ライフサイエンスコーパス: phosphate dehydrogenase

1 anufacturer control, GAPDH (glyceraldehyde-3-phosphate dehydrogenase).

2 e dehydrogenase and heat-denatured glucose-6-phosphate dehydrogenase.

3 rginine deiminase ArcA, and glyceraldehyde 3-phosphate dehydrogenase.

4 sphate transport system and an sn-glycerol-3-phosphate dehydrogenase.

5 a mutation of G6PD, which encodes glucose-6-phosphate dehydrogenase.

6 aminonicotinamide, an inhibitor of glucose-6-phosphate dehydrogenase.

7 within the endoplasmic reticulum by hexose-6-phosphate dehydrogenase.

8 -synuclein, synapsin-I, and glyceraldehyde-3-phosphate dehydrogenase.

9 derived from the metabolic enzyme glucose-6-phosphate dehydrogenase.

10 ), which is a potent inhibitor of glycerol-3-phosphate dehydrogenase.

11 inase and the rate-limiting enzyme glucose-6-phosphate dehydrogenase.

12 ific glycolytic proteins such as d-glucose-6-phosphate dehydrogenase.

13 ds to decreased activity of glyceraldehyde 3-phosphate dehydrogenase.

14 codes the glycolytic enzyme glyceraldehyde-3-phosphate dehydrogenase.

15 gresses from the host cell, glyceraldehyde-3-phosphate dehydrogenase 1 (GAPDH1), which is primary a g

16 malate/oxaloacetate shuttle and a glycerol-3-phosphate dehydrogenase 1(Gpd1p)-dependent shuttle are a

17 d with the PTS2-containing enzyme Glycerol-3-phosphate dehydrogenase 1, Gpd1.

18 ruvate dehydrogenase kinase 4 and glycerol 3-phosphate dehydrogenase 1, was acutely induced by a sing

19 in a conserved amino acid of the glycerol-3-phosphate dehydrogenase 1-like (GPD1-L) gene.

20 Here, we identify the enzyme glycerol-3-phosphate dehydrogenase 1-like (GPD1L) as a novel regula

21 of the mature miRNA, derepresses glycerol-3-phosphate dehydrogenase 1-like enzyme (GPD1L), a hypoxia

22 blocked the mutant A280V GPD1-L (glycerol-3-phosphate dehydrogenase 1-like) effect on reducing I(Na)

23 dentified by mass spectrometry as glycerol-3-phosphate dehydrogenase 2 (Gpd2).

24 lciparum , the bifunctional enzyme glucose-6-phosphate dehydrogenase-6-phosphogluconolactonase (PfGlu

25 inhibition of the parasite enzyme glucose-6-phosphate dehydrogenase-6-phosphogluconolactonase, a key

26 thway by ADPr inhibition of glyceraldehyde-3-phosphate dehydrogenase, a central enzyme of glycolysis.

27 for the nonphosphorylating glyceraldehyde-3-phosphate dehydrogenase, a cytosolic enzyme whose role i

28 rs of Plasmodium falciparum glyceraldehyde-3-phosphate dehydrogenase, a validated target for the trea

29 Also glucose-6-phosphate dehydrogenase activity and NADPH levels were i

30 hibited cylcooxgenase-2 and glyceraldehyde 3-phosphate dehydrogenase activity and triggered significa

31 NAD(+) pools sufficient for glyceraldehyde-3-phosphate dehydrogenase activity and Warburg metabolism.

32 by inducing maximal mitochondrial glycerol-3-phosphate dehydrogenase activity in rat liver while havi

33 Glucose phosphate dehydrogenase activity that gives rise to NADP

34 as also capable of inducing glyceraldehyde-3-phosphate dehydrogenase aggregation, but chondroitin sul

35 eS cluster binding, such as glyceraldehyde-3-phosphate dehydrogenase, aldehyde dehydrogenase, aconita

36 no acids) of serum albumin, glyceraldehyde-3-phosphate dehydrogenase, aldolase, and aspartate aminotr

37 stained with antibodies to glyceraldehyde-3-phosphate dehydrogenase, aldolase, phosphofructokinase,

38 A6, clathrin heavy chain 1, glyceraldehyde-3-phosphate dehydrogenase, alpha-enolase, filamin-A, and h

39 olism (triosephosphate isomerase, glycerol-3-phosphate-dehydrogenase, alpha enolase and L-lactate deh

40 Hexose-6-phosphate dehydrogenase and 11 beta-hydroxysteroid dehyd

41 Two NAD-dependent dehydrogenases (glucose-6-phosphate dehydrogenase and 6-phosphogluconate dehydroge

42 homeostasis, which is regulated by glucose-6-phosphate dehydrogenase and AMP kinase.

