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1 hest expression in the nervous system and in pharyngeal muscle.
2 ction coupling in the Caenorhabditis elegans pharyngeal muscle.
3 o specifically activate ceh-22 expression in pharyngeal muscle.
4 oline receptor subunit that functions in the pharyngeal muscle.
5 alcium-dependent processes in the C. elegans pharyngeal muscle.
6 MC, and it is required for MC stimulation of pharyngeal muscle.
7 dant to those of mlc-2 in both body-wall and pharyngeal muscle.
8 ly rescues a ceh-22 mutant when expressed in pharyngeal muscle.
9 as observed in motor neurons, intestine, and pharyngeal muscle.
10 r the hyperpolarizing effect of glutamate on pharyngeal muscle.
11 arynx are no longer dye-coupled to posterior pharyngeal muscles.
12 le precursors that form most of the juvenile pharyngeal muscles.
13 genitors of the second heart field (SHF) and pharyngeal muscles.
14 y activates neurotransmission from M4 to the pharyngeal muscles.
15 as L1 larvae lacking most or all ABa-derived pharyngeal muscles.
16 utants, ubc-9(RNAi) animals lack ABa-derived pharyngeal muscles.
17 ex muscles and indirectly by controlling the pharyngeal muscles.
18 kx2-5, which is expressed exclusively in the pharyngeal muscles.
19  is restricted to the somatic, visceral, and pharyngeal muscles.
20 atially restricted to somatic, visceral, and pharyngeal muscles.
21 he green fluorescent protein is expressed in pharyngeal muscles.
22 odermal and neuronal tissue and an excess of pharyngeal muscle, a Mex phenotype (muscle excess).
23 ults indicate that the mechanical effects of pharyngeal muscle activation depend not only on the regi
24  decerebrate cats to determine the effect of pharyngeal muscle activation on airway pressure-area rel
25 te cats to determine the effect of selective pharyngeal muscle activation on airway shape.
26 acheotomized cats to determine the effect of pharyngeal muscle activation on the pharyngeal airway.
27             Intraluminal airway pressure and pharyngeal muscle activity are widely recognized as majo
28 piratory arousal threshold without impairing pharyngeal muscle activity to reduce OSA severity, with
29                Extracellular recordings from pharyngeal muscle also demonstrate severe defects in syn
30       It is localized to the synapse between pharyngeal muscle and the main pharyngeal excitatory mot
31  is the earliest known gene expressed in the pharyngeal muscles and is likely regulated directly by f
32  response to environmental conditions within pharyngeal muscles and is recognized by the nuclear horm
33                      During this period, the pharyngeal muscles and marginal cells forming the isthmu
34 hermaphrodites, including body wall muscles, pharyngeal muscles and vulval muscles.
35 , a ceh-22 target normally expressed only in pharyngeal muscle, and a synthetic reporter construct co
36 gating enzymes are necessary for ABa-derived pharyngeal muscle, and we hypothesize that TBX-2 functio
37 s and subsequently in somatic, visceral, and pharyngeal muscles, and the dorsal vessel.
38 ement, termed DE3, is strongly active in the pharyngeal muscles, and we identified two short oligonuc
39  the posterior pharynx demonstrates that all pharyngeal muscles are dye-coupled in wild-type animals;
40 rotein aggregation in Caenorhabditis elegans pharyngeal muscles, but higher levels in body-wall muscl
41 ated cholinergic transmission from MC to the pharyngeal muscles by activating the Gsalpha signaling p
42  Tropomyosin I gene in somatic body-wall and pharyngeal muscles by binding to DNA sequences within th
43 glutamate-gated chloride channel (AVR-15) on pharyngeal muscle, causing complete pumping inhibition.
44 ody size, and knockdown of their receptor in pharyngeal muscle cells reduces growth.
45        SUP-37 is required within a subset of pharyngeal muscle cells to facilitate coordinated rhythm
46  successfully profiled proteins expressed in pharyngeal muscle cells, and in the process, identified
47  influx and hypercontraction of the head and pharyngeal muscle cells, ultimately resulting in rapid n
48 hape and duration of the action potential of pharyngeal muscle cells.
49 ), pharyngeal collapsibility (Vpassive), and pharyngeal muscle compensation (Vcomp)-were measured by
50 show that ectopic overexpression of slo-1 in pharyngeal muscle confers sensitivity of the muscle to e
51 nnel, EXP-2, to form a complex that controls pharyngeal muscle contractility.
52 ults indicate that the mechanical effects of pharyngeal muscle contraction depend on the airway level
53 olinergic neurons enabled photoinhibition of pharyngeal muscle contraction in live worms.
54 ne eat-5, which is required for synchronized pharyngeal muscle contractions, and find that it is a ne
55 0) hermaphrodites arrest as L1 larvae due to pharyngeal muscle defects.
56 utants retain ABa-derived marginal cells and pharyngeal muscles derived from MS.
