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1 ts SRC basal activation independently of its phosphatase activity.
2 ose production despite the loss of glucose-6-phosphatase activity.
3 membrane and nucleus without affecting lipid phosphatase activity.
4  calcium release on its way to activation of phosphatase activity.
5 ssed by staining for tartrate-resistant acid phosphatase activity.
6  human Treg cells and provides complementary phosphatase activity.
7 tion of purified PP2Ac with CIN85 suppressed phosphatase activity.
8 ons leads to significant or complete loss of phosphatase activity.
9  SUMOylated, yet is independent of its lipid phosphatase activity.
10 dependent platelet function by dampening the phosphatase activity.
11 f LC20 upon inhibition of myosin light chain phosphatase activity.
12 or and OC protein addition enhanced alkaline phosphatase activity.
13 e effect associated with intestinal alkaline phosphatase activity.
14 , which occurs independently of antagonizing phosphatase activity.
15 r factor-kappaB ligand (RANKL), and alkaline phosphatase activity.
16 ated PTEN mutations block its essential PIP3 phosphatase activity.
17 nd B subunits were stable and uncoupled from phosphatase activity.
18 ad deficient mineral apposition and alkaline phosphatase activity.
19  Asn394 as a critical amino acid involved in phosphatase activity.
20 e that help decipher the mechanism of glucan phosphatase activity.
21 phosphorylation of Shp2, which inhibits Shp2 phosphatase activity.
22 idative stress response through its NADPH 2' phosphatase activity.
23 ed ligands and inhibits its protein-tyrosine phosphatase activity.
24 brane electrical potential to inositol lipid phosphatase activity.
25 y with Ssu72 and strongly stimulates its CTD phosphatase activity.
26 ins and is concurrent with increases in PTEN phosphatase activity.
27 roximately 5-fold increase in Cps2B-mediated phosphatase activity.
28 nsport, PSRP1-sRNPC is stable against phloem phosphatase activity.
29 mplex and reduces but does not fully abolish phosphatase activity.
30 sociated with significant reduction in local phosphatase activity.
31 ase activity must be counteracted by protein phosphatase activity.
32  means for modulating the balance of CDK and phosphatase activity.
33 id kinase activity and stimulates Fig4 lipid phosphatase activity.
34 in the stimulation (6-fold) of phosphatidate phosphatase activity.
35 ensor domain that is expected to control the phosphatase activity.
36 XNWD motif, results in maximal activation of phosphatase activity.
37 chondrial membranes, which is independent of phosphatase activity.
38 and CnPpz1 in vitro but do not inhibit their phosphatase activity.
39 2) levels on endosomes, independently of its phosphatase activity.
40 adherin internalization independently of its phosphatase activity.
41 ain and membrane-targeting motif can restore phosphatase activity.
42 tiates TGF-beta signaling independent of its phosphatase activity.
43 LSF1 complex shows amylolytic but not glucan phosphatase activity.
44 atase (DSP) domain of LSF1 had no detectable phosphatase activity.
45 duced type 1 collagen secretion and alkaline phosphatase activity.
46 ority of them stained intensely for alkaline phosphatase activity.
47 iptional coactivators with intrinsic protein phosphatase activity.
48 ction networks depends on protein kinase and phosphatase activities.
49  SIK2-PP2A complex preserves both kinase and phosphatase activities.
50 se activity or both PTEN's lipid and protein phosphatase activities.
51 e loss of PTEN protein phosphatase and lipid phosphatase activities.
52 PTEN has mainly been attributed to its lipid phosphatase activity, a role for PTEN protein phosphatas
53                 Disruption of CovS kinase or phosphatase activities abolishes RocA function, consiste
54                           Together, the RVLM phosphatase activity acts tonically to attenuate the ERK
55 8(MAPK) determines signal amplitude, whereas phosphatase activity affects both signal amplitude and d
56 genic related markers (osteopontin, alkaline phosphatase activity, Alizarin red, and Von Kossa) compa
57 /-), which is associated with an impaired 5'-phosphatase activity, also leads to Parkinson's disease
58 osphorylation of Synj at S1029 enhances Synj phosphatase activity, alters interaction between Synj an
59 tional model that combines MAPK scaffold and phosphatase activities and is sufficient to account for
60 on in ICL repair is dependent on its protein phosphatase activity and ability to be SUMOylated, yet i
61               This was confirmed by alkaline phosphatase activity and Alizarin red and Von Kossa stai
62 e found an inverse relationship between root phosphatase activity and AM colonization in field-collec
63                             We measured root phosphatase activity and arbuscular mycorrhizal (AM) col
64 t and specific B-type subunits, and impaired phosphatase activity and C-terminal methylation.