43 er function through suppression of glucose-6-phosphate dehydrogenase and antioxidant defense systems.

44 on of the secretory protein glyceraldehyde-3-phosphate dehydrogenase and ATP synthase subunit alpha i

45 ncluding nonphosphorylating glyceraldehyde-3-phosphate dehydrogenase and beta-glucosidase.

46 Nuclear complex of glyceraldehyde-3-phosphate dehydrogenase and DNA repair enzyme apurinic/a

47 s response, especially the roles of hexose-6-phosphate dehydrogenase and ER-anchored RING finger E3 l

48 oplast-targeted Plasmodium yoelii glycerol 3-phosphate dehydrogenase and glycerol 3-phosphate acyltra

49 es that catalyze their synthesis, glycerol-1-phosphate dehydrogenase and heptaprenylglyceryl phosphat

50 ntent of glycolytic enzymes glyceraldehyde 3-phosphate dehydrogenase and pyruvate dehydrogenase subun

51 olving conversion to N(tz) ADPH by glucose-6-phosphate dehydrogenase and reoxidation to N(tz) ADP(+)

52 are sensitive to oxidation, glyceraldehyde-3-phosphate dehydrogenase and the sodium-potassium ATPase,

53 Evaluations for glucose-6-phosphate dehydrogenase and thiopurine S-methyltransfera

54 hosphate dehydrogenase, and glyceraldehyde-3-phosphate dehydrogenase) and their respective genetic va

55 glycerol synthesis gene, gpd1(+) (glycerol-3-phosphate dehydrogenase), and is independent of actin ca

56 l polymorphisms (ie, hemoglobin S, glucose-6-phosphate dehydrogenase, and alpha-thalassemia) were the

57 lucose 6-phosphate dehydrogenase, glycerol 3-phosphate dehydrogenase, and glucose oxidase.

58 mes (catechol-O-methyltransferase, glucose-6-phosphate dehydrogenase, and glyceraldehyde-3-phosphate

59 s including Tau, ubiquitin, glyceraldehyde-3-phosphate dehydrogenase, and glycosaminoglycans are rout

60 el such actin, tubulin, and glyceraldehyde-3-phosphate dehydrogenase are frequently used as endogenou

61 ycerol-3-phosphate shuttle (Gut2, glycerol-3-phosphate dehydrogenase) are novel longevity factors in

62 stable mRNAs (e.g., GAPDH [glyceraldehyde-3-phosphate dehydrogenase]) are rapidly degraded.

63 d soluble recombinant sperm glyceraldehyde-3-phosphate dehydrogenase as a heterotetramer with the Esc

64 Of the latter, we confirmed glyceraldehyde-3-phosphate dehydrogenase as a key target of IAA, specific

65 ntify the glycolytic enzyme glyceraldehyde-3-phosphate dehydrogenase as a potent inhibitor of caspase

66 ified glucose transport and glyceraldehyde-3-phosphate dehydrogenase as the most selective antiparasi

67 We found that the HKGs glyceraldehyde-3-phosphate dehydrogenase, beta actin and adenosine tripho

68 fied for the reaction catalyzed by glucose-6-phosphate dehydrogenase by comparing the 1-(13)C KIE wit

69 esin-1, gelsolin, tubulins, glyceraldehyde-3-phosphate dehydrogenase, calmodulin, ATP synthase, sperm

70 nd that the glycolytic enzyme glyceraldehyde phosphate dehydrogenase constitutes a major cellular hem

71 severe malaria data reported that glucose-6-phosphate dehydrogenase deficiency (G6PDd) was protectiv

72 olytic anaemia in individuals with glucose-6-phosphate dehydrogenase deficiency (G6PDd).

73 Other abnormal hemoglobins, glucose-6-phosphate dehydrogenase deficiency and pyruvate kinase d

74 idence interval {CI}, 0.52-0.90]), glucose-6-phosphate dehydrogenase deficiency in female children (R

75 ssemia, 0.3%; ABO group, 0.3%; and glucose-6-phosphate dehydrogenase deficiency, 0.5%) and the new po

76 eurotoxicity, such as prematurity, glucose-6-phosphate dehydrogenase deficiency, or hypoxia-ischemia.

