ライフサイエンスコーパス: pharyngitis

1 dence interval, 1.03-1.18, respectively, for pharyngitis).

2 genetic subpopulation of strains that cause pharyngitis.

3 misclassifying 18, 446 patients without GAS pharyngitis.

4 ing a nonhuman primate model of experimental pharyngitis.

5 sive infection compared with strains causing pharyngitis.

6 ble diagnostic techniques for F. necrophorum pharyngitis.

7 gnosis and management of patients with acute pharyngitis.

8 c selection for sinusitis, otitis media, and pharyngitis.

9 used to confirm group A streptococcus (GAS) pharyngitis.

10 nuated for colonization in a monkey model of pharyngitis.

11 used to diagnose group A streptococcal (GAS) pharyngitis.

12 ically carried between recurrent episodes of pharyngitis.

13 in 20 cynomolgus macaques with experimental pharyngitis.

14 llergic patients with Streptococcus pyogenes pharyngitis.

15 es or costs of diagnosis or treatment of GAS pharyngitis.

16 S) is one of the leading causes of bacterial pharyngitis.

17 ar EL phenotype, 55% of whom had a preceding pharyngitis.

18 ract infections, such as the common cold and pharyngitis.

19 r gene was highly expressed during human GAS pharyngitis.

20 cimens taken from 18 pediatric patients with pharyngitis.

21 nce, China, suffered from scarlet fever-like pharyngitis.

22 represented in the pool of subclones causing pharyngitis.

23 agent in approximately 10% of adult cases of pharyngitis.

24 racted separately for children with clinical pharyngitis.

25 internal jugular vein secondary to bacterial pharyngitis.

26 sive disease than in strains from those with pharyngitis.

27 s were randomly collected from patients with pharyngitis.

28 of group A Streptococcus from patients with pharyngitis.

29 development for prevention of streptococcal pharyngitis.

30 antibiotic treatment of acute streptococcal pharyngitis.

31 ated in cases of endemically occurring acute pharyngitis.

32 ompared to 4.2 in skin infections and 3.3 in pharyngitis.

33 cular test) or after 5 days in those without pharyngitis.

34 ccurately diagnose the etiologic agent(s) of pharyngitis.

35 in the management of individuals with acute pharyngitis.

36 olving state of diagnostic testing for acute pharyngitis.

37 rgency department with signs and symptoms of pharyngitis.

38 otics for acute otitis media, sinusitis, and pharyngitis.

39 ented with signs and symptoms similar to GAS pharyngitis.

40 cs only if they have confirmed streptococcal pharyngitis.

41 about this approach to diagnosing bacterial pharyngitis.

42 aryngitis clinically resembles streptococcal pharyngitis.

43 is used routinely to help diagnose and treat pharyngitis.

44 in adolescents and adults with suspected GAS pharyngitis.

45 at low risk for group A streptococcal (GAS) pharyngitis.

46 oropharynx of mice, and seldom caused human pharyngitis.

47 ity of its major regulatory target SpeB, and pharyngitis.

48 ediatric patients with group A streptococcal pharyngitis.

49 n and culture for the detection of bacterial pharyngitis?

50 is, 9; Helicobacter pylori, 1; Streptococcal pharyngitis 1; and posttransplant lymphoma, 1).

51 , 1.60; 95% confidence interval, 1.51-1.70), pharyngitis (1.48; 1.15-1.89), aphonia (1.81; 1.18-2.80)

52 es for the management of adult patients with pharyngitis: 1) observation without testing or treatment

53 Of 451 children with pharyngitis, 107 (24%) had group A beta-haemolytic strep

54 ral upper respiratory tract infection (47%), pharyngitis (18%), and allergy or asthma (11%).

55 rom patients with invasive disease (34%) and pharyngitis (21%) in the same period.