57 asic ontogenetic motif underlies cardiac and pharyngeal muscle development and evolution in chordates
58 s paper, we examine the role CEH-22 plays in pharyngeal muscle development and gene activation by (a)
59                                              Pharyngeal muscle development in the nematode Caenorhabd
60                     Moreover, Fgf18 supports pharyngeal muscle development in vivo and exogenous Fgf1
61                       Caenorhabditis elegans pharyngeal muscle development involves ceh-22, an NK-2 f
62 iously undescribed player during cardiac and pharyngeal muscle development.
63 xpression and that it is required for normal pharyngeal muscle development.
64 entially compensating pathways contribute to pharyngeal muscle differentiation.
65 tructive sleep apnea (OSA) require increased pharyngeal muscle dilator activation during wakefulness
66   Null and gain-of-function mutations affect pharyngeal muscle excitability in ways that are consiste
67 quiescence during DTQ results from a loss of pharyngeal muscle excitability, whereas feeding quiescen
68 lier/OLF/EBF) orchestrates the transition to pharyngeal muscle fate both by promoting an MRF-associat
69 agonism underlies a fundamental heart versus pharyngeal muscle fate choice that occurs in a conserved
70 ling maintains multipotency and promotes the pharyngeal muscle fate, whereas signal termination permi
71 ired for ABa-derived precursors to commit to pharyngeal muscle fate.
72 ely regulated directly by factors specifying pharyngeal muscle fate.
73 n factor tbx-2 is essential to form anterior pharyngeal muscles from the ABa blastomere.
74 d pha-1 have strongly synergistic effects on pharyngeal muscle gene expression; in addition, a pha-1
75                               Development of pharyngeal muscle in nematodes and cardiac muscle in ver
76 shes pumping, and optogenetic stimulation of pharyngeal muscle in these animals causes abnormal contr
77 esults indicate that selective activation of pharyngeal muscles in cats frequently results in greater
78 mV, induced by BWM action potentials, and in pharyngeal muscle, measured in simultaneous optical and
79 22 is necessary for neither formation of the pharyngeal muscles, nor for myo-2 expression.
80  Extracellular physiological recordings from pharyngeal muscle of hypomorphic mutants show alteration
81 netically encoded calcium indicators, in the pharyngeal muscle of the nematode worm Caenorhabditis el
82                       Gene expression in the pharyngeal muscles of Caenorhabditis elegans is controll
83 g UNC-89-A and -B primarily in body-wall and pharyngeal muscle, one internal promoter directing expre
84 s, intestinal epithelial cells, neurons, and pharyngeal muscle) or state-selective (heat-shock) promo
85 Mitoflash activity in Caenorhabditis elegans pharyngeal muscles peaked on adult day 3 during active r
86  forming first and second heart lineages and pharyngeal muscle precursors and characterize the molecu
87 soderm to distinct fate-restricted heart and pharyngeal muscle precursors.
88 esult in segregation of larval, cardiac, and pharyngeal muscle progenitors.
89                 For example, the cardiac and pharyngeal muscle programs are jointly primed in multipo
90 gh levels of signaling through GAR-3 inhibit pharyngeal muscle relaxation and impair feeding--but do
91 s of airway pressure, zolpidem did not alter pharyngeal muscle responsiveness during natural sleep.
92 indings that showed paradoxical increases in pharyngeal muscle responsiveness during transient manipu
93 s in arousal threshold without any change in pharyngeal muscle responsiveness, zolpidem does not alte
94 e giant UNC-89 isoforms, dis-organization of pharyngeal muscle, small body size, and reduced muscle f
95  accessibility, which govern later heart vs. pharyngeal muscle-specific expression profiles, demonstr
96 tro, CEH-22 binds the enhancer from myo-2, a pharyngeal muscle-specific myosin heavy chain gene.
97 olecular mechanisms that govern heart versus pharyngeal muscle specification within this lineage rema
98 rm an ancient conserved regulatory state for pharyngeal muscle specification, whereas their regulator
99 essential for development of the ABa-derived pharyngeal muscles, specification of neural cell fate in
100 ription similarly to DE3 specifically in the pharyngeal muscles, suggesting it may be an essential si
101 mal enhancer mediates expression in a single pharyngeal muscle, the donut-shaped m8 cell at the poste
102 k suggested that DAF-5 and DAF-3 function in pharyngeal muscle to regulate gene expression, but our a
103 nitors are primed to activate both heart and pharyngeal muscle transcriptional programs, which progre
104 nhancer activity is primarily limited to the pharyngeal muscles, we hypothesize that de209 also binds
105  our results and previous calcium imaging of pharyngeal muscles, we propose a model that explains how
106          In patients who had both ocular and pharyngeal muscle weakness, ptosis was just as likely to
107 entials, long-lasting depolarizations of the pharyngeal muscle, which are timed by neuronal input fro
108 d, starvation induces excessive autophagy in pharyngeal muscles, which in turn, causes damage that ma

 
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