77 alocytosis and two common forms of glucose-6-phosphate dehydrogenase deficiency.

78 relation with sickle cell trait or glucose-6-phosphate-dehydrogenase deficiency was observed.

79 pecies, the heparin-induced glyceraldehyde-3-phosphate dehydrogenase early oligomers might come in us

80 ncoded by glpK and suggest that a glycerol-3-phosphate dehydrogenase encoded by the upstream gpdABC o

81 Conditional expression of the glucose-6-phosphate dehydrogenase-encoding gene zwf, shown here to

82 Patients with glucose-6-phosphate dehydrogenase enzyme activity of less than 70%

83 licated falciparum malaria, normal glucose-6-phosphate dehydrogenase enzyme levels, and hemoglobin le

84 D or another oxidative PPP enzyme, glucose-6-phosphate dehydrogenase, exhibit non-immune hemolytic an

85 te for an enzyme, F(420)-dependent glucose-6-phosphate dehydrogenase (Fgd), found in few bacterial ge

86 er coenzyme for an F(420)-specific glucose-6-phosphate dehydrogenase (Fgd).

87 ly oligomers present in the glyceraldehyde-3-phosphate dehydrogenase fibrillation pathway promote alp

88 redoxin reductase (TR), and glyceraldehyde-3-phosphate dehydrogenase (G3PD) activities, as well as th

89 ldolase A and an isoform of glyceraldehyde 3-phosphate dehydrogenase (G3PDH) showed elevated oxidatio

90 ox polypeptide, beta-actin, glyceraldehyde 3-phosphate dehydrogenase (G3PDH), annexin A2, triose phos

91 itochondrial glutathione (GSH) and glucose 6-phosphate dehydrogenase (G6-PD) was restored by SAC trea

92 iphenyltetrazolium bromide] (MTT), glucose-6-phosphate dehydrogenase (G6DP), and calcein AM and ethid

93 rasites per microliter) and normal glucose-6-phosphate dehydrogenase (G6PD) activity (with normal act

94 isocitrate dehydrogenase (IDH) or glucose-6-phosphate dehydrogenase (G6PD) activity in a single gene

95 Glucose-6-phosphate dehydrogenase (G6PD) activity is essential for

96 ATM activation induces glucose-6-phosphate dehydrogenase (G6PD) activity, the limiting en

97 Unexpectedly, Glucose-6-phosphate dehydrogenase (G6PD) and 6-phosphogluconate de

98 xidation of G6P in the presence of glucose-6-phosphate dehydrogenase (G6PD) and nicotinamide adenine

99 egulated the protein expression of glucose-6-phosphate dehydrogenase (G6PD) and peroxiredoxin (PRDX).

100 ents (aged >=6 months) with normal glucose-6-phosphate dehydrogenase (G6PD) and presenting with uncom

101 tion, suggesting that intranuclear glucose-6-phosphate dehydrogenase (G6PD) can control NOX4 activity

102 ependent haemolysis in people with glucose-6-phosphate dehydrogenase (G6PD) deficiencies have limited

103 all patients should be tested for glucose-6-phosphate dehydrogenase (G6PD) deficiency (G6PDd) prior

104 Individuals with glucose 6-phosphate dehydrogenase (G6PD) deficiency are at risk fo

105 BACKGROUNDGlucose-6-phosphate dehydrogenase (G6PD) deficiency decreases the

106 halassaemia, ABO blood groups, and glucose-6-phosphate dehydrogenase (G6PD) deficiency encoded by the

107 Glucose 6-phosphate dehydrogenase (G6PD) deficiency is 1 of the co

108 Glucose-6-phosphate dehydrogenase (G6PD) deficiency is believed to

109 Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most co

110 Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most co

111 Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most co

112 The global prevalence of X-linked glucose-6-phosphate dehydrogenase (G6PD) deficiency is thought to

113 Despite glucose-6-phosphate dehydrogenase (G6PD) deficiency prevalence of

114 uce haemolysis in individuals with glucose-6-phosphate dehydrogenase (G6PD) deficiency, necessitating

115 by haemolysis in individuals with glucose-6-phosphate dehydrogenase (G6PD) deficiency.