56 highest for suppurative OM ($25.3 million), pharyngitis ($21.3 million), and viral URI ($19.1 millio

57 presented with serious complications of GAS pharyngitis: 29 (4.0%) had peritonsillar abscesses and 2

58 (2) increase optimal prescribing for AOM and pharyngitis, (3) provide clinician education on shorter

59 ing among children younger than 3 years with pharyngitis (3679 of 9785 [37.6%]), computed tomography

60 tibiotic prescriptions [95% CI, 41-54]), and pharyngitis (43 antibiotic prescriptions [95% CI, 38-49]

61 623 throat swab specimens from patients with pharyngitis (93.6% <18 years old, 54.3% female), the ref

62 but the annual spatiotemporal pattern of GAS pharyngitis across the United States is poorly character

63 United States are for acute sinusitis, acute pharyngitis, acute bronchitis, and nonspecific upper res

64 Humans with documented GAS infections (pharyngitis, acute rheumatic fever, and severe invasive

65 al specimens from individuals suffering from pharyngitis aids in the management and selection of anti

66 the diagnosis of group A Streptococcus (GAS) pharyngitis allows laboratories to adopt single-tiered t

67 es human diseases ranging from self-limiting pharyngitis (also known as strep throat) to severely inv

68 ial role for F. necrophorum as a pathogen of pharyngitis among young adults, but suggests that the pr

69 idemics, the sic gene was sequenced from 471 pharyngitis and 127 pyogenic and blood isolates recovere

70 m the throats of college students with acute pharyngitis and 5 strains isolated from patients with no

71 e results from 522 adult patients with acute pharyngitis and a positive ADT result; unexpectedly, 15%

72 on diagnosis (clinical signs and symptoms of pharyngitis and a positive rapid molecular test) or afte

73 pressed by some strains that cause impetigo, pharyngitis and acute glomerulonephritis.

74 but there were more instances of stomatitis/pharyngitis and hand-foot skin reaction in the continuou

75 esting, and office testing for streptococcal pharyngitis and Helicobacter pylori.

76 However, pharyngitis and impetigo are rarely observed prior to in

77 hat can manifest as a range of diseases from pharyngitis and impetigo to severe outcomes such as necr

78 The peak incidence for streptococcal pharyngitis and impetigo varies with season and locale,

79 ponsible for superficial infections, such as pharyngitis and impetigo, and severe invasive infections

80 e of most serotype M1 strains recovered from pharyngitis and invasive disease episodes in North Ameri

81 The pharyngitis and invasive groups were highly related to e

82 irulence gene profiles were identified among pharyngitis and invasive infection isolates.

83 subcutaneously with GAS and humans with GAS pharyngitis and invasive infections seroconverted to mos

84 Serotype M28 strains are a leading cause of pharyngitis and invasive infections, but little is known

85 ) is an important human pathogen that causes pharyngitis and invasive infections, including necrotizi

86 tion of the genetic relationship between GAS pharyngitis and invasive strains.

87 nd killing to cause human disease, including pharyngitis and necrotizing fasciitis (flesh-eating synd

88 streptococcus (GAS), the causative agent of pharyngitis and necrotizing fasciitis, secretes the pote

89 , and increase virulence in animal models of pharyngitis and necrotizing fasciitis.

90 more virulent in nonhuman primate models of pharyngitis and necrotizing fasciitis.

91 rapid, point-of-care testing method for GAS pharyngitis and obviate backup testing on negative resul

92 llance data into clinical guidelines for GAS pharyngitis and other communicable diseases should be co

93 Incident pharyngitis and pyoderma infections were captured.

94 mples were collected to detect carriage, and pharyngitis and pyoderma swab samples were taken to asse

95 developed clinical signs and symptoms of GAS pharyngitis and seroconverted to several GAS extracellul

96 t causes many types of infections, including pharyngitis and severe invasive diseases.