65 esulting in significantly increased alkaline phosphatase activity and calcium deposition of encapsula
66       The osteoblastic functions of alkaline phosphatase activity and calcium mineralization were als
67 ntration of cytoplasmic free zinc, increased phosphatase activity and decreased phosphorylation of si
68  cleaved by calpain-2, which inactivates its phosphatase activity and generates stable breakdown prod
69 terfaces, we show that D2 inhibits RPTPalpha phosphatase activity and identified a (405)PFTP(408) mot
70 R306, of EYA1 are essential for its in vitro phosphatase activity and in vivo function during Drosoph
71 nstrate that PTPN1 mutations lead to reduced phosphatase activity and increased phosphorylation of JA
72 d in partial inhibition of its ATP-dependent phosphatase activity and inhibited subsequent phosphoryl
73 9R-mutant endometrial cancer cells increases phosphatase activity and inhibits tumor growth in vivo F
74 al rearrangement reversal is correlated with phosphatase activity and is influenced by the membrane's
75 on of Pah1 stimulated both its phosphatidate phosphatase activity and its subsequent phosphorylation
76 were decreased, as evidenced by low alkaline phosphatase activity and matrix mineralization.
77 P-activated signaling pathway controls SHP-1 phosphatase activity and may regulate numerous receptor
78 r, the p85alpha homodimer enhances the lipid phosphatase activity and membrane association of PTEN.
79 rotein levels and induced increased alkaline phosphatase activity and mineralization in osteoblast cu
80 mber of committed osteoprogenitors, alkaline phosphatase activity and mineralization.
81  competition, however, AM colonization, root phosphatase activity and N2 fixation increased in the N2
82 formance remain unclear: biomass allocation, phosphatase activity and phosphorus-use efficiency did n
83 d on gamma glutamyl transferase and alkaline phosphatase activity and proliferative responses to secr
84  de-phosphorylation of PTEN, increased lipid-phosphatase activity and reduced Akt kinase activity in
85 show that FTY720-P enhances TNF-induced PP2A phosphatase activity and significantly represses TNF-ind
86 nt in polyP coincided with enhanced alkaline phosphatase activity and substitution of sulfolipids for
87  suggesting a functional correlation between phosphatase activity and systemic autoimmunity.
88 phate and tensin homolog through its protein phosphatase activity and that the increase in INPP4B is
89 acute pharmacological inhibition of the Eya2 phosphatase activity and the DNA damage checkpoint kinas
90 mains of Pah1 are required for phosphatidate phosphatase activity and the in vivo function of the enz
91 tion based on the maintenance of the Ser/Thr phosphatase activity and their neuroprotection against t
92 only tyrosine phosphatase activity, has dual phosphatase activities, and both the N- and C-terminal d
93 d and van Kossa stain and increased alkaline phosphatase activity, and by increased expression of ost
94 ulation of organic acids, enhanced secretory phosphatase activity, and depletion of ATP in overexpres
95 ant effects on protein synthesis or alkaline phosphatase activity, and drove discrete changes in a pa
96  disrupted sebocyte cell membranes, alkaline phosphatase activity, and significantly reduced sebocyte
97 ponent analysis shows that dehydrogenase and phosphatase activities are the vital variables contribut
98 tection from proteolysis, and stimulation of phosphatase activity are linked to oligomerization of Sp
99      Moreover, the PST/TPM and the threonine phosphatase activity are not required for in vitro inter
100               Both membrane localization and phosphatase activity are required for PPM5C's role in at
101 we report inherited dysregulation of protein phosphatase activity as a cause of intellectual disabili
102 EN protein has lipid phosphatase and protein phosphatase activity, as well as other functions in the
103                               Using alkaline phosphatase activity assays along with various biochemic
104 9D) constructs, in vitro binding assays, and phosphatase activity assays, we demonstrate that phospho
105                              Thus, a pool of phosphatase activity associated with a kinetochore-local
106 PHLPP phosphatase network allows coordinated phosphatase activities at the site of T-cell receptor ac
107 e His is responsible for the decrease in the phosphatase activity at acidic pH.