116 ata are limited in Southeast Asian glucose-6-phosphate dehydrogenase (G6PD) deficient (G6PD-) patient

117 and involved patients with normal glucose-6-phosphate dehydrogenase (G6PD) enzyme activity and femal

118 risk factors, and we also assessed glucose-6-phosphate dehydrogenase (G6PD) enzyme activity.

119 hondria, whereas chloroquine and a glucose-6-phosphate dehydrogenase (G6PD) inhibitor affected the cy

120 Glucose-6-phosphate dehydrogenase (G6PD) is a key enzyme in the pe

121 The enzyme glucose-6-phosphate dehydrogenase (G6PD) is a major contributor to

122 Glucose-6-phosphate dehydrogenase (G6PD) is critical in maintainin

123 The reaction catalyzed by glucose-6-phosphate dehydrogenase (G6PD) is the first, rate-limiti

124 Glucose 6-phosphate dehydrogenase (G6PD) is the most common defici

125 Glucose-6-phosphate dehydrogenase (G6PD) is the principal source o

126 Glucose-6-phosphate dehydrogenase (G6PD) is the rate-limiting enzy

127 Glucose-6-phosphate dehydrogenase (G6PD) modulates vascular functi

128 roscopy and had normal function of glucose-6-phosphate dehydrogenase (G6PD) on colorimetric quantific

129 s exacerbated in erythrocytes from glucose-6-phosphate dehydrogenase (G6PD) patients and rescued by a

130 ing ATM or its downstream effector glucose 6-phosphate dehydrogenase (G6PD) sensitizes AML cells to F

131 Glucose-6-phosphate dehydrogenase (G6PD) status was determined usi

132 ivo, pharmacological inhibition of glucose-6-phosphate dehydrogenase (G6PD) to decrease NADPH levels

133 tive capacity by overexpression of glucose-6-phosphate dehydrogenase (G6PD), a key enzyme for NADPH b

134 anthracyclines, and inhibitors of glucose-6-phosphate dehydrogenase (G6PD), an enzyme involved in gl

135 necessary for NET release because glucose-6-phosphate dehydrogenase (G6PD), an important enzyme from

136 ession and enzymatic activities of glucose-6-phosphate dehydrogenase (G6PD), glutathione reductase, a

137 owever, we found that knockdown of glucose-6-phosphate dehydrogenase (G6PD), the first oxiPPP enzyme,

138 rial pathogen Pseudomonas syringae Glucose-6-phosphate dehydrogenase (G6PD), the key enzyme of the ox

139 Here we show that glucose-6-phosphate dehydrogenase (G6PD), the rate-limiting enzyme

140 gulates stress tolerance by activating Glc-6-phosphate dehydrogenase (G6PD), which is essential for m

141 lase (HDAC) inhibitors (HDACis) in glucose-6-phosphate dehydrogenase (G6PD)-deficient cells reinstate

142 rotection from oxidative stress by glucose-6-phosphate dehydrogenase (G6PD).

143 lectively enhance transcription of glucose 6-phosphate dehydrogenase (G6PD).

144 NADPH by impairing the activity of glucose 6-phosphate dehydrogenase (G6PD).

145 In addition, glucose-6-phosphate-dehydrogenase (G6PD) deficiency, which is the

146 s in patients with a deficiency in glucose-6-phosphate dehydrogenase (G6PDd).

147 ake with a concomitant increase in glucose-6-phosphate dehydrogenase (G6PDH) activity, the key enzyme

148 Glucose-6-phosphate dehydrogenase (G6PDH) has been implicated in t

149 Moreover, higher activities of glucose 6-phosphate dehydrogenase (G6PDH), 6-phosphogluconate dehy

150 on enzymes such as NQO1 as well as glucose-6-phosphate dehydrogenase (G6PDH), a regulator of the pent

151 citrate dehydrogenase (NADP-ICDH), glucose-6-phosphate dehydrogenase (G6PDH), and, glutathione reduct

152 ibited high specificity for glyceraldehyde 3-phosphate dehydrogenase (GADPH) mRNA in live normal HEp-

153 n-1 (Hwp1); enolase (Enol); glyceraldehyde-3-phosphate dehydrogenase (Gap1); and phosphoglycerate kin

154 combined activities of the glyceraldehyde 3-phosphate dehydrogenases GapA/GapB and the malic enzymes