97 oup A Streptococcus (GAS) causes superficial pharyngitis and skin infections as well as serious autoi

98 hich may be the result of repeated precursor pharyngitis and skin infections that progressively boost

99 f infections in humans, from relatively mild pharyngitis and skin infections to life-threatening necr

100 Group A Streptococcus (GAS) commonly causes pharyngitis and skin infections.

101 Associations between GAS pharyngitis and specific food items were assessed among

102 overed from a single individual during acute pharyngitis and subsequent asymptomatic carriage.

103 visit-based prescribing rates decreased for pharyngitis and upper respiratory tract infection; howev

104 um is an emerging human pathogen that causes pharyngitis and wound infections.

105 of a non-drug-related serious adverse event (pharyngitis) and 1 because of lack of treatment efficacy

106 per 1000 person-years (31-84) for S pyogenes pharyngitis, and 263 per 1000 person-years (212-327) for

107 case of periodic fever, aphthous stomatitis, pharyngitis, and adenitis (PFAPA) syndrome, who had a to

108 and the periodic fever, aphthous stomatitis, pharyngitis, and adenopathy (PFAPA) syndrome.

109 or Raynaud syndrome, recurrent streptococcal pharyngitis, and an allergy to amoxicillin.

110 Periodic fever with aphthous stomatitis, pharyngitis, and cervical adenitis (PFAPA) is a relative

111 rome of periodic fever, aphthous stomatitis, pharyngitis, and cervical adenitis (PFAPA) is the most c

112 Periodic fever, aphthous stomatitis, pharyngitis, and cervical adenitis (PFAPA) syndrome is t

113 PFAPA (periodic fever, aphthous stomatitis, pharyngitis, and cervical adenitis) has been characteriz

114 accurate diagnosis of group A streptococcal pharyngitis, and laboratory testing for confirmation of

115 Fever, fatigue, pharyngitis, and proctitis were commonly reported.

116 ections, noninvasive soft tissue infections, pharyngitis, and rheumatic fever indicated that Slr is p

117 ections, noninvasive soft tissue infections, pharyngitis, and rheumatic fever indicated that these fo

118 s stomatitis, tongued tonsillitis with moss, pharyngitis, and submandibular lymphadenitis with tender

119 scripts were detectable in patients with GAS pharyngitis, and the levels increased significantly duri

120 each) were identified in which otitis media, pharyngitis, and urinary tract infection (UTI) were trea

121 te otitis media; 6746, group A streptococcal pharyngitis; and 4234, acute sinusitis), 4307 (14%) were

122 ute otitis media; 705, group A streptococcal pharyngitis; and 667, acute sinusitis), 868 (35%) were p

123 estimated 5.2 million outpatient visits for pharyngitis annually in the United States, with incidenc

124 opriate antibiotic use for adults with acute pharyngitis apply to immunocompetent adults without comp

125 Prompt diagnosis and treatment of GAS pharyngitis are cornerstones of the Rheumatic Fever Prev

126 GAS strains with a strong tendency to cause pharyngitis are distinct from strains that tend to cause

127 Real-time regional data on GAS pharyngitis are generally not yet available to clinician

128 on the host mucosal surface and cause acute pharyngitis are poorly understood.

129 rheumatic fever after group A streptococcal pharyngitis are still poorly understood.

130 ndividual patients at very high risk for GAS pharyngitis as assessed by a clinical decision rule.

131 s), Fusobacterium necrophorum causes endemic pharyngitis at a rate similar to that of group A beta-he

132 diatric patients presenting with symptoms of pharyngitis at any of three Lahey Clinic acute care faci

133 s can be confounded by a lack of symptoms of pharyngitis at the time of presentation and end-organ dy

134 n the number of symptomatic individuals with pharyngitis beginning in mid-1999.

135 69 of 76 865 (55.9%) clinical encounters for pharyngitis being optimal for antibiotic choice.

