived from wild-type (p47phox(+/+); phagocyte oxidase) mice with those from mice that lack p
34 tracheal myocytes from wild-type versus gp91 phagocyte oxidase null mice.
35 lation of the NOX2 component, p47phox (phox: phagocyte oxidase), on its mitogen-activated protein kin
36 glycemia induced Nox4, but not Nox1, and p22 phagocyte oxidase (p22phox) expression and IGF-I stimula
37 Herein, we evaluated humoral immunity in the phagocyte oxidase p47(phox)-deficient model of CGD and f
38 D phosphoprotein is the 67-kD subunit of the phagocyte oxidase (p67-phox).
39                The contribution of the NADPH phagocyte oxidase (phox) and inducible nitric oxide (NO)
40                       The roles of the NADPH phagocyte oxidase (phox) and inducible nitric oxide synt
41                           Mice deficient for phagocyte oxidase (Phox) and nitric oxide synthase 2 (NO
42 oduction of reactive oxygen intermediates by phagocyte oxidase (phox) and reactive nitrogen intermedi
43 ne that reversibly blocked the activation of phagocyte oxidase (phox) in human neutrophils in respons
44  which is dependent on the respiratory burst phagocyte oxidase (phox) is succeeded by a prolonged nit
45                                          The phagocyte oxidase (Phox) protein p40(phox) contains a Ph
46 lting in membrane translocation of cytosolic phagocyte oxidase (phox) proteins.
47 icating close functional similarities to the phagocyte oxidase (phox).
48        Here we have investigated the role of phagocyte oxidase (PHOX, gp91phox) in the production of
49 of the CYBB and NCF2 genes, which encode the phagocyte oxidase proteins gp91(PHOX) and p67(PHOX), res
50           The CYBB and NCF2 genes encode the phagocyte oxidase proteins gp91(PHOX) and p67(PHOX), res
51 nt in inducible nitric oxide synthase and/or phagocyte oxidase revealed that these two antimicrobial
52 ducible nitric oxide synthase (iNOS), or the phagocyte oxidase subunit p47.
53 1phox(-/-) mice that lack a functional NADPH phagocyte oxidase, suggesting that sigma(E) plays an imp
54 ologous to gp91(phox) (91-kDa subunit of the phagocyte oxidase), the electron-transporting subunit of
55 cleotide (phosphate) oxidases related to the phagocyte oxidase, the Nox/Duox family, provides additio
56 rget cells but at the same time activate the phagocyte oxidase to generate microbicidal products in i
57  platelet response to S protein via p47phox (phagocyte oxidase) upregulation.