155 The cytosolic enzyme glyceraldehyde-3-phosphate dehydrogenase (GAPC) catalyzes a key reaction

156 A cytosolic glyceraldehyde-3-phosphate dehydrogenase (GAPC) was identified to bind to

157 translocation of cytosolic glyceraldehyde-3-phosphate dehydrogenase (GAPC), but its nuclear function

158 dial glycolytic isoforms of glyceraldehyde-3-phosphate dehydrogenase (GAPCp) in photosynthetic and he

159 ytosolic glycolytic enzymes glyceraldehyde-3-phosphate dehydrogenases (GAPCs) interact with the plasm

160 ly used housekeeping genes, glyceraldehyde 3-phosphate dehydrogenase (GAPDH) and 18S ribosomal RNA (r

161 synergistic interaction of glyceraldehyde-3-phosphate dehydrogenase (GAPDH) and a major facilitator

162 Rab2 requires glyceraldehyde-3-phosphate dehydrogenase (GAPDH) and atypical protein kin

163 supramolecular complex with glyceraldehyde-3-phosphate dehydrogenase (GAPDH) and phosphoribulokinase,

164 ming a ternary complex with glyceraldehyde 3-phosphate dehydrogenase (GAPDH) and phosphoribulokinase.

165 sibly acetylated, including glyceraldehyde-3-phosphate dehydrogenase (GAPDH) and Rpa1177, a putative

166 by the interaction between glyceraldehyde-3-phosphate dehydrogenase (GAPDH) and the E3 ubiquitin lig

167 osphoribulokinase (PRK) and glyceraldehyde 3-phosphate dehydrogenase (GAPDH) are essential CB-cycle e

168 proaches, we identified the glyceraldehyde-3-phosphate dehydrogenase (GAPDH) as a C1q partner when ex

169 for RT-PCR and qrtPCR using glyceraldehyde-3-phosphate dehydrogenase (GAPDH) as an endogenous control

170 mammalian glycolysis enzyme glyceraldehyde 3-phosphate dehydrogenase (GAPDH) as an NleB-interacting p

171 We found that I/R induces glyceraldehyde-3-phosphate dehydrogenase (GAPDH) association with mitocho

172 y nitroso compounds inhibit glyceraldehyde 3-phosphate dehydrogenase (GAPDH) by forming a reduction r

173 Glyceraldehyde-3-phosphate dehydrogenase (GAPDH) from human pathogens Sta

174 single cell analysis of the glyceraldehyde 3 phosphate dehydrogenase (GAPDH) gene in human lymphocyte

175 Glyceraldehyde 3-phosphate dehydrogenase (GAPDH) has been found down-regu

176 Glyceraldehyde-3-phosphate dehydrogenase (GAPDH) has been hypothesized as

177 form molecular targeting of glyceraldehyde-3-phosphate dehydrogenase (GAPDH) in human hepatocellular

178 ocation and accumulation of glyceraldehyde-3-phosphate dehydrogenase (GAPDH) in the nucleus has close

179 Glyceraldehyde-3-phosphate dehydrogenase (GAPDH) is a glycolytic enzyme t

180 Glyceraldehyde 3-phosphate dehydrogenase (GAPDH) is a glycolytic enzyme t

181 Glyceraldehyde-3-phosphate dehydrogenase (GAPDH) is a key enzyme involved

182 Glyceraldehyde-3-phosphate dehydrogenase (GAPDH) is a multifunctional enz

183 Glyceraldehyde-3-phosphate dehydrogenase (GAPDH) is a ubiquitous and abun

184 NAD-dependent glyceraldehyde-3-phosphate dehydrogenase (GAPDH) is a ubiquitous enzyme i

185 Glyceraldehyde-3-phosphate dehydrogenase (GAPDH) is an abundant metabolic

186 Glyceraldehyde-3-phosphate dehydrogenase (GAPDH) is an enzyme best known

187 Glyceraldehyde-3-phosphate dehydrogenase (GAPDH) is often used as a stabl

188 lving nitric oxide (NO) and glyceraldehyde-3-phosphate dehydrogenase (GAPDH) mediates cocaine's trans

189 , superoxide dismutase, and glyceraldehyde 3-phosphate dehydrogenase (GAPDH) mRNA with a concomitant

190 de is a structural mimic of glyceraldehyde 3-phosphate dehydrogenase (GAPDH) on the sporozoite surfac

191 edly, the glycolytic enzyme glyceraldehyde-3-phosphate dehydrogenase (GAPDH) physically associates wi