136 Antibiotic treatment of adult pharyngitis benefits only those patients with GABHS infe

137 ococcus (GAS) is a common causative agent of pharyngitis, but the role of GAS in otitis media is unde

138 Diagnosis of GAS pharyngitis by a rapid antigen detection test (RADT) or

139 Diagnosis of GAS pharyngitis by clinician judgement alone is unreliable,

140 However, these 2 emm types caused 69% of the pharyngitis cases identified during training and represe

141 occus (GAS) causes the majority of bacterial pharyngitis cases in humans and is prone to persistently

142 in Minnesota and for a significant number of pharyngitis cases that also occurred during the outbreak

143 GAS and humans with invasive infections and pharyngitis caused by GAS seroconverted to Shp, indicati

144 dy of molecular events contributing to acute pharyngitis caused by GAS.

145 ctiveness of various strategies for managing pharyngitis caused by group A beta-hemolytic streptococc

146 an alternative regimen for the treatment of pharyngitis caused by group A beta-hemolytic streptococc

147 d antimicrobial regimen for the treatment of pharyngitis caused by group A beta-hemolytic streptococc

148 roat swabs of each of 20 patients with acute pharyngitis caused by serotype M1 GAS.

149 Humans with GAS pharyngitis caused by serotype M6 strains had antibody a

150 GAS pharyngitis causes 19.1 (95% confidence interval [CI], 1

151 GAS pharyngitis causes 93.2 (95% CI, 82.3-105.3) visits and

152 isorder periodic fever, aphthous stomatitis, pharyngitis, cervical adenitis (PFAPA) syndrome is unkno

153 the clinical picture, with acute symptomatic pharyngitis characterised by pharyngeal colonisation by

154 dent population, and F. necrophorum-positive pharyngitis clinically resembles streptococcal pharyngit

155 f GAS in throat swabs taken from humans with pharyngitis confirmed the findings.

156 PEA is only rarely associated with TSS after pharyngitis, despite being highly associated with TSS af

157 A patient-driven approach to pharyngitis diagnosis that uses this new score could sav

158 ecrotizing fasciitis) and noninvasive (e.g., pharyngitis) diseases.

159 Pharyngitis due to group A Streptococcus (GAS) represent

160 serious adverse events, hemolytic anemia and pharyngitis, each occurred in 2 patients (4%).

161 they choose to treat streptococcus-negative pharyngitis empirically.

162 r parent; both parent and child reported GAS pharyngitis episodes 3 weeks before the banquet.

163 ic research is needed on both F. necrophorum pharyngitis (especially clinical presentation) and the L

164 hargica (EL), a CNS disorder presenting with pharyngitis followed by sleep disorder, basal ganglia si

165 Clinically screen all adult patients with pharyngitis for the presence of the four Centor criteria

166 symptoms suggestive of group A streptococcal pharyngitis (for example, persistent fevers, anterior ce

167 olates recently recovered from patients with pharyngitis from 13 countries were characterized by emm

168 Many pharyngitis GAS strains circulate simultaneously.

169 While there are many causes of pediatric pharyngitis, group A streptococcal pharyngitis represent

170 Pharyngitis guidelines focus solely on group A beta-hemo

171 The frequency of antibiotic use for pharyngitis has greatly exceeded the prevalence of GABHS

172 The large majority of adults with acute pharyngitis have a self-limited illness, for which suppo

173 necrophorum causes at least 10% of cases of pharyngitis; however, few U.S. data exist.

174 roup A streptococcal diseases: uncomplicated pharyngitis, impetigo, and acute rheumatic fever.