108 ive estimates for the intracellular alkaline phosphatase activity at five different temperatures in d
109                    We assessed soil and root phosphatase activity between fixers and non-fixers in tw
110 ssion of OsHAD1 in rice resulted in enhanced phosphatase activity, biomass, and total and soluble P c
111 s no change in glycogen content or glucose 6-phosphatase activity but increased Slc2a2 glucose transp
112  of ethanol, caused no changes in BP or RVLM phosphatase activity but it produced significant increas
113  usually is attributed to myosin light chain phosphatase activity, but findings in non-VSM identified
114                  The PTEN-like domain has no phosphatase activity, but it can recognize phosphatidyli
115 ing (<=5 years) significantly increased soil phosphatase activity by 28%, long-term N loading had no
116                   Finally, inhibition of SHP phosphatase activity by NSC87877 abrogated B. pertussis
117               Inhibition of the RVLM ser/thr phosphatase activity by okadaic acid (OKA, 0.4 mug) or f
118 c homology 2 domain-containing phosphatase 2 phosphatase activity by scavenging reactive oxygen speci
119 burgdorferi CheD significantly enhances CheX phosphatase activity by specifically interacting with th
120 ggesting inhibition of SHP2 protein tyrosine phosphatase activity by this peptide.
121                           Moreover, alkaline phosphatase activity, calcium deposition and acidic poly
122  greatly promote MSC proliferation, alkaline phosphatase activity, calcium deposition and total prote
123 ent with this finding, inhibition of STRIPAK phosphatase activity causes cell morphology defects in m
124 BL) and enhanced the respiratory burst, acid phosphatase activity, chemotactic activity, and gene exp
125 emur length, and 30% elevated serum alkaline phosphatase activity compared to wild type.
126 inhibiting kinase activity while stimulating phosphatase activity, consistent with the selective inac
127                                     Impaired phosphatase activity contributes to the persistent activ
128                                    Its lipid phosphatase activity converts PIP3 to PIP2 and antagoniz
129 pects of disease pathology, with reduced DNA phosphatase activity correlating with neurodevelopmental
130                     BCR expression level and phosphatase activity could both contribute to such heter
131 sponse to TNF, and this increased GADD34-PP1 phosphatase activity, dephosphorylating eukaryotic trans
132 ations worldwide, that N stimulation of soil phosphatase activity diminishes over time.
133 ion, some dividing cells, and clear alkaline phosphatase activity (early bone marker).
134 or of STAT5 through liberation of endogenous phosphatase activity following NADPH oxidase (NOX) inhib
135 itant with a significant increase in protein phosphatase activity for two colon cancer cell lines in
136                              We enriched the phosphatase activity from a B. subtilis cell extract and
137  promoted DP markers and functional alkaline phosphatase activity from the DP cells.
138 tes, including bile acids transfer, alkaline phosphatase activity, gamma-glutamyl-transpeptidase acti
139  the N2 fixer with high N2 fixation and root phosphatase activity grew best on organic P, whereas the
140 , a GAS strain selectively deficient in CovS phosphatase activity had a distinct transcriptome relati
141 nd temporal regulation of protein kinase and phosphatase activities has to be achieved in the cell to
142                        Although in vitro CTD phosphatase activity has been established for some CPLs,
143 hich has been reported to have only tyrosine phosphatase activity, has dual phosphatase activities, a
144 we manipulated native loci to ablate hCDC14A phosphatase activity (hCDC14A(PD)) in untransformed hTER
145  Unexpectedly, we observed that inhibiting a phosphatase activity highly sensitive to OA caused an ab
146 he coronavirus macrodomain has ADP-ribose-1"-phosphatase activity; however, its function during infec
147         The values of soil dehydrogenase and phosphatase activities in the four wetlands follow the o
148 Chlamydomonas GPD2 showed both reductase and phosphatase activities in vitro and it can work as a bif
149 merize with wild-type PTEN and constrain its phosphatase activity in a dominant-negative manner.
150 A3 (RON3) protein as a regulator of the PP2A phosphatase activity in Arabidopsis thaliana.
151 hosphatase activity, a role for PTEN protein phosphatase activity in cell cycle regulation has also b
152                  Thus, TIMAP inhibits myosin phosphatase activity in ECs by competing with MYPT1 for
153 orylation in its activation and PTEN protein phosphatase activity in governing glycolysis and tumorig
154 Cs in the lesion cap, and increased alkaline phosphatase activity in lesions in the Ahr knockout in c
155                Upregulation of CD45 tyrosine phosphatase activity in MDSCs exposed to hypoxia in tumo
156                          We demonstrate that phosphatase activity in natural biofilm samples decrease
157 ion of OC protein partially rescued alkaline phosphatase activity in periodontal ligament (PDL) cells
158 cant increases in BP and inhibition of local phosphatase activity in rats treated with OKA or fostrie
159 ly members, serving as a platform to control phosphatase activity in response to diverse inputs.