192 Glyceraldehyde 3-phosphate dehydrogenase (GAPDH) plays a key regulatory f

193 METH also increases glyceraldehyde-3-phosphate dehydrogenase (GAPDH) protein in the crude ves

194 utoantigen protein p70, and glyceraldehyde 3-phosphate dehydrogenase (GAPDH) proteins and that the si

195 Using glyceraldehyde-3-phosphate dehydrogenase (GAPDH) silencing with small int

196 m of NADH-channeling from D-glyceraldehyde-3-phosphate dehydrogenase (GAPDH) to L-lactate Dehydrogena

197 horesis, and phosphorylated glyceraldehyde-3-phosphate dehydrogenase (GAPDH) was identified by matrix

198 mino acids with identity to glyceraldehyde-3-phosphate dehydrogenase (GAPDH) was obtained.

199 Arginine kinase (AK) and glyceraldehyde 3-phosphate dehydrogenase (GAPDH) were determined as the i

200 ble residues in the protein glyceraldehyde-3-phosphate dehydrogenase (GAPDH) were examined.

201 lves interaction of nuclear glyceraldehyde-3-phosphate dehydrogenase (GAPDH) with apurinic/apyrimidin

202 factor 1 alpha (EF1alpha), glyceraldehyde-3 phosphate dehydrogenase (GAPDH), 40 S ribosomal protein

203 Here, we demonstrate that glyceraldehyde 3-phosphate dehydrogenase (GAPDH), a conventional glycolyt

204 ctive covalent inhibitor of glyceraldehyde-3-phosphate dehydrogenase (GAPDH), a critical node in the

205 receptors for Plg including glyceraldehyde-3-phosphate dehydrogenase (GAPDH), a cytoplasmic enzyme th

206 f the key glycolytic enzyme glyceraldehyde-3-phosphate dehydrogenase (GAPDH), a modification that inh

207 Glyceraldehyde 3-phosphate dehydrogenase (GAPDH), a protein target used r

208 that specifically inhibits glyceraldehyde-3-phosphate dehydrogenase (GAPDH), a rate-controlling glyc

209 Glyceraldehyde-3-phosphate dehydrogenase (GAPDH), an important glycolytic

210 e, phosphoglycerate kinase, glyceraldehyde-3-phosphate dehydrogenase (GAPDH), and enolase, all of whi

211 o protect citrate synthase, glyceraldehyde-3-phosphate dehydrogenase (GAPDH), and the restriction end

212 Commonly used glyceraldehyde-3-phosphate dehydrogenase (Gapdh), beta-actin (Actb), or b

213 for total protein quantity, glyceraldehyde 3-phosphate dehydrogenase (GAPDH), citrate synthase (CS),

214 s, creatine kinase (CK) and glyceraldehyde-3-phosphate dehydrogenase (GAPDH), conformationally affect

215 elongation factor 2 (EF2), glyceraldehyde-3-phosphate dehydrogenase (GAPDH), hypoxanthine-guanine ph

216 n of the sulfhydryl enzyme, glyceraldehyde-3-phosphate dehydrogenase (GAPDH), in vitro and in tissues

217 Superoxide inhibits glyceraldehyde-3-phosphate dehydrogenase (GAPDH), which activates major p

218 vered that Escherichia coli glyceraldehyde-3-phosphate dehydrogenase (GAPDH), which is not known to b

219 olvement in this process of glyceraldehyde-3-phosphate dehydrogenase (GAPDH), which participates in i

220 s accumulate and inactivate glyceraldehyde 3-phosphate dehydrogenase (GAPDH).

221 a-2 microglobulin (B2M) and glyceraldehyde-3-phosphate dehydrogenase (GAPDH).

222 f the key glycolytic enzyme glyceraldehyde-3-phosphate dehydrogenase (GAPDH).

223 the L1 interaction partner glyceraldehyde-3-phosphate dehydrogenase (GAPDH).

224 ribosomal protein L13a, and glyceraldehyde-3-phosphate dehydrogenase (GAPDH).

225 osphoribulokinase (PRK) and glyceraldehyde-3-phosphate dehydrogenase (GAPDH).

226 nd nuclear translocation of glyceraldehyde 3-phosphate dehydrogenase (GAPDH).

227 osphate aldolase (FBPA) and glyceraldehyde-3-phosphate dehydrogenase (GAPDH).