175 ts for a variety of human diseases including pharyngitis, impetigo, toxic shock, and necrotizing fasc

176 elf-limiting diseases caused by GAS, such as pharyngitis, impose a significant economic burden on soc

177 are aphthous stomatitis in 70% of patients, pharyngitis in 72% of patients, and cervical adenitis in

178 Three outbreaks were studied: pharyngitis in a boarding school (serotype M5), cross-in

179 emm types causing asymptomatic carriage and pharyngitis in a closed population, we analyzed 675 isol

180 roup A beta-hemolytic streptococcal-positive pharyngitis in a student population, and F. necrophorum-

181 wabs of pediatric patients performed for GAS pharyngitis in a tertiary-care children's hospital netwo

182 ostic testing and treatment of suspected GAS pharyngitis in adults have very similar effectiveness an

183 expensive strategy at any prevalence of GAS pharyngitis in adults, although it may be reasonable for

184 the diagnosis of group A streptococcal (GAS) pharyngitis in adults.

185 streptococcus (GAS) species cause bacterial pharyngitis in both adults and children.

186 illion visits to ambulatory care centers for pharyngitis in children between the ages of 3 and 18 yea

187 and C/G streptococci as agents of bacterial pharyngitis in children.

188 Streptococcus pyogenes is a major cause of pharyngitis in humans and encodes several fibronectin-bi

189 has been implicated as a causative agent of pharyngitis in outbreak situations, but its role in ende

190 culture for the laboratory diagnosis of GAS pharyngitis in patients for whom testing is clinically i

191 s from an outbreak of erythromycin-resistant pharyngitis in Pittsburgh, PA, we found a correlation be

192 ic heart disease in children presenting with pharyngitis in urban primary care clinics in South Afric

193 Treating all children presenting with pharyngitis in urban primary care clinics in South Afric

194 nt-of-care testing for group A streptococcal pharyngitis, including rapid antigen detection tests and

195 pecific infections, i.e., skin infection and pharyngitis-induced acute rheumatic fever.

196 cine could prevent substantial incidences of pharyngitis infections and associated antibiotic prescri

197 aseline and changed little for streptococcal pharyngitis (intervention, from 4.4% to 3.4%; control, f

198 Since acute pharyngitis is a frequently occurring infectious disease

199 o acute pharyngitis strains; thus, childhood pharyngitis is a major reservoir for strains with invasi

200 Acute pharyngitis is a nonspecific symptom that can result fro

201 Group A streptococcal (GAS) pharyngitis is a particularly important condition in are

202 Group A Streptococcus (GAS) pharyngitis is a very common condition causing significa

203 Pharyngitis is an inflammatory condition of the pharynx

204 Adolescent and young adult pharyngitis is more complicated than previously consider

205 rred antibiotic for treatment of acute GABHS pharyngitis is penicillin, or erythromycin in a penicill

206 Streptococcal pharyngitis is still a major infectious disease seen in

207 Pharyngitis isolates represented 16 pulsed-field gel ele

208 Compared with pharyngitis isolates, invasive isolates were more likely

209 absent from a genetically related subset of pharyngitis isolates.

210 de misunderstanding for infectious recurrent pharyngitis, it is important to note that we should cons

211 , acute bronchitis (23%), otitis media (5%), pharyngitis, laryngitis, and tracheitis (11%), or more t

212 1 persons); soft-tissue superinfection (18); pharyngitis limiting oral intake (5); eye lesions (2); a

213 If vaccine prevention of GAS pharyngitis made the routine antibiotic treatment of pha

214 ost common DLTs were neutropenia, stomatitis/pharyngitis, myalgia, and arthralgia.

215 were met (fever/feverish, cough, congestion, pharyngitis, myalgias), staff obtained a mid-turbinate n

216 nfection for 22 (44%) of the children and by pharyngitis (no throat culture obtained) for 14 others (

217 orne outbreak of Group A Streptococcus (GAS) pharyngitis occurring among attendees of a high school d

218 Fusobacterium necrophorum-positive pharyngitis occurs more frequently than group A beta-hem

219 highly contagious purulent lymphadenitis and pharyngitis of members of the family Equidae.

220 ults were sensitive to the prevalence of GAS pharyngitis: OIA followed by culture was most effective

221 he routine primary evaluation of adults with pharyngitis or for confirmation of negative results on r

222 rum samples from patients with streptococcal pharyngitis or healthy control subjects.