160  Under laboratory conditions, BvgS shifts to phosphatase activity in response to modulators, notably
161 ate, showing that BvgS shifts from kinase to phosphatase activity in response to this modulator via a
162                              Inhibiting Shp1 phosphatase activity in the absence of Elp1 rescued NGF-
163 tion uncover a novel mechanism whereby lipid phosphatase activity in the nucleus can regulate mammali
164               Finally, the inhibition of CaN phosphatase activity in vascular smooth muscle cells, wh
165 5Thr mutation in SHP-1 caused a reduction in phosphatase activity in vitro, confirming the loss-of-fu
166 ed on the inhibition of immobilized alkaline phosphatase activity, in the presence of the phosphate i
167 ulate actin cytoskeleton dynamics and myosin phosphatase activity, including focal adhesion kinase, p
168                            Inhibiting serine phosphatase activity increased Ser(P)-IRS-2 and decrease
169           Furthermore, the inhibition of CaN phosphatase activity increased the basal phosphorylation
170 at MKK7gamma cytoplasmic retention by CaN is phosphatase activity independent.
171 strain, indicating that both CovS kinase and phosphatase activities influence the CovR phosphorylatio
172 al RNA-binding NSP2 octamer with nucleotidyl phosphatase activity is central to viroplasm formation a
173 esults indicate that scaffolding of inositol phosphatase activity is critical for maintaining PtdIns(
174                    Here, we report that PTEN phosphatase activity is inhibited via a transnitrosylati
175                  We also noted that alkaline phosphatase activity is rapidly restored when zinc is re
176 is study, we demonstrate that PTEN's protein phosphatase activity is required for epiblast epithelial
177 nd maintain 1 null allele, we show that PTEN phosphatase activity is required for preventing the emer
178                                       AtSAL1 phosphatase activity is suppressed by dimerization, intr
179 ab7, a recently identified substrate of PTEN phosphatase activity, is also a substrate of the innate
180 omplement lsf1 Thus, glucan binding, but not phosphatase activity, is required for the function of LS
181 h in vivo and in vitro, loss of EYA tyrosine phosphatase activity leads to defective assembly of gamm
182                     We find that loss of Ppz phosphatase activity leads to defects in ubiquitin homeo
183             In line with this, loss of PTPRK phosphatase activity leads to disrupted cell junctions a
184 well-investigated, little is known about its phosphatase activity, located in the N-terminal phosphat
185  confounders, such as inflammation, alkaline phosphatase activity, low serum albumin, renal function,
186 hanced differentiation indicated by alkaline phosphatase activity, mineral deposition, and transcript
187 entiation as indicated by increased alkaline phosphatase activity, mineralization, and up-regulation
188                                              Phosphatase activity must subsequently be restored to pr
189    These results indicate that neither Cps2B phosphatase activity nor Cps2D phosphorylation levels pe
190 concentrations shift the relative kinase and phosphatase activities of diphosphoinositol pentakisphos
191 ies of the bifunctional 5-InsP7 kinase/InsP8 phosphatase activities of full-length diphosphoinositol
192 ctively requires both the DNA kinase and DNA phosphatase activities of PNKP, and the fork-head associ
193               Moreover, we report that InsP8 phosphatase activities of PPIP5Ks are strongly inhibited
194        We hypothesize that the reductase and phosphatase activities of PSP-GPD multidomain enzymes ma
195 ere, we identified tumor inhibitory and RelA phosphatase activities of the protein phosphatase 2C (PP
196 , we show that the nuclease, polymerase, and phosphatase activities of yeast CPF are organized into t
197  stabilize beta-catenin while increasing the phosphatase activity of a Ppp2r2c-containing Pp2a comple
198 nteraction with AtMIA40 is necessary for the phosphatase activity of AtSLP2 and is dependent on the f
199 at dimerization is important for controlling phosphatase activity of CacyBP/SIP and for regulating th
200  N-terminal domain is indispensable for full phosphatase activity of CacyBP/SIP.
201                       alpha-syn enhanced the phosphatase activity of CN in both cell-free assays and
202              This was dependent on the 2'-5' phosphatase activity of DBR1, since it did not occur whe
203  with our previous results that the tyrosine phosphatase activity of Eya is dispensable for normal Ey
204 86Ser) transgenic mice demonstrates that the phosphatase activity of FIG4 has an essential role in vi
205 ed model has been postulated to regulate the phosphatase activity of HisKA HKs based on the pH-depend
206  the nuclear translocation and phosphatidate phosphatase activity of lipin-1.