228 We focused on one of these, glyceraldehyde-3-phosphate dehydrogenase (GAPDH).

229 (NO), which S-nitrosylates glyceraldehyde-3-phosphate dehydrogenase (GAPDH).

230 ansport into the cytoplasm; glyceraldehyde 3-phosphate dehydrogenase (GAPDH; a glycolysis enzyme); AT

231 protein (Ucp1) and mitochondrial glycerol-3-phosphate dehydrogenase (Gdm) result in mice with increa

232 et the histidine kinase and glyceraldehyde-3-phosphate dehydrogenase genes of B. dermatitidis and H.

233 s including c-myc, p21, and glyceraldehyde-3-phosphate dehydrogenase genes, indicating reduced promot

234 We studied the contribution of glucose-6-phosphate dehydrogenase (Glc-6-PD), an important regulat

235 Sn-glycerol-3-phosphate dehydrogenase (GlpD) is an essential membrane

236 ATP into glucose 6-phosphate), and glucose 6-phosphate dehydrogenase (glucose 6-phosphate into NADPH)

237 reaction utilizing three enzymes: glycerol 3-phosphate dehydrogenase, glycerol 3-phosphate acyltransf

238 s including alcohol dehydrogenase, glucose 6-phosphate dehydrogenase, glycerol 3-phosphate dehydrogen

239 phosphatase (PSP) motif fused to glycerol-3-phosphate dehydrogenase (GPD) domains.

240 ces cerevisiae has two homologous glycerol-3-phosphate dehydrogenases, Gpd1 and Gpd2, that are requir

241 e glycerol-producing PTS2 protein glycerol-3-phosphate dehydrogenase Gpd1p shows a tripartite localiz

242 lysaccharide (LPS), mitochondrial glycerol 3-phosphate dehydrogenase (GPD2) regulates glucose oxidati

243 Here we evaluate the effects of glycerol-3-phosphate dehydrogenase (Gpdh) and cytosolic malate dehy

244 of R269 lies at the surface of l-glycerol 3-phosphate dehydrogenase (GPDH) and forms an ion pair to

245 rginine or creatine kinase, glyceraldehyde-3-phosphate dehydrogenase (GPDH), calcium-binding protein,

246 Hexose-6-phosphate dehydrogenase (H6PD) generates NADPH, the cofa

247 Hexose-6-phosphate dehydrogenase (H6PD) is the initial component

248 hydrogenase type 1 (11betaHSD1) and hexose-6-phosphate dehydrogenase (H6PDH).

249 Sperm glyceraldehyde-3-phosphate dehydrogenase has been shown to be a successfu

250 xylase (ScOMPDC), and human liver glycerol 3-phosphate dehydrogenase (hlGPDH) for catalysis of reacti

251 The R269A mutation of glycerol-3-phosphate dehydrogenase (hlGPDH) results in a 9.1 kcal/m

252 r with the Escherichia coli glyceraldehyde-3-phosphate dehydrogenase in a ratio of 1:3 and have solve

253 -synuclein, synapsin-I, and glyceraldehyde-3-phosphate dehydrogenase in cultured hippocampal neurons,

254 nd formate, and overexpression of sorbitol-6-phosphate dehydrogenase in lactate dehydrogenase-deficie

255 f the pentose phosphate pathway by glucose-6-phosphate dehydrogenase inhibitors and siRNA suppressed

256 Glyceraldehyde-3-phosphate dehydrogenase is a glycolytic enzyme that can

257 PdxB (erythronate 4-phosphate dehydrogenase) is expected to be required for

258 vity of its ancillary subunit-the glycerol-3-phosphate dehydrogenase-like protein.

259 l 3-phosphate by mitochondrial sn-glycerol 3-phosphate dehydrogenase (mGPDH) is a major pathway for t

260 substrate glycolaldehyde (GLY) by glycerol 3-phosphate dehydrogenase (NAD (+), GPDH) saturated with N

261 ontrolled study, 16 malaria-naive, glucose-6-phosphate dehydrogenase-normal participants aged 20-42 y

262 chanism by which glycolytic glyceraldehyde-3-phosphate dehydrogenase of Arabidopsis thaliana (AtGAPC1

263 to the gpdC gene of the putative glycerol-3-phosphate dehydrogenase operon (gpdABC), based on the fi

264 observe changes in the activity of glucose-6-phosphate dehydrogenase or in the pentose phosphate path

265 of the Calvin cycle enzymes glyceraldehyde-3-phosphate dehydrogenase or ribulose-1,5-bisphosphate car

266 Cytosolic Oryza sativa glyceraldehyde-3-phosphate dehydrogenase (OsGAPDH), the enzyme involved i

267 towards the pentose phosphate and glycerol-3-phosphate dehydrogenase pathways.