223 r resulting from Group A Streptococcus (GAS) pharyngitis or impetigo in children and adolescents, whi

224 d GABHS infection, 60 (42%) with symptoms of pharyngitis or upper respiratory infection (no throat cu

225 eases ranging in severity from uncomplicated pharyngitis (or strep throat) to life-threatening infect

226 e most common infections involve the throat (pharyngitis) or skin (impetigo); however, the factors th

227 plications (subglottic stenosis, laryngitis, pharyngitis, or cancer) can occur without esophagitis.

228 ARTI diagnosis (acute bronchitis, sinusitis, pharyngitis, otitis media, allergic rhinitis, influenza,

229 Mycoplasma bovis causes pneumonia, pharyngitis, otitis, arthritis, mastitis, and reproducti

230 serotype M6 clone that was responsible for a pharyngitis outbreak in Pittsburgh, Pennsylvania, was co

231 Expanding the pharyngitis paradigm will have several important implica

232 In this foodborne outbreak of GAS pharyngitis, pasta was implicated as the vehicle.

233 on should be considered when clusters of GAS pharyngitis patients are encountered.

234 in vitro; however, analyses of sera from 155 pharyngitis patients revealed a strong correlation (P <

235 ver (ARF) and untreated scarlet fever and/or pharyngitis patients were reacted with streptococcal M p

236 a wide array of diseases in humans including pharyngitis, pneumonia, gastroenteritis, hemorrhagic cys

237 ive and most effective strategy when the GAS pharyngitis prevalence is 10%.

238 nt was the least effective strategy at a GAS pharyngitis prevalence of 10% (resulting in 0.41 lost qu

239 lowed by culture was most effective when GAS pharyngitis prevalence was greater than 20%.

240 Alleles shared among the pharyngitis, pyogenic, and blood samples were identified

241 d RLPP correlated with the likelihood of GAS pharyngitis (r(2) = 0.79; P < 0.001).

242 pediatric pharyngitis, group A streptococcal pharyngitis represents 15 to 30% of infections and is th

243 nd resolve without lasting effects; however, pharyngitis resulting from infection with Streptococcus

244 bronchitis," "respiratory tract infection," "pharyngitis," "rhinosinusitis," and "the common cold." H

245 additional person: 1.03, 1.00-1.05), as was pharyngitis risk (rainy season: 3.00, 1.10-8.22; househo

246 lineage (designated M1(UK))-with overlap of pharyngitis, scarlet fever, and invasive M1(UK) strains-

247 consecutive patients with symptoms of acute pharyngitis seen in two outpatient clinics in a large su

248 nctivitis from a bacterial etiology included pharyngitis (sensitivity range, 0.55-0.58; specificity r

249 atory toxicities included grade 4 stomatitis/pharyngitis, sepsis syndrome (one patient each), and gra

250 supernatant, and patients with streptococcal pharyngitis seroconverted to Sse, indicating that Sse wa

251 All patients with pharyngitis should be offered appropriate doses of analg

252 The diagnostic paradigm for adolescent pharyngitis should therefore be expanded to consider F.

253 s for presumptive treatment of streptococcal pharyngitis since bacterial culture and rapid diagnostic

254 iated with combination therapy were anxiety, pharyngitis, sinus congestion, and peripheral edema.

255 al infection (suppurative otitis media [OM], pharyngitis, sinusitis) or viral infection (influenza, v

256 Collectively, GAS-attributable pharyngitis, sinusitis, and AOM cause 26.9 (95% CI, 23.9

257 ratory tract encounters (acute otitis media, pharyngitis, sinusitis, presumed viral infection) after

258 he predicted amino acid sequences of the two pharyngitis strains were identical and were 88% homologo

259 e compared the genomes of 86 serotype M3 GAS pharyngitis strains with those of 215 invasive M3 strain

260 ionships between populations of invasive and pharyngitis strains.