207          Our data collectively show that the phosphatase activity of OsPP2A against OsRBR1 is regulat
208             The mechanisms that regulate the phosphatase activity of PGAM5 are poorly understood.
209 ular allosteric regulation mechanism for the phosphatase activity of PGAM5, in which the assembly of
210 hat function as allosteric regulators of the phosphatase activity of PGAM5.
211                       Furthermore, increased phosphatase activity of PP5 correlated with impaired pho
212 ever, Hsp70 binding to PP5 stimulates higher phosphatase activity of PP5 than the binding of Hsp90.
213            The protein rather than the lipid phosphatase activity of PTEN accounts for the reversal o
214                                  The protein phosphatase activity of PTEN dephosphorylates and inhibi
215 PTEN mutant constructs, we show that protein phosphatase activity of PTEN targets PTK6, with efficien
216  tumors, functionally caused the loss of the phosphatase activity of PTPRD, and were associated with
217 vels increased mRNA, protein expression, and phosphatase activity of SHP-1, which remained elevated d
218 steine residue, which inhibited the tyrosine-phosphatase activity of SHP-1.
219                                              Phosphatase activity of Siw14 was inhibited by oxidation
220                       We discovered that the phosphatase activity of STRIPAK reduces Slik phosphoryla
221         Phytohormone binding inactivates the phosphatase activity of the coreceptor, permitting phosp
222                              Pi inhibits the phosphatase activity of the enzyme.
223 oadly conserved mechanism that regulates the phosphatase activity of the largest family of bifunction
224  lacking the WDXNWD motif markedly increases phosphatase activity of the mutant protein.
225                                    The lipid phosphatase activity of the tumor suppressor phosphatase
226                 While the role of PTEN lipid-phosphatase activity on PtdIns(3,4,5)P3 and inhibition o
227 d, in this active conformation, exerts lipid phosphatase activity on PtdIns(3,4,5)P3.
228 ith PTEN mutants that lack only PTEN's lipid phosphatase activity or both PTEN's lipid and protein ph
229 enopus early embryonic extract revealed that phosphatase activity other than PP1 continuously suppres
230 table catalytic His with phosphotransfer and phosphatase activities over an effector response regulat
231 15 (P = 0.0001), and calcification (alkaline phosphatase activity, P < 0.01; osteocalcin, P < 0.05).
232 ere, we use fission yeast to investigate how phosphatase activity participates in this interplay duri
233 GAS mutants lacking SP-PTP revealed that the phosphatase activity per se positively regulates growth,
234              We also find that the threonine phosphatase activity plays only a minor role during Dros
235 acdc55 mutation affecting Cdc55-PP2A protein phosphatase activity prevented Orm dephosphorylation and
236 sion of a mutated form of PTEN with enhanced phosphatase activity prevented the TGF-beta-induced coll
237             These results imply that altered phosphatase activity promotes evolving phenotypes based
238 arterial hypertension and that EYA3 tyrosine phosphatase activity promotes the survival of these cell
239 1 directly binds to STRIPAK and inhibits its phosphatase activity, protecting MST2 activation-loop ph
240 fferences we observed in voltage-sensing and phosphatase activity provide a starting point for future
241 ed, the biological relevance of PTEN protein-phosphatase activity remains undefined.
242  Fig4 mutations predicted to inactivate FIG4 phosphatase activity rescue lysosome expansion phenotype
243 tracellular stores and increased calcineurin phosphatase activity, resulting in NFATc1 nuclear transl
244                       Within target tissues, phosphatase activity results in disassembly of PSRP1-sRN
245                              Moreover, PTPRO phosphatase activity shortened the half-life of ERBB2 by
246 c, and it possesses carbohydrate binding and phosphatase activity similar to human laforin.
247 rmacological inhibition of the EYA3 tyrosine phosphatase activity substantially reverses vascular rem
248 weight, long bone length, and serum alkaline phosphatase activity, suggesting that tooth dysfunction
249  function of PTEN is attributed to its lipid phosphatase activity that counters PI3K action.