268 and a member of the 4-hydroxy-l-threonine 4-phosphate dehydrogenase (PdxA) oxidative decarboxylase,

269 that inhibit Plasmodium falciparum glucose-6-phosphate dehydrogenase (PfG6PD).

270 cells includes glycolytic enzymes (glucose-6-phosphate dehydrogenase, phosphofructokinase), DNA repai

271 tants deficient in PavA, CodY, and glucose-6-phosphate dehydrogenase pointing to the robustness of th

272 se, malate dehydrogenase 1, glyceraldehyde-3-phosphate dehydrogenase, proteoglycan 4, minichromosome

273 eq to identify three genes (GLYCERALDEHYDE 3-PHOSPHATE DEHYDROGENASE (PvGAPC1), ORGANIC CATION TRANSP

274 sh L-lactate dehydrogenase, glyceraldehyde-3-phosphate dehydrogenase, pyruvate kinase, and glucose-6-

275 PDH) [small interfering RNA(glyceraldehyde 3-phosphate dehydrogenase)] reduces PLCbeta-mediated calci

276 e acyltransferase or GLY1-encoded glycerol-3-phosphate dehydrogenase restored 18:1 levels in HRT ssi2

277 somatic and sperm-specific glyceraldehyde-3-phosphate dehydrogenase revealed few differences at the

278 ediated by the nitric oxide-glyceraldehyde-3-phosphate dehydrogenase signaling pathway.

279 METHODS AND Patients with normal glucose-6-phosphate dehydrogenase status with symptomatic P. vivax

280 ockade of glycolysis at the glyceraldehyde 3-phosphate dehydrogenase step as the central metabolic ba

281 uation of glycolysis at the glyceraldehyde 3-phosphate dehydrogenase step due to the reduced availabi

282 rmediates before and at the glyceraldehyde 3-phosphate dehydrogenase step, promoting carbon overflow

283 tic intermediates after the glyceraldehyde 3-phosphate dehydrogenase step, thereby reducing carbon fl

284 glycolysis by blocking the glyceraldehyde 3-phosphate dehydrogenase step.

285 ailed analysis of the sperm glyceraldehyde-3-phosphate dehydrogenase structure revealed sites in the

286 ared with published somatic glyceraldehyde-3-phosphate dehydrogenase structures that could be exploit

287 gene, gpdA, encoding a homolog of glycerol-3-phosphate dehydrogenase subunit A, were upregulated (8-

288 p is a cytosolic NAD(+)-dependent glycerol 3-phosphate dehydrogenase that also localizes to peroxisom

289 peS6PDH encodes a NADPH-dependent sorbitol-6-phosphate dehydrogenase, the key enzyme for biosynthesis

290 ibitor (dehydroepiandrosterone) of glucose-6-phosphate dehydrogenase together established redundancy

291 ints in central metabolism (Glyceraldehyde 3-phosphate dehydrogenase, transaldolase, alcohol dehydrog

292 olytic enzymes, aldolase A, glyceraldehyde 3-phosphate dehydrogenase, triose phosphate isomerase, and

293 glycolaldehyde (GA) catalyzed by glycerol-3-phosphate dehydrogenase were determined over a 2100-fold

294 oxysteroid dehydrogenase type 1 and hexose-6-phosphate dehydrogenase were initially implicated in thi

295 osolic isocitrate dehydrogenase or glucose-6-phosphate dehydrogenase, which also produce cytosolic NA

296 a-actin, alpha-tubulin, and glyceraldehyde 3-phosphate dehydrogenase, which are usually used as contr

297 has >80% amino acid homology with glycerol-3-phosphate dehydrogenase, which is involved in NAD-depend

298 for the housekeeping enzyme glyceraldehyde-3-phosphate dehydrogenase, wild-type HSV infection reduced

299 lated lysine residue 184 in glyceraldehyde 3-phosphate dehydrogenase with glutamic acid, a malonyllys

300 er-expressing zwf gene (coding for glucose-6-phosphate dehydrogenase), WX-zwf, produced the highest g