261 pediatric GAS strains are identical to acute pharyngitis strains; thus, childhood pharyngitis is a ma

262 ts with group A beta-hemolytic streptococcal pharyngitis (strep throat) is an important task for clin

263 ignificance, causing infections ranging from pharyngitis (strep throat) to necrotizing fasciitis (fle

264 ng a diverse array of infections from simple pharyngitis ("strep throat") to invasive conditions, inc

265 s known about the genomes of strains causing pharyngitis (streptococcal "sore throat").

266 A STREPTOCOCCUS: (GAS) associated both with pharyngitis (streptococcal sore throat) and with invasiv

267 CI], 67.1%-91.7%) protection against typical pharyngitis symptoms among children reacquiring the same

268 When testing for GAS pharyngitis, the throat remains the optimum site for sam

269 he new score estimated the likelihood of GAS pharyngitis to be less than 10% instead of having clinic

270 t causes infections ranging in severity from pharyngitis to life-threatening invasive disease, such a

271 of humans, causing a range of diseases from pharyngitis to necrotizing fasciitis.

272 tomatically and cause illnesses ranging from pharyngitis to necrotizing fasciitis.

273 ns, from mild superficial infections such as pharyngitis to serious invasive infections such as necro

274 at causes a multitude of human diseases from pharyngitis to severe infections such as toxic shock syn

275 tions ranging in severity from self-limiting pharyngitis to severe invasive diseases that are associa

276 ranging from superficial skin infections and pharyngitis to severe invasive infections such as necrot

277 es diseases ranging from mild, self-limiting pharyngitis to severe invasive infections.

278 uses a wide spectrum of disease ranging from pharyngitis to streptococcal toxic shock syndrome.

279 ) causes diseases that range from mild (e.g. pharyngitis) to severely invasive (e.g. necrotizing fasc

280 AS) causes many different diseases including pharyngitis, tonsillitis, impetigo, scarlet fever, strep

281 ARF sera and sera from uncomplicated pharyngitis (UNC) reacted with C repeat region peptides

282 tis made the routine antibiotic treatment of pharyngitis unnecessary, up to 17.1% (95% CI, 15.0-19.6%

283 ratory Improvement Amendments, streptococcal pharyngitis, urinalyses, office stool examination, and i

284 Little is known why streptococcal pharyngitis usually does not lead to pneumonia and why t

285 d to assess their use since the frequency of pharyngitis varies.

286 osest estimate of the true prevalence of GAS pharyngitis was 19.7% (71/361 samples).

287 (illumigene assay) for the diagnosis of GAS pharyngitis was compared with that of a RADT and standar

288 Pharyngitis was diagnosed in 17 (85%; 95% CI 62-97) of 2

289 t the lower dose level (1-3 x 10(4) CFU/mL), pharyngitis was diagnosed in one of five participants.

290 um samples from children with GAS-associated pharyngitis were assayed, and a strong immune response t

291 Subjects with symptoms consistent with pharyngitis were enrolled across four sites in the Unite

292 egarding treatment of streptococcal tonsillo-pharyngitis were revised.

293 a-hemolytic streptococcal scarlet fever-like pharyngitis were studied.

294 Acute otitis media (AOM) and pharyngitis were the most common indications, with 85 63

295 information about the local incidence of GAS pharyngitis, which is necessary to calculate the new sco

296 f adult patients with clinically significant pharyngitis who can benefit from treatment.

297 needs to identify those patients with acute pharyngitis who require specific antimicrobial therapy a

298 States each year and 8500 patients with GAS pharyngitis who would have received antibiotics would no

299 ported to be an important cause of bacterial pharyngitis with a prevalence as high as that of group A

300 the red flags for adolescent and young adult pharyngitis: worsening symptoms or neck swelling (especi