250  dystrophies to defective phosphoinositide 5-phosphatase activity that is becoming increasingly recog
251         Furthermore, we show that YejM has a phosphatase activity that is dependent on the presence o
252        Thus, these results identify a 5'-tri-phosphatase activity that is involved in the biogenesis
253 a novel positive regulator of lipin 1beta PA phosphatase activity that is not achieved via altering i
254 use a significant reduction in phosphatidate phosphatase activity, the phosphorylation of Pah1p by pr
255 rovascular integrity by enabling VEC-related phosphatase activity, thereby preventing vascular leak d
256 e receptor kinase activity or enhancing CheC phosphatase activity, thereby regulating the levels of t
257 term through the initial stimulation of soil phosphatase activity, thereby securing P supply to suppo
258 revealed activation of PKA and inhibition of phosphatase activity thus explaining the increase in pho
259 antly, S-nitrosylation of PTEN decreases its phosphatase activity, thus promoting cell survival.
260 pair factor possessing both 5'-kinase and 3'-phosphatase activities to modify ends of a DNA break pri
261 iring holoenzyme formation and reducing PP2A phosphatase activity to promote endometrial cancer progr
262 s, which are rescued by loss of PTEN protein-phosphatase activity to restrain cell survival.
263          To evaluate the contribution of the phosphatase activity to the in vivo function of Fig4, we
264 sequesters sigma(S) and possesses kinase and phosphatase activities toward CrsA.
265 nal HisKA family, displaying both kinase and phosphatase activities toward their substrates.
266  inhibitors differentially affect MAP kinase phosphatase activity toward 2P-ERK2.
267 horylation-defective RPA2 subunit or lacking phosphatase activity toward RPA2.
268  addition, we find key differences in glucan phosphatase activity toward soluble and insoluble polygl
269                           Mutations impaired phosphatase activity toward the phosphoinositide phospha
270 the C-terminus of SCP1 was essential for its phosphatase activity towards c-Myc.
271                                              Phosphatase activity towards phosphorylated smooth muscl
272 high basal autophagy, possess stronger basal phosphatase activity towards ULK1 and require ULK1 for s
273 ation, starvation also causes an increase in phosphatase activity towards ULK1, an mTORC1 substrate w
274 genesis, the additional loss of PTEN protein-phosphatase activity triggered an extensive cell death r
275 rmational switch of HK853 that shuts off its phosphatase activity under acidic conditions.
276 cherichia coli similarly exhibited increased phosphatase activity under conditions of high-oxygen gro
277 m substrate trapping complexes with impaired phosphatase activity via increased recruitment of TIPRL1
278                                    Nem1-Spo7 phosphatase activity was dependent on the concentrations
279                                         CD45 phosphatase activity was determined by using a novel flo
280                                         CD45 phosphatase activity was increased by T-cell help both i
281 hosphorylated on IQGAP1 when phosphotyrosine phosphatase activity was inhibited in cells.
282 elate with phosphorus associations; however, phosphatase activity was most strongly down-regulated in
283 vitro and in vivo data demonstrated that the phosphatase activity was required for the inactivation o
284 fic functions, but how these domains control phosphatase activity was unknown.
285 uced Smad 1/5/8 phosphorylation and alkaline phosphatase activity were both enhanced by the addition
286 A on cell proliferation, migration, and PP2A phosphatase activity were investigated using ovarian and
287 genes, among which genes involved in protein phosphatase activity were overrepresented, suggesting a
288 te that Hsp70 recruits PP5 and activates its phosphatase activity which suggests dual roles for PP5 t
289 is governed by dynamic changes in kinase and phosphatase activities, which are difficult to assess wi
290                                      OCRL1 5-phosphatase activity, which is membrane curvature sensit
291 ressor activity depends largely on its lipid phosphatase activity, which opposes PI3K/AKT activation.
292 tion with CD45 on the T cell, inhibiting its phosphatase activity, which resulted in up-regulation of
293 atus of actin, as was its eIF2alpha-directed phosphatase activity, while localised G-actin depletion
294 d osteoblast differentiation marker alkaline phosphatase activity, while miR-138 inhibitor and OC pro
295 at feed into the PPP, and that CpgA has high phosphatase activity with 4PE.
296 tations in these allosteric clusters altered phosphatase activity with changes in kcat/KM ranging fro
297 oint mutations in cps2B demonstrated reduced phosphatase activity with corresponding increases in lev
298 n and septum formation and FgCdc14 possesses phosphatase activity with specificity for a subset of Cd
299                     Assessment of kinase and phosphatase activity within the myofilament fraction of
300 tion (activation) by regulating PTPN6 (Shp1) phosphatase activity within the signaling complex.

 
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